ライフサイエンスコーパス: angiopathy

1 of 'familial cerebellar ataxia with amyloid angiopathy'.

2 nic Alzheimer's disease and cerebral amyloid angiopathy.

3 t not Braak tangle stage or cerebral amyloid angiopathy.

4 agic neuroimaging marker of cerebral amyloid angiopathy.

5 th a particular emphasis on cerebral amyloid angiopathy.

6 gy of Alzheimer's disease and cerebral Abeta angiopathy.

7 Willis atherosclerosis, or cerebral amyloid angiopathy.

8 rominent Abeta38-containing cerebral amyloid angiopathy.

9 total plasma cholesterol or cerebral amyloid angiopathy.

10 Alzheimer disease but can also cause amyloid angiopathy.

11 ystem and the importance of cerebral amyloid angiopathy.

12 ry was significantly associated with amyloid angiopathy.

13 d enhancing astrocytosis or cerebral amyloid angiopathy.

14 le lobar CMBs suggestive of cerebral amyloid angiopathy.

15 se gene variants on risk of cerebral amyloid angiopathy.

16 ing Alzheimer's disease and cerebral amyloid angiopathy.

17 to significantly increased cerebral amyloid angiopathy.

18 ating neuroinflammation and cerebral amyloid angiopathy.

19 ial dissection, vasculitis, or postvaricella angiopathy.

20 amyloid diseases including cerebral amyloid angiopathy.

21 ated with familial forms of cerebral amyloid angiopathy.

22 lzheimer's disease (AD) and cerebral amyloid angiopathy.

23 myloid disease known as PrP cerebral amyloid angiopathy.

24 en, and 5) led to the development of amyloid angiopathy.

25 id deposits associated with cerebral amyloid angiopathy.

26 se familial forms of AD and cerebral amyloid angiopathy.

27 eningoencephalic vessels as cerebral amyloid angiopathy.

28 rentially mature senile plaques, and amyloid angiopathy.

29 ith Alzheimer's disease and cerebral amyloid angiopathy.

30 cored plaques, and mild or moderate amyloid angiopathy.

31 lay onset and retard progression of diabetic angiopathy.

32 d blood vessels severely affected by amyloid angiopathy.

33 ular degeneration in AD and cerebral amyloid angiopathy.

34 s causing familial forms of cerebral amyloid angiopathy.

35 logical hallmarks of AD and cerebral amyloid angiopathy.

36 morrhage is the hallmark of cerebral amyloid angiopathy.

37 o be associated with the severity of amyloid angiopathy.

38 of Alzheimer's disease and cerebral amyloid angiopathy.

39 lood vessels reminiscent of cerebral amyloid angiopathy.

40 s presenting with suspected cerebral amyloid angiopathy.

41 wo pathways associated with cerebral amyloid angiopathy.

42 rly for lobar hemorrhages related to amyloid angiopathy.

43 trast for Abeta plaques and cerebral amyloid angiopathy.

44 yloid disease, hereditary cystatin C amyloid angiopathy.

45 c peptide levels may delay progression of AD angiopathy.

46 luid (100% versus 58%; P = 0.02) and amyloid angiopathy (50% versus 12%; P = 0.03).

47 binding protein 43, 8.1% to cerebral amyloid angiopathy, 6.0% to atherosclerosis, and 5.2% to arterio

48 nted patients with probable cerebral amyloid angiopathy (69 +/- 10 years) and 29 similar aged control

49 merging as a key feature of cerebral amyloid angiopathy, a common and important age-related cerebral

50 In hereditary cystatin C amyloid angiopathy, a cystatin C variant is deposited in arteria

51 ons of Iowa-type hereditary cerebral amyloid angiopathy, a form of the disorder with little or no pla

52 yperphosphorylated tau; and cerebral amyloid angiopathy, a microangiopathy affecting both cerebral co

53 s, potentially resulting in cerebral amyloid angiopathy after several decades.

54 ementia can be secondary to cerebral amyloid angiopathy alone.

55 of candidate treatments for cerebral amyloid angiopathy, an untreatable cause of haemorrhagic stroke

56 ding a clue to the known association between angiopathies and diabetes and implying new gene candidat

57 wild-type mice, and formed cerebral amyloid angiopathy and Abeta plaques after a 12-month period of

58 ation, wound healing, scarring, angiogenesis/angiopathy and basement membrane physiology, as well as

59 0 and Abeta42 levels and accelerates amyloid angiopathy and cerebral beta-amyloidosis by diminishing

60 -tau pathology, severity of cerebral amyloid angiopathy and cortical microhaemorrhages.

61 walls was characteristic of cerebral amyloid angiopathy and did not co-localize with prion protein de

62 s, multiple system atrophy, cerebral amyloid angiopathy and elderly controls free of pathology.

63 ably increased the level of cerebral amyloid angiopathy and exacerbated cerebral amyloid angiopathy-r

64 Two persons had severe amyloid angiopathy and haemorrhagic stroke.

65 role in the pathogenesis of cerebral amyloid angiopathy and in the accumulation of Abeta in the brain

66 The severity of cerebral amyloid angiopathy and microhaemorrhages did not relate to any o

67 ears) with pathology-proven cerebral amyloid angiopathy and multiple microbleeds on in vivo clinical

68 Braak stage was five or six, amyloid angiopathy and neuritic plaques were common and more tha

69 Cerebral amyloid angiopathy and neurofibrillary tangles persist, however,

70 lationship between advanced cerebral amyloid angiopathy and neurologic dysfunction and that such larg

71 l vessel disease (lobar for cerebral amyloid angiopathy and non-lobar for arteriolosclerosis), we per

72 ved in an inherited form of cerebral amyloid angiopathy and readily form amyloid fibrils in vitro.

73 etween complement dysregulation and systemic angiopathy and suggest that complement activation may co

74 tter and in artery walls as cerebral amyloid angiopathy and tau protein accumulates as neurofibrillar

75 to be in part a protein elimination failure angiopathy and that this dysfunction is a feed-forward p

76 here a striking correlation between amyloid angiopathy and the location of mutation in PS-1 linked A

77 lmonary perfusion likely caused by pulmonary angiopathy and thrombosis.

78 39 with clinically probable cerebral amyloid angiopathy) and 47 age-matched controls.

79 not Abeta42) levels, cerebrovascular amyloid angiopathy, and ApoE4 allele frequency were all highly c

80 ic small vessel disease and cerebral amyloid angiopathy, and determined the Braak tangle stage.

81 been linked to HF through perfusion defects, angiopathy, and inflammation, whether patients with AD p

82 amyloid loading, increased cerebral amyloid angiopathy, and reduced insulin-signaling activities.

83 ta and in the mechanisms of cerebral amyloid angiopathy, and support the possibility that modulation

84 t cortical amyloid plaques, cerebral amyloid angiopathy, and tauopathy.

85 ic for microhaemorrhages in cerebral amyloid angiopathy, and that increasing the resolution of magnet

86 bleeds and microinfarcts in cerebral amyloid angiopathy, and to explore the pathological burden that

87 of Alzheimer's disease and cerebral amyloid angiopathy, APOE has been studied in other neurological

88 e pathophysiological level, cerebral amyloid angiopathy appears to be in part a protein elimination f

89 Synaptic dysfunction and microvascular angiopathy are confirmed as early progression mechanisms

90 roimaging markers of severe cerebral amyloid angiopathy are cortical microbleeds and microinfarcts.

91 gh chronic hypertension and cerebral amyloid angiopathy are the underlying cerebral vasculopathies ac

92 th TDP-43 proteinopathy and cerebral amyloid angiopathy associate with cognitive impairment of simila

93 ninvasively detect isolated cerebral amyloid angiopathy before overt signs of tissue damage such as h

94 viously designated familial cerebral amyloid angiopathy-British type, is an autosomal dominant disord

95 uction of Abeta plaques and cerebral amyloid angiopathy burden and a reduction of the total brain lev

96 ls where there was a severe cerebral amyloid angiopathy burden.

97 ociated with a reduction of cerebral amyloid angiopathy but not amyloid plaques.

98 enotype was associated with cerebral amyloid angiopathy, but not HS or arteriosclerosis.

99 effects in TgSwDI mouse, a cerebral amyloid angiopathy (CAA) and AD model.

100 -beta (Abeta) deposition in cerebral amyloid angiopathy (CAA) and Alzheimer disease (AD) is arguably

101 In cerebral amyloid angiopathy (CAA) and Alzheimer's disease (AD), the amylo

102 ent infarcts, haemorrhages, cerebral amyloid angiopathy (CAA) and arteriosclerosis, were examined.

103 Cerebral amyloid angiopathy (CAA) and beta-amyloid (Abeta) deposition in

104 yloid-beta (Abeta) plaques, cerebral amyloid angiopathy (CAA) and neurofibrillary tangles.

105 along cerebrovasculature as cerebral amyloid angiopathy (CAA) are hallmarks of AD.

106 ncephalopathy (CADASIL) and cerebral amyloid angiopathy (CAA) are two distinct vascular angiopathies

107 itis of the CNS (PACNS) and cerebral amyloid angiopathy (CAA) are unusual vasculopathies generally re

108 th pathologically diagnosed cerebral amyloid angiopathy (CAA) for evidence of an inflammatory respons

109 Although cerebral amyloid angiopathy (CAA) has important clinical implications, ou

110 Cerebral amyloid angiopathy (CAA) has never been more relevant.

111 Targeting Abeta to prevent cerebral amyloid angiopathy (CAA) has not been rigorously followed, altho

112 ebral haemorrhage (ICH) and cerebral amyloid angiopathy (CAA) in a systematic review of published neu

113 n selectively induces acute cerebral amyloid angiopathy (CAA) in neonatally-injected transgenic CRND8

114 Sporadic cerebral amyloid angiopathy (CAA) is a common age related cerebral small

115 Cerebral amyloid angiopathy (CAA) is a common cause of brain hemorrhage i

116 Cerebral amyloid angiopathy (CAA) is a common cause of symptomatic intrac

117 orm of neuritic plaques and cerebral amyloid angiopathy (CAA) is a key feature of Alzheimer's disease

118 Cerebral amyloid beta-protein angiopathy (CAA) is a key pathological feature of patien

119 emorrhagic stroke, advanced cerebral amyloid angiopathy (CAA) is also associated with ischemic lesion

120 Cerebral amyloid angiopathy (CAA) is an age-associated condition and a co

121 Cerebral amyloid angiopathy (CAA) is associated with lobar intracerebral

122 Cerebral amyloid angiopathy (CAA) is characteristically associated with m

123 Cerebral amyloid angiopathy (CAA) is characterized by deposition of amylo

124 Cerebral amyloid angiopathy (CAA) is characterized by the accumulation of

125 Cerebral amyloid angiopathy (CAA) is characterized by the deposition of a

126 Cerebral amyloid angiopathy (CAA) is common in Alzheimer's disease (AD) a

127 Cerebral amyloid angiopathy (CAA) is common in the ageing brain and is as

128 Cerebral amyloid angiopathy (CAA) is the accumulation of amyloid-beta pep

129 Cerebral amyloid angiopathy (CAA) may play a key role in determining the

130 ajor trends in the field of cerebral amyloid angiopathy (CAA) over six decades, from 1954 to 2014, us

131 Cerebral amyloid angiopathy (CAA) results from deposition of beta-amyloid

132 but has no direct effect on cerebral amyloid angiopathy (CAA) severity, whereas APOEepsilon4 is assoc

133 ngles, neuropil threads and cerebral amyloid angiopathy (CAA) similar to unimmunized AD, but lacked p

134 pertensive arteriopathy and cerebral amyloid angiopathy (CAA)) is an important cause of spontaneous i

135 ve cerebrovascular amyloid (cerebral amyloid angiopathy (CAA), and cardiovascular risk factors increa

136 compact amyloid plaques, congophilic amyloid angiopathy (CAA), and diffuse Abeta deposits.

137 We postulated that cerebral amyloid angiopathy (CAA), characterised by cortical vascular amy

138 beta), a condition known as cerebral amyloid angiopathy (CAA), is a common pathological feature of pa

139 ssels, a condition known as cerebral amyloid angiopathy (CAA), is a key pathological feature of Alzhe

140 beta), a condition known as cerebral amyloid angiopathy (CAA), is a key pathological feature of Alzhe

141 lls of brain blood vessels, cerebral amyloid angiopathy (CAA), is common in patients with Alzheimer's

142 cerebral arteries, known as cerebral amyloid angiopathy (CAA), occurs both in the setting of Alzheime

143 w (Tg2576) mice, a model of cerebral amyloid angiopathy (CAA), suggesting a pivotal role for Bim in A

144 Cerebral amyloid angiopathy (CAA), the deposition of cerebrovascular beta

145 Cerebral amyloid angiopathy (CAA), where beta-amyloid (Abeta) deposits ar

146 ead to a condition known as cerebral amyloid angiopathy (CAA), which impairs blood-brain barrier (BBB

147 is increasing evidence that cerebral amyloid angiopathy (CAA), which itself can cause haemorrhage (CA

148 -9), in the pathogenesis of cerebral amyloid angiopathy (CAA)-induced spontaneous hemorrhage.

149 been reported to exacerbate cerebral amyloid angiopathy (CAA)-related microhemorrhage in a transgenic

150 nchymal amyloid plaques and cerebral amyloid angiopathy (CAA).

151 pertensive arteriopathy and cerebral amyloid angiopathy (CAA).

152 e primarily associated with cerebral amyloid angiopathy (CAA).

153 rtex and in blood vessel as cerebral amyloid angiopathy (CAA).

154 of Alzheimer's disease and cerebral amyloid angiopathy (CAA).

155 els of soluble Abeta but no cerebral amyloid angiopathy (CAA).

156 including familial forms of cerebral amyloid angiopathy (CAA).

157 diffuse plaques and cerebrovascular amyloid angiopathy (CAA).

158 in blood vessels is called cerebral amyloid angiopathy (CAA).

159 alls of cerebral vessels as cerebral amyloid angiopathy (CAA).

160 lzheimer's disease (AD) and cerebral amyloid angiopathy (CAA).

161 arance of a core AD lesion, cerebral amyloid angiopathy (CAA).

162 e characteristic markers of cerebral amyloid angiopathy (CAA).

163 y might be early markers of cerebral amyloid angiopathy (CAA).

164 al distribution in sporadic cerebral amyloid angiopathy (CAA).

165 n cerebral blood vessels as cerebral amyloid angiopathy (CAA).

166 increasingly recognised in cerebral amyloid angiopathy (CAA).

167 e cerebrovasculature [i.e., cerebral amyloid angiopathy (CAA)] and a heightened risk of micro-hemorrh

168 n the cerebral vasculature [cerebral amyloid angiopathy (CAA)] in the majority of patients.

169 ascular amyloid deposition (cerebral amyloid angiopathy [CAA]) but not in patients with high parenchy

170 deposition of beta-amyloid (cerebral amyloid angiopathy [CAA]) is a major cause of hemorrhagic stroke

171 ar beta-amyloid deposition (cerebral amyloid angiopathy, CAA) is associated with cerebral microbleeds

172 tential outcome markers for cerebral amyloid angiopathy can be measured against the ideal criteria of

173 Cerebral amyloid angiopathy can cause life-threatening brain haemorrhages

174 e wall of cerebral vessels (cerebral amyloid angiopathy), can lead to weakness and rupture of the ves

175 rgeted sampling combined in cerebral amyloid angiopathy cases.

176 In cerebral amyloid angiopathy cohorts, cortical superficial siderosis is as

177 Cerebral angiopathy contributes to cognitive decline and dementia

178 microbleeds suggesting that cerebral amyloid angiopathy contributes to the relationship between amylo

179 TDP-43 and amyloid angiopathy correlated with coexistent Alzheimer patholog

180 olipoprotein E subtypes and cerebral amyloid angiopathy; deep intracerebral hemorrhage and the potent

181 ition is characteristic for cerebral amyloid angiopathy deposition and maturation in both humans and

182 has on reducing the rate of cerebral amyloid angiopathy deposition and restoring cerebral vascular he

183 ates atherosclerotic progression, peripheral angiopathy development, and arterial hypertension, all o

184 eimer's disease and related cerebral amyloid angiopathy disorders.

185 isease and related familial cerebral amyloid angiopathy disorders.

186 , several familial forms of cerebral amyloid angiopathy exist including the Dutch (E22Q) and Iowa (D2

187 Familial British dementia with amyloid angiopathy (FBD) is an autosomal dominant condition char

188 et symptomatic amyloid-beta cerebral amyloid angiopathy following childhood exposure to cadaveric dur

189 (CADASIL)and some forms of cerebral amyloid angiopathy have a genetic basis.

190 CMBs related to symptomatic cerebral amyloid angiopathy have abnormal vascular reactivity and cerebra

191 hanism, microembolism and pulmonary vascular angiopathy have been noted in rats.

192 P23-144, associated with a heritable amyloid angiopathy, have previously been shown to contain a comp

193 Hereditary cerebral amyloid angiopathy (HCAA) is a rare familial form of CAA in whic

194 s include severe widespread cerebral amyloid angiopathy, hippocampal plaques, and neurofibrillary tan

195 alysis to definite/probable cerebral amyloid angiopathy ICH uncovered a stronger effect.

196 ffuse and focal plaques and cerebral amyloid angiopathy in humans and nonhuman primates, whereas it i

197 issue from a patient with TBI showed amyloid angiopathy in meningeal vessels.

198 ase in the number of new plaques and amyloid angiopathy in the area immediately surrounding the infar

199 Autopsy revealed cerebral amyloid angiopathy in the complete absence of amyloid plaques or

200 e and ADan (Danish Amyloid) cerebral amyloid angiopathy in the mouse model of FDD.

201 rphologic features and the role of choroidal angiopathy in various disease states that, in the future

202 ypertensive arteriopathy or cerebral amyloid angiopathy) in a multicentre, cross-sectional study.

203 ial Alzheimer's disease and cerebral amyloid angiopathy, in which patients develop neuritic plaques a

204 ologies-amyloid plaques and cerebral amyloid angiopathy-in immunohistochemically-stained archival sli

205 r Abeta plaques versus cerebral beta-amyloid angiopathy, indicating that different amyloid types and

206 sized that ponatinib produces an endothelial angiopathy involving excessive endothelial-associated vo

207 Cerebral amyloid angiopathy is a common form of small-vessel disease and

208 Cerebral amyloid angiopathy is a common neuropathological finding in the

209 Sporadic cerebral amyloid angiopathy is a common, well-defined small vessel diseas

210 lts suggest that underlying cerebral amyloid angiopathy is a contributing factor to the occurrence of

211 As cerebral amyloid angiopathy is an almost invariable pathological finding

212 Hereditary cystatin C amyloid angiopathy is an autosomal dominant disorder in which a

213 consequences; for example, cerebral amyloid angiopathy is an important cause of haemorrhagic stroke.

214 Cerebral amyloid angiopathy is characterized by accumulation of the short

215 sgenic mouse model of human cerebral amyloid angiopathy is characterized by age-dependent cerebrovasc

216 From a clinical standpoint, cerebral amyloid angiopathy is characterized by individual focal lesions

217 Cerebral amyloid angiopathy is common among elderly patients, and is asso

218 Cerebral amyloid angiopathy is commonly associated with normal aging and

219 Prominent cerebral amyloid angiopathy is often observed in the brains of elderly in

220 the presence and burden of cerebral amyloid angiopathy is particularly important when planning to st

221 , its involvement in AD and cerebral amyloid angiopathy is poorly understood.

222 rebrovascular beta-amyloid (cerebral amyloid angiopathy) is complicated by the nearly universal overl

223 ) deposition, also known as cerebral amyloid angiopathy, is a common pathological feature of Alzheime

224 ature, a condition known as cerebral amyloid angiopathy, is increasingly recognized as an important c

225 eal, cortical and capillary cerebral amyloid angiopathy, large infarcts, lacunar infarcts, microhaemo

226 pathologies-leptomeningeal cerebral amyloid angiopathy, large infarcts, lacunar infarcts, microinfar

227 , arteriolar sclerosis, and cerebral amyloid angiopathy), Lewy bodies, transactive response DNA-bindi

228 ques, gross infarcts, microinfarcts, amyloid angiopathy, Lewy bodies, APOE epsilon4 presence, and cli

229 rovascular abnormalities of cerebral amyloid angiopathy (lobar structures) and hypertensive vasculopa

230 in locations suggestive of cerebral amyloid angiopathy (lobar with or without cerebellar microbleeds

231 Evidence suggests that choroidal angiopathy may coexist with retinal vascular damage.

232 ere small vessel disease or cerebral amyloid angiopathy may contribute in some cases, abnormal vascul

233 Fourteen autopsy cases with cerebral amyloid angiopathy (mean age at death 73 years, nine males) and

234 without adversely affecting cerebral amyloid angiopathy, microhemorrhages, myelination, or neuromuscu

235 rther in vivo evidence that cerebral amyloid angiopathy might be caused by transmission of amyloid-be

236 re occipital leptomeningeal cerebral amyloid angiopathy, moderate/severe arteriolosclerosis in occipi

237 in in an age-dependent manner, forms amyloid angiopathy morphologically similar to that seen in the h

238 sized that in patients with cerebral amyloid angiopathy, multiple small spatially distributed lesions

239 ere small vessel disease or cerebral amyloid angiopathy, neither VEGF nor MAG:PLP1 correlated signifi

240 ephaly, small vessel disease, and hereditary angiopathy, nephropathy, aneurysms, and cramps (HANAC) s

241 ons of the transcriptional repressor TDP-43, angiopathy, neuron loss and gliosis.

242 the brain and give rise to cerebral amyloid angiopathy, neuronal dysfunction, and cellular toxicity.

243 The term 'cerebral amyloid angiopathy' now encompasses not only a specific cerebrov

244 is abundant in plaques and cerebral amyloid angiopathy of AD and Down syndrome patients, colocalizin

245 her TDP-43 proteinopathy or cerebral amyloid angiopathy of sufficient severity to independently expla

246 Benign angiopathy of the central nervous system (BACNS) is a su

247 t of predominantly advanced cerebral amyloid angiopathy of the leptomeningeal vessels and may trigger

248 nd in the vasculature (cerebral beta-amyloid angiopathy) of beta-amyloid precursor protein transgenic

249 of Alzheimer's disease and cerebral amyloid angiopathy on the structure of the 21-30 fragment of the

250 logy (small vessel disease, cerebral amyloid angiopathy or VWF).

251 ial Alzheimer's disease, congophilic amyloid angiopathy, or hereditary cerebral hemorrhage with amylo

252 teriovenous shunts, biopsy-proven microrenal angiopathy, or thrombocytopenia and thus were diagnosed

253 n, generally, but also to facilitate amyloid angiopathy particularly in cases in which the mutation l

254 tion of fibrinogen lessened cerebral amyloid angiopathy pathology and reduced cognitive impairment in

255 ntrast, atherosclerosis and cerebral amyloid angiopathy pathology were associated with a lower level

256 protein deposits in human cystatin C amyloid angiopathy patients have also been found in tissues outs

257 t not Braak tangle stage or cerebral amyloid angiopathy predicted (11)C-Pittsburgh compound B standar

258 (PRNP), associated with PrP cerebral amyloid angiopathy (PrP-CAA) and Gerstmann-Straussler-Scheinker

259 neurofibrillary tangle and cerebral amyloid angiopathy ratings in the whole sample and within indivi

260 tionship between markers of cerebral amyloid angiopathy-related brain injury, network efficiency, and

261 ated patients with sporadic cerebral amyloid angiopathy-related hemorrhage found no other instances o

262 ever, their contribution to cerebral amyloid angiopathy-related hemorrhage remains unclear.

263 Cerebral amyloid angiopathy-related inflammation (CAA-ri) is an important

264 Cerebral amyloid angiopathy-related inflammation (CAA-ri) is characterize

265 three times more common in cerebral amyloid angiopathy-related intracerebral haemorrhage than in oth

266 was 9/39 (23%) in probable cerebral amyloid angiopathy-related intracerebral haemorrhage versus 6/75

267 angiopathy and exacerbated cerebral amyloid angiopathy-related microhemorrhage in APP23/ABCA1-/- mic

268 l wall (an advanced form of cerebral amyloid angiopathy-related vascular damage) in leptomeningeal ve

269 e who were previously enrolled in the Bypass Angiopathy Revascularization Investigation to compare in

270 d they had a higher risk of cerebral amyloid angiopathy (RR 6.6, 1.5-29.6).

271 48; 95% CI, 1.06-5.82), and cerebral amyloid angiopathy severity (p = 0.032; OR, 4.16; 95% CI, 1.13-1

272 s (P < 0.0001), but reduced cerebral amyloid angiopathy severity in cortical vessels (P = 0.048).

273 dense-core (senile) plaques, in the amyloid angiopathy surrounding diseased blood vessels, and in th

274 myloid-beta plaque load and cerebral amyloid angiopathy than early disease onset and tremor dominant

275 d angiopathy (CAA) are two distinct vascular angiopathies that share several similarities in clinical

276 s may facilitate development of beta-amyloid angiopathy that is more frequent in varepsilon 4/ vareps

277 Fibromuscular dysplasia is an uncommon angiopathy that occurs in young to middle-aged, predomin

278 telet adhesion and a secondary microvascular angiopathy that produces ischemic wall motion abnormalit

279 d from brain blood vessels with amyloid-beta angiopathy - the cells that were shown previously to acc

280 acerebral hemorrhage due to cerebral amyloid angiopathy, the result of beta-amyloid deposition in cer

281 uding microinfarcts, microhemorrhages, bland angiopathy, thrombotic angiopathy with platelet and fibr

282 in network in patients with cerebral amyloid angiopathy to healthy control subjects and examined the

283 nsgenic mice with prominent cerebral amyloid angiopathy to investigate the ability of ponezumab, an a

284 the candidate outcomes for cerebral amyloid angiopathy trials are probably applicable also to other

285 essels from subjects having cerebral amyloid angiopathy was 90 degrees out of phase from that in the

286 Instead, a global microvascular angiopathy was detected by immunohistochemistry and by i

287 Amyloid angiopathy was present in these cases and was readily de

288 S1 and APP/PS1/AI mice, but cerebral amyloid angiopathy was reduced in APP/PS1/AI mice.

289 s of hippocampal Abeta, and cerebral amyloid angiopathy was significantly more common compared with P

290 ffect of Apoa-I deletion on cerebral amyloid angiopathy, we measured insoluble Abeta isolated from ce

291 s, mainly according to the degree of amyloid angiopathy, were seen in the cerebellum.

292 at least partly, related to cerebral amyloid angiopathy, which belongs to the continuum of cerebral s

293 e have been associated with cerebral amyloid angiopathy, while those in the basal ganglia have been a

294 cause a hereditary form of cerebral amyloid angiopathy whilst cystatin B aggregates are found in cas

295 the proband revealed severe cerebral amyloid angiopathy, widespread neurofibrillary tangles, and unus

296 n with loss of cholinergic neurons, gliosis, angiopathy with mononuclear cell cuffing, progressive ca

297 isease, familial porencephaly and hereditary angiopathy with nephropathy aneurysm and cramps (HANAC)

298 crohemorrhages, bland angiopathy, thrombotic angiopathy with platelet and fibrin thrombi, neuronal ne

299 develop severe early-onset cerebral amyloid angiopathy with some of the related variants manifesting

300 e expected increase in prevalence of amyloid angiopathy with the ageing population, suggest that, in