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1 cal angiotensin II production and the type 2 angiotensin receptor.
2 asoconstriction via the activation of type 1 angiotensin receptors.
3 ing from elevated angiotensin II (AngII) and angiotensin receptor 2 (AT2R) producing increased plasma
4 1)-AA), stimulates sEng production via AT(1) angiotensin receptor activation in pregnant mice but not
5 covered that a circulating autoantibody, the angiotensin receptor agonistic autoantibody (AT(1)-AA),
6 onism of MAS1 receptors, beta-arrestin-based Angiotensin receptor agonists, and administration of sol
8 embranes, which is distinctly different from angiotensin receptors and key proteases processing angio
9 colleagues present evidence that implicates angiotensin receptors and the relocation of beta-catenin
10 tural basis of the distinct functions of the angiotensin receptors, and may guide the design of new s
11 f angiotensin-converting enzyme inhibitor or angiotensin receptor antagonist (88.3% versus 86.6%), an
13 ll three isoforms of TGF-beta), losartan (an angiotensin receptor antagonist), or a combination of th
14 tumor's inner core, AD was combined with an angiotensin receptor antagonist, Telmisartan (TS), which
15 Heart Failure) trial demonstrated that a new angiotensin receptor antagonist-neprilysin inhibitor was
16 ay lead to the development of a new class of angiotensin receptor antagonists with activities biased
17 rs, calcium channel blockers, diuretics, and angiotensin receptor antagonists), smoking status, alcoh
18 of angiotensin-converting enzyme inhibitors/angiotensin receptor antagonists, use of beta-blocker, a
21 n and termination of signaling of the type I angiotensin receptor (AT(1)-R) can lead to dynamic chang
24 of beta-arrestins in cross-talk between the angiotensin receptor (AT1aR) and a member of the transie
28 odocytes, which was prevented by concomitant angiotensin receptor blockade application and TRPC6 knoc
29 hat inhibition of TGF-beta signaling through angiotensin receptor blockade can attenuate CS-induced l
30 ly, the addition of neprilysin inhibition to angiotensin receptor blockade has been shown to be even
31 erglycemia and the antiproteinuric effect of angiotensin receptor blockade or angiotensin-converting
32 injections (RR 0.45; 95% CI 0.24-0.83), and angiotensin receptor blockade vs placebo (RR 0.65; 95% C
33 egradation inhibition through neprilysin and angiotensin receptor blockade, has led to groundbreaking
37 combination therapy with NEP-inhibitors and angiotensin-receptor-blockade, which has been shown bein
38 otensin-converting enzyme (ACE) inhibitor or angiotensin receptor blocker (ARB) affected the results.
39 ]: 1.04 to 1.10) and discharge ACE inhibitor/angiotensin receptor blocker (ARB) in LV dysfunction (64
40 tensin-converting enzyme inhibitor (ACEI) or angiotensin receptor blocker (ARB) influences disease ou
43 ology study uses claims data to characterize angiotensin receptor blocker (ARB) prescription trends t
44 ensin-converting enzyme inhibitor (ACEI) and angiotensin receptor blocker (ARB) should be used for se
45 ed in increased cancer risk, with a focus on angiotensin receptor blocker (ARB) therapy, as recent pu
46 giotensin-converting enzyme inhibitor (ACEI)/angiotensin receptor blocker (ARB) use and mortality in
47 otensin-converting enzyme (ACE) inhibitor or angiotensin receptor blocker (ARB) use, ESA use, dialysi
48 sing angiotensin-converting enzyme inhibitor/angiotensin receptor blocker (beta=0.36, P<0.001; CI: 0.
50 : high dose, 0.55; low dose, 0.72; both ACEi/angiotensin receptor blocker and beta-blocker: high dose
52 ate, angiotensin-converting enzyme inhibitor/angiotensin receptor blocker and oral anti-diabetic agen
54 angiotensin-converting enzyme inhibitor, or angiotensin receptor blocker as foundational therapy, wi
57 angiotensin-converting enzyme inhibitor and angiotensin receptor blocker groups resulted in two simi
58 ted, whereas acute HF and patients receiving angiotensin receptor blocker had higher plasma Ang II wi
59 been demonstrated that Valsartan (Val) as an angiotensin receptor blocker has renoprotective effects,
60 (angiotensin-converting enzyme) inhibitor or angiotensin receptor blocker if left ventricular ejectio
62 n angiotensin-converting enzyme inhibitor or angiotensin receptor blocker in persons with CKD to impr
67 giotensin receptor neprilysin inhibitor with angiotensin receptor blocker on Management Of heart fail
68 ting the renoprotective benefit of adding an angiotensin receptor blocker or a mineralocorticoid rece
70 d angiotensin-converting enzyme inhibitor or angiotensin receptor blocker therapy, 100% beta-blocker
71 d angiotensin-converting enzyme inhibitor or angiotensin receptor blocker therapy, 35% beta-blocker t
72 d angiotensin-converting enzyme inhibitor or angiotensin receptor blocker therapy, 51% beta-blocker t
79 r angiotensin-converting enzyme inhibitor or angiotensin receptor blocker usage during continuous flo
80 d angiotensin converting enzyme inhibitor or angiotensin receptor blocker usage were not significantl
81 .5%, angiotensin-converting enzyme inhibitor/angiotensin receptor blocker use in 24.9+/-1.9%, and asp
82 and angiotensin-converting enzyme inhibitor/angiotensin receptor blocker use in patients with an Ame
83 s in angiotensin-converting enzyme inhibitor/angiotensin receptor blocker use or anticoagulation for
84 , angiotensin-converting enzyme inhibitor or angiotensin receptor blocker use, and nonsmoking status-
85 Angiotensin-converting enzyme inhibitor or angiotensin receptor blocker use, beta-blocker use, anti
86 angiotensin-converting enzyme inhibitor use, angiotensin receptor blocker use, BMI z-score for age an
87 and angiotensin-converting enzyme inhibitor/angiotensin receptor blocker use-is well suited to provi
88 rketed by Novartis as Entresto) combines the angiotensin receptor blocker valsartan and the neprilysi
89 RNi with neprilysin inhibitor sacubitril and angiotensin receptor blocker valsartan on myocardial rem
90 loped by scientists at Novartis, combines an angiotensin receptor blocker with a neprilysin inhibitor
91 angiotensin converting enzyme inhibitor, and angiotensin receptor blocker), and N-terminal probrain n
92 nsin-converting enzyme) inhibitor or an ARB (angiotensin receptor blocker), at maximal or maximally t
93 tensin-converting enzyme inhibitor (ACEi, or angiotensin receptor blocker), beta-blocker, or both dru
94 f an angiotensin converting enzyme inhibitor/angiotensin receptor blocker, and hydrochlorothiazide 12
95 for angiotensin-converting enzyme inhibitor/angiotensin receptor blocker, beta-blocker, and mineralo
96 ent (angiotensin-converting enzyme inhibitor/angiotensin receptor blocker, beta-blocker, and mineralo
97 n patients receiving higher doses of ACEi or angiotensin receptor blocker, beta-blocker, or both (haz
98 an angiotensin-converting enzyme inhibitor, angiotensin receptor blocker, calcium channel blocker, o
99 DMT (angiotensin-converting enzyme inhibitor/angiotensin receptor blocker, or beta-blocker) at baseli
100 ngiotensin-converting enzyme inhibitor or an angiotensin receptor blocker, should generally be includ
106 rd ratio for eplerenone versus placebo, ACEi/angiotensin receptor blocker: high dose, 0.67; low dose,
107 sin Receptor-Neprilysin Inhibitor] with ARB [Angiotensin Receptor Blocker] Global Outcomes in HFpEF),
108 tly less likely after dual treatment with an angiotensin-receptor blocker (ARB) and an angiotensin-co
109 erting-enzyme inhibitor (lisinopril) plus an angiotensin-receptor blocker (telmisartan) or lisinopril
110 st orally active direct renin inhibitor, the angiotensin-receptor blocker losartan, and their combina
111 scle fibrosis and improved pain Losartan, an angiotensin-receptor blocker with anti-fibrotic abilitie
112 in-converting enzyme inhibitor) or losartan (angiotensin-receptor blocker) in FSGS mice stimulated th
114 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers (18.2% vs 16.9%, p = 1.000
115 for angiotensin-converting enzyme inhibitors/angiotensin receptor blockers (66% versus 68%; P=0.04) a
116 and angiotensin converting enzyme inhibitors/angiotensin receptor blockers (ACEI/ARB) in acute myocar
119 ensin-converting enzyme inhibitors (ACEI) or angiotensin receptor blockers (ARB) are generally well t
120 nsin-converting enzyme inhibitors (ACEI) and angiotensin receptor blockers (ARB) doses on outcomes in
121 ensin-converting enzyme inhibitors (ACEI) or angiotensin receptor blockers (ARB) initiated after myoc
122 tensin-converting enzyme (ACE) inhibitors or angiotensin receptor blockers (ARB) were associated with
123 otensin-converting enzyme inhibitors (ACEI), angiotensin receptor blockers (ARB), beta-blockers (BB),
125 tensin-converting enzyme (ACE) inhibitors or angiotensin receptor blockers (ARBs) and beta blockers.
126 nin angiotensin system pathways suggest that angiotensin receptor blockers (ARBs) are ideal drugs to
127 nsin-converting enzyme inhibitors (ACEIs) or angiotensin receptor blockers (ARBs) are widely prescrib
128 sin-converting-enzyme inhibitors (ACE-Is) or angiotensin receptor blockers (ARBs) both increased sign
131 ertensive patients (n = 469), patients using angiotensin receptor blockers (ARBs) did not show a decl
132 PURPOSE OF REVIEW: As their introduction, angiotensin receptor blockers (ARBs) have been widely pr
133 tensin-converting enzyme (ACE) inhibitors or angiotensin receptor blockers (ARBs) in patients with ty
134 has revealed that ACE inhibitors (ACEIs) or angiotensin receptor blockers (ARBs) may be beneficial i
135 sin-converting enzyme inhibitors (ACEis) and angiotensin receptor blockers (ARBs) may increase the ri
136 otensin-converting enzyme inhibitors (ACEIs)/angiotensin receptor blockers (ARBs) may make patients m
137 in-converting enzyme inhibitors (ACE-Is) and angiotensin receptor blockers (ARBs) on the composite of
138 iotensin-converting enzyme (ACE) inhibitors, angiotensin receptor blockers (ARBs), and direct renin (
139 iotensin converting enzyme (ACE) inhibitors, angiotensin receptor blockers (ARBs), beta blockers, and
140 otensin-converting enzyme inhibitors (ACEI), angiotensin receptor blockers (ARBs), beta-blockers, and
141 target the renin angiotensin system, such as angiotensin receptor blockers (ARBs), have been associat
142 in-converting enzyme inhibitors (ACE-Is) and angiotensin receptor blockers (ARBs), may be associated
144 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers (HR, 0.90; 95% CI, 0.79-1.
145 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers (ie, renin-angiotensin sys
146 for angiotensin-converting enzyme inhibitors/angiotensin receptor blockers (median, 85%; interquartil
147 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers (p = 0.029), higher biliru
149 tensin-converting enzyme inhibitors [ACEIs], angiotensin receptor blockers [ARBs], and beta-blockers
150 nsin-converting enzyme inhibitors [ACEIs] or angiotensin receptor blockers [ARBs], and cilostazol) an
151 iotensin-converting enzyme inhibitors and/or angiotensin receptor blockers achieves only partial reno
152 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers and 82% were receiving bet
153 expression is ameliorated by antiproteinuric angiotensin receptor blockers and angiotensin-converting
154 ith angiotensin-converting enzyme inhibitors/angiotensin receptor blockers and beta-blockers and an o
155 use angiotensin-converting enzyme inhibitors/angiotensin receptor blockers and beta-blockers compared
156 Angiotensin-converting enzyme inhibitors or angiotensin receptor blockers and beta-blockers were pre
158 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers at admission), beta-blocke
159 of angiotensin-converting enzyme inhibitors/angiotensin receptor blockers at discharge decreased in
160 and angiotensin-converting enzyme inhibitors/angiotensin receptor blockers decreased from 89%, 84.9%,
161 f angiotensin-convering enzyme inhibitors or angiotensin receptor blockers during the study and a his
162 cardiovascular examples, such as the use of angiotensin receptor blockers for chronic heart failure,
163 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers for more than 90 days betw
164 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers for secondary prevention a
165 (3) angiotensin-converting enzyme inhibitor/angiotensin receptor blockers for systolic dysfunction,
166 he therapeutic benefits of beta-blockers and angiotensin receptor blockers given the emerging concept
167 angiotensin-converting enzyme inhibitors and angiotensin receptor blockers have demonstrated benefici
170 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers in patients with COVID-19.
171 ngiotensin-converting enzyme inhibitors, and angiotensin receptor blockers in patients with heart fai
172 ngiotensin-converting enzyme inhibitors, and angiotensin receptor blockers increased by 23%, 57%, 31%
173 ensin converting enzyme (ACE) inhibitors and angiotensin receptor blockers increases the likelihood o
174 and angiotensin-converting enzyme inhibitors/angiotensin receptor blockers on outcome in patients wit
175 angiotensin-converting enzyme inhibitors and angiotensin receptor blockers on the risk and outcomes o
176 he renin-angiotensin-aldosterone system with angiotensin receptor blockers or angiotensin-converting
177 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers requires further investiga
178 for angiotensin-converting enzyme inhibitors/angiotensin receptor blockers therapy, 1.08 (95% confide
179 angiotensin-converting enzyme inhibitors and angiotensin receptor blockers use, was similar between c
181 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers was indicated in 18.1% of
183 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers with well controlled BP an
184 ensin-converting enzyme) inhibitors or ARBs (angiotensin receptor blockers) with beta-blockers (BBs)
185 ing angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, and 4.5 million not recei
186 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers, and aldosterone antagonis
187 aneurysmal disease, including beta-blockers, angiotensin receptor blockers, and angiotensin-convertin
188 ns, angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, and beta-blockers, respec
190 of angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, and more sunscreen use in
191 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers, and P2Y12 antagonists) re
192 rs, angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, and statins after acute m
193 rs, angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, and statins reduces cardi
194 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers, antiplatelet drugs, beta-
195 used in the clinic AT(1)R antagonist drugs (angiotensin receptor blockers, ARBs, or sartans) at prev
196 angiotensin-converting enzyme inhibitors or angiotensin receptor blockers, beta-blockers, aldosteron
197 of angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, beta-blockers, aldosteron
198 Angiotensin-converting enzyme inhibitors, angiotensin receptor blockers, beta-blockers, and aldost
199 ns, angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, beta-blockers, and dual a
200 ns, angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, beta-blockers, and dual a
201 sed angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, beta-blockers, and statin
202 (angiotensin-converting enzyme inhibitors or angiotensin receptor blockers, beta-blockers, spironolac
203 s, angiotensin-converting enzyme inhibitors, angiotensin receptor blockers, dihydropyridine calcium c
205 of angiotensin-converting enzyme inhibitors, angiotensin receptor blockers, or sacubitril-valsartan.
206 rs, angiotensin-converting enzyme inhibitors/angiotensin receptor blockers, P2Y12 inhibitors, and the
207 -blockers, angiotensin-converting inhibitors/angiotensin receptor blockers, statins, diabetic treatme
222 ing angiotensin-converting enzyme inhibitors/angiotensin receptors blockers, beta-blockers, and devic
223 (ACE) inhibitors (1.08, 1.02-1.15, p=0.008), angiotensin-receptor blockers (1.16, 1.07-1.25, p=0.0002
224 A potential association between the use of angiotensin-receptor blockers (ARBs) and angiotensin-con
227 ensin-converting-enzyme (ACE) inhibitors and angiotensin-receptor blockers (ARBs) decreases proteinur
228 sin-converting enzyme inhibitors (ACEIs) and angiotensin-receptor blockers (ARBs) in coronavirus dise
229 ensin-converting-enzyme (ACE) inhibitors and angiotensin-receptor blockers (ARBs) in this clinical co
230 ensin-converting enzyme (ACE) inhibitors and angiotensin-receptor blockers (ARBs) reduce cardiovascul
231 tensin-converting enzyme (ACE) inhibitors or angiotensin-receptor blockers (ARBs) who filled brand-na
232 ensin-converting-enzyme (ACE) inhibitors and angiotensin-receptor blockers (ARBs), 3-hydroxy-3-methyl
233 tensin-converting-enzyme (ACE) inhibitors or angiotensin-receptor blockers (ARBs), and beta blockers
234 iotensin-converting enzyme (ACE) inhibitors, angiotensin-receptor blockers (ARBs), beta blockers, and
235 allergic myocardial infarction, anaphylaxis, angiotensin-receptor blockers (ARBs), beta-adrenergic bl
237 ean BP reduction 12.9/7.7 mm Hg; p < 0.003), angiotensin-receptor blockers (mean BP reduction 13.3/7.
238 angiotensin-converting enzyme inhibitors or angiotensin-receptor blockers after SAVR for severe AS b
239 angiotensin-converting enzyme inhibitors or angiotensin-receptor blockers after surgical aortic valv
240 (angiotensin-converting enzyme inhibitors or angiotensin-receptor blockers and statins), and adverse
241 (angiotensin-converting enzyme inhibitors or angiotensin-receptor blockers and statins), and adverse
242 angiotensin-converting enzyme inhibitors and angiotensin-receptor blockers are not associated with gr
244 re angiotensin-converting enzyme inhibitors, angiotensin-receptor blockers, alpha-blockers, beta-bloc
245 irm the beneficial effect of ACE-inhibitors, angiotensin-receptor blockers, and diuretics and/or beta
246 of angiotensin-converting enzyme inhibitors, angiotensin-receptor blockers, beta blockers, calcium-ch
247 ng angiotensin-converting-enzyme inhibitors, angiotensin-receptor blockers, beta-blockers, and minera
248 ween previous treatment with ACE inhibitors, angiotensin-receptor blockers, beta-blockers, calcium-ch
249 ., angiotensin-converting enzyme inhibitors, angiotensin-receptor blockers, calcium channel blockers,
253 angiotensin-converting enzyme inhibitors or angiotensin receptor blocking agents 85.3% versus 77.4%
254 ssociated with increased activation of brain angiotensin receptors, enhanced sympathetic nervous syst
255 sms for sexual dimorphism of regional aortic angiotensin receptor expression and AAA formation are un
258 e findings suggest that activation of type 1 angiotensin receptors in the glomerulus is sufficient to
259 ies suggest that hyperactivation of brain AT angiotensin receptors is a major pathophysiological fact
260 In murine systems, there is a second type 1 angiotensin receptor isoform, AT1B, and its expression i
261 that are being probed include, among others, angiotensin receptors, matrix metalloproteinases, integr
263 e based on type 2 diabetes status, age, sex, angiotensin receptor neprilysin inhibitor (ARNI) treatme
264 icacy and safety of LCZ696, a first-in-class angiotensin receptor neprilysin inhibitor (ARNI), in pat
265 eptide and left atrial size suggest that the angiotensin receptor neprilysin inhibitor LCZ696 may red
266 ioN fracTion (PARAMOUNT) trial, in which the angiotensin receptor neprilysin inhibitor LCZ696 reduced
267 ents with heart failure (HF) treated with an angiotensin receptor neprilysin inhibitor lived longer w
270 rtality and Morbidity in Heart Failure), the angiotensin receptor neprilysin inhibitor sacubitril/val
271 on enrolled in the Prospective comparison of angiotensin receptor neprilysin inhibitor with angiotens
273 ACEI)/angiotensin II receptor blocker (ARB), angiotensin receptor-neprilysin inhibitor (ARNI), beta-b
275 randomized in the Prospective Comparison of Angiotensin Receptor-Neprilysin Inhibitor With an Angiot
276 from PARADIGM-HF (Prospective Comparison of Angiotensin Receptor-Neprilysin Inhibitor With Angiotens
277 ence in the rate of mortality when comparing angiotensin receptor-neprilysin inhibitor+BB+mineralocor
278 c defibrillator+ACE inhibitor or ARB+BB, and angiotensin receptor-neprilysin inhibitor+BB+mineralocor
279 be treated with a beta-blocker and one of an angiotensin receptor-neprilysin inhibitor, angiotensin-c
281 GM-HF trial (Prospective Comparison of ARNI [Angiotensin Receptor-Neprilysin Inhibitor] With ACEI [An
283 PARADIGM-HF (Prospective Comparison of ARNI [Angiotensin Receptor-Neprilysin Inhibitor] with ACEI [An
284 PARAGON-HF (Prospective Comparison of ARNI [Angiotensin Receptor-Neprilysin Inhibitor] with ARB [Ang
285 alocorticoid receptor antagonists (MRA), and angiotensin receptor-neprilysin inhibitors (ARNI), have
286 neralocorticoid receptor antagonists [MRAs], angiotensin receptor-neprilysin inhibitors [ARNIs], and
287 ive efficacy of pharmacological therapy with angiotensin receptor-neprilysin inhibitors and device th
289 ening in the Prospective Comparison of ARNI (angiotensin-receptor-neprilysin inhibitor) with ACEI (an
290 duced ejection fraction treatment with ARNI (angiotensin receptor/neprilysin inhibitor) therapy or le
291 ystems diacylglycerol produced during type 1 angiotensin receptor signaling can be converted to 2-ara
292 al mechanism for biased ligand action at the angiotensin receptor that can be exploited to rationally
293 th an animal model of PTSD and the selective angiotensin receptor type 1 (AT1) antagonist losartan, w
294 played decreased fibrosis in response to the angiotensin receptor type 1 blocker losartan showed decr
296 derlies increased placental HIF-1alpha in an angiotensin receptor type 1 receptor agonistic autoantib
298 eptors different from AT(1)R, in particular, angiotensin receptor type II (AT(2)R), resulting in biol
300 ance between angiotensin II, which activates angiotensin receptor types 1 and 2, and angiotensin 1-7