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1  pupil diameter, minimum pupil diameter, and anisocoria.
2 y, at baseline and after pilocarpine-induced anisocoria.
3    Sixty-three percent experienced new onset anisocoria.
4 ty between eyes also distinguished new onset anisocoria accompanied by objective evidence of abnormal
5                                    New onset anisocoria accompanied by objective evidence of abnormal
6                                    New onset anisocoria accompanied by objective evidence of abnormal
7  of abnormal pupil reactivity from new onset anisocoria accompanied by objective evidence of normal p
8 associated with midline shift, and new onset anisocoria accompanied by objective evidence of normal p
9                  The occurrence of new onset anisocoria accompanied by objective evidence of normal p
10 ristics associated with first-time new onset anisocoria and nonnew onset anisocoria at preceding obse
11                   In pediatric patients with anisocoria, apraclonidine is used off-label to exclude H
12 t-time new onset anisocoria and nonnew onset anisocoria at preceding observations using mixed effects
13                                     However, anisocoria correlated with RAPD only in subjects with da
14                                  Contraction anisocoria describes a phenomenon in which the pupil of
15 ly/specifically predicts clinically relevant anisocoria, enabling possible earlier treatments.
16 ifference distinguished first-time new onset anisocoria from nonnew onset anisocoria in up to four pr
17           Lastly, the localizing pitfalls of anisocoria--Horner and Raeder syndromes, physiologic ani
18 topical alpha-2 adrenergic agonist, reverses anisocoria in patients with Horner syndrome, a disruptio
19 -time new onset anisocoria from nonnew onset anisocoria in up to four preceding pupil observations (o
20 nvestigate the lateralization of contraction anisocoria in young female and male subjects.
21 ed to ascertain how a clinically significant anisocoria influences the relative afferent pupil defect
22 se results provide evidence that contraction anisocoria is more laterally asymmetric in males than in
23 uch right-side lateralization of contraction anisocoria is much greater in males than in females.
24 l fractures; unexplained neurologic deficit; anisocoria; lateral neck soft tissue injury; clinical su
25 d with narrow palpebral fissure, dysarthria, anisocoria (narrower pupil on the right side), unilatera
26                        Values of contraction anisocoria obtained by stimulating a subject's right or
27 tive pupil characteristics precede new onset anisocoria occurrence and may allow for earlier predicti
28 pil reactivity prior to first-time new onset anisocoria occurrence.
29                                    New onset anisocoria occurs in over 60% of patients with neurologi
30  in understanding the potential influence of anisocoria on the RAPD and also greater susceptibility o
31 des with elevated intracranial pressure (new anisocoria or intracranial pressure >20 mm Hg for >/=20
32 racteristic of clinically relevant new onset anisocoria phenotypes.
33                                      Induced anisocoria produced a significant change in RAPD from ba
34 ia--Horner and Raeder syndromes, physiologic anisocoria, pupil-involving third nerve palsy, and benig
35 ting the right eye led to larger contraction anisocoria than stimulating the left eye.
36 abetic patients with third-nerve palsies had anisocoria up to 2.5 mm.
37                                  Contraction anisocoria was calculated by subtracting relative constr
38  including maximum and minimum diameter, and anisocoria were measured.