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1  appropriate direction to compensate for the anisometropia.
2  refractive error, irregular astigmatism, or anisometropia.
3 ation was not associated with astigmatism or anisometropia.
4 e corneal power can lead to great amounts of anisometropia.
5 , or astigmatism, are more likely to develop anisometropia.
6 onstrated an improvement in the magnitude of anisometropia.
7  in children and that it appears to decrease anisometropia.
8 rs, 2593 children (7.7%) were diagnosed with anisometropia.
9 stent (SCS) devices, as well as incidence of anisometropia.
10  to lens opacity or the existence of a large anisometropia.
11 lity of exotropia, presence of amblyopia and anisometropia.
12 teria for myopia, hyperopia, astigmatism, or anisometropia.
13 ibromas, most commonly because of ptosis and anisometropia.
14 idual amblyopia resulting from strabismus or anisometropia.
15 terality, ocular dominance, and magnitude of anisometropia.
16 ignificant association with the magnitude of anisometropia.
17  >or=0.5 D of hyperopic, myopic, or cylinder anisometropia.
18 c anisometropia and >or=2 to 3 D of cylinder anisometropia.
19 dditional monkeys developed persistent axial anisometropias.
20                                Children with anisometropia 1.00 D, hyperopia +3.00 D, myopia -3.00D,
21 ts of SA (+/- 0.2 and +/- 0.4 mum) and fixed anisometropia (1.5 D).
22 r than +3 D, astigmatism 2 D or more, and/or anisometropia 2 D or more, were found in 25.6% of the ch
23 7%) had an amblyogenic factor, most commonly anisometropia (32.8%).
24 R lines) occurred, with >or=1 D of hyperopic anisometropia and >or=2 to 3 D of cylinder anisometropia
25 ed to assess variations in the prevalence of anisometropia and aniso-astigmatism by age group, with l
26  We describe the profile and associations of anisometropia and aniso-astigmatism in a population-base
27                                              Anisometropia and aniso-astigmatism were more common in
28  logistic regression used to compare odds of anisometropia and aniso-astigmatism with refractive stat
29 ia, particularly during the first 1.3 years; anisometropia and astigmatism also increased with age.
30 y to show an independent association between anisometropia and both spherical ametropia and astigmati
31 amage as well as from amblyopia arising from anisometropia and corneal opacification.
32 ulation, there is a high prevalence of axial anisometropia and corneal/axial aniso-astigmatism, assoc
33 hood commonly arising from strabismus and/or anisometropia and leading to dysfunctions in visual cort
34 is population to promptly diagnose and treat anisometropia and prevent potential visual complications
35 isometropia (n = 20) or associated with both anisometropia and strabismus (n = 33).
36 s (n = 34), anisometropia (n = 23), and both anisometropia and strabismus (n = 37).
37 tropia [n = 18]); strabismus [n = 22]); both anisometropia and strabismus [n = 32]) completed the occ
38 esolution fMRI to distinguish the impacts of anisometropia and strabismus amblyopia on the evoked ocu
39 nt hyperopia and astigmatism, in addition to anisometropia and strabismus, were the major amblyogenic
40 ecially when judged by its ability to detect anisometropia and the repeatability of the results.
41 emmetropia (hyperopia </=1.0 D; astigmatism, anisometropia, and myopia <1.0 D).
42 opic eyes of children with strabismus and/or anisometropia, and the associated poor stereoacuity prob
43 tween the 2 eyes, in addition to strabismus, anisometropia, and/or visual axis obstruction; bilateral
44 8 high-light-reared monkeys developed myopic anisometropias, and in 6 of these monkeys, the form-depr
45  the past year underscored the importance of anisometropia as a risk factor for amblyopia.
46 es were based on spherical equivalent (SEQ), anisometropia, astigmatism, and age (corrected for gesta
47                             Individuals with anisometropia, astigmatism, and hyperopia are more likel
48 status, birth weight, maternal age at birth, anisometropia, astigmatism, spherical equivalent, low vi
49              We studied the myopic shift and anisometropia at 10.6 (+/-0.3) years of age in the Infan
50                                The degree of anisometropia at 12 to 15 years was significantly associ
51                  To report the prevalence of anisometropia at age 5 years after unilateral intraocula
52 ajority of pseudophakic eyes had significant anisometropia at age 5 years.
53                       The threshold level of anisometropia at which unilateral amblyopia became signi
54 erocular difference in macular thickness and anisometropia, axial length, and VA.
55 en macular thickness and magnitude of myopic anisometropia, axial length, and visual acuity (VA) were
56  the use of stringent inclusion criteria for anisometropia, because otherwise a large proportion of t
57 hildren aged 1 to 6 years, initially without anisometropia but showing increasing severity of myopia,
58  patients with infantile onset strabismus or anisometropia can develop fusion maldevelopment nystagmu
59 tion, and higher prevalence of amblyopia and anisometropia compared to the intermittent cases.
60 nted to the dominant eye of patients without anisometropia, consistent with classical strabismic supp
61  across both sexes and when using a stricter anisometropia criterion.
62 he type and severity of refractive error and anisometropia development in preschool children.
63 dition to excessive eye growth may exist for anisometropia development, especially in hyperopia.
64   TMV was simulated with 1.5 diopters (D) of anisometropia (dominant eye at distance, nondominant eye
65 iopter contact lenses in one eye to simulate anisometropia during the first months of life, macaques
66 mal random-dot stereopsis, and hypermetropic anisometropia each pose a significant risk for the devel
67 y similar (treated eyes: F = 0.31, P = 0.74; anisometropia: F = 0.61, P = 0.59), but significantly di
68 as more useful as a screener, especially for anisometropia for which it was 91% sensitive and 92% spe
69 ucoma in the treated eye (n = 9) had greater anisometropia (glaucoma, median -8.25 D; IQR -11.38, -5.
70               Adjusted odds ratios (ORs) for anisometropia gradually increased with baseline refracti
71 eropia >/= 4.5 D, astigmatism >/= 2.0 D, and anisometropia &gt;/= 2.0 D.
72                            The prevalence of anisometropia &gt;/=1 diopters sphere (DS) did not differ s
73                    Spherical and cylindrical anisometropia &gt;/=1.00 D were each found in 26% of those
74                                Children with anisometropia &gt;=1.00 D, hyperopia >=+3.00 D, myopia >=-3
75 5 D), astigmatism (> 0.5 D of cylinder), and anisometropia (&gt; 1.0 D between eyes) increased with age.
76 o 74 years (OR, 0.14 [0.05-0.4], P < 0.001); anisometropia (&gt;1.0 diopters [D]: OR, 3.61 [2.32-5.62],
77 ical equivalent of < +3.00 D and significant anisometropia had a 7.8-fold increased risk for accommod
78 opia between the two groups, strabismus, and anisometropia had differential impacts on the OD bias, b
79 ies on the treatment of exotropia related to anisometropia have demonstrated less favorable outcomes.
80 , prematurity, smoking throughout pregnancy, anisometropia, hyperopia, and inheritance.
81 = 0.59), and better than the optometrist for anisometropia (ICC = 0.38).
82 logy of the MFS was esotropia in 58 (92.1%), anisometropia in 2 (3.2%), and exotropia in 3 patients (
83 ) made amblyopic by artificial strabismus or anisometropia in early life, as well as two visually nor
84 es, and the highest rates of astigmatism and anisometropia in Hispanics.
85 ical equivalent anisometropia or cylindrical anisometropia in identifying preschoolers with unilatera
86         At 12 to 15 years, the prevalence of anisometropia in the myopes was 9.64% and in the hyperop
87                                              Anisometropia in the severe ROP group increased approxim
88                            The prevalence of anisometropia increases between 5 and 15 years, when som
89 rate than that prior to age five years while anisometropia increases proportionally.
90 re myopic by another 10% if the magnitude of anisometropia is moderate.
91 rly monocular abnormalities of strabismus or anisometropia is proportional to the loss in cortical af
92 that often resulted in high myopia or severe anisometropia later in childhood.
93 t hyperopic meridian; astigmatism </=1.50 D; anisometropia &lt;/=1.0 D) and emmetropic status were deter
94 ian of at least 1 eye, astigmatism </=1.5 D, anisometropia &lt;/=1.0 D) or emmetropia (hyperopia </=1.0
95              Results: Among 20 children with anisometropia (mean [SD] age, 6.20 [2.16] years; 11 boys
96  than 1 line less than with full correction; anisometropia measured less than 1.0 diopter; and an app
97                      In spite of this visual anisometropia, monovision has been successfully used for
98 tigmatism were independently associated with anisometropia (myopes, P < 1.0E-61; hyperopes, P < 1.0E-
99 ivariable analysis: sex, age at examination, anisometropia, myopic and hyperopic refractive error (>/
100 yopia associated with strabismus (n = 32) or anisometropia (n = 20) or associated with both anisometr
101 blyopia associated with strabismus (n = 34), anisometropia (n = 23), and both anisometropia and strab
102 n (5-17 years old) with strabismus (n = 31), anisometropia (n = 29), or both conditions (n = 29).
103 10%), occlusion from ptosis (n = 9, 43%), or anisometropia (n = 9, 43%), or a combination of factors
104 s of age, mean +/- SD age 5.2 +/- 1.4 years (anisometropia [n = 18]); strabismus [n = 22]); both anis
105                                              Anisometropia occurred at POM1 in 2 patients (13.3%) in
106 D) or more, astigmatism of 1.0 D or more, or anisometropia of 0.5 D or more were present in 91% of ch
107 re affected in 64.4%, 28.9% showed spherical anisometropia of 1.0 D or more, and 19.5% showed astigma
108 nisometropia of more than 0.5 D, cylindrical anisometropia of more than 0.25 D, vertical and horizont
109 al amblyopia increased significantly with SE anisometropia of more than 0.5 D, cylindrical anisometro
110 dence for distinct impacts of strabismus and anisometropia on the mesoscale functional organization o
111 e is more accurate than spherical equivalent anisometropia or cylindrical anisometropia in identifyin
112               A subset of children with high anisometropia or isoametropia and neurobehavioral disord
113 4), astigmatism (OR, 5.7; 95% CI, 2.5-12.7), anisometropia (OR, 27.8; 95% CI, 11.2-69.3), and strabis
114 nly observed in individuals with strabismus, anisometropia, or amblyopia.
115 roximately 20/63) resulting from strabismus, anisometropia, or both were enrolled into a randomized c
116 e amblyopia or its risk factors (strabismus, anisometropia, or both).
117 /200, mean 20/63) resulting from strabismus, anisometropia, or both.
118 ular form deprivation developed myopic axial anisometropias outside the control range.
119  one or both eyes) have an increased risk of anisometropia (P < 0.05).
120  errors (myopia, hyperopia, astigmatism, and anisometropia; P<0.00001 for each) were associated indep
121      Thirty amblyopes with strabismus and/or anisometropia participated in the study.
122 th amblyopia risk factors (eg, strabismus or anisometropia), patching improved visual acuity of the a
123  Astigmatism ranged from 15.8% to 45.2%, and anisometropia ranged from 2.8% to 8.1%, depending on age
124                                          The anisometropia rate based on spherical equivalent differe
125  dominant eye of strabismic patients without anisometropia retained suppressive input from crossed bu
126       Third, monkeys recovering from induced anisometropias showed interocular alterations in choroid
127  best corrected visual acuity [BCVA]) due to anisometropia, strabismus, or both were eligible.
128  newly diagnosed with amblyopia secondary to anisometropia, strabismus, or both.
129 had less myopia, hyperopia, astigmatism, and anisometropia than did white persons.
130 o showed disruption of SA at lower values of anisometropia than were associated with increases in IAD
131   Monovision is therefore a form of acquired anisometropia that causes a superimposition of an in-foc
132             Using a cutoff of 1.00 D SER for anisometropia, the prevalence was 1.96%, 1.27%, and 5.77
133                  Sex was not associated with anisometropia to a clinically significant extent.
134 onventional correction modalities for myopic anisometropia, using spectacles, contact lenses, and/or
135                                Prevalence of anisometropia varied (P=0.009), with the lowest rate in
136 rs, specifically hyperopia, astigmatism, and anisometropia, varied by group, with the highest rate of
137  less than 3.0 D, and decreased below 70% if anisometropia was beyond 3.0 D.
138 ss than 4.0 D and decreased below 30% if the anisometropia was beyond 4.0 D.
139                                              Anisometropia was calculated at age 5 years.
140                                              Anisometropia was calculated in clinical notation (spher
141                                              Anisometropia was defined as a >=1 diopter interocular d
142                                              Anisometropia was defined as a 0.25-diopter (D) or more
143                                              Anisometropia was defined as a spherical equivalent (SE)
144                                     However, anisometropia was found to accompany both myopia and hyp
145                                              Anisometropia was greater in patients that developed gla
146 ye being more myopic increased to 80% if the anisometropia was less than 3.0 D, and decreased below 7
147 ng more myopic increased to above 40% if the anisometropia was less than 4.0 D and decreased below 30
148                                              Anisometropia was observed in 821 (19.34%) with constant
149 y different between eyes, and no significant anisometropia was observed.
150                                              Anisometropia was present in 13 subjects (14%).
151                                              Anisometropia was relatively stable between the ages of
152                                              Anisometropia was the presumed cause of amblyopia in 50.
153                            Larger amounts of anisometropia were associated with higher percentages of
154                   The presence and amount of anisometropia were associated with the presence of unila
155  D, lid-suture = -1.0 to -10.25 D) of myopic anisometropia were comparable in both treatment groups.
156 ataracts, glaucoma) other than strabismus or anisometropia were excluded.
157 This pattern was also evident in cylindrical anisometropia, where ORs increased with greater baseline
158 ous sensory manipulations (e.g., strabismus, anisometropia), which have been reported to alter the pe
159 the previously noted increased prevalence of anisometropia with age occurs later in hyperopes than in
160 ions between the prevalence and magnitude of anisometropia with age, sex, spherical power, and cylind

 
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