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1 intima-media thickness, carotid plaque, and ankle-brachial index).
2 ernal carotid intima-media thickness, or the ankle brachial index.
3 ned from cardiovascular risk factors and the ankle brachial index.
4 n increase in common femoral artery flow and ankle brachial index.
5 resistance, common femoral artery flow, and ankle brachial index.
6 ipheral revascularization and lower baseline ankle-brachial index.
7 dized testing for the presence of PAD by the ankle-brachial index.
8 lesterol, HDL cholesterol, leg symptoms, and ankle-brachial index.
9 ences in carotid intima-medial thickness and ankle-brachial index.
10 mean postprocedural increase of 0.23 in the ankle-brachial index.
11 , lipid and inflammatory biomarkers, and low ankle-brachial index.
12 l carotid artery intima-media thickness, and ankle-brachial index.
13 ignificant change in resting or postexercise ankle/brachial indexes.
15 le, 53% diabetic, 64% with tissue loss, mean ankle-brachial index 0.41, and mean toe pressure 26 mm H
16 68 +/- 10 years) with mild-to-moderate PAD (ankle-brachial index 0.69 +/- 0.14) had their most sympt
17 fe (-0.06; 95% CI: -0.17, 0.03; P = .20), or ankle-brachial index (0.03; 95% CI: -0.08, 0.14; P = .57
19 domized (mean age, 72.3 years [+/-7.1]; mean ankle brachial index, 0.66 [+/-0.15]), 40 (91%) complete
23 ticipants included 332 men and women with an ankle brachial index (ABI) <0.90 and 212 with ABI 0.90 t
24 ticipants included 392 men and women with an ankle brachial index (ABI) <0.90 and 249 with ABI 0.90 t
26 enome-wide association studies (GWAS) of the ankle brachial index (ABI) and PAD (defined as an ABI <
27 disease (PAD), specific leg symptoms and the ankle brachial index (ABI) are cross-sectionally related
28 ears) free of known cardiovascular (CVD) had ankle brachial index (ABI) assessment of their bilateral
29 erotic progression was assessed by computing ankle brachial index (ABI) at baseline (1,582 participan
30 s of subclinical atherosclerosis such as the ankle brachial index (ABI) could improve risk prediction
33 that defined peripheral artery disease as an ankle brachial index (ABI) lower than or equal to 0.90.
34 on of chronic kidney disease (CKD) with high ankle brachial index (ABI) measurement and to compare it
35 sought to determine the association of high ankle brachial index (ABI) measurements with left ventri
36 achial-ankle pulse wave velocity (baPWV) and ankle brachial index (ABI) were significantly higher but
37 associations of low (<0.90) and high (>1.40) ankle brachial index (ABI) with risk of all-cause and ca
38 months and changes in circulating PC levels, ankle brachial index (ABI), and walking impairment quest
41 purpose of this study is to determine if an ankle-brachial index (ABI) >or=1.40 is associated with r
42 -sectional association of PAD, defined as an ankle-brachial index (ABI) <0.9, and renal insufficiency
43 The authors studied associations between ankle-brachial index (ABI) and subclinical atheroscleros
45 ssible arteries (PCA) to those with a normal ankle-brachial index (ABI) and those with peripheral art
47 reviewed the evidence on the use of resting ankle-brachial index (ABI) as a screening test for PAD o
48 atherosclerotic progression, measured by the ankle-brachial index (ABI) at 3 consecutive time points
49 invasive test for diagnosis of LE-PAD is the ankle-brachial index (ABI) at rest and typically an ABI
50 s to determine whether use of an alternative ankle-brachial index (ABI) calculation method improves m
54 ed associations of borderline and low normal ankle-brachial index (ABI) values with functional declin
56 ine the association of both a low and a high ankle-brachial index (ABI) with incident cardiovascular
57 dentify genetic variants associated with the ankle-brachial index (ABI), a noninvasive measure of PAD
58 It can be noninvasively diagnosed with the ankle-brachial index (ABI), a ratio of Doppler-recorded
59 on between PAD, defined by low values of the ankle-brachial index (ABI), and future CVD risk has been
62 the coronary artery calcium (CAC) score, the ankle-brachial index (ABI), high-sensitivity C-reactive
72 1) young healthy subjects (YH) (n = 10; mean ankle-brachial index [ABI] 1.0 +/- 0.1, mean age 30 +/-
75 years, the 403 patients showed a significant ankle brachial index and toe brachial index deterioratio
76 isk factors, an inverse relationship between ankle-brachial index and cardiovascular events was obser
79 No difference in pain-free walking distance, ankle-brachial index and quality of life was found durin
80 and tertiary end points included changes in ankle-brachial index and quality-of-life assessments.
81 otid intimal medial thickness, stenosis, and ankle brachial index) and risk of dementia, CHD, and tot
84 fasting blood glucose, periodontal disease, ankle-brachial index, and carotid intima-media thickness
85 VM, LVM index, relative wall thickness, CAC, ankle-brachial index, and cIMT were more abnormal across
87 ed for age, gender, race, comorbidities, the ankle-brachial index, and other potential confounders.
88 is, pain score, pain-free walking distance, ankle-brachial index, and transcutaneous oxygen measurem
89 condary outcomes quality of life, rest pain, ankle-brachial index, and transcutaneous oxygen pressure
90 were independent of CVD risk factors or the ankle-brachial index, and VEGF treatment of ECs in vitro
93 artery disease (PAD) identified by screening ankle-brachial index benefit from preventive therapies t
94 CVD mortality, adjusting for age, sex, race, ankle-brachial index, body mass index, smoking, comorbid
95 ery calcium, carotid intima-media thickness, ankle-brachial index, brachial flow-mediated dilation, h
96 ical success was defined as improved resting ankle brachial index by > or =0.10, relief of resting pa
97 model without B-type natriuretic peptide and ankle-brachial index (C statistic, 0.79; 95% CI, 0.75-0.
98 and subclinical disease measures, including ankle-brachial index, carotid intimal-medial thickness,
100 Analyses were adjusted for age, sex, race, ankle brachial index, comorbidities, and other confounde
101 ace, body mass index, physical activity, the ankle brachial index, comorbidities, and other confounde
103 pported angioplasty for CLI and LLC improves ankle brachial indexes comparable to tibial bypass, heal
105 0%, 6%, 2.6 (95% CI, 1.4-4.8), and 39.2; for ankle-brachial index criteria, 0.6%, 9%, 5%, 2.3 (95% CI
108 S) 1999 to 2004 who underwent measurement of ankle brachial index, CRP, and fasting glucose and insul
109 sidered major progression, which was a -0.30 ankle brachial index decrease for LV-PAD and a -0.27 toe
110 on urine cadmium, potential confounders, and ankle brachial index determinations in the follow-up exa
111 s postoperatively with physical examination, ankle brachial index, duplex, and a quality-of-life ques
112 icity, comorbid conditions, body mass index, ankle-brachial index, education, leg symptoms, cigarette
114 ses and disease severity measures, including ankle-brachial index, forced expiratory volume, and exer
115 tors of poorer cognitive performance were an ankle brachial index greater than 1.30 (OR, 18.56 [95% C
116 indirect measures of arterial stiffness, an ankle brachial index greater than 1.30 and increased blo
117 cle characteristics, greater declines in the ankle brachial index, greater declines in lower extremit
118 89 patients) with critical limb ischemia and ankle brachial index >/=1.4 who underwent lower extremit
119 persisted after excluding participants with ankle brachial index >1.4 only as well as in subgroups d
120 brachial flow-mediated dilation >5% change, ankle-brachial index >0.9 and <1.3, high-sensitivity C-r
123 or a measure of subclinical atherosclerosis (ankle brachial index) had little impact on these associa
126 ion, baseline atrial fibrillation, and lower ankle-brachial index identify peripheral artery disease
129 score than carotid intima-media thickness or ankle-brachial index in subjects without and with CKD (H
131 dysfunction, microalbuminuria, and a reduced ankle-brachial index) in 2680 Framingham Study participa
132 common carotid intimal-medial thickness, and ankle-brachial index) in 5,172 US adults without clinica
133 ility of the first-line diagnostic test, the ankle-brachial index, in clinics; incorrect perceptions
134 clusion of carotid intima-media thickness or ankle-brachial index, inclusion of the coronary artery c
137 lantar flexion inversely correlated with the ankle-brachial index, indicating that patients with more
139 D risk: high-sensitivity C-reactive protein, ankle-brachial index, leukocyte count, fasting blood glu
140 class variable derived from body mass index, ankle-brachial index, low-density lipoprotein cholestero
142 .90, and severe prevalent PAD was defined as ankle brachial index </= 0.70, with both definitions als
146 Participants were free of PAD, defined as an ankle brachial index <0.9 or >1.4 at baseline, and had c
147 of 470 cases of incident PAD, defined as an ankle brachial index <0.9 or >1.4, were identified.
148 Participants were 384 men and women with an ankle brachial index <0.90 followed for a median of 47 m
149 e cumulative incidence of PAD, defined by an ankle brachial index <0.90 or a confirmed PAD event, wit
150 articipants were 370 men and women with PAD (ankle brachial index <0.90) and 231 without PAD.
151 pheral arterial disease (PAD) was defined by ankle brachial index <0.90, coronary artery calcificatio
154 en urinary BPA levels (in tertiles) and PAD (ankle-brachial index < 0.9, n = 63) using logistic regre
156 Patients were enrolled based on an abnormal ankle-brachial index </=0.80 or a previous lower extremi
159 ltivariable modeling, previous LER, baseline ankle-brachial index <0.50, surgical LER, and longer tar
162 Incident PAD was defined as a new onset of ankle-brachial index <0.9 assessed at regular examinatio
168 R 1.8, 95% CI 1.1-3.2, P=0.03), and baseline ankle-brachial index <=0.60 (HR 1.3 per 0.10 decrease, 9
175 Along with coronary artery calcium scanning, ankle-brachial index measurement, and carotid artery ult
176 isk factors, B-type natriuretic peptide, and ankle-brachial index (model 6) yielded modest improvemen
177 if they had intermittent claudication and an ankle brachial index of <0.85, or if they had a prior pe
178 Plasma was collected from PAD patients (ankle brachial index of <0.90; n=45) and subjects with r
181 n=3787) had a history of claudication and an ankle-brachial index of <0.85 or prior revascularization
182 ting vascular obstruction of 50% or greater, ankle-brachial index of less than 0.90, or physician-dia
184 taine stage IIa, able to walk >200 m) and an ankle-brachial index of less than or equal to 0.90 or to
186 cant differences in claudication onset time, ankle-brachial index, or quality-of-life measurements be
187 etABI study (German Epidemiological Trial on Ankle Brachial Index) participants undergoing ankle brac
188 d differences in pain-free walking distance, ankle-brachial index, quality of life, progression to cr
189 active protein, family history of ASCVD, and ankle-brachial index recommendations by the American Col
190 h critical limb ischemia and noncompressible ankle brachial index results, the prevalence of occlusiv
192 ation, but noninvasive measures, such as the ankle-brachial index, show that asymptomatic PAD is seve
194 , sex, race, comorbidities, body mass index, ankle brachial index, smoking, and walking exercise freq
195 djusted for age, sex, race, body mass index, ankle-brachial index, smoking, physical activity, and co
196 ent for age, sex, race, body mass index, the ankle-brachial index, smoking, physical activity, releva
197 iological measures including blood pressure, ankle-brachial index, spirometry, exhaled nitric oxide,
198 no carotid plaque, no family history, normal ankle-brachial index, test result <25th percentile (caro
199 Approximately 20% of patients undergoing ankle brachial index testing for critical limb ischemia
200 assessment of limb perfusion, including the ankle-brachial index, toe-brachial index, and other perf
201 en groups in secondary end points, including ankle-brachial index, toe-brachial index, pain relief, w
202 patients) precisely estimated the changes in ankle brachial index, transcutaneous oxygen tension, res
203 sted with carotid intima-media thickness and ankle-brachial index (two other measures of subclinical
207 event was not increased, even at the lowest ankle-brachial index values, and was the same as in a pa
209 , brachial flow-mediated dilation was 0.024, ankle-brachial index was 0.036, carotid intima-media thi