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1 proresolving mediators and proteins, such as annexin A1.
2  the structure of the N-terminally truncated annexin A1.
3 trophin-glycoprotein complex of proteins and annexin A1.
4 ng required the expression of caveolin 1 and annexin A1.
5 ntly changed in the tumor stroma that lacked annexin A1.
6  partitioning defective protein 3 (PAR3) and annexin A1.
7 d override the prosurvival effect of LPS via annexin A1.
8 ; and further investigation into the role of annexin A1, a downstream mediator of glucocorticoid acti
9 ulation were associated with the cleavage of annexin A1, a powerful anti-inflammatory protein known t
10            Furthermore, the protein level of Annexin A1, A2, A5 and A10 was increased from the early
11 cence and subcellular fractionation revealed Annexin A1, A2, and A5 in the cytoplasm and nuclei of tu
12       Within seconds of membrane disruption, annexins A1, A2, A5, and A6 formed a tight repair "cap."
13 domains, studies reported herein showed that annexins A1, A2, A5, and B12 could be divided into two g
14 ctinin-1, moesin, 14-3-3 protein zeta/delta, annexin A1/A3/A4/A5/A6, clathrin heavy chain 1, glyceral
15                                              Annexin A1 acts independently of G(alphai) to instruct t
16  tumor stroma and suggested a mechanism that annexin A1 affects tumor development and metastasis thro
17           In contrast, the nontrimer-forming annexin A1 and A2 group had the following Ca(2+)-depende
18            In conclusion, we have identified Annexin A1 and A5 as potentially useful early biomarkers
19  and sufficient for releasing EVs containing annexin A1 and GDE3 from the plasma membrane via Wiskott
20  we show that these contacts are tethered by annexin A1 and its Ca(2+)-dependent ligand, S100A11, and
21 that dysferlin normally associates with both annexins A1 and A2 in a Ca2+ and membrane injury-depende
22 lin and the Ca2+ and lipid-binding proteins, annexins A1 and A2, and define a role for dysferlin in C
23 ected as lipid mixing involved extracellular annexins A1 and A5 acting in a functionally redundant ma
24                                Appearance of annexins A1 and A5 at the cap is likewise diminished in
25 emmal repair through increased expression of annexins A1 and A6, which mediate myofiber repair.
26 tant-associated protein C (SP-C), L-plastin, annexin A1, and haptoglobin increased, whereas transferr
27 th I3C reduced the level of SP-C, L-plastin, annexin A1, and haptoglobin to that of untreated control
28      A post-translationally modified form of annexin A1 (AnnA1) is selectively concentrated in human
29 o experiments have identified involvement of Annexin A1 (Anx A1) in both these fusion processes.
30 the release of the anti-inflammatory protein Annexin-A1 (Anx-A1) from mast cells.
31                 Endogenous anti-inflammatory annexin-A1 (ANX-A1) plays an important role in preservin
32 o a decrease in the level of cell-associated annexin A1 (AnxA1) and cathelin-related antimicrobial pe
33 ncy was associated with an early increase of annexin A1 (AnxA1) and did not modify the course of neut
34  inflammation such as resolvins, protectins, annexin A1 (ANXA1) and galectins as potential targets fo
35 such target is the anti-inflammatory protein annexin A1 (AnxA1) and its receptor, FPR2/ALX.
36 gands, including the proresolution mediators annexin A1 (AnxA1) and lipoxin A(4), as well as the acti
37 upled with increased expression of mRNAs for annexin A1 (AnxA1) and the formyl peptide receptors [(Fp
38                                              Annexin A1 (ANXA1) and the NOD-like receptor family pyri
39 he proresolving properties of lipoxin A4 and annexin A1 (AnxA1) and the proinflammatory signals elici
40 cted role for the anti-inflammatory molecule annexin A1 (AnxA1) as a critical regulator of this proce
41  we identify the membrane-associated protein Annexin A1 (ANXA1) as an interactor of LGN in mammary ep
42 scle injury and repair, herein we identified annexin A1 (AnxA1) as the extracellular trigger of macro
43                       The endogenous protein annexin A1 (AnxA1) facilitates inflammation resolution v
44 tide receptor-1 (FPR1), which interacts with Annexin A1 (ANXA1) from dead cells.
45                                              Annexin A1 (ANXA1) has an important role in cell-cell co
46 2 decades of research, no clear function for annexin A1 (AnxA1) has been established.
47                                              Annexin A1 (AnxA1) is a glucocorticoid-regulated protein
48                                              Annexin A1 (AnxA1) is a glucocorticoid-regulated protein
49                                              Annexin A1 (ANXA1) is a mediator of glucocorticoid anti-
50                                              Annexin A1 (ANXA1) is a protein induced by glucocorticoi
51                                              Annexin A1 (AnxA1) is a protein involved in modulation a
52                                              Annexin A1 (AnxA1) is a protein that displays potent ant
53                                              Annexin A1 (AnxA1) is an effector of the resolution of i
54                                              Annexin A1 (AnxA1) is an endogenous glucocorticoid regul
55 e glucocorticoid anti-inflammatory messenger annexin A1 (ANXA1) is expressed in brain microvascular e
56 ytosis, to clear phagocytic apoptotic cells; annexin A1 (Anxa1) is key to efferocytosis, but its role
57                                  The protein annexin A1 (ANXA1) is key to the phagocytosis of apoptot
58                    The pro-resolving protein Annexin A1 (AnxA1) is known to counterbalance overexuber
59  The widely-expressed Ca(2+)-binding protein annexin A1 (ANXA1) is present in the nuclear envelope lu
60                                              Annexin A1 (AnxA1) is recognized as an endogenous anti-i
61 In this work, we demonstrate that endogenous annexin A1 (ANXA1) is released as a component of extrace
62                                              Annexin A1 (AnxA1) is the primary mediator of the anti-i
63 th reperfusion was performed in wild-type or annexin A1 (AnxA1) knockout (AnxA1(-/-)) mice.
64 eat me" signal on the LO surface, leading to annexin A1 (ANXA1) loading.
65                                              Annexin A1 (AnxA1) mainly acts through Formyl Peptide Re
66           We demonstrated high expression of annexin A1 (ANXA1) mRNA by MSCs and confirmed expression
67 mmatory pathway glucocorticoid receptor (GR)-annexin A1 (ANXA1) occurs.
68 tes the potential therapeutic treatment with annexin A1 (AnxA1) to induce cardiac repair after MI.
69                               Suppression of annexin A1 (ANXA1), a mediator of apoptosis and inhibito
70                                              Annexin A1 (ANXA1), a mediator of the anti-inflammatory
71 ion leads to cell surface externalization of Annexin A1 (AnxA1), an effector of endogenous anti-infla
72                                              Annexin A1 (ANXA1), an inflammation modulator, is a pote
73 at is activated by an endogenous FPR ligand, annexin A1 (ANXA1), and its cleavage product Ac2-26, whi
74                                              Annexin A1 (ANXA1), as an endogenous mediator, plays an
75  ALX/FPR2, which is activated by the protein annexin A1 (ANXA1), found in high abundance in inflammat
76  of the endogenous anti-inflammatory protein Annexin A1 (AnxA1), we investigated further this possibl
77 rtain anti-inflammatory molecules, including annexin A1 (ANXA1), which is an important mediator of gl
78         In this article, we engineered novel Annexin A1 (AnxA1)-based peptides, AnxA1(2-50), that dis
79                                              Annexin A1 (ANXA1, formerly termed lipocortin 1 or macro
80         In these studies, we have identified Annexin-A1 (ANXA1) as a novel regulator of TLR-induced I
81  activation of the p38/MAPKAP kinase-2/LIMK1/annexin-A1 (ANXA1) signaling axis.
82 vel therapeutic antibody developed to target annexin-A1 (ANXA1).
83                                  We identify annexin A1 as a specific marker for microvesicles that a
84                 Mechanistically, we identify annexin A1 as an endogenous inhibitor of integrin activa
85                       Our findings establish Annexin A1 as an upstream cortical cue that regulates LG
86  two of these proteins, aminopeptidase-P and annexin A1, as selective in vivo targets for antibodies
87 how that, coincident with a resealing event, annexin A1 becomes concentrated at disruption sites.
88 ntering through a disruption locally induces annexin A1 binding to membranes, initiating emergency fu
89  that annexin A6 binds mu1 and mu2, and that annexin A1 binds only mu1.
90                             Carbonylation of annexin A1 by endothelin-1 was followed by proteasome-de
91  Moreover, we show, for the first time, that annexin A1-dependent inhibition of adrenocorticotrophin
92                       Radio-immunotherapy to annexin A1 destroys tumours and increases animal surviva
93  follow-up study, we report that exposure of annexin A1 during secondary necrosis coincided with prot
94         Subcellular patterning is evident as annexin A1, dysferlin, diacylglycerol, active Rho, and a
95               Deletion of FPR2 or its ligand annexin A1 enhances atherosclerotic lesion formation, ar
96  Thus, altogether our findings indicate that annexin A1 externalization and its proteolytic processin
97 trol levels, and recently we could show that annexin A1 externalization during secondary necrosis pro
98  culture supernatants of secondary necrotic, annexin A1-externalizing cells induced chemoattraction o
99 nd mouse experimental systems to identify an annexin A1-formyl peptide receptor 1 (ANXA1-FPR1) bidire
100                              Instructing the annexin A1-FPR2 axis harbors a novel approach to target
101                            Specifically, the annexin A1 fragment Ac2-26 counteracts conformational ac
102 d DMF induce secretion of the 33-kDa form of annexin A1 from murine bone marrow-derived macrophages,
103                  We demonstrate here that an annexin A1 function-blocking antibody, a small peptide c
104 ed mRNA/protein expression of SOCS3, IL-1RA, annexin-A1, GST-alpha, HIF-1alpha.
105 f these lipids as well as by peptides (e.g., annexin A1), has been shown to be one of the receptors i
106   Pharmacological treatment with recombinant annexin A1 (hrANXA1) or reversion from a high-fat high-s
107 te the role of FPR2 and its resolving ligand annexin A1 in atherogenesis.
108 sis, and the previously known properties for annexin A1 in immune cells and inflammation, this study
109 t the X-ray structure of full-length porcine annexin A1 in the presence of calcium.
110          The signaling mechanism employed by annexin A1 in this process is uncertain, although we hav
111 ious finding of pro-angiogenic functions for annexin A1 in vascular endothelial cell sprouting, wound
112  activated by IAV, which harbors its ligand, annexin A1, in its envelope.
113 eported the structure of full-length porcine annexin A1 including the N-terminal domain in the absenc
114                                              Annexin A1 is a cytosolic protein that, when activated b
115                                              Annexin A1 is a key anti-inflammatory effector protein t
116 d inflammation, this study hypothesized that annexin A1 is a key functional player in tumor developme
117                                              Annexin A1 is a multi functional molecule which is invol
118         The glucocorticoid-regulated protein annexin A1 is a potent inhibitor of hormone exocytosis i
119                                              Annexin A1 is an intracellular calcium/phospholipid-bind
120 en tumors from annexin A1 wild type mice and annexin A1 knockout mice and found a list of genes that
121 etastasis, angiogenesis and wound healing in annexin A1 knockout mice.
122 f an N-terminally truncated version of human annexin A1 lacking the first 32 amino acid residues (PDB
123 ing the anti-inflammatory peptide Ac2-26, an annexin A1/lipocortin 1-mimetic peptide.
124  carbonylation and subsequent degradation of annexin A1 may play a role in endothelin-mediated cell g
125 nd treatment with FPR agonists: AnxA1Ac2-26 [Annexin A1 mimetic peptide (Ac-AMVSEFLKQAWFIENEEQEYVQTVK
126             Nine genes, PDGF A, Cathepsin L, annexin A1, Mm.112139, Est2 repressor factor, NrCAM, ZNF
127 rs Mcm5 and Brd4, phosphoinositide-3-kinase, annexin A1, mucosa-associated lymphoid tissue lymphoma t
128  peptide competitor, and a dominant-negative annexin A1 mutant protein incapable of Ca2+ binding all
129 tial of 2 proresolving endogenous mediators, annexin A1 N-terminal derived peptide (AnxA1Ac2-26) and
130 t implications for the inhibitory actions of annexin A1 on exocytosis in other endocrine and immune c
131 t to characterize the mechanism of action of annexin A1 on exocytosis using the release of adrenocort
132  neither method detected self-association of annexin A1 or A2 on bilayers.
133 lease from cells expressing either wild-type annexin A1 or mutant forms, we show a critical involveme
134 n of monocytes, which was clearly reduced in annexin A1- or ADAM10-knockdown cells.
135                                     As such, annexin A1 promotes the engulfment of dying cells and da
136 comprises a small bioactive peptide from the annexin A1 protein grafted into a sunflower trypsin inhi
137                                      Loss of Annexin A1 randomises mitotic spindle orientation, which
138                                              Annexin A1-regulated contacts function in the transfer o
139  We also show that this 4-OI- and DMF-driven annexin A1 secretion is NRF2-dependent and that other me
140 CA1, which has previously been implicated in annexin A1 secretion, is required for this process in ma
141 , mRNA level of HIF-1alpha, GPx, SOD1, SOD2, annexin-A1, SOCS3, IL-1RA, IL-1beta, IL-1R1, IL-1R2, TNF
142 he proapoptotic phospholipid-binding protein Annexin A1 that link early prostate development to early
143 ribution of FPR2 and its proresolving ligand annexin A1 to atherosclerotic lesion formation is largel
144                                This revealed annexin A1 to be an effective regulator in tumor stroma
145 N tumor-associated self Ags (TAA) and to the Annexin A1 tumor vascular Ag, even in mice in which aner
146 ine genes that included GPCR11, cadherin 11, annexin A1, vimentin, lactate dehydrogenase B (upregulat
147            Expression of the FPR2/ALX ligand Annexin A1 was also significantly increased in sub-acute
148 ated NF-kappaB signaling by interacting with annexin A1, which further induced Lys63-linked and Met1-
149  Fenton reaction-dependent manner, including annexin A1, which promotes apoptosis and suppresses cell
150 ared the gene expression between tumors from annexin A1 wild type mice and annexin A1 knockout mice a
151 h for the membrane aggregation properties of annexin A1 will be discussed.

 
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