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1 oblems such as word-finding difficulties and anomia.
2 d to explore the factors contributing to the anomia.
3 ts, fluent aphasia and a particularly severe anomia.
4 er reading or naming, resulting in alexia or anomia.
5 on for temporal lobe epilepsy had measurable anomia.
6 revealing an associative basis for retrieval anomia.
7 a, pure word blindness and category-specific anomias.
8 reated for one of three aphasia impairments: anomia, agrammatism, or dysgraphia.
9 nd after 30 h of aphasia treatment targeting anomia, an impairment in the ability to name common obje
10                     SD patients present with anomia and impaired word comprehension.
11 t anterior temporal lobe atrophy show severe anomia and verbal semantic deficits and meet criteria fo
12 sease starts with word-finding disturbances (anomia) and frequently proceeds to impair the grammatica
13 rk, leading to impairments of object naming (anomia) and word comprehension.
14 ation-induced speech arrest, 82 patients had anomia, and 23 patients had alexia.
15 d these deficits were modulated by degree of anomia, concept familiarity and item typicality.
16             In patients with the more severe anomias, conceptual maps were more extensively disrupted
17   Most stroke survivors with aphasia exhibit anomia (difficulties with naming common objects), but wh
18 rior temporal atrophy during which prominent anomia exists without word comprehension or object recog
19 but while many therapeutic interventions for anomia have been proposed, treatment effects are typical
20 re is progressive semantic deterioration and anomia in the face of relative preservation of other lan
21 ge support the argument that their prominent anomia is due to disrupted temporal lobe connections.
22 ion, an initial stage of relatively isolated anomia is followed by additional intra-category blurring
23 nsion deficits but with amnesia and variable anomia, leading some to conclude that semantic memory is
24                                          The anomia map revealed two clusters: one was in the posteri
25 causes global impairments such as multimodal anomia, neglect and amnesia, whereas their selective dis
26 ngaged in a computerised treatment for their anomia over a 6-week period.
27                                              Anomia/paraphasia data demonstrated foci not only within
28 tor) or negative (speech arrest, dysarthria, anomia, phonological and semantic paraphasias) findings
29 trong performance for behavioral measures in anomia (R(2) = 0.948, n = 28) and for fALFF predictors i
30 across all of the domains examined, but with anomia, reduced phonemic fluency and slowed speech rate
31 ct word in patients with mild but not severe anomia, reflecting a gradual intensification of the sema
32 ivalve tentacles in the common jingle shell, Anomia simplex.
33 te interruption of ongoing counting) and 423 anomia sites (inability to name or misnaming) were inclu
34 elations P < 0.05), and similar patterns for anomia (Spearman's coefficient r ranged from 0.37 to 0.8
35 e that individuals' variation in response to anomia treatment are, at least somewhat, systematic and
36 s preserved in one patient with postsemantic anomia, who could write the names of objects she could n