ライフサイエンスコーパス: anorexia nervosa

1 d behaviors, and muscle dysmorphia ("reverse anorexia nervosa").

2 inin (medium effect for ED, large effect for anorexia nervosa).

3 rtium Stage 1 and the Genetic Consortium for Anorexia Nervosa.

4 iety, depression, and the psychopathology of anorexia nervosa.

5 proach in patients with, or recovering from, anorexia nervosa.

6 it was their first admission to hospital for anorexia nervosa.

7 -8.4) for bulimia nervosa with no history of anorexia nervosa.

8 functioning raise the risk for mortality in anorexia nervosa.

9 developmental delay, autism, psychosis, and anorexia nervosa.

10 d mortality risks, similar to those found in anorexia nervosa.

11 ave been linked to suppression of eating and anorexia nervosa.

12 riods of extremely restricted food intake in anorexia nervosa.

13 ent and discharge criteria for patients with anorexia nervosa.

14 limia nervosa were unlikely to cross over to anorexia nervosa.

15 imia nervosa but were likely to relapse into anorexia nervosa.

16 cited risk of premature death in those with anorexia nervosa.

17 th eating disorders, particularly those with anorexia nervosa.

18 is little evidence for any rise in rates of anorexia nervosa.

19 n is frequently prescribed for patients with anorexia nervosa.

20 No specific recommendations were made for anorexia nervosa.

21 jor problem for research in the treatment of anorexia nervosa.

22 w randomized controlled treatment studies of anorexia nervosa.

23 amic amenorrhoea, and to clarify its role in anorexia nervosa.

24 nal nutritional status, eg, in patients with anorexia nervosa.

25 eir first discharge from hospitalization for anorexia nervosa.

26 adolescents, and 75 female adolescents with anorexia nervosa.

27 ization care and relapse prevention of adult anorexia nervosa.

28 inhibitors in the treatment of patients with anorexia nervosa.

29 ared with placebo for adult outpatients with anorexia nervosa.

30 ifetime weight and duration of amenorrhea in anorexia nervosa.

31 of obesity, dieting-induced weight gain, and anorexia nervosa.

32 degenerative diseases, obesity, bulimia, and anorexia nervosa.

33 insensitivity to these effects of hunger in anorexia nervosa.

34 or the treatment of eating disorders such as anorexia nervosa.

35 dynamics as a risk factor for arrhythmias in anorexia nervosa.

36 Ninety-seven individuals with anorexia nervosa, 282 with bulimia nervosa, and 293 with

37 Crude mortality rates were 4.0% for anorexia nervosa, 3.9% for bulimia nervosa, and 5.2% for

38 emor) = 39; n(treatment-resistant depression/anorexia nervosa) = 76) to identify the neuroanatomical

39 Anorexia nervosa affects 1-4% of women in United States

40 d circuit white matter fiber organization in anorexia nervosa after recovery could indicate a biologi

41 Anorexia nervosa also has large and significant genetic

42 The lifetime prevalence of DSM-5 anorexia nervosa among women might be up to 4%, and of b

43 anges in steroid metabolism in subjects with anorexia nervosa (AN) after weight gain have not been el

44 tate of the science, with a primary focus on anorexia nervosa (AN) and binge-eating behavior, and enc

45 Eating disorders, such as anorexia nervosa (AN) and bulimia nervosa (BN), have gen

46 Patients with anorexia nervosa (AN) and obesity (OB) were investigated

47 Both anorexia nervosa (AN) and obesity are complicated by aff

48 Anorexia nervosa (AN) and obsessive-compulsive disorder

49 Diagnoses for anorexia nervosa (AN) and other eating disorders (OED: b

50 tion between ADHD and various EDs, including anorexia nervosa (AN) and other EDs such as bulimia nerv

51 Anorexia nervosa (AN) and related eating disorders are c

52 sensitivity to reward, yet individuals with anorexia nervosa (AN) are not motivated to eat when star

53 ntless pursuit of thinness, individuals with anorexia nervosa (AN) engage in maladaptive behaviors (r

54 Individuals with anorexia nervosa (AN) engage in relentless restrictive e

55 al body weight is disrupted in patients with anorexia nervosa (AN) for prolonged periods of time.

56 Anorexia nervosa (AN) is a complex and heritable eating

57 Anorexia nervosa (AN) is a complex neuropsychiatric diso

58 Anorexia nervosa (AN) is a condition of severe undernutr

59 Anorexia nervosa (AN) is a devastating psychiatric illne

60 Anorexia nervosa (AN) is a serious disorder with high ra

61 Anorexia nervosa (AN) is a serious eating disorder chara

62 Anorexia nervosa (AN) is a serious eating disorder chara

63 Anorexia nervosa (AN) is a serious mental illness catego

64 Anorexia nervosa (AN) is a serious mental illness with m

65 Anorexia nervosa (AN) is a serious psychiatric illness a

66 Anorexia nervosa (AN) is a severe psychiatric disorder a

67 Anorexia nervosa (AN) is an eating disorder characterize

68 Anorexia nervosa (AN) is an eating disorder observed pre

69 Anorexia nervosa (AN) is characterized by a persistent r

70 Anorexia Nervosa (AN) is characterized by Diagnostic and

71 Anorexia nervosa (AN) is characterized by extremely low

72 Recovery from osteoporosis in anorexia nervosa (AN) is uncertain.

73 nts were included in the dataset as follows: Anorexia Nervosa (AN) n = 171; Bulimia Nervosa (BN) n =

74 Is starvation in anorexia nervosa (AN) or overeating in bulimia nervosa (

75 Individuals with anorexia nervosa (AN) override the drive to eat, forgoin

76 orts have been carried out on iron status in anorexia nervosa (AN) patients.

77 Individuals with anorexia nervosa (AN) restrict eating and become emaciat

78 ed a genome-wide association study (GWAS) of anorexia nervosa (AN) using a stringently defined phenot

79 dolescence is a common time for the onset of anorexia nervosa (AN), a condition associated with long-

80 e heightened tolerance to self-starvation in anorexia nervosa (AN), a hypothalamic dysregulation of e

81 prospectively correlate with future onset of anorexia nervosa (AN), bulimia nervosa (BN), binge eatin

82 isms was suggested in the pathophysiology of anorexia nervosa (AN), but the role of the endogenous me

83 ve implications for eating disorders such as anorexia nervosa (AN), in which there is a high prevalen

84 distribution has previously been studied in anorexia nervosa (AN), its influence in women with AN on

85 In anorexia nervosa (AN), taste and smell are believed to b

86 onsidered one of the core characteristics of anorexia nervosa (AN), the exact nature of this complex

87 Previous studies have shown that women with anorexia nervosa (AN), when ill and after recovery, have

88 n multiple psychiatric conditions, including anorexia nervosa (AN).

89 the complex eating-related behaviors seen in anorexia nervosa (AN).

90 that lead to inpatient medical treatment for anorexia nervosa (AN).

91 Body image distortions are a core feature of anorexia nervosa (AN).

92 sed physical activity is a common feature of anorexia nervosa (AN).

93 are characteristic features of patients with anorexia nervosa (AN).

94 ility, and genetic correlations (rg) between anorexia nervosa and 159 other phenotypes.

95 ratio was 4.37 (95% CI=2.4-7.3) for lifetime anorexia nervosa and 2.33 (95% CI=0.3-8.4) for bulimia n

96 Sixty-one adolescent female patients with anorexia nervosa and 45 age- and sex-matched healthy vol

97 me-wide association study of 16,992 cases of anorexia nervosa and 55,525 controls, identifying eight

98 Treatment of anorexia nervosa and atypical eating disorders has recei

99 examine DNA methylation across the genome of anorexia nervosa and binge-eating disorder patients.

100 e genetic correlations were observed between anorexia nervosa and body mass index, insulin, glucose,

101 wins were interviewed to assess diagnoses of anorexia nervosa and bulimia nervosa (per DSM-IV and bro

102 t as chronic as the well-validated disorders anorexia nervosa and bulimia nervosa and likely represen

103 unity level, 5-year recovery rates for DSM-5 anorexia nervosa and bulimia nervosa are 69 and 55%, res

104 Both anorexia nervosa and bulimia nervosa are associated with

105 Anorexia nervosa and bulimia nervosa are common and seve

106 ings support the longitudinal distinction of anorexia nervosa and bulimia nervosa but do not support

107 ell-characterized sample of individuals with anorexia nervosa and bulimia nervosa from the Price Foun

108 riatum volumes were reduced in the recovered anorexia nervosa and bulimia nervosa groups and predicte

109 Course and outcome studies of both anorexia nervosa and bulimia nervosa show that no signif

110 in particular was much higher in people with anorexia nervosa and bulimia nervosa than in a nonclinic

111 amine diagnostic crossover longitudinally in anorexia nervosa and bulimia nervosa to inform the valid

112 Participants who had recovered from anorexia nervosa and bulimia nervosa were studied to avo

113 agnosis EDNOS, by lowering the threshold for anorexia nervosa and bulimia nervosa, and adding BED as

114 ts suggest that eating disorders, especially anorexia nervosa and bulimia nervosa, are more common am

115 w is reflected by the diagnostic criteria of anorexia nervosa and bulimia nervosa, which emphasize in

116 n persons with an ED, including the subtypes anorexia nervosa and bulimia nervosa.

117 in patients with eating disorders, including anorexia nervosa and bulimia nervosa.

118 Similar patterns were found for anorexia nervosa and bulimia nervosa.

119 ldhood'; clarifications and modifications to anorexia nervosa and bulimia nervosa; and the inclusion

120 vosa, 282 with bulimia nervosa, and 293 with anorexia nervosa and bulimia were given the Structured C

121 conducted a genome-wide association study of anorexia nervosa and calculated genetic correlations wit

122 omen who had recovered from restricting-type anorexia nervosa and had 1 year of normal weight and reg

123 covered group (lower in women recovered from anorexia nervosa and higher in women recovered from buli

124 literature on the development and course of anorexia nervosa and interpreted critical features in li

125 he most established treatment for youth with anorexia nervosa and may be efficacious for youth with b

126 nalysis of the lumbar spine in patients with anorexia nervosa and normal-weight control subjects and

127 Women with anorexia nervosa and normal-weight control subjects were

128 tly are no significantly associated SNPs for anorexia nervosa and only three for educational attainme

129 uitry may contribute to restricted eating in anorexia nervosa and overeating in bulimia nervosa.

130 disorders: stabilization of the incidence of anorexia nervosa and possibly lower incidence rates of b

131 current first-line treatment for adolescent anorexia nervosa and promising for adolescent bulimia ne

132 of research on molecular genetic studies of anorexia nervosa and provide guidance for future researc

133 umes were increased on the right side in the anorexia nervosa and recovered anorexia nervosa groups a

134 results include genetic correlations between anorexia nervosa and schizophrenia, anorexia and obesity

135 e genetic correlations were observed between anorexia nervosa and schizophrenia, neuroticism, educati

136 processes are engaged in the development of anorexia nervosa and that stimulus-response learning (th

137 on exists against schizophrenia, autism, and anorexia nervosa and that these variants may be maintain

138 d/or agoraphobia (N=365) as there were about anorexia nervosa and/or bulimia nervosa (N=169).

139 number of empirical articles published about anorexia nervosa and/or bulimia nervosa and the number o

140 ttention-deficit/hyperactivity disorder, and anorexia nervosa) and 17 nonpsychiatric traits in more t

141 disorders (10 with bulimia nervosa, 16 with anorexia nervosa) and 19 healthy female comparison subje

142 80 females (0.70%) and 453 males (0.04%) had anorexia nervosa, and 3349 females (0.30%), and 61 males

143 aptured by 3 variables (any eating disorder, anorexia nervosa, and bulimia nervosa) identified by any

144 lian randomization identifies schizophrenia, anorexia nervosa, and higher education as causal for dec

145 rption syndrome, inflammatory bowel disease, anorexia nervosa, and intestinal pseudo-obstruction.

146 tinal bypass procedures, chronic alcoholics, anorexia nervosa, and restrictive diets.

147 xercise-induced hypothalamic amenorrhoea and anorexia nervosa are also associated with low concentrat

148 Acrocyanosis is common, and patients with anorexia nervosa are at risk of various arrhythmias.

149 -based treatment trials for adolescents with anorexia nervosa are few.

150 Individuals with anorexia nervosa are known to be ascetic and able to sus

151 s further encourage a reconceptualization of anorexia nervosa as a metabo-psychiatric disorder.

152 on of the dieting behavior characteristic of anorexia nervosa as a well-entrenched habit provides a b

153 We also found an enrichment of anorexia nervosa associated genes in the adult and fetal

154 hyperactivity disorder, alcohol dependence, anorexia nervosa, autism spectrum disorder, bipolar diso

155 ases to promote eating, yet individuals with anorexia nervosa avoid food despite emaciation.

156 t restricted eating and weight loss occur in anorexia nervosa because of a failure to accurately reco

157 purging behaviors (i.e., bulimia nervosa or anorexia nervosa, binge eating/purging type);14 with ano

158 This study examined the prevalence of anorexia nervosa, bulimia nervosa, and binge eating diso

159 Lifetime prevalence estimates of anorexia nervosa, bulimia nervosa, and binge-eating diso

160 uals born in 1975-1998 and followed them for anorexia nervosa, bulimia nervosa, and eating disorder n

161 goal of this study was to determine whether anorexia nervosa, bulimia nervosa, and eating disorder n

162 re divided into three diagnostic categories: anorexia nervosa, bulimia nervosa, and the atypical eati

163 w, and the probable four new categories are: anorexia nervosa, bulimia nervosa, binge eating disorder

164 rs are now recognised in diagnostic systems: anorexia nervosa, bulimia nervosa, binge eating disorder

165 hors found little evidence that the risk for anorexia nervosa, bulimia nervosa, or disordered eating

166 6.9; 95% confidence interval, 15.3-145.7) in anorexia nervosa but not for death (1.3; 95% confidence

167 moderately-to-severely ill adolescents with anorexia nervosa, but it is costly, and the risks of rel

168 placebo on weight in adult outpatients with anorexia nervosa, but no significant benefit for psychol

169 Anorexia nervosa, but not bulimia nervosa, has one of th

170 ves our understanding of the neurobiology of anorexia nervosa by suggesting disturbances in subcortic

171 rmulation in which the marked persistence of anorexia nervosa can be usefully understood as a well-in

172 in 12 case-control cohorts comprising 3,495 anorexia nervosa cases and 10,982 controls, the authors

173 hat genes from an induced stem cell study of anorexia nervosa cases are expressed at higher levels in

174 sted how brain reward learning in adolescent anorexia nervosa changes with weight restoration.

175 quired deficiencies, including patients with anorexia nervosa, cystic fibrosis, patients receiving lo

176 n patients with chronic treatment-refractory anorexia nervosa, DBS is well tolerated and is associate

177 Patients with anorexia nervosa exhibit abnormal myocardial repolarizat

178 Over 7 years, the majority of women with anorexia nervosa experienced diagnostic crossover: more

179 fluoxetine in the treatment of patients with anorexia nervosa following weight restoration.

180 .6 (95% CI=3.2-12.1) for those with lifetime anorexia nervosa for >15 to 30 years (10/67 died).

181 (95% CI=0.9-8.3) for patients with lifetime anorexia nervosa for 0 to 15 years (4/119 died), and 6.6

182 lled female patients (aged 11-18 years) with anorexia nervosa from six centres in Germany.

183 ignment in 32 weight-recovered subjects with anorexia nervosa from the New York site of the Fluoxetin

184 Here we combine data from the Anorexia Nervosa Genetics Initiative (ANGI)(8,9) and the

185 amen functional connectivity in the remitted anorexia nervosa group compared with the control group.

186 Compared with the control group, the anorexia nervosa group exhibited greater brain response

187 sponse in the control group and the remitted anorexia nervosa group, with an increase and a decrease,

188 avoidance among participants in the remitted anorexia nervosa group.

189 t side in the anorexia nervosa and recovered anorexia nervosa groups and on the left side in the buli

190 nce of breast cancer; nulliparous women with anorexia nervosa had a 23% (95% CI, 79% higher to 75% lo

191 ower) lower incidence, and parous women with anorexia nervosa had a 76% (95% CI, 13%-97%) lower incid

192 Results Women with anorexia nervosa had higher skewness and kurtosis, lower

193 Conclusion Patients with anorexia nervosa had increased skewness and kurtosis and

194 Although anorexia nervosa has a high mortality rate, our understa

195 Anorexia nervosa has been consistently associated with i

196 Anorexia nervosa has the highest mortality rate of any p

197 Anorexia nervosa has the highest mortality rate of any p

198 he authors hypothesize that individuals with anorexia nervosa have an imbalance in information proces

199 y, several large population-based studies of anorexia nervosa have been conducted in twins; it is pos

200 of the prevalence, incidence, and outcome of anorexia nervosa have been limited to cases detected thr

201 ubset of patients suffering from restrictive anorexia nervosa have enhanced habit formation compared

202 e levels with clinical outcome in women with anorexia nervosa in a relapse-prevention trial.

203 trongest evidence base for effectiveness for anorexia nervosa in adolescents.

204 erapy as a posthospitalization treatment for anorexia nervosa in adults.

205 nervosa subtype predicted fatal outcome for anorexia nervosa in males.

206 at birth had an independent association with anorexia nervosa in males.

207 sought to describe the onset and outcomes of anorexia nervosa in the general population.

208 ler discusses two trials of individuals with anorexia nervosa in which deep brain stimulation of diff

209 The incidence of anorexia nervosa in women between 15 and 19 years of age

210 Predictors of mortality in anorexia nervosa included severity of alcohol use disord

211 cterized primarily by a low body-mass index, anorexia nervosa is a complex and serious illness(1), af

212 Anorexia nervosa is a complex eating disorder with genet

213 Anorexia nervosa is a complex heritable phenotype for wh

214 Anorexia nervosa is a life-threatening complex psychiatr

215 Anorexia nervosa is a life-threatening illness.

216 Anorexia nervosa is a perplexing illness marked by low b

217 Anorexia nervosa is a psychiatric disorder of unknown et

218 Anorexia nervosa is a severe psychiatric disorder associ

219 Treatment of patients with anorexia nervosa is a topic that continues to spark deba

220 Anorexia nervosa is a well characterized disorder with r

221 Anorexia nervosa is an important cause of physical and p

222 A core feature of anorexia nervosa is an over-estimation of body size.

223 ological impact of exercise in patients with anorexia nervosa is complex, and exercise may have a neg

224 Anorexia nervosa is prevalent in adolescents and young a

225 In recently weight-restored women with anorexia nervosa, lower percent body fat was associated

226 Individuals who have recovered from anorexia nervosa may have difficulties in differentiatin

227 The study included 30 women with anorexia nervosa (mean age +/- standard deviation, 26 ye

228 bulimia nervosa (mean=5.8 years, SD=9.1) or anorexia nervosa (mean=5.9 years, SD=7.4).

229 isorder, depression, suicide, schizophrenia, anorexia nervosa, migraine, dementia, and PD.

230 The genetic architecture of anorexia nervosa mirrors its clinical presentation, show

231 and no black women met lifetime criteria for anorexia nervosa; more white women (N=23, 2.3%) than bla

232 articles and conference abstracts addressing anorexia nervosa (n = 13), bulimia nervosa (n = 6), and

233 (N = 246) diagnosed as having either DSM-IV anorexia nervosa (n = 136) or bulimia nervosa (n = 110)

234 omparison women (N=14), women recovered from anorexia nervosa (N=14) had significantly diminished and

235 o-controlled trial of adult outpatients with anorexia nervosa (N=152, 96% of whom were women; the sam

236 over 8 to 25 years in 1,885 individuals with anorexia nervosa (N=177), bulimia nervosa (N=906), or ea

237 isorder (N=131), bulimia nervosa (N=17), and anorexia nervosa (N=18).

238 lumes in women with current restricting-type anorexia nervosa (N=19), women recovered from restrictin

239 METHOD: Female adolescents with anorexia nervosa (N=21; mean age, 16.4 years [SD=1.9]) u

240 N=19), women recovered from restricting-type anorexia nervosa (N=24), women with bulimia nervosa (N=1

241 ixteen deaths (6.5%) were recorded (lifetime anorexia nervosa, N=14; bulimia nervosa with no history

242 sa, N=14; bulimia nervosa with no history of anorexia nervosa, N=2).

243 For adults with anorexia nervosa, no one specialist treatment has been s

244 enetic correlation between the Eyes Test and anorexia nervosa, openness (NEO-Five Factor Inventory),

245 aging studies of adolescents and adults with anorexia nervosa or bulimia nervosa that, together, impl

246 A total of 216 women with a diagnosis of anorexia nervosa or bulimia nervosa were followed for 7

247 , 246 treatment-seeking female patients with anorexia nervosa or bulimia nervosa were interviewed eve

248 ey are a vulnerability factor for developing anorexia nervosa or bulimia nervosa.

249 ourished subjects or clinical states such as anorexia nervosa or cancer cachexia.

250 In humans with anorexia nervosa or kwashiorkor, ghrelin and growth horm

251 premature death among patients with lifetime anorexia nervosa peaked within the first 10 years of fol

252 t, particularly for adolescent patients with anorexia nervosa, point to the benefits of specialised f

253 nts were 7303 Swedish women hospitalized for anorexia nervosa prior to age 40 years between 1965 and

254 h general population, women hospitalized for anorexia nervosa prior to age 40 years had a 53% (95% co

255 Of the five male anorexia nervosa probands, only one was from an opposite

256 anuary 2000 until May 2005, 93 patients with anorexia nervosa received intensive inpatient or day-pro

257 hanges in cerebral glucose metabolism in key anorexia nervosa-related structures at both 6 months and

258 The pathophysiology of anorexia nervosa remains obscure, but structural brain a

259 Patients with anorexia nervosa reported higher levels of perceived coe

260 were aged 20-60 years and had a diagnosis of anorexia nervosa (restricting or binge-purging subtype)

261 nervosa, binge eating/purging type);14 with anorexia nervosa, restricting type; and 13 healthy compa

262 Research in Anorexia Nervosa (RIAN) is a 2-group (FBT and SyFT) rand

263 studies have consistently demonstrated that anorexia nervosa runs in families.

264 learning-based interventions for addiction, anorexia nervosa, schizophrenia, and depression.

265 Two patterns emerge: (1) anorexia nervosa, schizophrenia, obsessive-compulsive di

266 care in adolescent patients with non-chronic anorexia nervosa seems no less effective than IP for wei

267 Physicians treating patients with anorexia nervosa should carefully assess patterns of alc

268 Anorexia nervosa shows a stronger genetic correlation wi

269 I), mood, anxiety, affective regulation, and anorexia nervosa-specific behaviours at 12 months after

270 ggesting a combination of re-nourishment and anorexia nervosa-specific psychotherapy is most effectiv

271 and lower BMI at admission, and restrictive anorexia nervosa subtype predicted fatal outcome for ano

272 effect of percent body fat, body mass index, anorexia nervosa subtype, waist-to-hip ratio, and serum

273 gh rates of diagnostic "crossover" among the anorexia nervosa subtypes and bulimia nervosa may reflec

274 een the restricting and binge eating/purging anorexia nervosa subtypes over time; one-third crossed o

275 a and bulimia nervosa but do not support the anorexia nervosa subtyping schema.

276 Similar results were found for anorexia nervosa (suicide attempts: crude, 4.42 [4.12-4.

277 Brain circuits believed to drive anorexia nervosa symptoms can be accessed with surgical

278 higher lifetime prevalence and incidence of anorexia nervosa than reported in previous studies, most

279 ions may represent a phenotype of adolescent anorexia nervosa that does not respond well to treatment

280 contribute information about bone health in anorexia nervosa that is independent of that provided wi

281 ings of altered striatal dopamine binding in anorexia nervosa, the authors sought to assess the respo

282 aking to eat is crucial for survival, but in anorexia nervosa, the brain persistently supports reduce

283 usal genes from the largest genetic study of anorexia nervosa to date were enriched for expression in

284 This model helps explain the resistance of anorexia nervosa to interventions that have established

285 ed meal, 26 women who were in remission from anorexia nervosa (to avoid the confounding effects of ma

286 Adults with anorexia nervosa too have a realistic chance of achievin

287 Fluoxetine to Prevent Relapse in Women With Anorexia Nervosa trial.

288 The h(2)SNP of anorexia nervosa was 0.20 (SE=0.02), suggesting that a s

289 The lifetime prevalence of DSM-IV anorexia nervosa was 2.2%, and half of the cases had not

290 Anorexia nervosa was independently predicted by multiple

291 Anorexia nervosa was inversely associated with the devel

292 win pairs, the prevalence of DSM-IV or broad anorexia nervosa was not significantly different than th

293 Historically, anorexia nervosa was viewed as a disorder primarily infl

294 ncidence rates, and 5-year recovery rates of anorexia nervosa were calculated on the basis of data fr

295 16 patients with treatment-refractory anorexia nervosa were enrolled between September, 2011,

296 ter hospitalization, 33 patients with DSM-IV anorexia nervosa were randomly assigned to 1 year of out

297 tem responsiveness is elevated in adolescent anorexia nervosa when underweight and after weight resto

298 closer to understanding the neurobiology of anorexia nervosa, which still remains a mystery and pose

299 The authors found that individuals with anorexia nervosa, who make maladaptive food choices to t

300 Given the progress of genomic discovery in anorexia nervosa, with the identification of the first g