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1 he distal motor nerve up to the level of the anterior horn cell.
2 s and nuclei and in the neuropil surrounding anterior horn cells.
3 ron" pattern: a symmetric involvement of the anterior horn cells.
4 iquitin-positive intraneuronal aggregates in anterior horn cells.
5 7% in Purkinje cells to 80.6 +/- 2.8% in the anterior horn cells.
6 corticospinal tract, without involvement of anterior horn cells.
7 udy is to investigate the involvement of the anterior horn cells (AHC) in the early post-stroke perio
9 ed atrophic spinal cords with marked loss of anterior horn cells and degeneration of corticospinal tr
10 d disorders characterized by degeneration of anterior horn cells and progressive muscle weakness.
12 atrophy, there is selective degeneration of anterior horn cells but a normal corticospinal tract.
15 non-immune forms of PNH that include toxins, anterior horn cell degeneration in motor neurone disease
17 re syndrome 1 and lethal arthrogryposis with anterior horn cell disease are autosomal recessive fetal
19 41 million (95% CI, $1132-$1351 million) for anterior horn cell disease; $75 million (95% CI, $57-$92
20 Virus antigen was localized predominantly to anterior horn cells in infected IFN-gamma(-/-) H-2(q) mi
23 of the central grey matter, with predominant anterior horn-cell involvement, and nine (75%) children
25 only to the primary cells of the periphery (anterior horn cells, motor axons and primary afferent se
26 oma-spinal cord-34 motor neurons and primary anterior horn cell neurons showed that IL-6 exerted a ne
33 erebral and brainstem neurons to spinal cord anterior horn cells; thus, severe poliomyelitis, but not
34 aracterized by selective degeneration of the anterior horn cells with subsequent weakness and atrophy