戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 have ectopic Shh and Ptch1 expression in the anterior limb.
2 Hand2 likely mediates Gli3 repression in the anterior limb.
3 ssively by power-strokes of each of the more anterior limbs.
4 ns in TBX5, a related and linked gene, cause anterior limb abnormalities in Holt-Oram syndrome.
5 ctyly phenotype with loss of digits from the anterior limb, although the development of more proximal
6 bers descend in the internal capsule (SMA in anterior limb and genu; M1 in posterior limb) and traver
7 ing activity, leading to derepression of the anterior limb and induction of polarizing zone marker ge
8 ed to connections with the internal capsule (anterior limb) and the pallidum.
9 n mouse led to ectopic Shh expression in the anterior limb bud and a preaxial polydactyly (PPD) skele
10 nd-independent Shh signaling, was induced in anterior limb bud cells resulting in extra digits.
11 duced extra, sometimes multiple digits, from anterior limb bud demonstrating the negative role in Shh
12 is required to inhibit Shh expression in the anterior limb bud mesenchyme.
13 of SMAD4 in upregulating target genes in the anterior limb bud mesenchyme.
14 nges in the regulation of development of the anterior limb bud resulting in ectopic expression of sig
15     Ectopic Ihh expression in the distal and anterior limb bud results in the ectopic activation of s
16   The polydactyly was preceded by unexpected anterior limb bud transcription of Shh, so one function
17 al cells, including mesenchymal cells in the anterior limb bud, and mice homozygous for targeted disr
18 topic sonic hedgehog (Shh) expression in the anterior limb bud.
19 F signaling to inhibit Shh expression in the anterior limb bud.
20  of ptc1 is to repress Shh expression in the anterior limb bud.
21  promoting precocious cell-cycle exit in the anterior limb bud.
22                                 In addition, anterior limb buds show aberrant Gli3 processing, consis
23 function abrogates ectopic Shh expression in anterior limb buds, limits overexpression in the zone of
24 ons in the Shh limb enhancer lead to similar anterior limb defects, highlight the importance of Shh r
25 , results in a severe truncation of proximal-anterior limb elements.
26  the pathways were widespread, involving the anterior limb, genu and posterior limb with the M3 proje
27 ough the middle and posterior portion of the anterior limb (ICa).
28 actyly exhibit ectopic Shh expression at the anterior limb margin.
29   Beads soaked in Shh-N do not induce Shh in anterior limb mesenchyme ruling out direct propagation o
30 ch are known repressors of Shh expression in anterior limb mesenchyme, strongly enhances the anterior
31  Msx-2 during normal limb development in the anterior limb mesoderm, the posterior necrotic zone, and
32 dulating the level of Gli3 processing in the anterior limb mesoderm.
33 integrity in the fibers that are part of the anterior limb of internal capsule (ALIC) in MDD and diab
34           Lower fractional anisotropy in the anterior limb of internal capsule at discharge and in ge
35 ve disorder who underwent DBS to the ventral anterior limb of internal capsule with subsequent progra
36 ubcortical, 188 x 10(-5) mm(2)/sec at birth; anterior limb of internal capsule, 130 x 10(-5) mm(2)/se
37 and/or maintain continued development of the anterior limb of the developing heart.
38  (-0.01; p = .02) of the corpus callosum and anterior limb of the internal capsule (-0.02; p =.01) at
39 al globus pallidus (GPe), nucleus accumbens, anterior limb of the internal capsule (ALIC) and anterio
40            Surgical procedures targeting the anterior limb of the internal capsule (aLIC) can be effe
41                         Disconnection of the anterior limb of the internal capsule (ALIC) was assesse
42  patients (N = 50) that underwent DBS to the anterior limb of the internal capsule (ALIC), the nucleu
43 c; right hemisphere, 1.17 x10(3)mum(2)/sec), anterior limb of the internal capsule (left, 1.11 x10(3)
44 ec; right hemisphere, 1.17 x10(3)um(2)/sec), anterior limb of the internal capsule (left, 1.11 x10(3)
45 dus internus (GPi) (n=47), nucleus accumbens/anterior limb of the internal capsule (n=4) or a combina
46  around the nucleus accumbens (P = .03), and anterior limb of the internal capsule (P = .02).
47 trial, received bilateral DBS of the ventral anterior limb of the internal capsule (vALIC) between Ap
48 cy and safety of DBS targeted at the ventral anterior limb of the internal capsule (vALIC) in 25 pati
49  study of DBS targeting the ventral striatum/anterior limb of the internal capsule (VS/ALIC) in 10 pa
50 1 year to evaluate the effects of DBS in the anterior limb of the internal capsule and nucleus accumb
51 uced fractional anisotropy (FA) in the right anterior limb of the internal capsule and right uncinate
52 ets have been used, mostly in and around the anterior limb of the internal capsule and ventral striat
53  OCD received DBS of the ventral part of the anterior limb of the internal capsule and were followed
54  OCD received DBS of the ventral part of the anterior limb of the internal capsule and were followed
55                   In addition, reduced FA in anterior limb of the internal capsule correlated signifi
56  are modulated by stimulation of the ventral anterior limb of the internal capsule for obsessive-comp
57 nteers revealed that fiber tracts within the anterior limb of the internal capsule have a significant
58 sed ultrasound (MRgFUS) trials targeting the anterior limb of the internal capsule have shown promisi
59 evidence that DBS of the ventral part of the anterior limb of the internal capsule is effective and t
60 CD patient cohorts (N = 50) receiving DBS to anterior limb of the internal capsule or subthalamic nuc
61 ateral lesions in the ventral portion of the anterior limb of the internal capsule over a 20-year per
62        Deep brain stimulation of the ventral anterior limb of the internal capsule rapidly improves m
63 Brown Obsessive Compulsive Scale, one in the anterior limb of the internal capsule that overlapped wi
64 n multiple regression, associations with the anterior limb of the internal capsule, a subcortical whi
65 primates form a well defined and topographic anterior limb of the internal capsule, the specific loca
66                            These include the anterior limb of the internal capsule, the ventral stria
67 re also associated with abnormalities in the anterior limb of the internal capsule, the white matter
68 mental manipulations of the striatum and the anterior limb of the internal capsule.
69  be viewed as the rat homolog of the primate anterior limb of the internal capsule.
70 mbic connection following the fornix and the anterior limb of the internal capsule.
71 side irradiated at the ventral border of the anterior limb of the internal capsule.
72  DBS electrodes bilaterally implanted in the anterior limb of the internal capsule.
73  chronic deep brain stimulation of the human anterior limb of the internal capsule/NAcc region for tr
74 dergoing deep brain stimulation (DBS) of the anterior limb of the internal capsule/NAcc region for tr
75 ergoing deep brain stimulation targeting the anterior limb of the internal capsule/nucleus accumbens.
76 , a small region of increased atrophy in the anterior limb of the left internal capsule adjacent to t
77  to sham deep brain stimulation (DBS) in the anterior limb of the ventral capsule/ventral striatum (V
78 esults explain why GLI3 is required only for anterior limb patterning and why GLI2 can compensate for
79 nd Shh gene dosage improves the integrity of anterior limb structures, validating the importance of t
80 he involvement of inhibitory pathways in the anterior limb that prevent secondary polarizing zone for