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1 birth date and are dispersed throughout the anterior lobe.
2 enchymal transition, is absent in the dorsal anterior lobe.
3 Rathke's pouch to cell specification in the anterior lobe.
4 yed recall also showed associations with the anterior lobe.
5 s highly expressed in the rostral tip of the anterior lobe.
6 er projection pattern in lobule VIII and the anterior lobe.
7 al granular layer of the mature meander tail anterior lobe.
8 round the portal venous branch supplying the anterior lobe.
9 e folliculostellate cells of the pituitary's anterior lobe.
10 erably lower amounts of staining observed in anterior lobes.
11 nal granular layer between the posterior and anterior lobes.
12 E-LPV) and/or the concentration of DA in the anterior lobe (AL) are inversely related to the secretio
14 The secretion of prolactin (PRL) from the anterior lobe (AL) of the pituitary gland is tonically i
17 nce (ME), decreased in the outer zone of the anterior lobe (AL-OZ), as well as the intermediate (IL)
18 eously from Purkinje cells in the paravermal anterior lobe and from muscles of the hand and arm in th
19 ells revealed widespread degeneration in the anterior lobe and in limited areas of the posterior lobe
22 pe, corticotrope and lactotrope cells in the anterior lobe and the intermediate lobe melanotropes.
24 of the paravermal C1 zone in lobule V of the anterior lobe and the rostral folia of the paramedian lo
26 motor cortex, bilateral putamen and insula, anterior lobe and vermis of the cerebellum and superior
27 gin, with the oral ectoderm contributing the anterior lobe, and the neural ectoderm generating the po
28 s they transition from Rathke's pouch to the anterior lobe appears to be essential for their movement
30 llum is greater than the number of PC in the anterior lobe, as classically defined by the primary fis
31 , and 3) the defect is not restricted to the anterior lobe but involves a portion of the posterior lo
32 tact in Rathke's pouch, the precursor to the anterior lobe, but the anterior lobe was hypoplastic.
33 expressed by folliculo-stellate cells in the anterior lobe, by a group of astrocyte-like cells and by
36 minant caudal pons projections to cerebellar anterior lobe, contrasted with associative-predominant r
37 ss the pro-opiomelanocortin (POMC) gene, the anterior lobe corticotropes, producing adrenocorticotrop
38 s inactivated the cerebellar nuclei, lateral anterior lobe, crus I, rostral crus II, and lobule HVI i
39 ound, in addition to the marked depletion of anterior lobe granule cells ( > 90%), there were also si
40 the neural ectoderm was sufficient to impair anterior lobe growth, but not the differentiation of hor
41 e pouch of conditional embryos led to severe anterior lobe hypoplasia with drastically reduced expres
43 pathology was confined to lobules I-V of the anterior lobe in patients with sporadic ALS in contrast
44 while the reduction of granule cells in the anterior lobes is substantial, there is an almost comple
45 had chronic hyperprolactinemia and developed anterior lobe lactotroph hyperplasia without evidence of
46 g from pairs of C1 zone sites located in the anterior lobe (lobule V) and C1 or C3 zone sites in rost
47 bjects had greater activity in the bilateral anterior lobe of cerebellum, premotor area, parietal cor
48 with, the caudal extent of the disorganized anterior lobe of meander tail and the rostral extent of
49 genitors transplanted into the granuloprival anterior lobe of neonatal mea mutants differentiated int
53 nditioning results in plasticity in both the anterior lobe of the cerebellar cortex and in the anteri
54 logical analysis suggests that damage to the anterior lobe of the cerebellar cortex is necessary and
57 near-total depletion of granule cells in the anterior lobe of the cerebellum, as well as aberrantly l
60 1 causes a morphological defect in which the anterior lobe of the pituitary gland protrudes through t
62 ground develop high-penetrance tumors of the anterior lobe of the pituitary, a class of tumors estima
64 Luteinizing hormone (LH), produced in the anterior lobe of the pituitary, is a member of the hypot
65 progressive, dose-dependent loss of the most anterior lobe of the vermis in mice lacking Fgf17 and in
66 ranule cells that predominantly populate the anterior lobes of the adult cerebellum and later, those
67 right superior temporal gyrus, posterior and anterior lobes of the cerebellum, and bilateral parahipp
68 right superior temporal gyrus, posterior and anterior lobes of the cerebellum, bilateral parahippocam
69 lexics exhibited significantly smaller right anterior lobes of the cerebellum, pars triangularis bila
71 ells were identified in the intermediate and anterior lobes of the pituitary, the thyroid and parathy
72 lly mea/mea granule cells are present in the anterior lobe or, unexpectedly, in the posterior lobe.
74 However in-depth analysis has focused on the anterior lobe progenitors (AP), ignoring the posterior p
75 te mouse gestation and the postnatal period, anterior lobe progenitors re-enter the cell cycle and ex
77 , loose the majority of granule cells in the anterior lobe resulting in few axons and atypical Purkin
78 ypoplastic with an abnormal branching of the anterior lobe, revealing a role for microRNAs in pituita
79 from the central vermis, and analysis of the anterior lobe reveals several missing zebrin II- bands.
82 ults in accelerated progression of pituitary anterior lobe tumors and medullary thyroid carcinomas.
84 e territory providing climbing fibres to the anterior lobe was centred more laterally than the territ
86 ellar vermis and left lobule V of cerebellar anterior lobe were additionally activated for dual-task
88 diate lobe and over a subset of cells in the anterior lobe, whereas CRF-R2 transcripts were expressed
89 ngs developed accelerated enlargement of the anterior lobe with predominantly TSH cell hyperplasia.