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1 between the immobilized IgG with a suitable anti-IgG.
2 and brlf1 mRNAs can be detected 1.5 h after anti-IgG.
3 e edge sites for subsequent amide linkage to anti-IgG.
4 phorylated following stimulation with either anti-IgG Abs or pervanadate in the murine B cell lymphom
7 of Akata cells reactivated from latency with anti-IgG and a lymphoblastoid cell line (LCL) reactivate
10 r system, we demonstrated 100% detachment of anti-IgG and IgG bound beads (which is on the same order
11 both, secondary and primary nanogold probes (anti-IgG and IgG coupled to AuNP, on double and single-a
14 , which blocked subsequent responses to OVA, anti-IgG, and anti-kappa, but also competed for binding
15 vation of EBV in Akata cells, in response to anti-IgG, and in Raji cells, in response to tetradecanoy
16 own to be expressed by 1 h after addition of anti-IgG, and their expression preceded that of bzlf1 an
18 he nonspecific interaction between AuNPs and anti-IgG antibodies to realize sensitive detection of Ig
19 ncept, we designed a mass-based sensor where anti-IgG antibodies were coated on a quartz crystal micr
24 of a B lymphoma cell line, A20, with intact anti-IgG antibody induced a direct, SH2-mediated associa
28 zacytidine), B cell receptor engagement with anti-IgG antibody, hydrogen peroxide, and the proteosome
30 odel sandwich assay system with biotinylated anti-IgG as the capture antibody, rabbit IgG as the anti
31 acrylate copolymer, an antibody/antigen (IgG/anti-IgG) assay was carried out to assess the performanc
34 d-phase binding assay that utilizes magnetic anti-IgG beads to capture CAP18(106-138)-IgG (and bound
36 ulin G (IgG) covalent immobilization, an IgG/anti-IgG bioassay was implemented along the grating regi
37 ected before 2 h in Akata cells treated with anti-IgG, but both long- and short-duration stimuli requ
38 s required up to 1.25 h after application of anti-IgG; bzlf1 and brlf1 mRNAs can be detected 1.5 h af
39 o anti-human and anti-rabbit immunoglobulin (anti-IgG) concentrations less than 100 nM using only 10
42 ivity was further enhanced using a secondary anti-IgG detection antibodies to give a limit of detecti
44 pecific binding and have been able to detect anti-IgG even in the presence of 100-fold larger concent
45 In contrast, stimulation of A20 cells with anti-IgG F(ab')2 resulted in little increase in the asso
46 fluorescein-5'-isothiocyanate (FITC)-labeled anti-IgG F(ab) antibodies to the recycling endosomes in
47 ) using nanomagnetic beads capture probe and anti-IgG functionalized-fluorescence nanoparticles as th
48 ity constants reported in the literature for anti-IgG/IgG binding pairs and provides intrinsic detect
51 n of the cells with either F(ab')2 or intact anti-IgG induced very similar changes in levels of tyros
54 ERS), which utilizes a Fe3O4@Ag@streptavidin@anti-IgG nanocomposite with strong magnetic properties a
55 selectivity was achieved by the presence of anti-IgG on the surface of silver nanoparticles coupled
58 aling of protein kinase A was blocked by CM, anti-IgG, or by specific inhibitors of the beta-adrenerg
59 ry antigen bound to microtitration wells and anti-IgG- plus anti-IgM-coated indicator erythrocytes as
60 over the location of anti-immunoglobulin G (anti-IgG) proteins bound to Au nanoplates formed on glas
61 ure antibody, rabbit IgG as the antigen, and anti-IgG-R-phycoerythrin as the reporter antibody, we de
64 e alkali hydrolysis of the FITC@SiO(2)-NH(2)-anti-IgG released FITC molecules, leading to an amplifie
65 (r approximately 0.85; P < 0.00001) with the anti-IgG results but showed no advantage over measuring
66 g of anti-MOG.MOG complexes with a secondary anti-IgG results in the lipid raft-dependent phosphoryla
73 c cycle activation occurs very rapidly after anti-IgG treatment, de novo protein synthesis is also re
75 tion of Cy5 labeled IgG and Alexa633 labeled anti-IgG using a single laser source (636 nm excitation)
76 horylation of p40/42 in A20 cells induced by anti-IgG was rapid and transient, peaking at 2 min after
77 ntrol zone, antibody of S. typhi O901 and an anti-IgG were dotted on the nitrocellulose membrane, res
79 died the interaction of fluorescently tagged anti-IgG with surface immobilized IgG controlled by elec