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1 e production in RAW264.7 cells (a measure of anti-inflammation).
2 possible regulatory pathways of MC1-R beyond anti-inflammation.
3  applied to antimutation, antityrosinase and anti-inflammation.
4 compounds i.e. anti-cancer, anti-allergy and anti-inflammation.
5  for its pharmacological functions including anti-inflammation.
6 resistance by reorienting macrophages toward anti-inflammation.
7 anced cellular DNA repair, antioxidation and anti-inflammation.
8 f miR-33 that mediates simvastatin-triggered anti-inflammation.
9 nnexin A1 (AnxA1), an effector of endogenous anti-inflammation.
10 cal activities including anticoagulation and anti-inflammation.
11 ects in vasodilation, anti-angiogenesis, and anti-inflammation.
12 bitors with in vivo antineuropathic pain and anti-inflammation.
13 o M1-type immune functions but also boosting anti-inflammation.
14 Progesterone treatment showed no significant anti-inflammation action on cytokine concentrations.
15 -trained monocytes can effectively propagate anti-inflammation activity to neighboring monocytes thro
16 ty quickly after the arthritis induction and anti-inflammation analgesic treatment by intra-articular
17 ced neurodegeneration involving antioxidant, anti-inflammation and anti-apoptotic mechanisms.
18                                          The anti-inflammation and anti-infection functions for Vitam
19          The involvement of Vitamin D/VDR in anti-inflammation and anti-infection represents a newly
20 ovel cyclooxygenase inhibitors, may serve as anti-inflammation and anticancer agents, and may contrib
21                        Berberine has several anti-inflammation and anticancer biologic effects.
22 nt of green tea extract, exhibits effects of anti-inflammation and antioxidation on periodontal infla
23 sts that WLT can be applied to antimutation, anti-inflammation and antityrosinase.
24 ialized proresolving mediators in regulating anti-inflammation and antiviral response is also discuss
25 atins have shown pleiotropic effects such as anti-inflammation and bone stimulation.
26 ontributing to the development of a state of anti-inflammation and dichotomy of immunologic polarizat
27 -infected cell corpses suppresses macrophage anti-inflammation and efficient tissue repair programs a
28 senting atheroprotective flow, maintains the anti-inflammation and homeostatic phenotype of ECs, but
29 n important role in endogenous mechanisms of anti-inflammation and pro-resolution.
30 sal antimicrobial responses demonstrate that anti-inflammation and proresolution are different respon
31  Se via metabolic reprograming to facilitate anti-inflammation and proresolution.
32 tasis and modulate immune function to effect anti-inflammation and resolution.
33    There is an important distinction between anti-inflammation and resolution; anti-inflammation is p
34 d protective processes: Th2 cytokine-induced anti-inflammation and ROS reduction.
35 ompound including anticancer, antimicrobial, anti-inflammation, and anti-diabetic activities, as well
36 ysiological effects, including vasodilation, anti-inflammation, and cardioprotection.
37 l in vitro screening, such as anti-diabetic, anti-inflammation, and in silico analysis.
38 the effects of MSC therapy on injury volume, anti-inflammation, and neurogenesis.
39 ced KLF2 and eNOS expression as well as flow anti-inflammation, and suggest that HDAC5 could be a pot
40  signaling, cell migration, tissue survival, anti-inflammation, and T-cell-mediated cellular immunity
41                 This could stem from in situ anti-inflammation, antioxidation, and nerve protection.
42 tiatherogenic effects of HDL (antioxidation, anti-inflammation, antithrombotic effects, endothelial s
43 d injury model through a unique mechanism of anti-inflammation-based fibrotic scar reduction.
44 acid-stimulated neutrophils, consistent with anti-inflammation but not COX inhibition (IC50s = 1-8 mM
45 pha transcription and suggest a mechanism of anti-inflammation by the antioxidants through control of
46       It has many health benefits, including anti-inflammation, cancer prevention, and anti-diabetes.
47 efficacy in lung function, QOL, and possibly anti-inflammation compared with BUD plus Tulo treatment.
48 ic functions for this effector of endogenous anti-inflammation could be unveiled by studying collagen
49 tissue homeostasis, in part by secreting the anti-inflammation cytokine interleukin-10 (IL-10).
50 ory cytokines and antibodies while increased anti-inflammation cytokines, finally leading to restored
51 haracterize the balance of systemic pro- and anti-inflammation early after burn and inhalation injury
52 2-nHP66 scaffold exerts potent antibacterial/anti-inflammation effects in vivo and promotes bone form
53  CRISPR or PKA inhibitor (H89) abolished the anti-inflammation effects of INT-777, on NLRP3-ASC infla
54 s but also performed excellent antitumor and anti-inflammation efficacy for intestinal disease treatm
55 )-induced metabolic diseases due to enhanced anti-inflammation engineered by lowering receptor intera
56 ranscriptional regulator for antioxidant and anti-inflammation enzymes that binds to its endogenous i
57                     Our study identified the anti-inflammation functional domain of SMAD7 and suggest
58 a suggest positive effects on antioxidation, anti-inflammation, hepatoprotection, intestinal protecti
59 a-induced genes featuring cell migration and anti-inflammation, highlighting intertumoral diversity o
60 ta has been shown to play a critical role in anti-inflammation; however, the signaling mechanisms of
61                                              Anti-inflammation immunotherapy has been successfully ap
62 10 was a critical mediator for PGRN-mediated anti-inflammation in collagen-induced arthritis by using
63 s model to explore the importance of dynamic anti-inflammation in promoting resolution of infection a
64 on between anti-inflammation and resolution; anti-inflammation is pharmacologic intervention in infla
65 in the microglial cells while downregulating anti-inflammation M2 phenotype.
66 o be a key pathway by which immunomodulation/anti-inflammation may operate.
67 on the resultant cellular anti-oxidation and anti-inflammation (NO, IL-8 secretion).
68 tic animal model for AF valuable for testing anti-inflammation or other therapies for AF.
69 tosterol showed a strong Th2-inclination and anti-inflammation potential in vitro.
70 sting that these terpenoid compounds have an anti-inflammation potential through the inhibition of T-
71 in the 1980s demonstrated that pharmacologic anti-inflammation prevented and slowed the progression o
72 3 eicosapentaenoic acid that displays potent anti-inflammation/pro-resolution actions in vivo.
73 ein A-I mimetic peptide with antioxidant and anti-inflammation properties on a high-fat or normal cho
74 -beta1 is a pleiotropic cytokine with potent anti-inflammation property.
75 n (EPRAP), whose function in PGE(2)-mediated anti-inflammation remains undefined.
76 tion and bone loss, suggesting that enhanced anti-inflammation status is an active process.
77 ng in extra-hepatic tissues by statins as an anti-inflammation strategy.
78 ng Gbetagamma-regulation may be an effective anti-inflammation strategy.
79 e addiction state and how microglia-targeted anti-inflammation therapies affect addiction are reviewe
80    To investigate the molecular mechanism of anti-inflammation, we analyzed the regulation of tumor n
81 lockade up-regulated myeloid CXCL10-mediated anti-inflammation with decreased CXCL14-mediated myeloid