ライフサイエンスコーパス: anti-inflammatory

1 Thus, the anti-inflammatory action of Glp1r agonism was tested in

2 Furthermore, the anti-inflammatory action of Treg cells on innate immune

3 ple mechanisms potentially contribute to the anti-inflammatory actions of methotrexate, including the

4 onists (VDRAs) may have nephroprotective and anti-inflammatory actions, but their mechanisms of actio

5 egulates a shift between proinflammatory and anti-inflammatory activation during ER stress-induced in

6 es possessing multiple chemokine-binding and anti-inflammatory activities can be developed from the c

7 We investigated whether the anti-inflammatory activities of aspirin (acetylsalicylic

8 o toxicity, antioxidant, antinociceptive and anti-inflammatory activities of faveleira seed oil.

9 solid proofs of keeping both antioxidant and anti-inflammatory activities of peptides from cooked pro

10 tent in Ismailia 1 cultivar and improved the anti-inflammatory activities through inhibiting cyclooxy

11 r light boosted the antioxidant capacity and anti-inflammatory activities through inhibiting cyclooxy

12 s and enzymes as well as the antioxidant and anti-inflammatory activities were determined in laser-tr

13 ng, respectively for optimal antioxidant and anti-inflammatory activities with favorable consumer's a

14 nd known to be involved in antimicrobial and anti-inflammatory activities, showed a twofold increase

15 prebiotic effect, protection from pathogens, anti-inflammatory activity and reduction of the risk of

16 Our results indicate that PRF holds an anti-inflammatory activity and shifts the macrophage pol

17 ty, anti-inciting action, and suppression of anti-inflammatory activity may limit their development a

18 lysis to reduce cytotoxicity and improve the anti-inflammatory activity of the parental peptides.

19 other proteasome inhibitors, exhibits potent anti-inflammatory activity.

20 Topical nonsteroidal anti-inflammatory agents (NSAIDs) proved to have the gre

21 the disease, including antifibrotic agents, anti-inflammatory agents and antioxidants.

22 failure, as well as TD139 and Arglabin, two anti-inflammatory agents new to cardiac diseases, to pre

23 posing or engineering Evasins as therapeutic anti-inflammatory agents.

24 tory activity may limit their development as anti-inflammatory agents.

25 e, reinforcing the use of EP2 antagonists as anti-inflammatory agents.

26 t the use of catalytic inhibitors of USP7 as anti-inflammatory agents.

27 ent a highly promising class of antiviral or anti-inflammatory agents.

28 We found that the frequency of CD163(+) anti-inflammatory AM was decreased, whereas CD163(-)CCR7

29 nities for assessing the in vivo efficacy of anti-inflammatory and anti-cancer therapeutics.

30 Drugs with anti-inflammatory and anti-fibrotic effects are of promi

31 tic acid and hydroxystearic acid (PAHSA) are anti-inflammatory and antidiabetic lipokines that connec

32 oisomerization of colchicine deactivates its anti-inflammatory and antimitotic properties.

33 dy, we have used Sinomenine (Sino), a potent anti-inflammatory and antioxidant drug conjugated to hyd

34 bition of BD1 domains is sufficient to drive anti-inflammatory and antiproliferative effects.

35 Apo M exerts anti-inflammatory and cardioprotective effects in animal

36 rom mycobacterial Cpn60.1 has a long-lasting anti-inflammatory and immunomodulatory activity which ma

37 Treatments for IPF are limited to anti-inflammatory and immunomodulatory agents, which are

38 cesses in the heart failure setting, such as anti-inflammatory and immunomodulatory strategies.

39 Our data indicate that anti-inflammatory and immunosuppressive mechanisms are n

40 EP2 antagonist 1 (TG6-10-1), which displayed anti-inflammatory and neuroprotective actions in rodent

41 Anti-inflammatory and other cytokines, as well as chemok

42 e macrolides have been shown to also possess anti-inflammatory and other therapeutic activities in ma

43 ylethanolamine-oxazoline (PEA-OXA) possesses anti-inflammatory and potent neuroprotective effects.

44 ion between Th2 and Tregs subsets creates an anti-inflammatory and proreparative milieu associated wi

45 an endogenous lipid mediator with compelling anti-inflammatory and proresolution properties.

46 Taking also into considerations its anti-inflammatory and thrombin-inhibitory actions, a bio

47 ties, such as antioxidant, anti-proliferate, anti-inflammatory, and antimicrobial activities.

48 , anticancer, antihyperlipidemia, antiulcer, anti-inflammatory, and hepatoprotective activities.

49 nificant pharmaceutical compounds, including anti-inflammatory, anti-depressant, anti-bacterial, anti

50 biological activities, including analgesic, anti-inflammatory, anti-HIV, antidiabetic, antitumor, an

51 tioxidant activity, also presenting moderate anti-inflammatory, anti-proliferative, and antimicrobial

52 vities including antioxidant, antimicrobial, anti-inflammatory, anticancer, antidiabetic, wound heali

53 can substantially enhance their antioxidant, anti-inflammatory, antimutagenic, antitumoral or chemopr

54 determine whether N-acetylcysteine (NAC), an anti-inflammatory antioxidant, attenuates programmed sus

55 of all features, focusing on compounds with anti-inflammatory, antioxidant or antimicrobial properti

56 Based on the anti-inflammatory benefits of the upregulation of the AC

57 trometry measurements of a panel of pro- and anti-inflammatory bioactive lipids, we measure the profi

58 reported to act as both antidepressants and anti-inflammatories, but the mechanisms for these effect

59 milar antifungal activity but did not reveal anti-inflammatory capacity or cytotoxicity to the tested

60 Here, we report that NMP is a bioactive anti-inflammatory compound well tolerated in vivo, that

61 aol metabolites, M2 and M13, are more potent anti-inflammatory compounds than 6-shogaol itself; NL na

62 ated epoxy fatty acids (EpFA) are endogenous anti-inflammatory compounds, which are converted by the

63 data demonstrates that over-expression of an anti-inflammatory cytokine and a decoy chemokine recepto

64 IL-35 is an anti-inflammatory cytokine and is thought to be produced

65 IL-10 is a potent anti-inflammatory cytokine capable of suppressing a numb

66 macrophage infiltration, proinflammatory and anti-inflammatory cytokine expression, and fibrosis alon

67 Surprisingly, the anti-inflammatory cytokine IL-10, but not the pro-inflam

68 correlation between MARCO expression and the anti-inflammatory cytokine IL37.

69 oliferating cell nuclear antigen (PCNA), the anti-inflammatory cytokine interleukin (IL) 10, and the

70 egions that are the main local source of the anti-inflammatory cytokine interleukin-10 (IL-10) and th

71 posure of PMN to Mtb LAM did elicit pro- and anti-inflammatory cytokine production and release in a T

72 ts, and normalization of proinflammatory and anti-inflammatory cytokine production by macrophages.

73 ur results also show that rolipram increases anti-inflammatory cytokine production.

74 10 responses, and consequently with high pro/anti-inflammatory cytokine ratios.

75 STAT proteins can regulate both pro- and anti-inflammatory cytokine signaling.

76 Interleukin 10 (IL-10) is an anti-inflammatory cytokine that may be protective agains

77 ssion of Interleukin-10 (IL-10), a classical anti-inflammatory cytokine, extends lifespan in the SOD1

78 microglia, along with elevated levels of the anti-inflammatory cytokine, IL-10 were observed in the h

79 mad4 is sufficient for the expression of the anti-inflammatory cytokine, IL-10, in T(H)17 cells.

80 eceptor antagonists (IL-1Ra, IL-36Ra), and 2 anti-inflammatory cytokines (IL-37, IL-38).

81 The anti-inflammatory cytokines (interleukin-4 [IL-4], IL-6,

82 of pro-inflammatory cytokines (negative) and anti-inflammatory cytokines (positive) allow for the sel

83 ry responses are potentially counteracted by anti-inflammatory cytokines and type 2 responses.

84 TNF-alpha, IFN-gamma, and IL-2 more than the anti-inflammatory cytokines IL-4 and IL-10 in tonsil and

85 ition of NFkappaB signaling, increase of the anti-inflammatory cytokines IL10 and IL12P40, and increa

86 e induction of mRNA transcripts which encode anti-inflammatory cytokines that promote inflammation re

87 Overall, the association of mostly anti-inflammatory cytokines with less tau pathology sugg

88 fection has been established, the effects of anti-inflammatory cytokines, such as IL-10, in TB are le

89 t of M1, since they did not produce pro- and anti-inflammatory cytokines.

90 We aimed to investigate if an anti-inflammatory diet reduces disease activity in patie

91 ed Trem2 expression, thus preventing TREM2's anti-inflammatory drive.

92 hip between early postoperative nonsteroidal anti-inflammatory drug (NSAID) administration and postop

93 hydrase inhibitor in 6 eyes and nonsteroidal anti-inflammatory drug (NSAID) drops in 2 eyes.

94 Multitarget nonsteroidal anti-inflammatory drug (NSAID)-carbonic anhydrase inhibi

95 ither diagnose or differentiate nonsteroidal anti-inflammatory drug (NSAID)-exacerbated respiratory d

96 Nonsteroidal anti-inflammatory drug (NSAID)-exacerbated respiratory d

97 The anti-inflammatory drug candidate, 6-shogaol, has demonst

98 % of patients with a suspected non-steroidal anti-inflammatory drug hypersensitivity (NSAIDH), challe

99 Leflunomide is an anti-inflammatory drug primarily used for treating rheum

100 The anti-inflammatory drug sulfasalazine programs macrophage

101 ly active, not smoke, and avoid nonsteroidal anti-inflammatory drug use except aspirin prescribed for

102 ebroventricular infusion of indomethacin, an anti-inflammatory drug, mitigates the effect of ALI on f

103 We examined the role of RGS4 in nonsteroidal anti-inflammatory drug-associated bronchoconstriction by

104 jury induced by indomethacin, a nonsteroidal anti-inflammatory drug.

105 d by topical steroids (86%) and nonsteroidal anti-inflammatory drugs (66%), and accounted for 26%, 37

106 treatments such as antibiotics (rifaximin), anti-inflammatory drugs (mesalazine) and probiotics, alo

107 gressive consumption growth of non-steroidal anti-inflammatory drugs (NSAIDs) has progressively raise

108 Hypersensitivity to nonsteroidal anti-inflammatory drugs (NSAIDs) is of great concern bec

109 ated the remarkable efficacy of nonsteroidal anti-inflammatory drugs (NSAIDs) such as ibuprofen, napr

110 Although nonsteroidal anti-inflammatory drugs (NSAIDs) target primarily cycloo

111 The use of nonsteroidal anti-inflammatory drugs (NSAIDs) with oral anticoagulant

112 llions of people worldwide take nonsteroidal anti-inflammatory drugs (NSAIDs), often in conjunction w

113 ate response or intolerance to non-steroidal anti-inflammatory drugs (NSAIDs).

114 treated melanoma transplants with nonsteroid anti-inflammatory drugs and show that higher melanoma di

115 Since anti-inflammatory drugs are beneficial as adjuvants to c

116 be responsive to glucocorticoid, a class of anti-inflammatory drugs commonly used in DMD patients.

117 the development of IL-37 and IL-38 as novel anti-inflammatory drugs has not been considered adequate

118 h ionized and neutral states of nonsteroidal anti-inflammatory drugs must be considered to clarify th

119 mmatory diseases involves a long-term use of anti-inflammatory drugs such as corticosteroids and nons

120 utrophils as a universal platform to deliver anti-inflammatory drugs to promote tissue regeneration i

121 d supplementation of cells with nonsteroidal anti-inflammatory drugs was able to restore the level of

122 tment involves intravenous administration of anti-inflammatory drugs which have limited clinical outc

123 adverse effects, glucocorticoids (GC, potent anti-inflammatory drugs) are used extensively in clinica

124 ingestion of aspirin and other nonsteroidal anti-inflammatory drugs, a disorder termed aspirin-exace

125 ugs such as corticosteroids and nonsteroidal anti-inflammatory drugs, often accompanied by dose-depen

126 quid), and potential cofactors (nonsteroidal anti-inflammatory drugs, protonic pump inhibitors, exerc

127 ied include aspirin, nonaspirin nonsteroidal anti-inflammatory drugs, statins, agents that target met

128 treatment of pericarditis is represented by anti-inflammatory drugs.

129 d as a starting point for the development of anti-inflammatory drugs.

130 epresent an important family of nonsteroidal anti-inflammatory drugs.

131 of toxicity and to ultimately develop safer anti-inflammatory drugs.

132 The anti-inflammatory effect associated with flavonoids cont

133 romising alternative strategy to harness the anti-inflammatory effect exerted by activation of the AH

134 This anti-inflammatory effect is in part dependent on the TGR

135 the actions of M2 and M13 mostly confer the anti-inflammatory effect of 6-S-NL.

136 e upregulation, suggesting a VDR-independent anti-inflammatory effect of paricalcitol.

137 ous effects of LPS, suggesting a synergistic anti-inflammatory effect of PS/drug formulations.

138 rmed increased PPAR-target expression and an anti-inflammatory effect with saroglitazar.

139 ment to infection foci, and 3) production of anti-inflammatory effectors IL-10 and thioredoxin 1.

140 a innate immunity pathway is associated with anti-inflammatory effects and a reduced risk of recurren

141 AdipoRon also exerted anti-inflammatory effects by reducing microglial and ast

142 periments implicated a humoral factor in the anti-inflammatory effects generated by endothelial PHD2/

143 IAP also exhibits anti-inflammatory effects in a Toll-like Receptor-4 (TLR

144 ha (PPAR-alpha) agonist that exerts profound anti-inflammatory effects in animal models.

145 VDR activation exerts anti-inflammatory effects in immune cells.

146 The anti-inflammatory effects of alpha-MSH were blocked by s

147 n of several spleen immune cell subsets, the anti-inflammatory effects of Delta9-THC were preserved i

148 P may induce autophagy which may mediate the anti-inflammatory effects of IAP.

149 ages; however, NRF2 was not required for the anti-inflammatory effects of OI.

150 The anti-inflammatory effects of the hybrid peptide were mai

151 The anti-inflammatory effects of vagus nerve stimulation are

152 How MVs exert their anti-inflammatory effects or whether their tolerance-ind

153 ons drives the vagal-adrenal axis, producing anti-inflammatory effects that depend on NPY(+) adrenal

154 yphenolic compound with redox scavenging and anti-inflammatory effects, and poly(N-vinylpyrrolidone)

155 Artemisinins have also shown anti-inflammatory effects, including inhibition of inter

156 s a pleotropic cytokine with potent pro- and anti-inflammatory effects.

157 NSAIDs have analgesic, opioid-sparing, and anti-inflammatory effects.

158 rachidonic acid cascade leads to synergistic anti-inflammatory effects.

159 lly administrated 6-S-NL exhibits a superior anti-inflammatory efficacy likely due to the controlled

160 tabolites of 6-shogaol may contribute to the anti-inflammatory efficacy of 6-S-NL.

161 frontal cortex expression of the antioxidant/anti-inflammatory enzyme heme oxygenase-1 (HO-1) and inc

162 lpha, IL-1beta, and IL-6) and up-regulate an anti-inflammatory factor (IL-10) in inflamed Raw 264.7 c

163 n: they can acquire both a pathogenic and an anti-inflammatory fate.

164 mmals, IL-10 appears to have a more striking anti-inflammatory function than IL-4-like cytokines and

165 Both pro- and anti-inflammatory functions have been described, but the

166 selective access to IL-2 that supports their anti-inflammatory functions in vivo.

167 Beyond anti-inflammatory functions, miR-146a is a known tumor s

168 dependent epigenetic reprogramming to elicit anti-inflammatory gene expression.

169 sed energy utilization and the expression of anti-inflammatory genes.

170 o-inflammatory genes and the upregulation of anti-inflammatory genes.

171 n DTPs and thereby confers susceptibility to anti-inflammatory glucocorticoids (GCs).

172 To demonstrate the presence of these anti-inflammatory, growth-promoting macrophages in a hum

173 physical exercise, and certain non-steroidal anti-inflammatories have been shown to alter kynurenine

174 the highest phenolic compounds content, and anti-inflammatory (IC(50) = 72 ug/mL) and cytotoxic (GI(

175 Secondly, Pf increased anti-inflammatory IL-1RA and reduced neutrophil degranul

176 y cytokines IL-6, IL-18, and MIG, as well as anti-inflammatory IL-2 that were upregulated in a correl

177 Moreover, we show that anti-inflammatory IL-2C can deliver survival signals to

178 nflammatory proteins upregulated by pro- and anti-inflammatory IL-2C treatment and uncover a pattern

179 w that treatment of mice with a prototypical anti-inflammatory IL-2C, JES6-1-IL-2C, best known to ind

180 metabolism dependency of proinflammatory and anti-inflammatory ILC2 phenotypes.

181 perimental studies investigating the complex anti-inflammatory, immunomodulatory, and tissue-repair p

182 Consistent with the increased anti-inflammatory/immunosuppressive phenotype in vitro,

183 ctivities in primary human tissues, activate anti-inflammatory interleukin-10 and improve colitis sym

184 Vamorolone is a first-in-class anti-inflammatory investigational drug that has shown ev

185 and the switch from a pro-inflammatory to an anti-inflammatory lesion environment.

186 the inhibitory effect of arctigenin, a novel anti-inflammatory lignan, on prostate cancer in obese co

187 iomarkers of inflammation, oxidative stress, anti-inflammatory lipid mediators, tissue injury and rep

188 urther, ACE10 macrophages presented distinct anti-inflammatory (low inducible nitric oxide synthase a

189 ase (5-LO) pathways and concurrently reduces anti-inflammatory LXA4 secretion to maintain KSHV latenc

190 crophages can phagocytose tumor cells, while anti-inflammatory M2 macrophages such as tumor-associate

191 to either a proinflammatory (M1) state or an anti-inflammatory (M2) state.

192 nflammatory-M1) and alternatively activated (anti-inflammatory-M2) phenotypes.

193 activating pulp stem cells and promoting an anti-inflammatory macrophage response.

194 In fact, pro- and anti-inflammatory macrophages differ in the expression o

195 a 4T1 mouse model additionally revealed that anti-inflammatory macrophages promoted HIF-associated va

196 rved a significant shift favoring reparatory/anti-inflammatory macrophages with L-AZM formulation.

197 ed short-acting beta-agonists (SABAs), while anti-inflammatory maintenance with inhaled corticosteroi

198 Of note, heparin increased the anti-inflammatory markers arginase 1 and interleukin-10

199 old difference in the expression of pro- and anti-inflammatory markers.

200 tion in vitro and in vivo and its underlying anti-inflammatory mechanisms.

201 Nitro-fatty acids are electrophilic anti-inflammatory mediators which are generated during m

202 -inflammatory factors, without alteration of anti-inflammatory mediators, and significantly attenuate

203 eas their presence in NPH was accompanied by anti-inflammatory mediators.

204 ggests that targeting the immune system with anti-inflammatory medication holds the potential to infl

205 potential means for targeting treatment with anti-inflammatory medications.

206 lammation and to NAAA itself as a target for anti-inflammatory medicines.

207 d from GLA and result in proinflammatory and anti-inflammatory metabolites, respectively.

208 miR-146a was originally identified as an anti-inflammatory microRNA that targets signaling protei

209 called efferocytosis, and thereby promote an anti-inflammatory milieu.

210 ignificant increase in the expression of the anti-inflammatory molecule ubiquitin A20 and significant

211 re realized for the sustained release of two anti-inflammatory molecules, the natural polyphenol curc

212 ies including antioxidant, antimicrobial and anti-inflammatory ones.

213 ptor BLT2 is a receptor involved in pro- and anti-inflammatory pathways and can be activated by vario

214 ment for E-DII, suggesting the importance of anti-inflammatory pathways.

215 In the present study, a novel hybrid anti-inflammatory peptide that combines the active cente

216 could thus inspire the development of novel anti-inflammatory peptides that therapeutically target t

217 d amaranth on the release of antioxidant and anti-inflammatory peptides was evaluated.

218 D(2) and 11-dehydro-TBX(2) ) was balanced by anti-inflammatory PGE(2) .

219 GH reprograms inflammatory macrophages to an anti-inflammatory phenotype and improves resolution duri

220 n skew the patient's immune system toward an anti-inflammatory phenotype and therefore could provide

221 Macrophages of the anti-inflammatory phenotype express high levels of CD163

222 their polarization toward a more tolerogenic anti-inflammatory phenotype.

223 ng a number of the genes up-regulated in the anti-inflammatory phenotype.

224 y protecting against ER stress and promoting anti-inflammatory polarization.

225 One of the suspected mechanisms is the anti-inflammatory potential of GLP-1 receptor (Glp1r) ag

226 both P. pellucida and C. sinensis teas, the anti-inflammatory potential of P. pellucida was signific

227 Additionally, their cytotoxicity and anti-inflammatory potential were assessed in cellular mo

228 Sepsis initiates simultaneous pro- and anti-inflammatory processes, the pattern and intensity o

229 n and maintenance of Th1 and Th17/Th 22, and anti-inflammatory/profibrogenic cytokines.

230 veloped and proven to have antibacterial and anti-inflammatory properties for the treatment of period

231 hat Sirt1 knockdown or inhibition blunts the anti-inflammatory properties of LDN in vitro Using numer

232 ases, the question arises weather UDCA holds anti-inflammatory properties on periodontal health.

233 ecent member of the IL-1 superfamily and has anti-inflammatory properties similar to those of IL-37 b

234 duce hyperexcitability and neuron loss, have anti-inflammatory properties, and are well tolerated by

235 Importantly, CysC may have anti-inflammatory properties, and circulating CysC level

236 The mammalian homolog, PUF60, also displays anti-inflammatory properties, and its levels swiftly dec

237 bitors of gastric acid production, also have anti-inflammatory properties, protect mice from sepsis,

238 he management of periodontitis through their anti-inflammatory properties.

239 n due to their pleotropic lipid lowering and anti-inflammatory properties.

240 selenium pharmaceutical with antioxidant and anti-inflammatory properties.

241 tion in cardiovascular medicine owing to its anti-inflammatory properties.

242 al clades with additional benefits for their anti-inflammatory properties.

243 kinase inhibitor, has been shown to exert an anti-inflammatory property.

244 , which are known by their potent beneficial anti-inflammatory, proresolving, and tissue-modifying pr

245 A20 is an anti-inflammatory protein that is strongly linked to hum

246 review of the evidence supporting the use of anti-inflammatory reliever therapy in mild asthma and th

247 in patients with mild asthma have shown that anti-inflammatory reliever therapy with budesonide-formo

248 hat macrophages from inflamed gut acquire an anti-inflammatory/reparative profile.

249 previously demonstrated that the Salmonella anti-inflammatory response activator SarA (Stm2585, GogC

250 Derepression of SOCS3 enhances the anti-inflammatory response by inhibiting JAK/STAT-signal

251 clearance mechanisms and/or promotion of an anti-inflammatory response may slow or prevent disease p

252 response syndrome, followed by compensatory anti-inflammatory response syndrome (CARS).

253 rved among participants with the most robust anti-inflammatory response, an effect corroborated in fo

254 c forms of neutrophil death typically elicit anti-inflammatory responses and promote healing, pathway

255 t EpOMEs play a crucial role in facilitating anti-inflammatory responses in S. exigua.

256 Interestingly, animals with anti-inflammatory responses including high IL-10:IL-6 an

257 hrough a distinct cleavage site and promotes anti-inflammatory responses, prosurvival, and endothelia

258 Together, our results reveal a new anti-inflammatory role for JAK3 in innate immune cells a

259 pro-inflammatory role of neutrophils and the anti-inflammatory role of macrophages.

260 d for the p65 subunit to further support the anti-inflammatory role of UDCA.

261 that this balance between the dual pro- and anti-inflammatory roles of C5aR2 ultimately dictates dis

262 elial cell protein C receptor (EPCR) induces anti-inflammatory signaling and protects vascular barrie

263 that canagliflozin augments antioxidant and anti-inflammatory signaling involving AMP-activated prot

264 ow that canagliflozin stimulates antioxidant/anti-inflammatory signaling pathways involving AMP-activ

265 ctions by promoting multiple antioxidant and anti-inflammatory signaling.

266 astrocyte protein markers associated with an anti-inflammatory state, suggesting that the biological

267 ted macrophages (TAMs) can exist in pro- and anti-inflammatory states.

268 polarization to either a pro-inflammatory or anti-inflammatory status is controlled by activating inf

269 Anti-inflammatory steroids and bronchodilators are the g

270 olysaccharide (LPS) pro-inflammatory or IL-4 anti-inflammatory stimulation revealed the importance of

271 Anti-inflammatory TAMs (also referred to as M2-polarized

272 We found that anti-inflammatory TAMs promoted a metabolic state in bre

273 Anti-inflammatory TAMs secreted the cytokine TGF-beta th

274 pressant effects of standard or novel (e.g., anti-inflammatory) therapeutic approaches.

275 , the recent success of clinical trials with anti-inflammatory therapies and the accelerated atherosc

276 Current anti-inflammatory therapies are often insufficient in pr

277 Antiviral/anti-inflammatory therapies could attenuate bronchial in

278 19 patients and sepsis and describe proposed anti-inflammatory therapies for coronavirus disease 2019

279 mediators, the expectation is that targeted anti-inflammatory therapies may improve prognosis in pat

280 t failure, including antidiabetic therapies, anti-inflammatory therapies, and novel immunometabolic a

281 the latest evidence from clinical trials of anti-inflammatory therapies.

282 ich reinforces the use of an early intensive anti-inflammatory therapy to prevent neurodegeneration i

283 Anti-inflammatory therapy with canakinumab significantly

284 9 were diagnosed with HAM, with 10 receiving anti-inflammatory therapy.

285 Although the Canakinumab Anti-Inflammatory Thrombosis Outcomes Study (CANTOS) est

286 In the Canakinumab Anti-inflammatory Thrombosis Outcomes Study (CANTOS), in

287 to prevent autoimmune disease and promote an anti-inflammatory tissue environment.

288 ssion and that UV exposure induces a broadly anti-inflammatory transcriptional program dependent on R

289 NA methylation and represses antioxidant and anti-inflammatory transcriptional programs.

290 apping multilineage phenotypes with pro- and anti-inflammatory transcripts of T(H)1, T helper cell ty

291 CD200 is known as an anti-inflammatory transmembrane glycoprotein in the immu

292 C3 might prevent organ damage and provide an anti-inflammatory treatment for vascular and kidney dise

293 Responses to activation and anti-inflammatory treatment in mice may not always be pr

294 f murine cells to inflammatory activation or anti-inflammatory treatment is predictive of the respons

295 tors is essential for the development of new anti-inflammatory treatment strategies.

296 ctable presymptomatically and be amenable to anti-inflammatory treatment.

297 ally triggered release of embedded drugs for anti-inflammatory treatment.

298 Anti-inflammatory treatments vary, however, in both effe

299 tracts on the cytotoxicity and regulation of anti-inflammatory tristrataprolin (TTP) family gene expr

300 ll cell lung cancer (NSCLC), accumulation of anti-inflammatory tumor-associated macrophages (TAM) is