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1 the three-dimensional structure of the virus-antibody complex.
2 iew of antigen flexibility within an antigen:antibody complex.
3 ed the molecular heterogeneity of the hapten-antibody complex.
4 rease in the dissociation rate of the hapten-antibody complex.
5 the interaction and stability of the antigen-antibody complex.
6 the crystal structure of the alpha-hemolysin:antibody complex.
7 yclophosphamide (CyP), and IAC (IL-2/JES6-1) antibody complex.
8 y the formation of a surface aptamer-protein-antibody complex.
9 individual antigens, antibodies, and antigen/antibody complexes.
10  cytometry after stimulation with RSV or RSV-antibody complexes.
11 otected from inflammation induced by antigen-antibody complexes.
12 odies via differential catabolism of antigen-antibody complexes.
13 bodies and interaction interfaces of antigen-antibody complexes.
14  for predicting structural models of antigen-antibody complexes.
15 complement activity induced by other antigen-antibody complexes.
16 ough activation of effector cells by antigen-antibody complexes.
17 g produced the increased affinity of antigen-antibody complexes.
18 from the deformed states of gp120 in certain antibody complexes.
19 t GVHD by coadministering rapamycin and IL-2 antibody complexes.
20 re capable of using metals to bridge antigen:antibody complexes.
21 eneic donor by treatment with IL-2/anti-IL-2 antibody complexes.
22 namically characterize high-affinity antigen/antibody complexes.
23 erably increases the loss induced by antigen-antibody complexes (AACs) via the amplification effect o
24 d, to define the k(d) of this stable antigen/antibody complex accurately, the highest PSA concentrati
25 otting factor in such a way that the protein-antibody complex acquires a unique function.
26 nsplantation, the mechanism by which antigen-antibody complexes activate complement to induce lung da
27 alyzed an internalization process of antigen-antibody complexes after binding of RSV-specific antibod
28 ng Treg cell numbers by delivering IL-2:IL-2 antibody complexes after stroke improved white matter in
29  humans, the resulting formation of IL-1beta-antibody complexes allowed the detection of in vivo-prod
30 athy (MN) results from deposition of antigen-antibody complexes along the glomerular basement membran
31 nearly indistinguishable, whereas on antigen-antibody complexes, AlphaFold3 is significantly superior
32 ay of complement activated by, e.g., antigen-antibody complexes, also recognizes the C4 C345C domain
33 0 min of room temperature incubation for the antibody complex and after 18 min for the protein comple
34 count the stoichiometry of the mAbs/antidrug antibody complex and the bivalent properties of the two
35                            Use of bispecific antibody complexes and (99m)Tc-DTPA-succinyl-polylysine
36                                   Bispecific antibody complexes and (99m)Tc-labeled negatively charge
37 feres with thrombotic properties of beta2GPI/antibody complexes and does not affect normal thrombus f
38 asured by computational mutagenesis of spike-antibody complexes and mutation frequency calculated fro
39      These defects were due to viral antigen-antibody complexes and not the chronic infection per se,
40   Precipitation of the radiolabeled receptor-antibody complexes and scintillation counting enabled qu
41 L-63 in the crystal structure of the antigen-antibody complex, and 10 HyHEL-63 residues in contact wi
42                       HSP70 protein, antigen-antibody complexes, and complement were prevalent in IPF
43 cytokines bound to their receptors, allergen-antibody complexes, and innate immune receptors with the
44                                    The toxin-antibody complex anti-d(beta)h-saporin (DSAP) selectivel
45 tal scans or three-dimensional structures of antibody complexes are available.
46 esults show that neither B cells nor antigen-antibody complexes are essential for the maintenance of
47 ariety of situations in which stable antigen-antibody complexes are formed in the presence of nonreac
48                 Because internalized PAM/PAM-antibody complexes are returned to secretory granules, t
49 re of the Gla domain in the Factor IX-(1-47)-antibody complex at 2.2 A is similar to the structure of
50  determined the crystal structure of the RBD-antibody complex at 2.3-A resolution.
51 ay blockade, the organization of the antigen-antibody complexes at the cell surface, and opportunitie
52 s the first to provide evidence that antigen-antibody complexes bind specifically to apoptotic neutro
53 inity, allowing simple separation of antigen-antibody complex by thermal precipitation.
54                    Disruption of PF4-VWF-HIT antibody complexes by drugs that prevent or block VWF ol
55                     Thus, different cytokine-antibody complexes can be used to selectively boost or i
56  cells as soluble shed antigen or as antigen-antibody complexes, causing impairment in the activation
57 utic induction of basophilia by IL3/anti-IL3 antibody complexes, combined with transfer of CD8(+) T c
58 dies, deposition of anti-double-stranded DNA antibody complexes, complement activation, and immune co
59  one in this issue, demonstrate that antigen-antibody complexes containing RNAs activate B lymphocyte
60 er expanding Tregs in vivo with an IL-2/IL-2 antibody complex could be safe, well tolerated and effic
61 ngstrom crystal structure of a beta-tryptase/antibody complex coupled with biochemical studies reveal
62 mation and selective precipitation of cyclic antibody complexes created by binding to trivalent hapte
63                                  For antigen-antibody complexes, DARS is slightly better than a numbe
64 cryo-electron microscopy structures of spike-antibody complexes demonstrated targeting of conserved R
65 ion of the kinetics and stoichiometry of Env-antibody complexes demonstrated the applicability of our
66                                  The antigen-antibody complex demonstrates a high association constan
67      It describes the structures of four AAV-antibody complexes determined by cryo-electron microscop
68 and HBoV4 capsids using structures of capsid-antibody complexes determined using cryo-electron micros
69             In mice, low-dose IL-2-anti-IL-2 antibody complexes drove group 2 innate lymphoid cells (
70 y translate to a lack of toxicity of antigen-antibody complexes during the course of infections with
71 tudies of B41 in complex with a B41-specific antibody complex elucidate the molecular basis of this s
72                           Surprisingly, aSyn-antibody complexes enhanced rather than suppressed infla
73 -stain EM to model the PCSK9 antigen-J16 Fab antibody complex, followed by validation of the model by
74 influence on the affinity of the beta(2)-GPI antibody complex for the membrane vesicles.
75 formed a comparative analysis of all protein-antibody complexes for which structures have been experi
76                                          The antibody complexed form of sHLA was ineffective in the i
77 tion of a functional, full-length monoclonal antibody complex from transgenic Nicotiana tabacum roots
78 tation), associated with shedding of antigen-antibody complexes from endothelial cells.
79 dic cluster affects exit of internalized PAM-antibody complexes from late endosomes; internalized PAM
80  antibodies led to the release of the capsid/antibody complexes from the cell surface and their accum
81                                    Uptake of antibody-complexed GAD65 was Fc receptor (FcR)-mediated
82 ore this measurement can be made, the ligand-antibody complex generally has to be separated from bulk
83 ron microscopy, multiple structures of Spike-antibody complexes have distinct binding modes that not
84                                   Monoclonal antibody complexes have proven very useful in the study
85                      Two of these were virus-antibody complexes having contrasting transcriptional ca
86 e Ig crystallizable fragment (Fc) in antigen-antibody complexes held on FDCs decreases the activation
87 e ions covering the HA antigen versus the HA.antibody complex highlights regions with suppressed back
88 ed antigens as periodically arranged antigen-antibody complexes (ICs).
89 ng of the antibody structure into the virion/antibody complex identifies two conformations of the ant
90 hile the impacts of systemic IL-2: anti-IL-2 antibody complex (IL-2C) administration are well-defined
91 cell (Treg)-biased IL-2/anti-IL-2 monoclonal antibody complexes (IL-2c) can preferentially deliver IL
92 PS vaccines with CD122-biased IL-2/anti-IL-2 antibody complexes (IL-2cx) drives ~3-fold expansion of
93 sh fluorescence detection of the GFP antigen-antibody complex in a similar manner.
94 acer enzyme and used to detect the herbicide-antibody complex in an ELISA format.
95 orescence analyses revealed granular antigen-antibody complexes in a subepithelial location along the
96  understand the presence and duration of NS1 antibody complexes in clinical dengue infections.
97 EMS-based mass spectrometry by analysing IgM antibody complexes in real time.
98 ight into this process, we characterized PrP-antibody complexes in solution using a fast protein liqu
99 s studies demonstrated that specific antigen-antibody complexes in the sera of patients with LD could
100 nhibits prothrombotic properties of beta2GPI/antibody complexes in wild-type mice after acute infusio
101  binding (hormone-receptor and neuraminidase-antibody complexes), in one case the net effect is close
102  upon UV photoactivation of HA and of the HA.antibody complex indicates the elimination of some seque
103                  Immunization with chlamydia-antibody complexes induced elevated and protective Th1 r
104 n of Fc-receptor-bearing effector cells with antibody-complexed infected cells is important in reduci
105 ide insights into how nanopatterning antigen-antibody complexes influence the activation of the C1 co
106 ncer cells followed by tracking the receptor-antibody complex internalization by confocal microscopy.
107               One the one hand, this antigen-antibody complex internalization could result in an anti
108 activation in human serum induced by antigen-antibody complexes is a major hurdle for monitoring ther
109            Overall, DEPC labeling of antigen-antibody complexes is found to depend on both changes in
110         Platelet adhesion to the PF4-VWF-HIT antibody complexes is inhibited by antibodies that block
111 cination with PD1 blockade or IL-2/anti-IL-2 antibody complexes led to complete disease eradication a
112 e demonstrate that on recognition of antigen-antibody complexes, lining macrophages undergo significa
113         X-ray crystallography of an allergen-antibody complex localizes in detail amino acid residues
114 roscopy reveals that endocytosis of the PSMA-antibody complex occurs via clathrin-coated pits.
115 e current study we demonstrate that cytokine:antibody complexes of IL-2 and anti-IL-2 mAb reduce the
116 tection to directly count individual protein-antibody complexes of protein G or herpes simplex virus
117 tation by B cells and persistence of antigen-antibody complexes on follicular dendritic cells (FDC) h
118 and the capacity to form multivalent antigen-antibody complexes on target cells were key determinants
119 ary infection, there were indeed NS1 antigen-antibody complexes on the admission day during the febri
120 CL) with exposure to X-ray film, the antigen-antibody complexes on the blot are reacted with a chromo
121 a detects the pattern of fluorescent antigen:antibody complexes on the sensor surface.
122 n the mobile phase eluted the benzodiazepine-antibody complexes onto a C-18 restricted access media (
123 e formation of citrullinated protein antigen-antibody complexes or other forms of ICs.
124 surement of the concentration of the protein-antibody complexes over 3 orders of magnitude down to th
125 iated with limited proteolysis of an antigen-antibody complex particularly in the vicinity of the bin
126                     Accelerated clearance of antibody-complexed pegylated drugs by Kupffer cells may
127 n each platform with three different antigen/antibody complexes possessing nanomolar to picomolar equ
128                             Models of spacer-antibody complexes predicted that residues R568, L591, F
129                                      Antigen-antibody complexes provide useful models for analyzing t
130                                      Antigen-antibody complexes provide useful models for studying th
131                Comparison with structures of antibody complexes provides new insights on immune neutr
132 three-dimensional reconstruction of the RecA-antibody complex reveal that the lobe containing the epi
133        Finally, electron microscopy of TRPM1-antibody complexes revealed a large particle that can ac
134          Detailed molecular mapping of spike-antibody complexes revealed epitopes that were different
135  cocrystal structure of a TGFB2-anti-TGFB2/3 antibody complex reveals an allosteric isoform-selective
136 ultimers, peptide-protein complexes, antigen-antibody complexes, RNA, RNA multimers, and protein-nucl
137             Structural studies on the hapten-antibody complex show that the active site contains two
138 ope chemistries differed, all 16 gp120-CD4bs antibody complexes showed geometric similarity, with ant
139 on microscopic analysis of two neuraminidase-antibody complexes shows that the conserved neuraminidas
140  on extensive structural analyses of antigen-antibody complexes.Single-particle electron cryomicrosco
141               In contrast to the case of IAV-antibody complexes, SP-D-IAV complexes attached to and a
142 Bacteria samples, treated with a fluorescent antibody complex specific to Streptococcus pyogenes, wer
143 ine design efforts, as well as sets of spike-antibody complexes, spike sequence variability, and know
144 nation of genetic cell ablation and cytokine-antibody complex stimulation, we show that NK cell funct
145        Epitome consists of all known antigen/antibody complex structures, a detailed description of t
146 n includes new tables dedicated to TCR mimic antibody complex structures, TCR-CD3 complexes and annot
147 igenic interactions within the known antigen-antibody complex structures.
148 lead to the production of auto-antigen: auto-antibody complexes that are the hallmark of systemic lup
149                                       Unlike antibody complexes that are usually rapidly cleared, the
150 ed to Sepharose were used to isolate antigen-antibody complexes that contained few contaminating host
151 , we report three crystal structures of TcdB-antibody complexes that reveal how antibodies could spec
152 l structures of wild-type and cross-reactive antibodies complexed to BoNT/A1 at resolutions up to 2.6
153 e antigens that enhance access of the virion-antibody complex to FcgammaR-bearing cells.
154 P-cadherin turnover and targets the cadherin-antibody complex to the lysosome.
155 submicrogram quantities of goat anti-hamster antibody complexed to mAb N418 elicited goat antibody-sp
156 to couple receptors for antigens and antigen-antibody complexes to adaptive and innate immune respons
157 egy for in-depth characterization of antigen-antibody complexes to enable the identification of prote
158 l properties of poly-GA and to shift poly-GA/antibody complexes to more rapidly sedimenting ones.
159  atomic force microscopy imaging of receptor-antibody complexes to show that whereas the GluA1/GluA2
160            We show here that binding of 135S-antibody complexes to the Fc receptor (CDw32) increases
161 mpartment with fluorescent primary-secondary antibody complexes, total internal reflection fluorescen
162 ylococcal protein A-Sepharose indicated that antibody-complexed tracer was released from the liver 20
163 of these depend on the detection of a ligand-antibody complex using some kind of optical technique, e
164 nology for switchable assembly of functional antibody complexes using chemically induced dimerization
165 ian, 558 IU/ml; range, 42 to 135,000 IU/ml), antibody-complexed virus, free specific IgG, and potenti
166 ype GFP and the formation of the GFP antigen-antibody complex was monitored.
167                  We also found that an Abeta/antibody complex was present in both the plasma and the
168 y (EIS) in which the formation of an antigen-antibody complex was quantified as a function of charge
169                     The stability of Bet v 1-antibody complexes was compared by ELISA and by surface
170  X-ray crystal structure of a gp120-CD4-CD4i antibody complex, we introduced changes into gp120 that
171 iakinumab and by leveraging additional IL-23:antibody complexes, we propose a mechanistic paradigm fo
172 cally with the capture arm of the bispecific antibody complex were used to demonstrate the feasibilit
173 tion of IgG sufficient to inhibit HA, virion-antibody complexes were monodispersed and not aggregated
174   Similarly, cells treated with Nef/anti-Nef antibody complexes were protected from Nef-induced apopt
175 d antibodies were capable of forming antigen-antibody complexes which could be isolated by gel filtra
176  these subpopulations of pAbs formed antigen-antibody complexes which could be isolated by gel-filtra
177           Further, incubation of the antigen-antibody complex with 11-cis-retinal failed to regenerat
178 of a covalently linked pentameric monoclonal antibody complex with a mass of ~800 kDa reveals several
179 es: crystalline GB1 and the precipitated GB1-antibody complex with a molecular weight of more than 30
180 e enhancement of 3.5 x 10(4) in favor of the antibody complex with an effective molarity of 76.7 M, r
181  are imported into the nucleus as an antigen-antibody complex with coilin.
182      Crystallization of one IgM+ MBC-derived antibody complexed with antigen defined a linear epitope
183 his study, the effect of anti-E. chaffeensis antibody complexed with E. chaffeensis on the expression
184                          Anti-E. chaffeensis antibody complexed with E. chaffeensis significantly enh
185 line heavy-chain/mature light-chain chimeric antibody complexed with HIV-1 gp120.
186                         The C-pro alpha 2(I) antibody complexed with more cell proteins.
187  with hen egg white lysozyme (HEL), the D1.3 antibody complexed with the anti-lysozyme antibody E5.2,
188 tructure of a Fab fragment derived from this antibody complexed with the factor VIII C2 domain was de
189 he size distribution and affinity of protein-antibody complexes with picomolar KD values.
190 ic studies coupling the structure of antigen-antibody complexes with their antiviral function has beg
191 51, a tyrosine-sulfated coreceptor-mimicking antibody, complexed with a CD4-bound open HIV-1 native-l

 
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