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1 fetal loss in a mouse pregnancy model due to anticardiolipin antibodies.
2 ding; and those who had detectable levels of anticardiolipin antibodies.
4 e (APAS) is characterized by the presence of anticardiolipin antibodies (ACA) in association with thr
6 trongly that lupus anticoagulants (LACs) and anticardiolipin antibodies (ACAs) are antibodies to prot
7 lls treated with a range of human monoclonal anticardiolipin antibodies (aCL) (as anti-beta2-glycopro
9 anti-beta2-glycoprotein I (anti-beta2GPI) or anticardiolipin antibodies (aCL) may be implicated in th
15 rotein AI to OxCL-coated wells increased the anticardiolipin antibody (aCL) binding from APS sera tha
16 uble-stranded DNA (anti-dsDNA) antibody, and anticardiolipin antibody (aCL), as well as proteinuria a
17 La, anti-RNP, lupus anticoagulant (LAC), and anticardiolipin antibody [aCL]) were selected for cluste
18 d lupus anticoagulant (LA) or high titers of anticardiolipin antibodies (ACLAs), either immunoglobuli
20 otein(a), homocysteine, lupus anticoagulant, anticardiolipin antibodies and genotyping of factor V Le
21 f CL is needed to generate epitopes for many anticardiolipin antibodies and that some of these epitop
22 atients with stroke associated with elevated anticardiolipin antibodies, and we discuss their managem
23 ter IgG or IgM anticardiolipin antibody, IgA anticardiolipin antibody, and anti-beta2-glycoprotein I
27 iciency, activated protein C resistance, and anticardiolipin antibodies) at predictably greater risk
28 Ls were measured longitudinally: 3 in serum (anticardiolipin antibodies, beta2-glycoprotein I, and an
30 nd 1 patient each with protein C deficiency, anticardiolipin antibodies, factor VII mutation, factor
32 uld be proposed in men, aged >40 years, with anticardiolipin antibodies >60 GPLU when TTE is inconclu
37 strated that human monoclonal and polyclonal anticardiolipin antibodies have thrombogenic properties
38 aris, thrombocytopenia, low-titer IgG or IgM anticardiolipin antibody, IgA anticardiolipin antibody,
40 ted the possible association between HAT and anticardiolipin antibodies in adult patients who underwe
43 luded if they had moderate or high levels of anticardiolipin antibodies, lupus anticoagulant, or a hi
44 of Raynaud's phenomenon, as well as elevated anticardiolipin antibodies, lupus anticoagulant, or a hi
45 e not generalizable to women with high-titer anticardiolipin antibodies, lupus anticoagulant, or prev
47 ity for lupus anticoagulant (P < 0.0001) and anticardiolipin antibodies of the IgG (P = 0.035) or the
48 and the presence of the lupus anticoagulant, anticardiolipin antibodies, or anti-beta(2)-glycoprotein
49 in lymphocyte count or serum immunoglobulin, anticardiolipin antibody, or rubella IgG antibody concen
50 of 35 (63%) liver recipients had a positive anticardiolipin antibody test (either IgG or IgM titer >
51 include measurements of lupus anticoagulant, anticardiolipin antibodies, thyroid function, and a tran
52 either the lupus anticoagulant titer or the anticardiolipin antibody titer on two occasions, separat
54 ng anticoagulant therapy in patients with an anticardiolipin antibody was 1.53 (95% CI, 0.76-3.11), a
55 coagulable state associated with circulating anticardiolipin antibodies, which prompted discontinuati