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1 the most cold sensitive was lamotrigine, an anticonvulsant drug.
2 sed as local anesthetic, antiarrhythmic, and anticonvulsant drugs.
3 in producing the mood-stabilizing effects of anticonvulsant drugs.
4 eses for the antimanic action of lithium and anticonvulsant drugs.
5 ful reduction of CNS excitability exerted by anticonvulsant drugs.
6 We have, therefore, studied the effects of anticonvulsant drugs acting via use-dependent block of v
9 to anticonvulsant drugs, those unexposed to anticonvulsant drugs but with a maternal history of seiz
12 with epilepsy is associated with the use of anticonvulsant drugs during pregnancy, rather than with
13 others had a history of epilepsy but took no anticonvulsant drugs during the pregnancy did not have a
16 scussed in light of the effectiveness of the anticonvulsant drug gabapentin, which is a specific inhi
22 ld more potent than the clinically effective anticonvulsant drug lamotrigine (IC50=27.7 microM), a pr
23 that are associated with the binding of the anticonvulsant drug Lamotrigine and batrachotoxin are al
26 lth except for seizure disorder, with stable anticonvulsant drug levels in the upper half of the ther
27 efractory convulsive status epilepticus, the anticonvulsant drugs levetiracetam, fosphenytoin, and va
28 o examine the effects of retigabine, a novel anticonvulsant drug, on the electroresponsive properties
29 ve agents, estrogens, progestins, sedatives, anticonvulsant drugs, or drugs that may form N-nitroso d
31 efficacy of topiramate, a structurally novel anticonvulsant drug recently shown to attenuate AMPA/kai
32 Bath application of dynamin inhibitors or anticonvulsant drugs rescued TBI-induced tauopathy and c
34 izures and can be refractory to conventional anticonvulsant drugs, suggesting an age-specific form of
35 thy was higher in 223 infants exposed to one anticonvulsant drug than in 508 control infants (20.6 pe
36 higher in 93 infants exposed to two or more anticonvulsant drugs than in the controls (28.0 percent
39 administration of anxiolytic, sedative, and anticonvulsant drugs that act through the GABAA receptor
40 on to die of their disease respond poorly to anticonvulsant drug therapy, suggesting a need for new t
41 intake, less exposure to ultraviolet light, anticonvulsant-drug therapy, renal dialysis, nephrotic s
42 fy three groups of infants: those exposed to anticonvulsant drugs, those unexposed to anticonvulsant
43 nt of such co-morbidity; the contribution of anticonvulsant drugs towards the development of psychiat
44 included continued attacks despite adequate anticonvulsant drug treatment (n = 36) or uncertainty ab
48 fy the most sensitive therapeutic target for anticonvulsant drugs, which could be critical to pharmac
49 id is a short branched fatty acid used as an anticonvulsant drug whose therapeutic action has been pr
50 history of seizures, and those unexposed to anticonvulsant drugs with no maternal history of seizure