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1 g no medications; as well as between the two anticonvulsants.
2 tients had seizures, which were treated with anticonvulsants.
3 40 (5.6%) of 716 patients received anticonvulsants.
4 (p=0.26) was found among patients receiving anticonvulsants.
5 ng effects on block by local anesthetics and anticonvulsants.
6 ncephalopathies that are resistant to modern anticonvulsants.
7 -benzyl-2-acetamidoacetamides (2) are potent anticonvulsants.
8 tics, class I antiarrhythmic drugs, and some anticonvulsants.
9 sts for antipsychotics, antidepressants, and anticonvulsants.
10 lopmental quotient (DQ) > 30 did not require anticonvulsants.
11 ccupied by traditional local anesthetics and anticonvulsants.
12 nce seizure generation and responsiveness to anticonvulsants.
13 acting drugs, including antidepressants and anticonvulsants.
14 t spontaneous convulsions (>1 per hour) with anticonvulsants.
15 g-sensitive (BS) mutants as a tool to screen anticonvulsants.
16 by hypoxia can be refractory to conventional anticonvulsants.
17 logical conditions and is the target of some anticonvulsants.
18 Its pharmacology seems unaffected by anticonvulsants.
20 %), CCB (14.8% versus 14.4% versus 8.0%) and anticonvulsants (13.4% versus 7.7% versus 6.4%) with OR
22 h other opioid-involved overdose deaths (eg, anticonvulsants: 18.6% [95% CI, 17.0%-20.3%] vs 5.4% [95
24 to the hypothesis that lithium and antimanic anticonvulsants act by targeting parts of the "arachidon
26 i); and Bn, Et (7j)] are also very effective anticonvulsants against seizures induced by maximal elec
27 psychotropic classes (e.g., antidepressants, anticonvulsants), although they may be safer options.
28 ues in patients treated with enzyme-inducing anticonvulsants, although this did not reach statistical
33 ly important compounds, including diuretics, anticonvulsants and antidepressants, many of which have
35 ation suggest that concurrent treatment with anticonvulsants and dexamethasone enhances drug clearanc
36 er combinations appear to be the mixtures of anticonvulsants and lithium, particularly valproate plus
38 rmined that the recommendations from the CNS anticonvulsants and steroids guidelines, published Janua
40 on-beta-lactam antimicrobials, 9 (6.0%) were anticonvulsants, and 5 (3.3%) were calcium channel block
42 es occur frequently, are often refractory to anticonvulsants, and are associated with considerable mo
44 low-potency phenothiazines, lithium, certain anticonvulsants, and benzodiazepines may increase the re
45 inistration of uterotonics, antibiotics, and anticonvulsants, and clean birth environments) would acc
46 mptomatic treatment with corticosteroids and anticonvulsants, and definitive therapy in the form of w
47 asses of clinical agents (e.g., anxiolytics, anticonvulsants, and general anesthetics) that act as po
51 etrovirals, antidepressants, antipsychotics, anticonvulsants, and immunosuppressants for transplant p
52 neurological symptoms, with gabapentin-type anticonvulsants, and is among the first in nonepileptic
55 r-bipolar subtype (N = 15) for whom lithium, anticonvulsants, and neuroleptics had been ineffective,
57 sychotics, benzodiazepines, antidepressants, anticonvulsants, and opioids followed the onset of the C
58 reatments include tricyclic antidepressants, anticonvulsants, and opioids, depending on the severity
59 Their therapeutic potential as anxiolytics, anticonvulsants, and sedative/hypnotics is limited by ra
62 n prescriptions (analgesics or antipyretics, anticonvulsants, antidepressants, and nonsteroidal anti-
63 utic drugs and supportive-care drugs-such as anticonvulsants, antiemetics, uric-acid-lowering compoun
64 esthetics, antiarrhythmics, antidepressants, anticonvulsants, antihistamines, antihypertensives, anti
65 17-3.81; P for trend < .001), whereas use of anticonvulsants, antipsychotics, or antidepressants was
66 ulate GABAA receptors have potential uses as anticonvulsants, anxiolytics, and sedative-hypnotic agen
72 ochloride (B = 0.033; SE = 0.010; P < .001), anticonvulsants (B = 0.014; SE = 0.006; P = .03), and op
73 s to specific treatments, including sedative anticonvulsants (barbiturates and benzodiazepines) and E
75 de: antidepressants, anti-adrenergic agents, anticonvulsants, benzodiazepines, atypical antipsychotic
77 dditional commonly used Na(+) channel-acting anticonvulsants, both in control and epileptic animals.
78 se findings advance the development of novel anticonvulsants by validating an influence of alkyl chai
80 ies and other risk factors, among individual anticonvulsants compared with topiramate and secondarily
82 .80-3.42; P for trend < .001), as was use of anticonvulsants (definite CKD, 1-2 prescriptions: HR = 1
83 unds represent novel leads in the search for anticonvulsants devoid of sedative, ataxic, and amnestic
84 ic and clinical seizures with currently used anticonvulsants did not significantly reduce the rate of
85 a is provided by modern antipsychotics, some anticonvulsants (divalproex and carbamazepine), and lith
86 rograms, placebo, opioids, psychostimulants, anticonvulsants, dopamine agonists, antidepressants, ant
87 zyl-3-ethyl lactam 7j are the most effective anticonvulsants (ED50 = 46 and 42 mg/kg, respectively) a
88 s included antiinfectives (eg, fluconazole), anticonvulsants (eg, valproate), antihypertensives (eg,
89 d in patients receiving p450 enzyme-inducing anticonvulsants (EIACs) by 73%, 47%, and 50%, respective
90 aging or EEG), prophylactic antipyretics and anticonvulsants far outweigh their potential benefits.
91 sia and eclampsia; and short-term parenteral anticonvulsants for seizures associated with encephalopa
94 ncy O-methylated analogue 18 are both potent anticonvulsants in a mouse maximal electroshock-induced
95 ll have surprisingly high in vivo potency as anticonvulsants in a mouse maximal electroshock-induced
98 ge underlying precipitant causes, administer anticonvulsants in rapid succession until seizures have
99 ation, and for prophylactic antipyretics and anticonvulsants, in the majority of children with simple
101 onatal seizures do not respond to first-line anticonvulsants, including phenobarbital, which enhances
102 the most teratogenic of commonly prescribed anticonvulsants, increasing the risk in humans of major
103 ds, acetaminophen, anti-inflammatory agents, anticonvulsants, ketamine, clonidine, mexiletine, antide
105 re antidepressants, atypical antipsychotics, anticonvulsants, lithium, and other medications used in
106 pregnancy and that increasing doses of these anticonvulsants may be needed throughout the course of p
110 sed to assess the prognostic significance of anticonvulsants on event-free survival and risk of haema
111 sychotics, benzodiazepines, antidepressants, anticonvulsants, opioids, antibiotics, angiotensin recep
112 ds for chronic noncancer pain, compared with anticonvulsants or cyclic antidepressants, was associate
113 for long-acting opioids or either analgesic anticonvulsants or low-dose cyclic antidepressants (cont
114 ity under anesthesia, but clinical trials of anticonvulsants or neural system suppressors have, so fa
115 ) or were nonambulatory and either receiving anticonvulsants or not; all had developmental delays.
117 psychotics, combinations of antidepressants, anticonvulsants, or "other") for >/=60 consecutive days
118 , which was similar to that observed for the anticonvulsants phenytoin (slowly binds to the fast-inac
120 eceived treatment for 30 days or longer with anticonvulsants (phenytoin, phenobarbital, carbamazepine
123 ng at very high rates, suggesting that these anticonvulsants should cause impaired GABAergic inhibiti
124 TCAs (SMD, -0.78 [CrI, -1.24 to -0.33]), and anticonvulsants (SMD, -0.67 [CrI, -0.97 to -0.37]) were
125 epinephrine reuptake inhibitors (SNRIs) than anticonvulsants (standardized mean difference [SMD], -0.
126 ng-term relapse prevention; the evidence for anticonvulsants such as divalproex and lamotrigine is le
127 lectroencephalographic response to GABAergic anticonvulsants such as phenobarbital and benzodiazepine
129 includes mood stabilizers, such as lithium, anticonvulsants, such as valproate and lamotrigine, and
131 CBZ) and oxcarbazepine (OXC) are widely used anticonvulsants that are extensively metabolized in the
132 activity in the cortex is not suppressed by anticonvulsants that block the transmission of seizure a
133 tribute to resistance of seizure activity to anticonvulsants that increase GABAergic function, and ma
137 des because of the increased availability of anticonvulsants, the ketogenic diet has re-emerged as a
138 yl-N'-(4-methoxynaphth-1-yl)guanidine (3) as anticonvulsants through blockade of sodium channels.
142 to treatment with tricyclic antidepressants, anticonvulsants (topiramate), coenzyme Q-10, and L-carni
143 differences in sex distribution or number of anticonvulsants used between patients with bruising/blee
144 , attempted suicides, and violent deaths for anticonvulsants used in at least 100 treatment episodes
147 reased rate of end-stage CKD, whereas use of anticonvulsants was (1-2 prescriptions, HR = 0 [95% CI,
149 rtication at age 8.5 years and withdrawal of anticonvulsants when he was more than 9 years old, Alex
150 nonketotic hyperglycinemia required multiple anticonvulsants, whereas patients with developmental quo
151 ribute an important component to binding for anticonvulsants, which compensates energetically for the
152 ical conditions, such as antidepressants and anticonvulsants, which were found empirically to be effe
153 compound was directed to identifying potent anticonvulsants with a long duration of action and a fav
154 tassium channels, we have docked these three anticonvulsants with residues identified by mutagenesis
155 eatment step and a higher cumulative dose of anticonvulsants within the first period of treatment wer
156 mnolence and dizziness with TCAs, SNRIs, and anticonvulsants; xerostomia with TCAs; and peripheral ed
157 have a long history of use as folk medicine anticonvulsants, yet the underlying mechanisms often rem