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1 sis due to troglitazone (a thiazolidinedione antidiabetic agent).
2 y mass index, use of insulin, and other oral antidiabetic agents).
3 ntroquinonol indicate that it is a potential antidiabetic agent.
4 ium, which is a proposed insulin mimetic and antidiabetic agent.
5 tential role for EGCG, or derivatives, as an antidiabetic agent.
6 nstrate the importance of IF1 as a potential antidiabetic agent.
7 which supports the role of camel milk as an antidiabetic agent.
8 sing candidate for further development as an antidiabetic agent.
9 hypoglycemia, and can be combined with other antidiabetic agents.
10 eed the in vitro activity of known synthetic antidiabetic agents.
11 ent, whereas 7 (28%) of 25 controls required antidiabetic agents.
12 receptor pharmacology that may lead to novel antidiabetic agents.
13 long-acting insulin analogues and noninsulin antidiabetic agents.
14 ery and design of GK activators as potential antidiabetic agents.
15 ctivators represent a potential new class of antidiabetic agents.
16 t passive GR antagonists may have utility as antidiabetic agents.
17 es-a group of potent PPAR gamma agonists and antidiabetic agents.
18 ailability of several unique classes of oral antidiabetic agents.
19 thiazolidinedione (TZD) and certain non-TZD antidiabetic agents.
20 lated compounds may represent a new class of antidiabetic agents.
21 R PAMs may become clinically useful as novel antidiabetic agents.
22 Such chemical entities may prove useful as antidiabetic agents.
23 th other forms of therapy, for example, oral antidiabetic agents.
24 nimal models and have promise as potent oral antidiabetic agents.
25 iet and exercise therapy and the use of oral antidiabetic agents.
26 using Click chemistry to identify potential antidiabetic agents.
27 metformin while remaining sensitive to other antidiabetic agents.
28 in different food formulations as functional antidiabetic agents.
29 The main outcome was the receipt of antidiabetic agents.
32 ssant, antihyperlipidemic, antiulcerant, and antidiabetic agents--also demonstrated significant price
33 e and significant increase in the receipt of antidiabetic agents among all people with diabetes durin
34 ile basal insulin remains the most effective antidiabetic agent and substantially reduces the risk of
35 lipid-lowering drugs, diabetes and/or use of antidiabetic agents, and hypertension and/or use of anti
38 azone and pioglitazone), a new class of oral antidiabetic agents, are "insulin sensitizers" and exert
39 binders, and thiazolidinediones, a class of antidiabetic agents, are particularly susceptible to thi
40 e, I discuss a rational basis for the use of antidiabetic agents as novel and potentially effective t
42 15d-PGJ2), or members of a new class of oral antidiabetic agents, e.g. BRL49653 and ciglitizone, has
43 t group) or short-acting insulin and/or oral antidiabetic agents for blood glucose >/=180-250 mg/dl (
44 idase-4 (DPP-4) inhibitors are commonly used antidiabetic agents for patients with advanced-stage chr
45 enosine, glucose-insulin-potassium, statins, antidiabetic agents, FX06, iron chelation, and ranolazin
47 r-gamma (PPARgamma) agonists, a new class of antidiabetic agents, have been shown to possess antiinfl
48 peptidase-4 inhibitors (DPP-4is) as a third antidiabetic agent in patients with type 2 diabetes mell
52 mediate-acting insulin analogues) with other antidiabetic agents is urgently required to guide approp
54 tion) in the presence of hyperglycemia, this antidiabetic agent may prove to have significant salutar
55 liver dysfunction is no longer valid; novel antidiabetic agents may provide adequate glucose control
56 some of the beneficial effects of PPARgamma antidiabetic agents may result, at least in part, from t
62 ning the effects of intrauterine exposure to antidiabetic agents on longitudinal growth, and the impo
65 ency in the thiazolidinedione (TZD) class of antidiabetic agents provided a dramatic increase in the
68 T2D patients are routinely treated with oral antidiabetic agents such as sulfonylureas or dipeptidyl
70 lar target of the thiazolidinedione class of antidiabetic agents suggested a key role for PPAR-gamma
77 ones (TZDs) such as BRL 49653 are a class of antidiabetic agents that are agonists for the peroxisome
78 r 2 (SGLT-2) inhibitors are a novel class of antidiabetic agents that have been linked to an increase
79 ter 2 inhibitors (SGLT2i) were first used as antidiabetic agents that lower the blood glucose levels
81 t, as well as members of a new class of oral antidiabetic agents, the thiazolidinediones, and a varie
82 s devoid of the side effects of the marketed antidiabetic agents thiazolidinediones and the dual agon
84 Thus, PPARgamma activation in VSMCs, via the antidiabetic agent troglitazone or naturally occurring l
86 r agonists to individuals treated with other antidiabetic agents using a Cox regression with inverse
87 and inhibition assays for canagliflozin, an antidiabetic agent verified its effective binding to bot
88 7, troglitazone, the first thiazolidinedione antidiabetic agent, was found to cause life-threatening
92 ptidyl peptidase-4 inhibitor sitagliptin, an antidiabetic agent, which lowers blood glucose levels, a
94 nsulin sensitizers and incretin mimetics are antidiabetic agents with vastly different mechanisms of