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1 hereas others (raloxifene and GW7604) remain antiestrogenic.
2 :ER complex from estrogen-like to completely antiestrogenic.
3 enase 2 and are therefore antiandrogenic and antiestrogenic.
4 that Pak1 overexpression interfered with the antiestrogenic action of tamoxifen upon the ER transacti
5 were lost in these stable transfectants, but antiestrogenic action was retained.
6 eceptor (ER), all of these compounds exhibit antiestrogenic action when the side chain length is n >
7 taneously preventing mammary carcinogenesis (antiestrogenic action).
8 targets for receptor-mediated estrogenic and antiestrogenic action.
9 posed that tissue-specific estrogenic and/or antiestrogenic actions of certain xenoestrogens may be a
10 apies to exert tissue-specific estrogenic or antiestrogenic activities, this knowledge will be crucia
11  activity in addition to its well-documented antiestrogenic activity and raises the possibility that
12                               MCDF displayed antiestrogenic activity at higher concentrations in MCF-
13 addition, CPC showed concentration-dependent antiestrogenic activity half maximal effective concentra
14 sterol and bone metabolism while maintaining antiestrogenic activity in the uterus.
15 have investigated at the molecular level the antiestrogenic activity of 2,3,7,8-tetrachlorodibenzo-p-
16                                          The antiestrogenic activity of CPC, BAK, rotenone, and tricl
17 ential AhR-ER cross-talk is evidenced by the antiestrogenic activity of MCDF and the degradative effe
18 brief, the characterization of structure and antiestrogenic activity of MTA1s peptide highlight its t
19 with aspartate 351 makes this the key to the antiestrogenic activity of raloxifene.
20 ancer is limited by the mixed estrogenic and antiestrogenic activity of selective estrogen receptor m
21 ete agonist activity on the one hand or pure antiestrogenic activity on the other.
22 In all three cases, there was overlap of the antiestrogenic activity with the mitochondrial inhibitor
23 oxifen is a tamoxifen metabolite with potent antiestrogenic activity.
24 raloxifene in breast cancer depends on their antiestrogenic activity.
25 iverse biological effects including striking antiestrogenic activity.
26        Our findings indicate that TBC, as an antiestrogenic agent, may contribute to the pathogenesis
27                         Soy isoflavones have antiestrogenic and anticancer properties but also posses
28 nes--a class of phytoestrogens that has both antiestrogenic and anticarcinogenic properties.
29 stewater induced antiandrogenic, estrogenic, antiestrogenic, and retinoid-like activity.
30 de and of similar action as BPA (estrogenic, antiestrogenic, androgenic, and antiandrogenic) in vitro
31 r BPA and 11 BPA analogs exhibit estrogenic, antiestrogenic, androgenic, or antiandrogenic effects by
32 investigate the activities of estrogenic and antiestrogenic compounds in yeast Saccharomyces cerevisi
33 ary target of air pollution, the presence of antiestrogenic compounds like TBC in atmospheric particu
34 macological behaviour of many estrogenic and antiestrogenic compounds, including the naturally occurr
35 to investigate the effects of estrogenic and antiestrogenic compounds.
36 er that is nonresponsive to estrogen and the antiestrogenic drug tamoxifen.
37 otypes with increased resistance to standard antiestrogenic drugs.
38 n and thus may contribute toward the loss of antiestrogenic effect of tamoxifen.
39  to understand the mechanisms underlying the antiestrogenic effect of this prototype SERM, we perform
40                                          The antiestrogenic effect of transfected PR also provides a
41                                          The antiestrogenic effect was also observed at the level of
42  decreases in ER are not responsible for the antiestrogenic effect.
43     This finding suggests that TCDD may have antiestrogenic effects in the uterine myometrium, in con
44                                              Antiestrogenic effects of cerulenin and C75 were observe
45 ty rates in Asian countries and the putative antiestrogenic effects of isoflavones have fueled specul
46 research on the mitochondrial inhibitory and antiestrogenic effects of QUATS, including CPC and BAK.
47   However, tumors that are refractory to the antiestrogenic effects of selective ER modulators often
48             The regulation of estrogenic and antiestrogenic effects of selective estrogen receptor mo
49 esults provide a molecular mechanism for the antiestrogenic effects of TCDD and demonstrate the prese
50                                              Antiestrogenic effects of the compounds were measured by
51    In mechanistic studies, we found that the antiestrogenic effects of the MTA1s peptide were due to
52                                          The antiestrogenic effects of transfected PR are associated
53 lective estrogen receptor modulator that has antiestrogenic effects on breast and endometrial tissue
54 n experiments conducted with Ishikawa cells, antiestrogenic effects were confirmed by dose-dependent
55                     Similar mixed estrogenic/antiestrogenic effects were noted with S30 cells, wherea
56 ed thus far in fish have known estrogenic or antiestrogenic effects, and as such cause the inappropri
57   Proposed mechanisms include estrogenic and antiestrogenic effects, induction of cancer cell differe
58                                   As well as antiestrogenic effects, there were proestrogenic actions
59 ain flavonoids exhibit antiproliferative and antiestrogenic effects.
60 or, also showed mitochondrial inhibitory and antiestrogenic effects.
61 ds and neutralizes the Asp-351 to produce an antiestrogenic ERalpha complex.
62 -analogues of estradiol have tissue specific antiestrogenic/estrogenic actions, characteristics of se
63 to genistein treatments, suggesting that the antiestrogenic function of genistein is unrelated to the
64 of estrogen can be blocked completely by the antiestrogenic ICI 182,780.
65 uired for this activity as TCDD is no longer antiestrogenic in a mutant cell line that is deficient i
66                                       Though antiestrogenic in the breast, some antiestrogens have es
67 aken together, these results suggest a novel antiestrogenic mechanism for specific polyamine analogue
68 droxy-N-desmethyltamoxifen), the most potent antiestrogenic metabolite, are reduced in women whose CY
69                 A number of fractions showed antiestrogenic or antiandrogenicity potency, with the ne
70 s (PCBs) have been shown to have estrogenic, antiestrogenic, or antiandrogenic properties; as a resul
71                         We asked whether the antiestrogenic phytochemical indole-3-carbinol (I3C), fo
72 estrant (ICI182,780) display a more complete antiestrogenic profile.
73 rast, a mutation at AA 400 does not, and the antiestrogenic properties of raloxifene are retained.
74                              In light of the antiestrogenic properties of TBC, we administrated estra
75 -Tetrachlorodibenzo-p-dioxin (TCDD) exhibits antiestrogenic properties, including the inhibition of e
76 as been reported to have weak estrogenic and antiestrogenic properties, to be an antioxidant, to inhi
77 tably expressing either SULT1A1*1 or *2, the antiestrogenic response to OHT was found to be allele-sp
78 nsiveness to estradiol (E2) and also retains antiestrogenic responsiveness to both raloxifene and ICI
79 l AI/SERM, while in breast cancer tissue the antiestrogenic SERM activity of a dual AI/SERM could act
80 hydroxyl on the phenyl ring that contains an antiestrogenic side chain in endoxifen.
81 ars to be critical for interactions with the antiestrogenic side chain of antiestrogens.
82 utions on the TPE phenyl ring containing the antiestrogenic side chain of endoxifen or a free hydroxy
83  No substitution at the site occupied by the antiestrogenic side chain of endoxifen results in early
84 eir biology depends on an interaction of the antiestrogenic side chain with amino acid 351.
85 ne (TPE) derivative of tamoxifen without the antiestrogenic side chain.
86 -targeting compounds restored sensitivity to antiestrogenic therapy.
87 pendent growth often leads to the failure of antiestrogenic tumor therapies.
88 nsteroidal anti-inflammatory) and tamoxifen (antiestrogenic widely used in breast cancer therapy and