戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ucidate the interaction and stability of the antigen-antibody complex.
2 a rare view of antigen flexibility within an antigen:antibody complex.
3 nt is protected from inflammation induced by antigen-antibody complexes.
4 ic antibodies via differential catabolism of antigen-antibody complexes.
5 nity through activation of effector cells by antigen-antibody complexes.
6 tic antibodies and interaction interfaces of antigen-antibody complexes.
7 employed for predicting structural models of antigen-antibody complexes.
8 valuate complement activity induced by other antigen-antibody complexes.
9 sslinking produced the increased affinity of antigen-antibody complexes.
10 thermodynamically characterize high-affinity antigen/antibody complexes.
11 tion of individual antigens, antibodies, and antigen/antibody complexes.
12 bodies are capable of using metals to bridge antigen:antibody complexes.
13 h considerably increases the loss induced by antigen-antibody complexes (AACs) via the amplification
14    Second, to define the k(d) of this stable antigen/antibody complex accurately, the highest PSA con
15 lung transplantation, the mechanism by which antigen-antibody complexes activate complement to induce
16 e, we analyzed an internalization process of antigen-antibody complexes after binding of RSV-specific
17  nephropathy (MN) results from deposition of antigen-antibody complexes along the glomerular basement
18 ces are nearly indistinguishable, whereas on antigen-antibody complexes, AlphaFold3 is significantly
19 al pathway of complement activated by, e.g., antigen-antibody complexes, also recognizes the C4 C345C
20              These defects were due to viral antigen-antibody complexes and not the chronic infection
21 ith HyHEL-63 in the crystal structure of the antigen-antibody complex, and 10 HyHEL-63 residues in co
22                               HSP70 protein, antigen-antibody complexes, and complement were prevalen
23  these results show that neither B cells nor antigen-antibody complexes are essential for the mainten
24 e to a variety of situations in which stable antigen-antibody complexes are formed in the presence of
25 of pathway blockade, the organization of the antigen-antibody complexes at the cell surface, and oppo
26 report is the first to provide evidence that antigen-antibody complexes bind specifically to apoptoti
27 high affinity, allowing simple separation of antigen-antibody complex by thermal precipitation.
28 tor on T cells as soluble shed antigen or as antigen-antibody complexes, causing impairment in the ac
29  (1) and one in this issue, demonstrate that antigen-antibody complexes containing RNAs activate B ly
30                                          For antigen-antibody complexes, DARS is slightly better than
31                                          The antigen-antibody complex demonstrates a high association
32  mice may translate to a lack of toxicity of antigen-antibody complexes during the course of infectio
33 es (adaptation), associated with shedding of antigen-antibody complexes from endothelial cells.
34 B) by the Ig crystallizable fragment (Fc) in antigen-antibody complexes held on FDCs decreases the ac
35 ltimerized antigens as periodically arranged antigen-antibody complexes (ICs).
36 t diminish fluorescence detection of the GFP antigen-antibody complex in a similar manner.
37 mmunofluorescence analyses revealed granular antigen-antibody complexes in a subepithelial location a
38  Previous studies demonstrated that specific antigen-antibody complexes in the sera of patients with
39 ata provide insights into how nanopatterning antigen-antibody complexes influence the activation of t
40                       One the one hand, this antigen-antibody complex internalization could result in
41 plement activation in human serum induced by antigen-antibody complexes is a major hurdle for monitor
42                    Overall, DEPC labeling of antigen-antibody complexes is found to depend on both ch
43 model, we demonstrate that on recognition of antigen-antibody complexes, lining macrophages undergo s
44 n presentation by B cells and persistence of antigen-antibody complexes on follicular dendritic cells
45 antigen and the capacity to form multivalent antigen-antibody complexes on target cells were key dete
46 f secondary infection, there were indeed NS1 antigen-antibody complexes on the admission day during t
47 cence (ECL) with exposure to X-ray film, the antigen-antibody complexes on the blot are reacted with
48 CD camera detects the pattern of fluorescent antigen:antibody complexes on the sensor surface.
49  with the formation of citrullinated protein antigen-antibody complexes or other forms of ICs.
50 ms associated with limited proteolysis of an antigen-antibody complex particularly in the vicinity of
51 formed on each platform with three different antigen/antibody complexes possessing nanomolar to picom
52                                              Antigen-antibody complexes provide useful models for ana
53                                              Antigen-antibody complexes provide useful models for stu
54 rotein multimers, peptide-protein complexes, antigen-antibody complexes, RNA, RNA multimers, and prot
55  depends on extensive structural analyses of antigen-antibody complexes.Single-particle electron cryo
56  the antigenic interactions within the known antigen-antibody complex structures.
57                Epitome consists of all known antigen/antibody complex structures, a detailed descript
58 n A linked to Sepharose were used to isolate antigen-antibody complexes that contained few contaminat
59  system to couple receptors for antigens and antigen-antibody complexes to adaptive and innate immune
60 ed strategy for in-depth characterization of antigen-antibody complexes to enable the identification
61 h wild-type GFP and the formation of the GFP antigen-antibody complex was monitored.
62 ctroscopy (EIS) in which the formation of an antigen-antibody complex was quantified as a function of
63 three of these subpopulations of pAbs formed antigen-antibody complexes which could be isolated by ge
64  refolded antibodies were capable of forming antigen-antibody complexes which could be isolated by ge
65                   Further, incubation of the antigen-antibody complex with 11-cis-retinal failed to r
66 tibodies are imported into the nucleus as an antigen-antibody complex with coilin.
67 nd genetic studies coupling the structure of antigen-antibody complexes with their antiviral function