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1 determinant for hypertension and response to antihypertensive therapy.
2 %-0.6%) also met the guideline threshold for antihypertensive therapy.
3 risk, contributing to low use of statin and antihypertensive therapy.
4 ts are often managed with intensification of antihypertensive therapy.
5 ssociated with nonadherence to statin and/or antihypertensive therapy.
6 le to where hypertension is not treated with antihypertensive therapy.
7 lion (95% CI, 2.02 million-3.21 million) for antihypertensive therapy.
8 ween perceived discrimination and suboptimal antihypertensive therapy.
9 se these drugs in patients already receiving antihypertensive therapy.
10 atient was discharged with anticoagulant and antihypertensive therapy.
11 equired further intensification of discharge antihypertensive therapy.
12 RDN emerged as an innovative interventional antihypertensive therapy.
13 or antagonist, or AngII infusion with triple-antihypertensive therapy.
14 ther help advance personalized approaches to antihypertensive therapy.
15 mm Hg diastolic and treated with intravenous antihypertensive therapy.
16 bo required the initiation of or a change in antihypertensive therapy.
17 t is resistant to most forms of conventional antihypertensive therapy.
18 t the study, most were on antithrombotic and antihypertensive therapy.
19 number needed to treat for both statins and antihypertensive therapy.
20 the score components of diabetes and use of antihypertensive therapy.
21 ossibility of targeting the EP1 receptor for antihypertensive therapy.
22 e provider in improving patient adherence to antihypertensive therapy.
23 ians than in African Americans regardless of antihypertensive therapy.
24 he physiologic evidence of renoprotection by antihypertensive therapy.
25 cal cardiovascular disease and not receiving antihypertensive therapy.
26 pine, or placebo in addition to conventional antihypertensive therapy.
27 sease and should be preferred for first-line antihypertensive therapy.
28 kers, should no longer be used as first-line antihypertensive therapy.
29 on who were under evaluation for a change in antihypertensive therapy.
30 disease and those receiving anticoagulant or antihypertensive therapy.
31 those adherent to statin, but nonadherent to antihypertensive, therapy.
32 potential for designing and evaluating novel antihypertensive therapies.
33 versus <=145 mm Hg (standard treatment) with antihypertensive therapies.
34 rovide a potential target for individualized antihypertensive therapies.
35 wer risk of combined MI or stroke than other antihypertensive therapies.
36 59) for those nonadherent both to statin and antihypertensive therapy, 1.82 (95% CI: 1.43 to 2.33) fo
38 olds could reduce eligibility for statin and antihypertensive therapy among 15.8 million US adults.
39 tions between 1953 and 1957 and were free of antihypertensive therapy and cardiovascular disease.
40 ovide validation for TMEM16A as a target for antihypertensive therapy and demonstrate the efficacy of
42 ians at all exercise workloads regardless of antihypertensive therapy and had over a 90% higher likel
43 kers, represents the backbone of recommended antihypertensive therapy and intense debate about their
44 and of all-cause mortality in the setting of antihypertensive therapy and regression of ECG left vent
45 ith statins and other lipid-lowering agents, antihypertensive therapies, and antihyperglycemic treatm
46 those non-adherent to statin but adherent to antihypertensive therapy, and 1.30 (95% CI: 0.53 to 3.20
47 ead time, reduced effectiveness of intensive antihypertensive therapy, and increased relative risk re
49 sk categorization, eligibility for statin or antihypertensive therapy, and projected occurrences of m
50 ramework for addressing patient adherence to antihypertensive therapy, and to propose future directio
51 ludes diuretics as part of the first step of antihypertensive therapy, and updated analysis does not
52 f lifetime discrimination on the underuse of antihypertensive therapy appears partially mediated by d
53 ust, 2010, for randomised clinical trials of antihypertensive therapy (ARBs, angiotensin-converting-e
56 d ACEI/ARB and 24,001 (61.1%) received other antihypertensive therapy at year 1 after transplantation
57 multivariable models adjusting for age, sex, antihypertensive therapy, body mass index, heart rate, t
58 nzyme (ACE) inhibitors are used primarily in antihypertensive therapy but also are known to improve w
59 ing fixed-combination, low-dose, triple-pill antihypertensive therapy (consisting of amlodipine, telm
61 terol, estimated glomerular filtration rate, antihypertensive therapy, diabetes mellitus, and smoking
62 sence of renal failure, and documentation of antihypertensive therapy, diabetic status, proteinuria s
63 further reducing the diastolic pressure with antihypertensive therapy, especially in patients with co
65 lyzed according to whether ACEI/ARB or other antihypertensive therapy (excluding diuretics) was admin
67 Large-scale trials to assess the value of antihypertensive therapy for older patients with SBP of
68 te, free of coronary heart disease (CHD) and antihypertensive therapy, from the Chicago Heart Associa
70 hough black patients received more intensive antihypertensive therapy, Hispanics were undertreated.
71 the pathophysiology, risks, and benefits of antihypertensive therapies in the patient with intracran
73 aptured year-by-year adherence to statin and antihypertensive therapy in both study groups and estima
74 e endothelial, cardiac, and renal effects of antihypertensive therapy in hypertension and may explain
75 hypotension; the effectiveness of nocturnal antihypertensive therapy in patients with coexistent neu
76 fined a new risk threshold for initiation of antihypertensive therapy in patients with stage 1 hypert
77 cal trials from 1965 through October 2010 of antihypertensive therapy in patients with type 2 diabete
78 ding the biological mechanisms and choice of antihypertensive therapy in pregnancy should be carefull
82 aracteristics associated with an increase in antihypertensive therapy included increased levels of bo
83 nell product electrocardiographic LVH during antihypertensive therapy is associated with a lower like
84 nell product electrocardiographic LVH during antihypertensive therapy is associated with fewer hospit
85 uct and Sokolow-Lyon voltage criteria during antihypertensive therapy is associated with lower likeli
88 ough literature on reduction of LV mass with antihypertensive therapy is extensive, little informatio
90 re have been advances in management, such as antihypertensive therapy, magnesium sulphate, and fluid
92 dies have suggested that evening dosing with antihypertensive therapy might have better outcomes than
93 blind candesartan or placebo with open-label antihypertensive therapy (mostly thiazide diuretics) add
96 f incident dementia; however, the effects of antihypertensive therapy on cognitive function in contro
99 combined outcome of MI and stroke than other antihypertensive therapies (OR, 0.49; 95% CI, 0.32-0.77)
100 diabetes mellitus, systolic blood pressure, antihypertensive therapy, prior coronary disease, and le
102 hest approved dosage of losartan and optimal antihypertensive therapy reduces albuminuria over 6 mo a
104 with hypertension who were not receiving any antihypertensive therapy (relative hazard, 0.91; 95 perc
106 baseline severity of ECG LVH, losartan-based antihypertensive therapy resulted in greater regression
108 sis was managed by withholding pre-apheresis antihypertensive therapy, saline prehydration, and reduc
113 ave less regression of CP LVH in response to antihypertensive therapy than patients without diabetes,
116 relative to those who adhered to statins and antihypertensive therapy, the odds ratio at the year of
117 or age, sex, systolic blood pressure, use of antihypertensive therapy, total and high-density lipopro
118 tics will rightfully remain a cornerstone in antihypertensive therapy, we should remember (as we were
124 derate evidence to support initial or add-on antihypertensive therapy with an angiotensin-converting
125 nsion (ACCOMPLISH) trial showed that initial antihypertensive therapy with benazepril plus amlodipine
126 HEP) trial, conducted between 1985 and 1990, antihypertensive therapy with chlorthalidone-based stepp
129 partitioning to assess the probability that antihypertensive therapy would be increased at a given c