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1  a new target of indomethacin, a widely used antiinflammatory drug.
2  the thyroid) can be handled by nonsteroidal antiinflammatory drugs.
3  of death was 0.68 for users of nonsteroidal antiinflammatory drugs.
4 , even in the absence of recent nonsteroidal antiinflammatory drugs.
5 herapy for paucibacillary leprosy along with antiinflammatory drugs.
6  are the therapeutic targets of nonsteroidal antiinflammatory drugs.
7 th chemical gastritis caused by nonsteroidal antiinflammatory drugs.
8 ynthesis and are the targets of nonsteroidal antiinflammatory drugs.
9 d is potently inhibited by some nonsteroidal antiinflammatory drugs.
10 patients following ingestion of nonsteroidal antiinflammatory drugs.
11 both low-dose aspirin and other nonsteroidal antiinflammatory drugs.
12 get for the discovery and development of new antiinflammatory drugs.
13 RA) and its response to chronic nonsteroidal antiinflammatory drugs.
14 tential targets for the development of novel antiinflammatory drugs.
15  additional approaches to the development of antiinflammatory drugs.
16 OX-1 inhibition associated with nonsteroidal antiinflammatory drugs.
17  measure the in vivo potency of nonsteroidal antiinflammatory drugs.
18 target of antineoplastic, antimicrobial, and antiinflammatory drugs.
19  interfere with PGE2 synthesis, as effective antiinflammatory drugs.
20 a prime therapeutic target for analgesic and antiinflammatory drugs.
21 to treatment with allopurinol, steroids, and antiinflammatory drugs.
22 ts this emerging issue in patients requiring antiinflammatory drugs.
23 s of the indomethacin family of nonsteroidal antiinflammatory drugs.
24 esthetics block neurons, but are also potent antiinflammatory drugs.
25  patients received prescription nonsteroidal antiinflammatory drugs, 58% received opioids, and 50% re
26                                              Antiinflammatory drugs achieve their therapeutic actions
27 ain mimic suggests an intracellular site for antiinflammatory drug action.
28 acrophage inhibitory cytokine-1/nonsteroidal antiinflammatory drug-activated gene-1 promoter by 2-5A
29 acrophage inhibitory cytokine-1/nonsteroidal antiinflammatory drug-activated gene-1, a TGF-beta super
30 onservative treatment (CT) with nonsteroidal antiinflammatory drugs alone.
31 tic drugs, corticosteroids, and nonsteroidal antiinflammatory drugs, alone or in combination.
32  analogues of diclofenac (1), a nonsteroidal antiinflammatory drug and known inhibitor of transthyret
33 corticoids have great clinical importance as antiinflammatory drugs and can act as potent inducers of
34  are pharmacological targets of nonsteroidal antiinflammatory drugs and cyclooxygenase (COX) 2 inhibi
35          In contrast, opioids, non-steriodal antiinflammatory drugs and cyclooxygenase-2 inhibitors h
36 ns for the perioperative use of nonsteroidal antiinflammatory drugs and glucocorticoids have the most
37 ths and inadequate responses to nonsteroidal antiinflammatory drugs and glucocorticoids) to the anti-
38 properties yet differs from the nonsteroidal antiinflammatory drugs and inhibitors of prostaglandin H
39               Stable dosages of nonsteroidal antiinflammatory drugs and low-dose prednisone were allo
40  discontinued; stable dosage of nonsteroidal antiinflammatory drugs and oral corticosteroids (</=10 m
41 ll count, and use of nonaspirin nonsteroidal antiinflammatory drugs and other medications, subjects w
42 ction of 15-LOX-1 expression by nonsteroidal antiinflammatory drugs and related agents.
43 y in cells were detected: three nonsteroidal antiinflammatory drugs and the thyroid hormone 3,3',5-tr
44                    Therapy with nonsteroidal antiinflammatory drugs and traditionally used disease-mo
45 g medications in chronic gout, 2) the use of antiinflammatory drugs, and 3) counseling on lifestyle m
46 iovascular risk factors, use of nonsteroidal antiinflammatory drugs, and a history of gastric or duod
47  some medications, particularly nonsteroidal antiinflammatory drugs, and has been associated with aut
48  of visiting a physician, using analgesic or antiinflammatory drugs, and having arthroplasty (P < 0.0
49 n-converting enzyme inhibitors, nonsteroidal antiinflammatory drugs, and immunosuppressive drugs.
50 g)/height (m)2), never-users of nonsteroidal antiinflammatory drugs, and those with lower intake of d
51                    We found that widely used antiinflammatory drugs antagonize both biochemical and b
52 enocarcinoma but did agree that nonsteroidal antiinflammatory drugs are associated with a cancer risk
53                                 Nonsteroidal antiinflammatory drugs are used in rheumatoid arthritis
54                       High-dose nonsteroidal antiinflammatory drugs are usually the first-line treatm
55 otentially game-changing in the nonsteroidal antiinflammatory drug arena.
56                                   The common antiinflammatory drug aspirin can reduce inflammasome ac
57 esis and are the targets of the nonsteroidal antiinflammatory drugs aspirin and ibuprofen and the COX
58 s at 100 mug/mL was similar to non-steroidal antiinflammatory drugs aspirin, ibuprofen and naproxen,
59 ygenases, the central action of nonsteroidal antiinflammatory drugs (but not a prominent effect of st
60 ncer chemoprotective effects of nonsteroidal antiinflammatory drugs by defining a mechanism through w
61                                 Nonsteroidal antiinflammatory drugs can cause regression of adenomas,
62 h prednisolone, methotrexate, 3 nonsteroidal antiinflammatory drugs (celecoxib, diclofenac, and indom
63 hways and the target of cytokine-suppressing antiinflammatory drugs (CSAIDs).
64 ctive lipid mediators via COX-2-nonsteroidal antiinflammatory drug-dependent oxygenations and cell-ce
65                       Important nonsteroidal antiinflammatory drug derivatives such as (+/-)-naproxen
66 rimental concentration depth profiles of the antiinflammatory drug dexamethasone in human skin, we mo
67  human skin, we model the penetration of the antiinflammatory drug dexamethasone into the skin layers
68 -inflammatory drug naproxen or the steroidal antiinflammatory drug dexamethasone.
69 T is considered to be a promising target for antiinflammatory drug discovery.
70          Moreover, treatment of animals with antiinflammatory drugs during epileptogenesis prevented
71                                 Nonsteroidal antiinflammatory drug enteropathy is a stepwise process
72 aemia are useful indications of nonsteroidal antiinflammatory drug enteropathy.
73                                 Nonsteroidal antiinflammatory drugs, especially Voltaren, may offer e
74 ow that the R-enantiomer of the nonsteroidal antiinflammatory drug etodolac inhibited tumor developme
75                Given that clinical trials of antiinflammatory drugs for LUTS have been largely unsucc
76  corticosteroids - normally highly effective antiinflammatory drugs - has little therapeutic benefit.
77                                         Most antiinflammatory drugs have been associated with an incr
78                   Glucocorticoids are potent antiinflammatory drugs; however, the mechanisms by which
79 e catalysis, and a known chemopreventive and antiinflammatory drug, i.e., curcumin, is used for the m
80 a- and gamma-cyclodextrins with nonsteroidal antiinflammatory drugs ibuprofen, naproxen, and flurbipr
81 nitor the therapeutic effectiveness of novel antiinflammatory drugs in future human trials.
82 reate concerns about the use of nonsteroidal antiinflammatory drugs in patients with skeletal injury.
83 re as effective as nonselective nonsteroidal antiinflammatory drugs in relieving pain and inflammatio
84 is unique among clinically used nonsteroidal antiinflammatory drugs in that it irreversibly inactivat
85                                 Nonsteroidal antiinflammatory drugs, including ibuprofen, are among t
86 is unclear whether nonselective nonsteroidal antiinflammatory drugs increase or decrease the rate of
87  three solid-state forms of the nonsteroidal antiinflammatory drug indomethacin with ammonia gas.
88 istinguishable from that of the nonsteroidal antiinflammatory drug indomethacin, which blocked the pr
89 ing inflammatory bowel disease, nonsteroidal antiinflammatory drug-induced gut injury, and chemothera
90 e endoscopy studies demonstrate nonsteroidal antiinflammatory drug-induced small bowel erosions, but
91                                 Nonsteroidal antiinflammatory drugs inhibit the development of prosta
92 n colon cancer cells by certain nonsteroidal antiinflammatory drugs involves increased expression of
93 blockade by two analgesics (the nonsteroidal antiinflammatory drug ketoprofen and the mu-opioid agoni
94 perative, administration of the nonsteroidal antiinflammatory drug ketorolac and/or resolvins, a fami
95 rate that preoperative administration of the antiinflammatory drug ketorolac or specialized proresolv
96           Surprisingly, certain nonsteroidal antiinflammatory drugs known to shift the site of proteo
97  the chemopreventive effects of nonsteroidal antiinflammatory drugs may be related to their inhibitio
98                        Existing nonsteroidal antiinflammatory drugs may be safer than originally thou
99 nticoagulants like heparin and non-steroidal antiinflammatory drugs may have a solid basis.
100     This class of nonsteroidal, GR-dependent antiinflammatory drugs may offer a safer alternative to
101 ests that rectally administered nonsteroidal antiinflammatory drugs may reduce the incidence of pancr
102  inhibit PG production, such as nonsteroidal antiinflammatory drugs, may be deleterious for muscle gr
103 idely used to treat OA, such as nonsteroidal antiinflammatory drugs, may have limited efficacy once j
104 oped for the quantitation of the anti-cancer/antiinflammatory drug methotrexate (MTX) and its major m
105 d nature of interactions of two nonsteroidal antiinflammatory drugs, namely aspirin and ibuprofen, in
106 cal studies have shown that the nonsteroidal antiinflammatory drug naproxen may be useful in the trea
107 er and amide derivatives of the nonsteroidal antiinflammatory drug naproxen was designed to have both
108 r novel activities such as hydroxylating the antiinflammatory drug naproxen.
109 r Helicobacter pylori-negative, nonsteroidal antiinflammatory drug-negative ulcer patients?
110 ncer cells with and without the nonsteroidal antiinflammatory drug NS-398 or the histone deacetylase
111 itis: 1) a generic nonselective nonsteroidal antiinflammatory drug (NSAID(NS)) alone; 2) NSAID(NS) pl
112 nt a washout of their analgesic/nonsteroidal antiinflammatory drug (NSAID) agents (duration 5 half-li
113 tudy compared the effect of the nonsteroidal antiinflammatory drug (NSAID) diclofenac, which is the m
114 e in zebrafish larvae using the nonsteroidal antiinflammatory drug (NSAID) Glafenine.
115 stine is a more common site for nonsteroidal antiinflammatory drug (NSAID) toxicity than the well-rec
116 les but only modestly decreased nonsteroidal antiinflammatory drug (NSAID) turnover while maintaining
117 ors conducted a cohort study of nonsteroidal antiinflammatory drug (NSAID) use and risk of symptomati
118                                 Nonsteroidal antiinflammatory drug (NSAID) use is a known risk factor
119  the present study, sulindac, a nonsteroidal antiinflammatory drug (NSAID), was tested for protection
120  of diflunisal, an FDA-approved nonsteroidal antiinflammatory drug (NSAID), were synthesized and eval
121                                 Nonsteroidal antiinflammatory drug (NSAID)-associated gastropathy is
122                        Aspirin, nonsteroidal antiinflammatory drugs (NSAID), and acetaminophen are co
123 tiveness of both currently used nonsteroidal antiinflammatory drugs (NSAIDs) (diclofenac) and novel C
124               Among nonusers of nonsteroidal antiinflammatory drugs (NSAIDs) (Pinteraction = 0.055),
125 whether use of aspirin or other nonsteroidal antiinflammatory drugs (NSAIDs) affects risk.
126  to assess the relation between nonsteroidal antiinflammatory drugs (NSAIDs) and 1-year mortality in
127  risk, studies of nonselective, nonsteroidal antiinflammatory drugs (NSAIDs) and acetaminophen have b
128             Previous studies on nonsteroidal antiinflammatory drugs (NSAIDs) and breast cancer have p
129 ted an inverse relation between nonsteroidal antiinflammatory drugs (NSAIDs) and colon cancer, but fe
130 nt preferences for nonselective nonsteroidal antiinflammatory drugs (NSAIDs) and cyclooxygenase-2 (CO
131 iation between long-term use of nonsteroidal antiinflammatory drugs (NSAIDs) and non-Hodgkin lymphoma
132 data on the association between nonsteroidal antiinflammatory drugs (NSAIDs) and ovarian cancer risk
133 ns have identified a variety of nonsteroidal antiinflammatory drugs (NSAIDs) and structurally related
134 nac or other competitively acting nonsteroid antiinflammatory drugs (NSAIDs) and the enzyme activity
135  a concurrent administration of nonsteroidal antiinflammatory drugs (NSAIDS) and/or low-dose corticos
136 es and because apparently safer nonsteroidal antiinflammatory drugs (NSAIDs) are being produced, we s
137                                 Nonsteroidal antiinflammatory drugs (NSAIDs) are commonly used and fr
138                        Although nonsteroidal antiinflammatory drugs (NSAIDs) are frequently used for
139                        Although nonsteroidal antiinflammatory drugs (NSAIDs) are ineffective in treat
140                                 Nonsteroidal antiinflammatory drugs (NSAIDs) block prostanoid biosynt
141 reas treatment with 2 different nonsteroidal antiinflammatory drugs (NSAIDs) blocked neuronal CCEs an
142                                 Nonsteroidal antiinflammatory drugs (NSAIDs) can inhibit colorectal t
143 stablished that taking multiple nonsteroidal antiinflammatory drugs (NSAIDs) can lead to serious gast
144                             The nonsteroidal antiinflammatory drugs (NSAIDs) continue to be important
145  have shown that regular use of nonsteroidal antiinflammatory drugs (NSAIDs) decreases bladder cancer
146                                 Nonsteroidal antiinflammatory drugs (NSAIDs) form a paradigm for the
147                                 Nonsteroidal antiinflammatory drugs (NSAIDs) have been reported to an
148 of all its substrates, certain non-steroidal antiinflammatory drugs (NSAIDs) have been shown to selec
149          Here, we show that the nonsteroidal antiinflammatory drugs (NSAIDs) ibuprofen and indomethac
150                        Although nonsteroidal antiinflammatory drugs (NSAIDs) in general are passed in
151                             All nonsteroidal antiinflammatory drugs (NSAIDs) inhibit the cyclooxygena
152 gic studies suggest that use of nonsteroidal antiinflammatory drugs (NSAIDs) is associated with a red
153 ase-inhibiting (COX-inhibiting) nonsteroidal antiinflammatory drugs (NSAIDs) is often complicated by
154    The cardiovascular safety of nonsteroidal antiinflammatory drugs (NSAIDs) may be influenced by int
155 nal prostaglandin inhibition by nonsteroidal antiinflammatory drugs (NSAIDs) may decrease renal funct
156 ing the possibility that use of nonsteroidal antiinflammatory drugs (NSAIDs) may inhibit the developm
157 dies have suggested that use of nonsteroidal antiinflammatory drugs (NSAIDs) may reduce the risk of b
158 otentially protective effect of nonsteroidal antiinflammatory drugs (NSAIDs) on prostate cancer may o
159 nee monarthritis were compared: nonsteroidal antiinflammatory drugs (NSAIDs) only, NSAID trial follow
160 res of high-risk prescribing of nonsteroidal antiinflammatory drugs (NSAIDs) or selected antiplatelet
161 al studies suggests that use of nonsteroidal antiinflammatory drugs (NSAIDs) reduces risk of colon an
162 ave shown that long-term use of nonsteroidal antiinflammatory drugs (NSAIDs) reduces the risk of deve
163                        Although nonsteroidal antiinflammatory drugs (NSAIDs) show great promise as th
164                Both traditional nonsteroidal antiinflammatory drugs (NSAIDs) that inhibit COX-1 and C
165 ntestinal liabilities common to nonsteroidal antiinflammatory drugs (NSAIDs) that might be reduced by
166                                 Nonsteroidal antiinflammatory drugs (NSAIDs) transcriptionally up-reg
167 etermine whether daily users of nonsteroidal antiinflammatory drugs (NSAIDs) were at lower risk than
168                       Different nonsteroidal antiinflammatory drugs (NSAIDs) were used to examine the
169  including coxibs, nonselective nonsteroidal antiinflammatory drugs (NSAIDs), and meloxicam, were pre
170 ostaglandin production, such as nonsteroidal antiinflammatory drugs (NSAIDs), and pharmacologic nitri
171               Acid suppression, nonsteroidal antiinflammatory drugs (NSAIDs), and statins may play a
172 any reason did not differ among nonsteroidal antiinflammatory drugs (NSAIDs), but discontinuations du
173                     Many of these nonsteroid antiinflammatory drugs (NSAIDs), but in particular diclo
174 ugs, including D-penicillamine, nonsteroidal antiinflammatory drugs (NSAIDs), calcium channel blocker
175 nhibitors and aspirin, or other nonsteroidal antiinflammatory drugs (NSAIDs), in combination as a str
176 , as compared with nonselective nonsteroidal antiinflammatory drugs (NSAIDs), remains uncertain.
177 teoarthritis (OA) pain, such as nonsteroidal antiinflammatory drugs (NSAIDs), simple analgesics, and
178                                 Nonsteroidal antiinflammatory drugs (NSAIDs), such as aspirin, freque
179 4-eicosatetraynoic acid, and in nonsteroidal antiinflammatory drugs (NSAIDs), such as indomethacin an
180 al hemorrhage than nonselective nonsteroidal antiinflammatory drugs (NSAIDs), there has been concern
181 mmon undesirable effects of the nonsteroidal antiinflammatory drugs (NSAIDs).
182 rapeutic target for widely used nonsteroidal antiinflammatory drugs (NSAIDs).
183 proportion of prescriptions for nonsteroidal antiinflammatory drugs (NSAIDs).
184 urally occurring flavonols) and nonsteroidal antiinflammatory drugs (NSAIDs).
185 ds is more limited than that on nonsteroidal antiinflammatory drugs (NSAIDs).
186 d are the molecular targets for nonsteroidal antiinflammatory drugs (NSAIDs).
187 effects than currently marketed nonsteroidal antiinflammatory drugs (NSAIDs).
188 on was inhibited by aspirin and nonsteroidal antiinflammatory drugs (NSAIDs).
189 1 (PGHS-1) is the target of the nonsteroidal antiinflammatory drugs (NSAIDs).
190    Both isoforms are targets of nonsteroidal antiinflammatory drugs (NSAIDs).
191 MTX), oral glucocorticoids, and nonsteroidal antiinflammatory drugs (NSAIDs).
192 ng the use of aspirin and other nonsteroidal antiinflammatory drugs (NSAIDs).
193 al cancer in relation to use of nonsteroidal antiinflammatory drugs (NSAIDs).
194 itors (coxibs) and nonselective nonsteroidal antiinflammatory drugs (NSAIDs).
195 ommon active internal controls (nonsteroidal antiinflammatory drugs [NSAIDs], naproxen) were determin
196 re both targets of nonselective nonsteroidal antiinflammatory drugs (nsNSAIDs) including aspirin wher
197 ent only among those not taking nonsteroidal antiinflammatory drugs (odds ratio = 0.49, 95% CI: 0.25,
198  cancer in individuals who take nonsteroidal antiinflammatory drugs on a regular basis compared with
199 persons taking aspirin or other nonsteroidal antiinflammatory drugs on a regular basis.
200 To evaluate the effects of antimicrobial and antiinflammatory drugs on oviductal pathology in chronic
201 hylaxis for leishmaniasis or even the use of antiinflammatory drugs or antibiotics may be considered
202 dicated that sulindac and other nonsteroidal antiinflammatory drugs or cyclooxygenase inhibitors have
203 tinue their background doses of nonsteroidal antiinflammatory drugs or disease-modifying antirheumati
204                          Use of nonsteroidal antiinflammatory drugs (OR = 1.7, 95% CI 1.0, 2.6), angi
205 term users of aspirin, ethanol, nonsteroidal antiinflammatory drugs, or a combination of these substa
206 tween the use of aspirin, other nonsteroidal antiinflammatory drugs, or acetaminophen and PD risk.
207  to determine whether steroids, nonsteroidal antiinflammatory drugs, or both can prevent or treat the
208 were allowed to continue taking nonsteroidal antiinflammatory drugs, oral corticosteriods (< or =10 m
209 rcinomas, and its inhibition by nonsteroidal antiinflammatory drugs protects against colorectal cance
210 as diminished by an analog of a nonsteroidal antiinflammatory drug (R-etodolac), at concentrations th
211                 Tor proteins, targets of the antiinflammatory drug rapamycin, mediate a conserved sig
212 laboratory studies suggest that nonsteroidal antiinflammatory drugs reduce the risk of colon cancer a
213                                 Nonsteroidal antiinflammatory drugs reduce the risk of colon cancer,
214 ger among participants who used nonsteroidal antiinflammatory drugs regularly (highest vs. lowest qua
215 the start of therapy: age, sex, nonsteroidal antiinflammatory drug-related risk factors for upper gas
216 ition of the COX-2 component by nonsteroidal antiinflammatory drugs restored the apoptotic response.
217 o study the interactions of two nonsteroidal antiinflammatory drugs, salicylate and ibuprofen, with v
218 to the analysis of a mixture of nonsteroidal antiinflammatory drugs separated by reversed-phase HPLC.
219                           Whenever possible, antiinflammatory drugs should be given in monotherapy an
220                                 Nonsteroidal antiinflammatory drugs still remain mainstays of treatme
221 logically important targets for nonsteroidal antiinflammatory drugs, such as aspirin and newer COX-2
222 rsion of carboxylate-containing nonsteroidal antiinflammatory drugs, such as indomethacin, to esters
223 bryos to disordered inflammation; therefore, antiinflammatory drugs, such as low-dose aspirin (LDA),
224 l-2-isopropylpyrazolo-[1,5-a]pyridine) is an antiinflammatory drug that was initially developed for t
225 -neuronal cells are blocked by non-steroidal antiinflammatory drugs that are derivatives of diphenyla
226  measure the in vivo potency of nonsteroidal antiinflammatory drugs that competitively inhibit arachi
227 e or nonuse of aspirin or other nonsteroidal antiinflammatory drugs; the presence or absence of physi
228  In contrast, when mice were treated with an antiinflammatory drug to prevent microglial activation,
229 cin (INDO) was one of the first nonsteroidal antiinflammatory drugs to be characterized as a time-dep
230 e may be a more common site for nonsteroidal antiinflammatory drug toxicity than the gastroduodenal m
231                                 Nonsteroidal antiinflammatory drug toxicity to the small intestine is
232                        The lack of effective antiinflammatory drug treatment for COPD has thus shifte
233 viously received treatment with nonsteroidal antiinflammatory drugs underwent stratification accordin
234 ession analysis revealed age and non-steroid antiinflammatory drug usage were risk factors for divert
235 es in New Jersey, the effect of nonsteroidal antiinflammatory drug use on 1-year all-cause mortality
236         We investigated whether nonsteroidal antiinflammatory drug use was associated with a lower ri
237  physical activity, aspirin and nonsteroidal antiinflammatory drug use, alcohol consumption, and inta
238           Two-thirds of regular nonsteroidal antiinflammatory drug users develop subclinical small bo
239 s (primarily fluoroquinolones), nonsteroidal antiinflammatory drugs (Voltaren and ketorolac), and new
240 d has been shown to be a molecular target of antiinflammatory drugs, we sought to characterize the fu
241 dy medications and avoidance of nonsteroidal antiinflammatory drugs were excellent.
242  of fluids ingested, and use of nonsteroidal antiinflammatory drugs were not.
243 story of inadequate response to nonsteroidal antiinflammatory drugs were randomized to receive 40 mg
244                                 Nonsteroidal antiinflammatory drugs, which inhibit COX-2, prevent col
245                            As an established antiinflammatory drug with neuroprotective properties, m

 
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