1 of a fluorine atom at C3 of 3DUrd shifts its antimetabolic action to inhibition of the orotidylate de

2 (s)(t)ide mimetics as potential antiviral or antimetabolic agents.

3 s, ivermectin shows antiviral, antimalarial, antimetabolic, and anticancer effects.

4 rmore, glutamine deprivation, as well as the antimetabolic drugs 2-deoxyglucose and metformin, also p

5 ity allows pathogens to evolve resistance to antimetabolic drugs by acquiring new metabolic pathways

6  = 23.3 muM) coupled with the absence of any antimetabolic effect (CC50 > 224 muM; SI > 28) in a cell

7 f F2,6BP sensitizes transformed cells to the antimetabolic effects of PFK2/FBPase inhibition.

8 on of hindbrain catecholamine neurons by the antimetabolic glucose analogue, 2-deoxy-D-glucose (2DG;

9  volumes of 5-thio-D-glucose (5TG), a potent antimetabolic glucose analogue, were studied at 142 hind

10 tabolic stress through intrinsic or acquired antimetabolic stress responses to maintain homeostasis a

11 ility to monitor biomarkers for responses to antimetabolic therapy in real-time, paving the way for c

12  results offer a rationale to evaluate novel antimetabolic treatment strategies being developed in ot