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1 ue, vague cognitive defects, and a low titer antinuclear antibody.
2                Three patients had a positive antinuclear antibody.
3      Sera were evaluated for the presence of antinuclear antibodies.
4 ive autoimmune B cells to produce pathogenic antinuclear antibodies.
5 , P-ANCA was associated with the presence of antinuclear antibodies.
6 mphoid organs spontaneously develop specific antinuclear antibodies.
7  thickening, and high titers of anti-DNA and antinuclear antibodies.
8  increased in humans and mice, combined with antinuclear antibodies.
9 dating autoantibody expression in SLE beyond antinuclear antibodies.
10  disease, such as arthritis, lymphopenia, or antinuclear antibodies.
11 hermore, about one-third of the mice develop antinuclear antibodies.
12 mes clinically detectable by the presence of antinuclear antibodies.
13 ), that are characterized by the presence of antinuclear antibodies.
14 c mice and was accompanied by an increase in antinuclear antibodies.
15 omenon (RP) and the presence of SSc-specific antinuclear antibodies.
16 ortion of autoreactive antibodies, including antinuclear antibodies.
17  immunofluorescence studies that demonstrate antinuclear antibodies.
18 tis and small joint involvement, and express antinuclear antibodies.
19 ts produced in vitro anti-GBM antibodies and antinuclear antibodies.
20  the majority of MZ twins are concordant for antinuclear antibodies.
21 cular dryness in the presence of circulating antinuclear antibodies.
22           Sera from 9/12 patients containing antinuclear antibodies (10/12 meeting diagnostic criteri
23 fold capillaroscopy and laboratory tests for antinuclear antibodies (ANA) and ANA subsets were obtain
24 indirect immunofluorescence assays to detect antinuclear antibodies (ANA) and antineutrophil cytoplas
25 ficient in this molecule develop spontaneous antinuclear antibodies (ANA) and fatal glomerulonephriti
26  in children may be associated with positive antinuclear antibodies (ANA) and may progress to systemi
27 say were compared with those of an assay for antinuclear antibodies (ANA) and with the severity of th
28                                              Antinuclear antibodies (ANA) are important in diagnosis
29   Screening only for antibodies to actin and antinuclear antibodies (ANA) failed to establish the dia
30 cted mixtures in relation to the presence of antinuclear antibodies (ANA), a widely used biomarker of
31  A majority of these autoantibodies are true antinuclear antibodies (ANA), and 50% of the ANAs are al
32 active protein, leukocyte count, presence of antinuclear antibodies (ANA), presence of human leukocyt
33  (NCWS), and investigated whether they carry antinuclear antibodies (ANA).
34  A locus Adnz1 on chromosome 4 was linked to antinuclear antibody (ANA) and anti-double stranded DNA
35    Here we report high titers of spontaneous antinuclear antibody (ANA) in C4(-/)- mice.
36 suggests that patients with in vivo speckled antinuclear antibody (ANA) patterns have high titers of
37  associations between mercury biomarkers and antinuclear antibody (ANA) positivity and titer strength
38 IA patients, including female preponderance, antinuclear antibody (ANA) positivity, and certain human
39 three positive answers were to attend for an antinuclear antibody (ANA) test; and those testing posit
40 orescence methods with bead-based assays for antinuclear antibody (ANA) testing is a new clinical opt
41 ected over a 5-year period in a rheumatology antinuclear antibody (ANA) testing laboratory.
42 A were identified, and serologic studies for antinuclear antibody (ANA), rheumatoid factor, and antib
43 DNA) and double-stranded DNA (anti-dsDNA) in antinuclear antibody (ANA)-positive type 1 autoimmune he
44 ic arthritis, spinal pain, fever, psoriasis, antinuclear antibodies [ANA], and rheumatoid factor).
45 E Disease Activity Index (SLEDAI), serology (antinuclear antibody [ANA] and anti-double-stranded (ds)
46 case-control study confirmed the presence of antinuclear antibodies (ANAs) and other autoantibodies i
47                                          IgG antinuclear antibodies (ANAs) are a feature of several a
48                                     Although antinuclear antibodies (ANAs) are detected in many autoi
49         Two outstanding questions concerning antinuclear antibodies (ANAs) in lupus involve their pat
50 nt (SLC-/-) mice harbored elevated levels of antinuclear antibodies (ANAs) in their serum and showed
51 s (SLE) is characterized by high-avidity IgG antinuclear antibodies (ANAs) that are almost certainly
52 ototypic autoimmune disease characterized by antinuclear antibodies (ANAs) that form immune complexes
53                                  Presence of antinuclear antibodies (ANAs) was also a risk factor for
54                              The presence of antinuclear antibodies (ANAs) was determined using indir
55 -12 survey), laboratory parameters including antinuclear antibodies (ANAs), and SARS-CoV-2 antibody l
56 ti-citrullinated protein antibodies (ACPAs), antinuclear antibodies (ANAs), anti-tissue transglutamin
57 ototypic autoimmune disease characterized by antinuclear antibodies (ANAs), including pathogenic spec
58 bers according to the presence or absence of antinuclear antibodies (ANAs), the ANA staining patterns
59 systemic autoimmune disease characterized by antinuclear antibodies (ANAs).
60 in forming a pre-BCR, leads to production of antinuclear antibodies (ANAs).
61 yed a breached immune tolerance and secreted antinuclear antibodies (ANAs).
62                                              Antinuclear antibodies and AHAs, but not ssDNA abs, were
63 els, E6446 slowed development of circulating antinuclear antibodies and had a modest effect on anti-d
64 omparable in patients with and those without antinuclear antibodies and in both early- and late-onset
65   Female NZB/WF1 mice produce high titers of antinuclear antibodies and invariably succumb to severe
66 between fibromyalgia patients with low titer antinuclear antibodies and primary SS remains difficult.
67            Serum IgE, rheumatoid factor, and antinuclear antibodies and skin sensitivity to common al
68 itis, and laboratory abnormalities including antinuclear antibody and rheumatoid factor positivity.
69  SLE was based on the presence of arthritis, antinuclear antibodies, and double-stranded DNA, and a c
70 ore transplant of idiopathic cardiomyopathy, antinuclear antibodies, and HLA-B8, DR3 haplotypes.
71 ted diagnoses, discoid lupus, positivity for antinuclear antibodies, and/or false-positive syphilis t
72                      Smooth muscle antibody, antinuclear antibody, and antibody to liver/kidney micro
73  presence of EBOV-specific immunoglobulin G, antinuclear antibodies, anti-cyclic citrullinated peptid
74 udies were similar, including positivity for antinuclear antibodies, anti-native DNA, and anti-double
75 ion markers included (1) autoimmunity signs (antinuclear antibody, antihistone antibody, antithyroglo
76                                              Antinuclear antibodies are a hallmark feature of general
77                        Most polyreactive and antinuclear antibodies are removed from the human antibo
78 etric analysis of salivary glands, and serum antinuclear antibody assay were performed to assess vari
79 rbent assay, autoantigen proteome array, and antinuclear antibody assay.
80                         These sera contained antinuclear antibodies, but lacked antibodies to double-
81 ed with the presence of rheumatoid factor or antinuclear antibodies, but not with positive allergen s
82 ccinee (n = 14), and one specimen containing antinuclear antibody cross-reacted with the assay.
83       C-reactive protein, rheumatoid factor, antinuclear antibody, cytoplasmic antineutrophil cytopla
84     Loci linked with anti-gp70 compared with antinuclear antibodies demonstrated the strongest linkag
85 to the drug developed in 8% of patients, and antinuclear antibodies developed in 2%.
86 en patients with (Ab(+)) and without (Ab(-)) antinuclear antibody during adulthood and an age- and se
87             Although SLE is characterized by antinuclear antibody expression, a significant proportio
88                      This study explores the antinuclear antibody fine specificities and underlying c
89 ss offspring uncovered 5 suggestive loci for antinuclear antibody formation, consisting of 3 dominant
90                              Sera containing antinuclear antibodies from patients with systemic lupus
91  regimen eluted severalfold more IgG and IgM antinuclear antibodies from the kidneys of nephritic SNF
92 n(KO) mice is characterized by production of antinuclear antibodies, glomerulonephritis, impaired B c
93 et of 321 serologically active SLE patients (antinuclear antibodies >/=1:80 and/or anti-double-strand
94 ly, H2(d/z) (compared with H2(d/d)) enhanced antinuclear antibodies in a coordinate fashion but did n
95 ther compounded by the finding of "positive" antinuclear antibodies in a high percent of the general
96 ssociations between selected xenobiotics and antinuclear antibodies in the National Health and Nutrit
97 hocytic infiltration of the lacrimal glands, antinuclear antibodies in the serum, and elevated titer
98  serum anti-CCP, rheumatoid factor (RF), and antinuclear antibody in a defined sample of 9,988 of bla
99                    Two patients had positive antinuclear antibodies, including one with a history of
100  positive results because of the presence of antinuclear antibodies, is not a specific marker of anti
101 lambda(s) 11.0), anemia (lambda(s) 1.7), and antinuclear antibodies (lambda(s) 1.3).
102   Serologic markers showed increased IgG and antinuclear antibody levels in seven of 12 patients.
103 ers such as serum immunoglobulin G (IgG) and antinuclear antibody levels were available in 12 patient
104 active protein level, rheumatoid factor, and antinuclear antibody levels were within normal limits.
105 ith DSRD were more likely to have a positive antinuclear antibody, low complement 3, abnormal cytokin
106     Sera were tested using immunofluorescent antinuclear antibodies on HEp-2 cell slides, by anti-RNA
107 -like syndrome with joint manifestations and antinuclear antibodies: one had anti-RNP and antiphospho
108 sy-proven lupus nephritis in the presence of antinuclear antibodies or anti-double-stranded DNA antib
109 n-experienced autoreactive B cells producing antinuclear antibody or rheumatoid factor and exhibit th
110                All six patients had positive antinuclear antibody or smooth muscle antibody titers.
111 )/L: OR 2.15, 95% CI 1.63-2.83), presence of antinuclear antibodies (OR 2.87, 95% 1.57-5.24), and tre
112 ficantly associated with: i/ the presence of antinuclear antibodies (OR=5.9, P=0.04) ii/ a more sever
113       Analysis of his serum was negative for antinuclear antibody (or ANA), cytoplasmic antineutrophi
114 fferences in EBOV-specific immunoglobulin G, antinuclear antibody, or anti-cyclic citrullinated pepti
115 s (P = 0.01) and to have increased titers of antinuclear antibodies (P = 0.04).
116  before the diagnosis, were found later than antinuclear antibodies (P=0.06) and earlier than anti-nu
117 e weakness and congestive heart failure, was antinuclear antibody positive and had muscle biopsy find
118                           Five children were antinuclear antibody positive.
119 on between the effects of rheumatoid factor, antinuclear antibodies, positive skin allergen tests, or
120                              We selected 196 antinuclear antibody-positive adult SLE patients (age >
121 r, randomized, placebo-controlled trial, 819 antinuclear antibody-positive or anti-double-stranded DN
122                                        Seven antinuclear antibody-positive, nephritic female (SWR x N
123  most frequent combination was arthritis and antinuclear antibody positivity followed by malar rash a
124 llin 1 antibodies and active skin disease or antinuclear antibody positivity were found.
125                                              Antinuclear antibody positivity, early onset of disease,
126 subjects in this cohort had similar rates of antinuclear antibody positivity, whereas lupus-specific
127                       We further demonstrate antinuclear antibody production is influenced by the pre
128                                     However, antinuclear antibody production was increased compared w
129 dermatomyositis, the mice developed a strong antinuclear antibody response, characteristic of autoimm
130 ash, autoimmune thyroiditis, the presence of antinuclear antibodies, sinopulmonary infections, and co
131              Serum analysis was negative for antinuclear antibody, Sjogren syndrome antigens A and B,
132  range of autoantibodies (rheumatoid factor, antinuclear antibody, smooth muscle autoantibody, mitoch
133                                              Antinuclear antibody status was a predictor for the deve
134              Male sex, independent of age or antinuclear antibody status, is associated with increase
135                                              Antinuclear antibodies test results were negative.
136                    The indirect fluorescence antinuclear antibody test on Hep-2 cells demonstrated an
137                                      Routine antinuclear antibody testing has a low positive predicti
138 ccordingly, the positive predictive value of antinuclear antibody testing in diagnosing SLE in a pati
139  is needed to establish the value of routine antinuclear antibody testing in patients with uveitis.
140     The positive predictive value of routine antinuclear antibody testing was 2.9% (95% CI, 2.65%-3.1
141                                      Routine antinuclear antibody testing, for example, is not recomm
142 e negative for anti-SSA/Ro, anti-SSB/La, and antinuclear antibodies than women (36% men vs 11% women,
143 up (71.5%) of serologically active patients (antinuclear antibody titer >/=1:80 and/or anti-double-st
144 ents with lcSSc or Raynaud's syndrome and an antinuclear antibody typical of lcSSc as part of the bas
145 elet volume, positive rheumatoid factor, and antinuclear antibodies were all significantly more preva
146 s syndrome with high levels of antineural or antinuclear antibodies were infused bilaterally into the
147                                              Antinuclear antibodies were present at low titers in 4 p
148                                              Antinuclear antibodies were present in 23 (69.9%) of pat
149                                              Antinuclear antibodies were present in 4 of 55 sera test
150                                              Antinuclear antibodies were present in 78 percent (at a
151                                              Antinuclear antibodies were present in all patients.
152 nofluorescence immunoreactants and low titer antinuclear antibodies were variably present.
153 s with systemic autoimmune disease featuring antinuclear antibodies with IgG4 elevation uncovered an

 
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