ライフサイエンスコーパス: antioxidant vitamin

1 onger correlates of lung function than other antioxidant vitamins.

2 All of these changes were prevented by antioxidant vitamins.

3 ive role for supplemental vitamin E or other antioxidant vitamins.

4 ions of lipid hydroperoxides and liposoluble antioxidant vitamins.

5 ingredients - polyphenols, carotenoids, and antioxidant vitamins.

6 PANTS: Within an ongoing randomized trial of antioxidant vitamins, 5442 women who were US health prof

7 cans were poorer and had lower levels of the antioxidant vitamins A and C and alpha-carotene.

8 Dietary intake, especially of antioxidant vitamins A, C, E, and the carotenoids, has b

9 ients, especially folate, calcium, iron, and antioxidant vitamins, affect the risk of colorectal neop

10 mg pyridoxine, and 0.02 mg cyanocobalamin), antioxidant vitamins alone (150 mg ascorbic acid, 67 mg

11 A series of novel derivatives of potent antioxidant vitamin, alpha-lipoic acid, and related anal

12 as to determine the plasma concentrations of antioxidant vitamins and carotenoids in hemodialysis pat

13 Antioxidant vitamins and carotenoids may be protective b

14 ic evidence on the association of individual antioxidant vitamins and cognition is inconsistent.

15 dation is maintained for longer than that of antioxidant vitamins and could thus be more protective.

16 cant trends between plasma concentrations of antioxidant vitamins and either measure of carotid ather

17 lind, randomized trial, Supplementation with Antioxidant Vitamins and Minerals (SU.VI.MAX) Study (199

18 f participants from the Supplementation with Antioxidant Vitamins and Minerals (SU.VI.MAX) Study usin

19 patic iron may promote liver injury, whereas antioxidant vitamins and minerals may inhibit it, but fe

20 eficial effects of oral supplementation with antioxidant vitamins and minerals on the development of

21 5 years after a randomized clinical trial of antioxidant vitamins and minerals was completed.

22 nt effects on plasma concentrations of other antioxidant vitamins and minerals, glutathione peroxidas

23 The association of antioxidant vitamins and trace elements from foods and s

24 fects of the AREDS and AREDS2 supplements of antioxidant vitamins and zinc (plus copper) for reducing

25 Treatment with antioxidant vitamins and zinc can reduce the risk of dev

26 tic components, and higher amounts of fibre, antioxidants, vitamins and other beneficial components.

27 ke and AMD risk by smoking status, intake of antioxidant vitamins, and body fatness.

28 nd the effects of individual phytochemicals, antioxidant vitamins, and minerals.

29 tudy suggest a moderate beneficial effect of antioxidant, vitamin, and zinc supplementation in reduci

30 Deficiencies of antioxidants, vitamins, and microelements, as well as ph

31 a ketogenic diet and an increased intake of antioxidants, vitamins, and minerals, in reducing inflam

32 Accumulating evidence suggests that dietary antioxidant vitamins are positively associated with lung

33 Both lipid-modifying therapy and antioxidant vitamins are thought to have benefit in pati

34 Antioxidant vitamins are thought to play a role in ather

35 of mortality in the general population, and antioxidant vitamins are thought to positively influence

36 Hormone replacement therapy (HRT) and antioxidant vitamins are widely used for secondary preve

37 re simultaneously assigned to receive either antioxidant vitamins (beta-carotene, ascorbic acid, and

38 .15) mm/y with HRT placebo (P =.17); and for antioxidant vitamins by 0.044 (0.15) mm/y and with vitam

39 mage and mutagenesis, whereas addition of an antioxidant (vitamin C) can counteract the induced effec

40 each eddy, where it mixes and reacts with an antioxidant (vitamin C) present in the bulk solution.

41 sside were repeated with a coinfusion of the antioxidant vitamin C (24 mg min(-1)).

42 as measured before and after infusion of the antioxidant vitamin C (25 mg/min i.a.) to determine the

43 side (SNP) and a co-infusion of ACh with the antioxidant vitamin C (ACh+VitC).

44 Moreover, depletion of a murine endogenous antioxidant vitamin C (VC), by genetic disruption of VC-

45 lower in vivo HP (13)C DHA reduction to the antioxidant vitamin C (VitC), prior to histological evid

46 Pretreatment of cells with the antioxidant vitamin C abolished apoptosis, measured as t

47 These results indicate that the antioxidant vitamin C favourably affects the capacity of

48 We sought to determine whether the antioxidant vitamin C improves endothelium-dependent vas

49 This activity was reversed by the antioxidant vitamin C indicating the prooxidant effect o

50 etermine whether acute administration of the antioxidant vitamin C modifies sympathetic nerve activit

51 sons who self-select for supplemental use of antioxidant vitamin C or E or multivitamins, large reduc

52 Co-infusion of acetylcholine with the antioxidant vitamin C partially restored the total forea

53 ter pylori have abnormally low levels of the antioxidant vitamin C, which protects against the format

54 humans is restored by administration of the antioxidant vitamin C.

55 jects before and after administration of the antioxidant vitamin C.

56 tress mechanisms and can be inhibited by the antioxidant vitamin C.

57 acute hypoxaemia of fetal treatment with the antioxidant vitamin C.

58 (n = 8), or HC diet supplemented daily with antioxidant vitamins C (1 g) and E (100 IU/kg) (HC + vit

59 tenosis, n = 7), RVD daily supplemented with antioxidant vitamins C (1 g), and E (100 IU/kg) (RVD+Vit

60 atorvastatin to reduce LDL to <80 mg/dL plus antioxidant vitamins C (1000 mg/d) and E (800 mg/d) (n=1

61 nd, placebo-controlled trial, the effects of antioxidant vitamins C (500 mg/d) and E (400 IU/d) for 6

62 n pill taking was determined in the Trial of Antioxidant Vitamins C and E (TRACE) Study, a factorial

63 e tested whether long-term administration of antioxidant vitamins C and E improves coronary and brach

64 Supplementation with antioxidant vitamins C and E retards the early progressi

65 be significantly reduced by the addition of antioxidant vitamins C and E to the culture medium.

66 We postulated that treatment with antioxidant vitamins C and E would retard the progressio

67 lated Eye Disease Study (AREDS) formulation (antioxidant vitamins C and E, beta carotene, and zinc) h

68 his decrease is blocked by pretreatment with antioxidant vitamins C and E, suggesting an oxidative me

69 to humans, we tested the hypothesis that the antioxidant, vitamin C, could improve endothelium-depend

70 lementation of subjects with the nutritional antioxidant, vitamin C, influences the ability of lympho

71 tus can be improved by administration of the antioxidant, vitamin C.

72 Antioxidants (vitamin C and selenium) and trace elements

73 qual to 70mM was very high compared to usual antioxidants (vitamin C and trolox).

74 he administration dose, combinations with co-antioxidants (vitamin C, caffeic acid, chlorogenic acid,

75 Substituting for UA, alternative antioxidants (vitamin C, DTT, and N-acetylcysteine) also

76 In combination with antioxidants (vitamin C, gallic acid, caffeic acid, trol

77 rms of AREDS supplement components: placebo, antioxidants (vitamin C, vitamin E, beta-carotene), zinc

78 of lutein, 4 mg/day of zeaxanthin, and other antioxidants (vitamin C, vitamin E, zinc, copper) for 16

79 e for 5 months with a diet supplemented with antioxidants (vitamins C and E, selenium, and N-acetylcy

80 Short-term treatment with the antioxidants vitamin C (10 micromol/L) or deferoxamine (

81 Pretreatment of KB cells with antioxidants vitamin C and N-acetylcysteine or the pharm

82 ht to address whether intakes of the dietary antioxidants, vitamin C, alpha-tocopherol, and beta-caro

83 Two olive oil meals also contained antioxidant vitamins (C and E) or foods (balsamic vinega

84 minantly from in vitro studies suggests that antioxidant vitamins can prevent lipid peroxidation and

85 Antioxidant vitamins can protect LDL from oxidation, and

86 Here, we evaluated the antioxidant vitamin composition of dry and germinating s

87 deficiencies that result in homocysteinemia, antioxidants, vitamin D, and iodine.

88 able consumption on plasma concentrations of antioxidant vitamins, daily fruit and vegetable intake,

89 e after one year of therapy with statins and antioxidant vitamins, despite significant lipid-lowering

90 to an LDL level of 80 mg/dL, with or without antioxidant vitamins, does not provide any further benef

91 n of carnosic compared with a reference diet antioxidant (vitamin E), the animals were slaughtered an

92 s investigated the association between serum antioxidant (vitamins E, C, A, carotenoids, selenium) le

93 nt supplementation of the HFHC diet with the antioxidant vitamin E (250, 500, or 1000 IU/kg diet) for

94 Here, we demonstrate that the antioxidant vitamin E (90% alpha-tocopherol) moderately

95 nt in reactive oxygen detoxification via the antioxidant vitamin E (tocopherol).

96 treatment for three days with the endogenous antioxidant vitamin E (Vit-E, 100 mg/kg, i.p.) had an ev

97 ion, treatment of cells with a lipid-soluble antioxidant vitamin E blocked TAM-induced caspase-3 and

98 tert-butyl-alpha-phenylnitrone (PBN) and the antioxidant vitamin E established the involvement of fre

99 Co-administration of the antioxidant vitamin E negated the therapeutic benefit of

100 ent study, we investigated the effect of the antioxidant Vitamin E on the AD-like phenotype when give

101 ptotic cell death that were prevented by the antioxidant vitamin E or mitochondrial catalase overexpr

102 Pretreatment with the antioxidant vitamin E or the PAF-R antagonists WEB 2086

103 Pretreatment with the antioxidant vitamin E significantly protected against UV

104 Men taking antioxidant vitamin E supplements have increased prostat

105 , as well as the inability of the lipophilic antioxidant vitamin E to abrogate MAPK phosphorylation,

106 mice receiving a diet supplemented with the antioxidant vitamin E, higher levels of RPE bisretinoid

107 ignature of final structure is erased via an antioxidant vitamin E, subsequent to guided self-assembl

108 gnificantly reduced by pretreatment with the antioxidant vitamin E.

109 ydropyridine Ca2+ blocker amlodipine and the antioxidant vitamin E.

110 n = 7) diet, HC diet supplemented daily with antioxidant vitamins E (100 IU/kg) and C (1000 mg; HC+vi

111 The effects of antioxidant vitamins E and C added to the PC diet were a

112 viduals who regularly use supplements of the antioxidant vitamins E and C have a lower risk of ALS th

113 x vivo results, we depleted mice of a second antioxidant, vitamin E, which is normally absent under e

114 ination, on the concentrations of lipophilic antioxidants (vitamin E and gamma-oryzanol), soluble (in

115 here and vacuum skin packaging - MAPVSP) and antioxidants (vitamin E enriched beef).

116 n of protein kinase C (H-7), the addition of antioxidants (vitamin E or superoxide dismutase), or a p

117 Various antioxidants (vitamin E, vitamin C, beta-carotene, catal

118 We separately analyzed trials using antioxidants (vitamins E and C, beta-carotene, or seleni

119 gle-nucleotide polymorphisms for circulating antioxidants (vitamins E and C, retinol, B-carotene, and

120 of cats with a high dose combination of the antioxidants vitamin E (200 I.U./day) and selenium (50 m

121 The effects of PM were compared with the antioxidants vitamin E (VE) and R-alpha-lipoic acid (LA)

122 ists CV-6209 and WEB 2086, as well as by the antioxidants vitamin E and 1,1,3,3-tetramethyl-2-thioure

123 The antioxidants vitamin E and N-acetylcysteine (NAC) blocke

124 Interestingly, the antioxidants vitamin E and N-acetylcysteine abolished bo

125 The antioxidants vitamin E, vitamin C, or trolox prevented t

126 Mass spectrometry revealed that the antioxidants vitamins E and C, butylated hydroxytoluene,

127 A combination of antioxidants, vitamin E, and butylated hydroxytoluene (B

128 In particular, n-3 fatty acids, antioxidant vitamins (especially vitamins E and C), foli

129 revention trial of the benefits and risks of antioxidant vitamins, folic acid, vitamin B6, and vitami

130 Consumption of antioxidant vitamins from diet or supplement sources did

131 The timing of administration of antioxidant vitamins has variable effects on markers of

132 tioxidant hypothesis of atherosclerosis than antioxidant vitamins have done.

133 In contrast, clinical trials with antioxidant vitamins have not produced consistent result

134 Lipid-lowering therapy and antioxidant vitamins improve endothelium-dependent vasod

135 n regimen nor the combination of statins and antioxidant vitamins improved flow-mediated vasodilation

136 Combined neonatal dexamethasone with antioxidant vitamins improved postnatal survival, restor

137 y intakes of magnesium, vitamin C, and other antioxidant vitamins in a general population cohort in N

138 n in vivo, and its modulation by aspirin and antioxidant vitamins in chronic cigarette smokers.

139 e results suggest that beneficial effects of antioxidant vitamins in coronary artery disease are rela

140 Few data are available on the importance of antioxidant vitamins in earlier stages of atherogenesis.

141 tive way to obtain an optimal composition of antioxidant vitamins in foodstuffs such as salads.

142 ions of lipid hydroperoxides and liposoluble antioxidant vitamins in healthy volunteers.

143 The role of dietary antioxidant vitamins in preventing coronary heart diseas

144 The use of antioxidant vitamins in this setting must be questioned.

145 To assess the role of ascorbate (AA), an antioxidant vitamin, in modulating striatal activity, si

146 To assess the relationship between antioxidant vitamin intake and all-cause mortality in ol

147 The association between dietary antioxidant vitamin intake and the risk of breast cancer

148 oth free radical and oxidant exposure, while antioxidant vitamin intake is positively related to lung

149 e association of self-selected supplementary antioxidant vitamin intake on the rate of progression of

150 y symptoms in adults that are independent of antioxidant vitamin intake, but little is known about fi

151 re lower in children with inadequate dietary antioxidant vitamin intake.

152 We examined the association between antioxidant vitamin intakes and death from stroke.

153 Combined neonatal treatment with antioxidant vitamins is an effective intervention.

154 wever, epidemiological evidence to implicate antioxidant vitamins is weak, and data on the role of fl

155 iatum contains a high level of ascorbate, an antioxidant vitamin known to play a key role in behavior

156 Socioeconomic factors and antioxidant vitamin levels accounted for an additional 7

157 with claims that consider vitamin A to be an antioxidant vitamin, like vitamins C and E.

158 Our findings support the hypothesis that antioxidant vitamins may play a role in respiratory heal

159 Antioxidant vitamins may play a role in the prevention o

160 tamin C, or supplementation of the diet with antioxidant vitamins, may inhibit oxidation and the proc

161 deficit in the release of ascorbate (AA), an antioxidant vitamin, occurs in the striatum of R6/2 mice

162 termine the long-term effects of statins and antioxidant vitamins on flow-mediated vasodilation of th

163 Reliable data on whether antioxidant vitamins or trace minerals decrease the deve

164 ncer risk, nor was intake of macronutrients, antioxidant vitamins, or other micronutrients.

165 d 9 mg beta-carotene), B-group vitamins with antioxidant vitamins, or placebo.

166 Antioxidant vitamin pretreatment altered neither respons

167 n in injured vessels and to demonstrate that antioxidant vitamins reduce O2- production.

168 A combination of antioxidant vitamins reduced the myocardial oxidative st

169 Antioxidant vitamins reduced tissue oxidative stress in

170 levels (p < 0.001); however, neither statin, antioxidant vitamin regimen nor the combination of stati

171 When they analyzed all of these antioxidant vitamins simultaneously, they observed the s

172 tigated the cross-sectional relation between antioxidant vitamin status and carotid atherosclerosis i

173 A high antioxidant vitamin status may help to prevent the initi

174 Antioxidant vitamin status was assessed by measuring fas

175 authors examined whether self-selection for antioxidant vitamin supplement use affects the incidence

176 assessed the effects of B-group vitamins and antioxidant vitamin supplementation on homocysteine conc

177 Large trials of antioxidant vitamin supplementation to prevent cancer su

178 uggest an increase in overall mortality with antioxidant vitamin supplementation, at least in populat

179 oxidant system, through n-3 fatty acids plus antioxidant vitamin supplementation, could reduce the in

180 ies have demonstrated a beneficial impact of antioxidant vitamin supplements on lipid peroxidation, t

181 women with coronary disease, neither HRT nor antioxidant vitamin supplements provide cardiovascular b

182 Vitamin C is an antioxidant vitamin that has been hypothesized to antago

183 his correlates with depletion of liposoluble antioxidant vitamins that are consumed during lipid pero

184 antihypertensives, aspirin, B-vitamins, and antioxidant vitamins) that are of proven or suspected be

185 Antioxidant vitamin therapy improved FMD of the brachial

186 rpose of this study was to determine whether antioxidant vitamin therapy improves endothelial functio

187 ttes and switching to nicotine patches or by antioxidant vitamin therapy.

188 We studied simvastatin-niacin and antioxidant-vitamin therapy, alone and together, for car

189 We administered antioxidant vitamins to rabbits with pacing-induced card

190 Using antioxidant vitamins to reset ROS levels in Nrf2(-/-) iD

191 observed associations between mortality and antioxidant vitamin use after adjustment for confounders

192 zed, placebo-controlled studies suggest that antioxidant vitamins (vitamin E, vitamin C, beta-caroten

193 The relationship between intake of antioxidant vitamins (vitamins A, C, D, and E) and indiv

194 eopenia and fractures; and low levels of the antioxidant vitamins (vitamins A, E, and C) may increase

195 -adjusted intake of fruit and vegetables and antioxidant vitamins was estimated using a semi-quantita

196 When all of these antioxidant vitamins were analyzed simultaneously in a m

197 sue endogenous oxygen radical scavengers and antioxidant vitamins were depleted and LDL oxidizability

198 ions of lipid hydroperoxides and liposoluble antioxidant vitamins were determined by using the FOX 2