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1 if parathyroid hormone is not followed by an antiresorptive agent.
2 y, whose appearance could be prevented by an antiresorptive agent.
3 a T score of <=-2.5 for BMD), followed by an antiresorptive agent.
4 in the presence of zoledronic acid, a potent antiresorptive agent.
5 coincident with a decline in the use of the antiresorptive agents.
6 effect in patients undergoing treatment with antiresorptive agents.
7 pounds for the development of a new class of antiresorptive agents.
8 impact of a locally or systemically applied antiresorptive agent (alendronate) on simvastatin-induce
13 10-year absolute fracture risk (eg, >=20%), antiresorptive agents (bisphosphonates or, if contraindi
15 se findings suggest that, among women taking antiresorptive agents, greater increases in BMD are asso
16 ation, vitamin K therapy, calcimimetics, and antiresorptive agents had conflicting or inconclusive ou
19 e effects of these pharmacologically diverse antiresorptive agents on gene expression in bone has not
21 thyroid hormone in the future, the choice of antiresorptive agent should be carefully considered.
24 ciated bone disease are available, including antiresorptive agents such as denosumab and bisphosphona
25 nstay of treatment for osteoporosis has been antiresorptive agents (such as bisphosphonates), which h
26 be used either alone or in combination with antiresorptive agents to build new bone and reduce fract
27 rapy approaches with parathyroid hormone and antiresorptive agents to optimize efficacy outcomes.
28 reatment (parathyroid hormone followed by an antiresorptive agent), which aims to maintain or build o
30 e that combining parathyroid hormone with an antiresorptive agent will enhance its effect on bone min
31 is also possible that the combination of an antiresorptive agent with an anabolic agent could be mor
32 fonamides like 55 form a new class of potent antiresorptive agents with possible therapeutic use in d