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1 denoted treatment with none or 1 intravenous antiseizure drug.
2 noted treatment with more than 1 intravenous antiseizure drug.
3 ynchrony is modulated by epileptogenesis and antiseizure drugs.
4 sy contributes to seizures and resistance to antiseizure drugs.
5 st in 35% of patients despite optimal use of antiseizure drugs.
6 cant differences in plasma concentrations of antiseizure drugs.
7 s fail to become seizure-free in response to antiseizure drugs.
8 /ED50) that compared favorably with clinical antiseizure drugs.
11 tives and antipsychotic, antidepressant, and antiseizure drugs as well as drugs of abuse, such as coc
13 Despite the availability of approximately 30 antiseizure drugs (ASDs), epilepsy remains a major unmet
15 s indicated by the clinical finding that the antiseizure drug carbamazepine (CBZ) relieves pain becau
17 euronal excitability and highlight potential antiseizure drug development strategies based on the Na(
20 eractions with targets reported for marketed antiseizure drugs, indicative of a novel and unprecedent
22 denoted treatment with 1 or more intravenous antiseizure drug, more than 1 intravenous anesthetic, an
23 etam; PET imaging tracer UCB-J; experimental antiseizure drug padsevonil; and allosteric modulator UC
24 acterized by seizures that respond poorly to antiseizure drugs, psychomotor delay, and cognitive and
28 CANCE STATEMENT PMP is a regulatory approved antiseizure drug used for refractory partial-onset and g
32 les include neuroinhibitory medications like antiseizure drugs, which are used against epilepsy and n
33 30% of focal epilepsies remain resistant to antiseizure drugs, with effective drug development imped