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1 vironment and are generally considered to be antitumorigenic.
2 chidonic acid possibly shifts from producing antitumorigenic 15-LOX-1 and 15-LOX-2 products to produc
3                                          The antitumorigenic action of GH-RH antagonists could be par
4 ever, the molecular mechanism underlying the antitumorigenic action of p100 remains poorly understood
5 vation of BAI1 expression underlies EPZ-6438 antitumorigenic action, and provide preclinical proof-of
6 ay a wider role in the antiproliferative and antitumorigenic actions of IGFBP-3.
7 ignant growth of cultured ECs, overcomes the antitumorigenic actions of RA, and is selectively expres
8 N response is required for effective in vivo antitumorigenic actions of the DNMTi 5-azacytidine (AZA)
9 eptor (GPCR) antagonists that have potential antitumorigenic activities, although the mechanism(s) ar
10  clues for explaining their proapoptotic and antitumorigenic activities.
11 nt of ginger, exhibits anti-inflammatory and antitumorigenic activities.
12 at the compound, BMH-21, has wide and potent antitumorigenic activity across NCI60 cancer cell lines
13       Our data suggest that SLPI may possess antitumorigenic activity by virtue of its ability to int
14  to and repress the Smad proteins, while the antitumorigenic activity can be mediated by both Smad-de
15                                 However, its antitumorigenic activity has not been elucidated in vivo
16  this study provides a new mechanism for the antitumorigenic activity of AHPN.
17 ing a role for this protein in mediating the antitumorigenic activity of DIM-C-pPhCF3 in bladder canc
18                                          The antitumorigenic activity of nonsteroidal anti-inflammato
19 on variants of MYC and demonstrated that the antitumorigenic activity of SMAPs depends on MYC degrada
20             Similarly, lenalidomide exhibits antitumorigenic activity paralleled by increased miR-181
21 (NAG-(Tg+)) were characterized, and then the antitumorigenic activity was evaluated with 2 colorectal
22 ptor gamma (PPARgamma) agonists that exhibit antitumorigenic activity.
23 ompounds, particularly as anti-infective and antitumorigenic agents, have led to a large body of publ
24 iver X receptor agonists may have utility as antitumorigenic agents.
25  gastrointestinal disorders, as it possesses antitumorigenic, analgesic, anti-inflammatory and antiox
26 etic oleanolic acid derivative that displays antitumorigenic and anti-inflammatory activities, and we
27 cture and as such may be responsible for the antitumorigenic and antiangiogenic activities of ACTIBIN
28 e RNA to 3' mononucleotides and also possess antitumorigenic and antiangiogenic activities.
29    These results establish angiostatin as an antitumorigenic and antiangiogenic agent through a mecha
30 sent report, we review recent studies on the antitumorigenic and antiangiogenic effects of exogenousl
31 finity chromatography and found to have both antitumorigenic and antiangiogenic properties.
32           Thus, Ac-PHSCN-NH2 may be a potent antitumorigenic and antimetastatic agent for postsurgica
33 umorigenic protein in vitro, induced by many antitumorigenic and chemopreventive drugs including cycl
34 hese actions of ursolic acid may mediate its antitumorigenic and chemosensitizing effects.
35 myeloid cells as well as activators of their antitumorigenic and immunostimulating functions.
36                          In vivo, MV-Edm was antitumorigenic and inhibited the establishment of myelo
37 -activated gene 1 (NAG-1), a protein with an antitumorigenic and proapoptotic activity that could in
38      These data demonstrate that NAG-1 is an antitumorigenic and proapoptotic protein, and its regula
39 eduction of TR1 levels in malignant cells is antitumorigenic and suggest that the enzyme is a prime t
40 s report, we show that the expression of the antitumorigenic and/or pro-apoptotic gene NAG-1 (nonster
41 8S-LOX plays a role as a prodifferentiating, antitumorigenic, and tumor suppressing gene in mouse ski
42 ctor (PEDF) is a multifunctional serpin with antitumorigenic, antimetastatic, and differentiating act
43 mor growth was 50 microg/kg/day, and TAM was antitumorigenic at a dose of 100 microg/kg/day.
44  exercise mediating changes that promote the antitumorigenic characteristics of the gut microbiota.
45 4) as contextually active protumorigenic and antitumorigenic contributors in this communication ecosy
46 sema3C may be further developed into a novel antitumorigenic drug.
47 ct on PI3K, LY294002 has been shown to exert antitumorigenic effect in vivo and in vitro.
48   Furthermore, TREM1 inhibition enhanced the antitumorigenic effect of anti-PD-1 treatment, in part,
49                                          The antitumorigenic effect of downregulation of HSF1 was ass
50                 Ormeloxifene potentiated the antitumorigenic effect of gemcitabine by 75% in PDAC xen
51 mature myeloid cells, indicating a potential antitumorigenic effect of histamine.
52 pathway may be, in part, responsible for the antitumorigenic effect of LY294002 in human colorectal c
53 ng MAT1A induction significantly reduced the antitumorigenic effect of miR-495 siRNA, whereas maintai
54               These results suggest that the antitumorigenic effect of n-3 PUFA may be mediated, in p
55                  At the molecular level, the antitumorigenic effect of SOX30 is mediated by directly
56 ponse and p53 activation, which mediate this antitumorigenic effect.
57 lecular mechanisms by which they exert their antitumorigenic effects are unknown.
58  of sulindac that is believed to mediate its antitumorigenic effects by inducing apoptosis.
59                             These apparently antitumorigenic effects can be reversed by a dominant-ne
60 sults demonstrate that miR-34a exerts potent antitumorigenic effects in vitro and in vivo and suggest
61 f genes that may be critical for the in vivo antitumorigenic effects of AhR agonists.
62                                          The antitumorigenic effects of AR-R17779 were linked to an a
63 , indicating the relative specificity of the antitumorigenic effects of AS gastrin RNA expression.
64       Although the mechanisms underlying the antitumorigenic effects of exercise remain to be defined
65                                          The antitumorigenic effects of FJ9 were pronounced, includin
66 or promoting lung tumorigenesis and that the antitumorigenic effects of PPARgamma are mediated in par
67 associated phenotypes and by reinforcing the antitumorigenic effects of the SASP.
68 ions may contribute to the observed combined antitumorigenic effects of these compounds.
69 ent for innate immune cells in mediating the antitumorigenic effects of USP6.
70 wever, the precise mechanisms underlying its antitumorigenic effects remain unclear.
71  AVE 0991 has orexigenic, anticachectic, and antitumorigenic effects, identifying it as a promising a
72  our results suggest that NSAIDs exert their antitumorigenic effects, in part, via interference with
73 ct on cellular processes, and their pro- and antitumorigenic effects.
74 ce more cancer due to lower CX3CL1-dependent antitumorigenic effects.
75 ic microenvironments, exerting both pro- and antitumorigenic effects.
76 rimary mechanism by which NSAIDs exert their antitumorigenic effects.
77                                 IFNgamma has antitumorigenic effects; however, the findings of IFNgam
78 C, can be used as a biomarker to predict the antitumorigenic efficacy of patritumab and whether the d
79 ronment is complex, containing both pro- and antitumorigenic elements, and remains to be fully charac
80 ubsets as a key instructing factor directing antitumorigenic eosinophil activities.
81 ulting in the proteolytic destruction of the antitumorigenic factor thrombospondin-1 (Tsp-1).
82 inhibition neither consistently enhanced all antitumorigenic factors nor suppressed all protumorigeni
83  functions, including both protumorignic and antitumorigenic features.
84  the EPHB3 tumor suppressor and argue for an antitumorigenic function of Notch signaling in advanced
85                                         PL's antitumorigenic function was causally linked to its Stat
86  the proteolytic cleavage-dependent pro- and antitumorigenic functions of ANGPTL4 and identifies and
87 interactions contribute to both the pro- and antitumorigenic functions of JAM.
88 s likely unwise given the protumorigenic and antitumorigenic functions of various family members.
89 hemokines associated with immunostimulatory, antitumorigenic functions, including CXCL10 and CCL5.
90 r differentiation state, can exhibit pro- or antitumorigenic functions.
91 tion, with it being desirable to retain some antitumorigenic functions.
92 nd examine their diverse pro-tumorigenic and antitumorigenic functions.
93 lumes and neoangiogenesis and an increase in antitumorigenic GAM infiltrate.
94  types for reprogramming MDSCs and TAMs into antitumorigenic immune cells using a drug that would oth
95 veloping malignant lesions are eliminated by antitumorigenic immune cells.
96 e effects were associated with a shift to an antitumorigenic immune microenvironment.
97 that it could be proleukemic in children and antitumorigenic in adults.
98 hway can be both proleukemic in children and antitumorigenic in adults.
99 wever, this phosphatase was also shown to be antitumorigenic in HCC.
100 eroidal anti-inflammatory drugs (NSAIDs) are antitumorigenic in humans as well as in animal models of
101 hough data indicate that RARgamma agonism is antitumorigenic in oral cavity squamous cell carcinoma (
102 tic target for future studies to enhance the antitumorigenic inflammatory response in HNSCC and other
103 tive sphingolipid ceramide has emerged as an antitumorigenic lipid, and treatment with short-chain C6
104  myeloid leukocyte responses and compromises antitumorigenic lymphocyte activity, ultimately supporti
105 eshaped the immunosuppressive TME, promoting antitumorigenic M1 polarized macrophages and reducing my
106 st in response to DNA damage is an important antitumorigenic mechanism.
107 NA binding proteins, to rid themselves of an antitumorigenic miRNA.
108  pro-invasive MMP1 gene and upregulating the antitumorigenic MMP8 gene, resulting in reduced invasive
109                       Preclinical studies on antitumorigenic or therapeutic introduction of FHIT were
110 trate, and Nrp1-depleted BMDM adopted a more antitumorigenic phenotype relative to wild-type GAMs wit
111 differentiation of the macrophages toward an antitumorigenic phenotype, depletion of tumor-promoting
112        The reactions exhibited both pro- and antitumorigenic potential and primarily corresponded to
113     These observations led us to examine the antitumorigenic potential of miR-1 in lung cancer cells.
114 with the normal microenvironment to overcome antitumorigenic pressures.
115 gle key splicing event can manifest powerful antitumorigenic properties and validating endogenous spl
116 h has been postulated to carry both pro- and antitumorigenic properties depending on tissue context.
117 tion, whereas SRC overexpression rescued the antitumorigenic properties mediated by miR-34a.
118 GR-1 may provide a novel explanation for the antitumorigenic properties of LY294002 in human colorect
119 g a novel mechanism to explain, in part, the antitumorigenic properties of some NSAIDs.
120 pled receptor with potent antiangiogenic and antitumorigenic properties that is mutated in certain ca
121 kinase inhibitor that has antiangiogenic and antitumorigenic properties with potential efficacy for t
122 umor-derived cytokines that suppress initial antitumorigenic properties, causing them to support tumo
123 by which the TME trains eosinophils to adopt antitumorigenic properties, which may lead to the develo
124 gnaling and exhibits both antimetastatic and antitumorigenic properties.
125 ral MMPs including MMP-9 exert both pro- and antitumorigenic properties.
126 astatic, neuronotrophic, antiangiogenic, and antitumorigenic properties.
127 AG-1 may provide a novel explanation for the antitumorigenic property of TGZ.
128 ne (NAG-1) was identified as a proapoptotic, antitumorigenic protein in vitro, induced by many antitu
129 lts are consistent with a more than additive antitumorigenic response for the low dose group (25 + 25
130                         These data reveal an antitumorigenic role for 5-LO products in the microenvir
131 ormation in Apc(Min)(/+) mice, suggesting an antitumorigenic role for PAF in settings characterized b
132           Thus, in these cells, Ski plays an antitumorigenic role.
133    Thus, SnoN plays both pro-tumorigenic and antitumorigenic roles at different stages of mammalian m
134  light on this duality of protumorigenic and antitumorigenic roles of DYRK2.
135  mechanistic explanation for the paradoxical antitumorigenic roles of E+P in breast cancer by showing
136  The decorin interactome commands a powerful antitumorigenic signal by potently repressing and attenu
137 sues places several stromal cell types in an antitumorigenic state, directly inhibiting EC proliferat
138 tic regulation of the expression of pro- and antitumorigenic target genes.
139 f the TGFbeta pathway and its change from an antitumorigenic to a protumorigenic pathway.
140 ng leads to functional diversification, from antitumorigenic to immunosuppressive phenotypes.
141     Transition of macrophage phenotype, from antitumorigenic to protumorigenic, occurs before tumorig
142             The (-)-deuterated enantiomer is antitumorigenic, whereas the (+)-deuterated enantiomer h

 
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