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1 e principally recognized as key mediators of antiviral immunity.
2 l infections and acts as a potent trigger of antiviral immunity.
3 e functional integration of SFKs into innate antiviral immunity.
4 which are present in proteins implicated in antiviral immunity.
5 Type III IFNs are important mediators of antiviral immunity.
6 we analyzed the effects of RG-101 therapy on antiviral immunity.
7 pe I IFNs are key cytokines mediating innate antiviral immunity.
8 o have critical immune functions, but not in antiviral immunity.
9 he induction of T cell responses and mucosal antiviral immunity.
10 RNAs are at the vanguard of cell-autonomous antiviral immunity.
11 l dysbiosis on infant CD8(+) T cell-mediated antiviral immunity.
12 ignaling protein (MAVS) that is critical for antiviral immunity.
13 FN) signaling by producing cGAMP to initiate antiviral immunity.
14 ensing of nucleic acids lies at the heart of antiviral immunity.
15 n mice have demonstrated a role for Isg15 in antiviral immunity.
16 e virus in HIV controllers despite effective antiviral immunity.
17 s are critical mediators of mammalian innate antiviral immunity.
18 als in epithelia, thereby inducing localized antiviral immunity.
19 TLR7 recognize viral ssRNA motifs and induce antiviral immunity.
20 motes type IIFNsignaling as well as cellular antiviral immunity.
21 RNA virus infection to initiate and modulate antiviral immunity.
22 portant role for RIPK3-mediated apoptosis in antiviral immunity.
23 in dNK can provide both for NK tolerance and antiviral immunity.
24 to assess whether pollen may interfere with antiviral immunity.
25 is critical for innate immune activation and antiviral immunity.
26 combination therapies based on modulation of antiviral immunity.
27 t cells but not in cells unable to establish antiviral immunity.
28 T cell expansion was the limiting factor in antiviral immunity.
29 s-encoded immune evasion mechanisms and host antiviral immunity.
30 aptation typical of host factors involved in antiviral immunity.
31 persist and display functional hallmarks of antiviral immunity.
32 cumvent respiratory epithelial cell-specific antiviral immunity.
33 poptotic caspases regulate the activation of antiviral immunity.
34 s have recently emerged as key regulators of antiviral immunity.
35 ic cells may provide a means of potentiating antiviral immunity.
36 ype I IFN response to RNA and DNA viruses in antiviral immunity.
37 ype I interferon system is integral to human antiviral immunity.
38 ongly influenced by the nature of the host's antiviral immunity.
39 gistic roles in CD4+ and CD8+ T cells during antiviral immunity.
40 on of type I interferons (IFN-I) involved in antiviral immunity.
41 der in controlling and regulating the innate antiviral immunity.
42 tivated macrophages, could partially restore antiviral immunity.
43 f mechanisms to evade interferon (IFN)-based antiviral immunity.
44 ic cells (DCs), are critically important for antiviral immunity.
45 type I IFNs (IFN-Is), important mediators of antiviral immunity.
46 host and viral factors that impair effective antiviral immunity.
47 ection as a consequence of a failure in host antiviral immunity.
48 NK cells play a critical role in mediating antiviral immunity.
49 cross-species signals in the meal to trigger antiviral immunity.
50 g CD4(+) T cells with cytotoxic potential in antiviral immunity.
51 rus replication and establish cell-intrinsic antiviral immunity.
52 nown about the effects of WASP deficiency on antiviral immunity.
53 ecific T-cell response and may contribute to antiviral immunity.
54 and considerations central to studying plant antiviral immunity.
55 immune T cell responses without compromising antiviral immunity.
56 e also been shown to play important roles in antiviral immunity.
57 dendritic cells (pDC) are key regulators of antiviral immunity.
58 survival and proliferation to chemotaxis and antiviral immunity.
59 an NFkappaB family transcription factor, in antiviral immunity.
60 tion that is essential for the generation of antiviral immunity.
61 une evasion, providing targets for restoring antiviral immunity.
62 ng that their regulation by miR-155 promotes antiviral immunity.
63 nt of a RIG-I translocon required for innate antiviral immunity.
64 implicate VCP as an important host factor in antiviral immunity.
65 terferon (IFN)-regulated gene expression and antiviral immunity.
66 Kp44-NKp44L signaling pathway contributes to antiviral immunity.
67 ncovers a critical role for DR3 in mediating antiviral immunity.
68 ctional consequences of this recognition for antiviral immunity.
69 and reveal a novel role for TRIM56 in innate antiviral immunity.
70 ce for CD4(+) T cells as direct effectors in antiviral immunity.
71 a critical role for TAPE in linking RLRs to antiviral immunity.
72 subsets critical in the generation of strong antiviral immunity.
73 ng its abundance as key regulators of innate antiviral immunity.
74 cytokine production and in the generation of antiviral immunity.
75 ced capacity to activate innate and acquired antiviral immunity.
76 tute a crucial platform for the induction of antiviral immunity.
77 critical functions in autoimmune disease and antiviral immunity.
78 tides and plays a key role in anticancer and antiviral immunity.
79 sistent with this gene playing a key role in antiviral immunity.
80 present genetic approaches to enhance their antiviral immunity.
81 CC), which might be explained by its role in antiviral immunity.
82 suggesting that BMP/SMAD activity influences antiviral immunity.
83 t viral RNAi suppressors may completely mask antiviral immunity.
84 via a stress-induced SUMO switch to augment antiviral immunity.
85 uced viral particles yet strongly stimulated antiviral immunity.
86 echanisms for cellular dsRNA homeostasis and antiviral immunity.
87 NV impacting some, but not all, mediators of antiviral immunity.
88 d to viral delivery, spread, resistance, and antiviral immunity.
89 stimulated by type I IFNs to further enhance antiviral immunity.
90 iral strategies to avoid, evade, or suppress antiviral immunity.
91 ral errors in protein translation to provide antiviral immunity.
92 3 and thus allows timely induction of innate antiviral immunity.
93 ates with mtDNA to evade the STING-dependent antiviral immunity.
94 a pathway of T cell migration that sustains antiviral immunity.
95 e, NF-kappaB, and JAK-STAT pathways underlie antiviral immunity.
96 ferentiation that can be targeted to improve antiviral immunity.
97 replication and acts as a potent trigger of antiviral immunity.
98 so promotes transgenerational inheritance of antiviral immunity.
99 FN-alpha, suggesting a potential weakness in antiviral immunity.
100 es exacerbates Tfr cell responses to subvert antiviral immunity.
101 rotein translation, autophagy, apoptosis and antiviral immunity.
102 gnalosome required for RIG-I-mediated innate antiviral immunity.
103 rtholog, and this transport is important for antiviral immunity.
104 ggesting that NS1 evades IFN-lambda-mediated antiviral immunity.
105 Because cure is accompanied by recovery of antiviral immunity, a combination of direct-acting antiv
106 ay provide mechanistic insights into hepatic antiviral immunity, a prerequisite for the development o
108 that NIK1, which positively regulates plant antiviral immunity, acts as an important negative regula
109 ortance of avoiding GVHD when reconstructing antiviral immunity after BMT, and highlight the mechanis
110 ed CD8(+) regulatory T cells (Treg cells) to antiviral immunity after infection by lymphocytic chorio
111 operty by enhancing both innate and adaptive antiviral immunity against a variety of viral pathogens,
115 nosuppressive medications result in impaired antiviral immunity and a propensity for cytomegalovirus
116 of recent progress in understanding mosquito antiviral immunity and advances in the strategies by whi
117 namic adaptations to infection may reinforce antiviral immunity and at the same time serve to limit p
122 acerbation; however, only anti-IL-33 boosted antiviral immunity and decreased viral replication.
123 on of IL-33 as a potent suppressor of innate antiviral immunity and demonstrate that IL-33 contribute
124 studies reveal a novel function for CFTR in antiviral immunity and demonstrate that the DeltaF508 mu
125 fies NFkappaB2 as a target for IKKepsilon in antiviral immunity and describes, for the first time, a
126 and maintaining a protective balance between antiviral immunity and excessive inflammation within the
127 st widespread 2'3'-cGAMP signaling in insect antiviral immunity and explain how a family of cGAS-STIN
130 ll-like receptor (TLR) activation stimulates antiviral immunity and has been shown to induce HIV from
131 Interferon (IFN)-Is are crucial mediators of antiviral immunity and homeostatic immune system regulat
133 toid dendritic cells (pDCs) are important in antiviral immunity and in maintaining tolerance to inert
134 d with plasma levels of soluble mediators of antiviral immunity and inflammation such as IP-10, TNF,
136 findings may provide a means of potentiating antiviral immunity and leading to novel vaccines for PRR
137 Thus, symbiotic intestinal bacteria modulate antiviral immunity and levels of circulating alphaviruse
138 Plasmacytoid dendritic cells (pDCs) initiate antiviral immunity and might determine outcomes of HBV i
139 17 in persistent viral infection may promote antiviral immunity and prevent progression to cancer.
141 that have been demonstrated to modulate both antiviral immunity and regulate direct host-virus intera
142 llenge the view that tadpoles have defective antiviral immunity and suggest, rather, that their antiv
143 s a conserved role of selective autophagy in antiviral immunity and suggests the evolvement of viral
144 dapted poxvirus MCV can so effectively evade antiviral immunity and suppress inflammation to persist
145 t into the molecular mechanisms that control antiviral immunity and the development of autoimmunity.
146 of the immune signaling cascades controlling antiviral immunity and the development of immune memory.
149 in the context of other links between innate antiviral immunity and type I interferon mediated diseas
150 ssembly of large MAVS aggregates and healthy antiviral immunity and underlay nutrient-triggered mitoc
151 Helminth coinfection resulted in impaired antiviral immunity and was associated with changes in th
152 r the age of patients with CHB affects their antiviral immunity and whether children and young adults
153 However, little is known about amphibian antiviral immunity and, specifically, type I interferons
154 diversifies NK cell effector function during antiviral immunity, and how avidity selection might serv
155 e invertebrate model for viral infection and antiviral immunity, and is a focus for studies of insect
156 ection of mice with gammaHV68 elicits robust antiviral immunity, and long-term protection from gammaH
157 g viral replication are the main trigger for antiviral immunity, and mutations that disrupt nucleic a
158 de inhibitors that block cGAS-STING-mediated antiviral immunity, and that modulation of this pathway
159 gnificant up-regulation of genes involved in antiviral-immunity, and a down-regulation of genes invol
161 RNA persistence and only marginal changes in antiviral immunity, arthritic disease was substantially
162 LRs), is essential for STING-mediated innate antiviral immunity as well as pro-protozoal responses.
163 her tissue-resident lymphocytes confer early antiviral immunity at local sites of primary infection p
164 cytic cell activation statuses interact with antiviral immunity, because it directly infects subsets
165 tion of DNA is crucial for the initiation of antiviral immunity but can also cause autoimmunity in th
166 Type I interferon (IFN-I) provides effective antiviral immunity but can exacerbate harmful inflammato
167 a and IFN-beta are the central regulators of antiviral immunity but little is known about their roles
168 nflammatory cytokines that are essential for antiviral immunity but whose overexpression is associate
169 ISG15, therefore, is not only redundant for antiviral immunity, but is a key negative regulator of I
170 killer (NK) cells play an essential role in antiviral immunity, but knowledge of their function in s
171 at human ISGylation is largely redundant for antiviral immunity, but that ISG15 plays an essential ro
173 e I interferons (IFN-Is) are fundamental for antiviral immunity, but their role in bacterial infectio
174 s of foreign nucleic acids are essential for antiviral immunity, but these same sensors can cause aut
176 erve as a substrate for ISGylation-dependent antiviral immunity, but to ensure USP18-dependent regula
181 oy different strategies to elicit functional antiviral immunity by both antibody isotypes in the muco
182 llular pathway that can contribute to innate antiviral immunity by delivering viruses to lysosomes fo
183 Here we report that EV-D68 inhibits innate antiviral immunity by downregulation of interferon regul
185 nstrated role in shaping innate and adaptive antiviral immunity by inducing the expression of IFN-sti
187 4-3-3varepsilon serves a crucial function in antiviral immunity by mediating the cytosol-to-mitochond
188 I) molecules play a central role in adaptive antiviral immunity by presenting viral peptides to CD4(+
189 (DRs) of the TNFR superfamily contribute to antiviral immunity by promoting apoptosis and regulating
190 ster more rapid reconstitution of protective antiviral immunity by reducing graft-vs-host directed al
191 Because HLA-E plays an important role in antiviral immunity by regulating natural killer and CD8(
193 tive regulator of the mitochondrial-mediated antiviral immunity, by interacting with mitochondrial an
194 These results establish that intracellular antiviral immunity can be redirected against host-origin
197 the mechanisms by which virus infection and antiviral immunity contribute to the development of auto
198 was associated with IFN-gamma signaling and antiviral immunity controlled by T cells (T(H)1 and CD8(
199 necroptosis has been shown to contribute to antiviral immunity, death-independent roles for RIPK3 in
201 type I interferons (IFN-I) are essential to antiviral immunity derives from studies on animal models
202 limits seroconversion, and enhances cellular antiviral immunity despite persistence of infection in l
207 d dendritic cells (pDCs), prominent cells of antiviral immunity, exhibit proinflammatory or tolerogen
210 terference (RNAi) plays an important role in antiviral immunity, gene regulation and protection from
211 against Candida, but mechanisms of impaired antiviral immunity have not previously been examined.
212 Type I interferons (IFN-I) are critical for antiviral immunity; however, chronic IFN-I signaling is
213 using several immunomodulators for boosting antiviral immunity, immunotherapy that is able to induce
214 l CD8(+) T cell-mediated immunity, including antiviral immunity, implying that selective pharmacologi
215 these TCR-mediated processes is limiting for antiviral immunity in a mouse strain with reduced expres
219 strate that NK cells can negatively regulate antiviral immunity in chronic HBV infection and illustra
221 observed that latent-infected cells trigger antiviral immunity in dendritic cells (DCs) through sele
224 dysfunction as a potential cause of impaired antiviral immunity in graft-versus-host disease (GVHD).
228 ore, transcriptional pausing is critical for antiviral immunity in insects because NELF and P-TEFb ar
231 athway and has emerged as a key mechanism of antiviral immunity in metazoans, including the selective
233 ized mechanism whereby IL-36 promotes innate antiviral immunity in mouse and human models of herpes s
236 novel insights into mechanisms that preserve antiviral immunity in patients undergoing chemotherapy a
237 findings have implications for understanding antiviral immunity in patients with T cell deficiencies.
238 ting Tregs may be necessary to confer robust antiviral immunity in the context of mAb-based therapy.
241 support a novel axis of type I IFN-dependent antiviral immunity in the virus-infected brain that is d
242 CNS tissues, we examined the development of antiviral immunity in wild-type (WT) and T-bet knockout
243 ncing effect of delayed NK cell depletion on antiviral immunity, in contrast to early NK cell depleti
244 suffer from elevated EBV load and activated antiviral immunity, in particular in skin lesions that a
245 utophagy plays a paramount role in mammalian antiviral immunity including direct targeting of viruses
246 The ability of pollen to suppress innate antiviral immunity, independent of allergy, suggests tha
247 essential immunological processes including antiviral immunity, inflammasome activation and antibody
252 type I IFN system is critical for amplifying antiviral immunity, it has been shown to play a homeosta
256 The species-specific gain-of-function in antiviral immunity observed in ISG15 deficiency is expla
257 nt of class Ib molecules for development and antiviral immunity of a mammalian iNKT or mucosal-associ
259 examined whether cocaine targets the innate antiviral immunity of CD4+ T cells mediated by cellular
260 here that RIPLET, an essential E3 ligase in antiviral immunity, promotes the antiviral signaling act
261 CD8(+) T cells are essential effectors in antiviral immunity, recognizing short virus-derived pept
262 hese processes were associated with impaired antiviral immunity, reduced retinoic acid-inducible gene
264 tions, one of the major mechanisms for plant antiviral immunity relies on RNA silencing, which is oft
266 are innate-like T cells that play a role in antiviral immunity, specifically in controlling viral re
268 t in defense, showing proteins implicated in antiviral immunity, stress response, and ubiquitination/
269 t orthogonal therapies designed to stimulate antiviral immunity, such as therapeutic vaccines or broa
270 ptor agonists are potent enhancers of innate antiviral immunity that can also improve the adaptive im
271 IFITM3 represents a checkpoint regulator of antiviral immunity that controls cytokine production to
272 virus-infected cells reveals determinants of antiviral immunity that might underlie the human respons
273 h CD4(+) T cell "help" is crucial to sustain antiviral immunity, the mechanisms by which CD4(+) T cel
274 importance of IFN-lambda in tissue-specific antiviral immunity, the molecular mechanisms responsible
276 with RIG-I yet plays essential functions in antiviral immunity through distinct specificity for vira
277 y reported role of TKFC in regulating innate antiviral immunity through suppression of MDA5, we specu
278 lls (pDCs) play a crucial role in triggering antiviral immunity through their ability to capture and
279 estrate, sustain, and potentially regenerate antiviral immunity throughout persistent viral infection
281 ating the mechanisms by which CHIKV subverts antiviral immunity to establish and maintain a persisten
284 tion over time, potentially undermining host antiviral immunity to the transmitted viral strain.
285 ddition to their functions in cell-intrinsic antiviral immunity, type III IFNs protect epithelial bar
286 binding protein that initiates and amplifies antiviral immunity, unveiling a new facet of DNA recogni
287 ing gene, to explore CD8(+) T cell-dependent antiviral immunity using the lymphocytic choriomeningiti
290 Notably, helminth-induced impairment of antiviral immunity was evident in germ-free mice, but ne
292 hypothesized that a primary defect of innate antiviral immunity was responsible for unusually severe
294 ition, in which the cGAS-STING axis triggers antiviral immunity, whereas AIM2 triggers inflammasome a
295 rtant role for BMPs and activins in cellular antiviral immunity, which acts independently of, and mod
296 f interest in attempts to restore functional antiviral immunity, which is critical for the control of
297 the importance of cell type heterogeneity in antiviral immunity will aid in the identification of uni
299 stem for the study of viral pathogenesis and antiviral immunity within the central nervous system (CN
300 ctivation statuses and functionally regulate antiviral immunity within the framework of the activatio