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1 ifferences of these cells within the gastric antrum.
2 a of the lower esophagus, gastric corpus and antrum.
3 cts with ICC-IM in the corpus but not in the antrum.
4 e wild type occupied both the corpus and the antrum.
5 pression normally is confined to the gastric antrum.
6 re absent from the longitudinal layer of the antrum.
7 ture and in the longitudinal axis toward the antrum.
8 ive propagation of slow waves in the gastric antrum.
9 M) layers of the isolated guinea-pig gastric antrum.
10 arations made from the circular layer of the antrum.
11 xing and emptying of contents in the gastric antrum.
12 ed from the circular layer of the guinea pig antrum.
13 he increase was significantly greater in the antrum.
14 ltiple anatomic abnormalities of the pyloric antrum.
15 vs 4.77 +/- 0.27, P < 0.001) staining in the antrum.
16 urrents in myocytes isolated from the murine antrum.
17 gastric muscle sheets containing corpus and antrum.
18 ne produced by G-cells in the normal gastric antrum.
19 d to alpha-BGT that project to the fundus or antrum.
20 networks in the laryngeal esophagus and the antrum.
21 most severe changes occurring in the gastric antrum.
22 all of these, corpus, corpus and antrum, and antrum.
23 to the roof or other portions of the cupular antrum.
24 ch, binds to epithelial cells of the gastric antrum.
25 al cells except for those lining the gastric antrum.
26 iferate, stratify and develop a fluid-filled antrum.
27 e pooling of red blood cells in the follicle antrum.
28 ons and nodularity in the gastric cardia and antrum.
29 C activation in Lgr5(+) cells of the gastric antrum.
30 narrower ablative lesions in the left atrial antrum.
31 astritis and gastric stenosis in the gastric antrum.
32 hich contains the fundic epithelium, and the antrum.
33 environment of increased inflammation in the antrum.
34 d exclusively in the lesser curvature of the antrum.
35 profound mucosal hyperplasia of the gastric antrum.
36 near the junction of the gastric corpus and antrum.
37 ic cancer primarily in the distal stomach or antrum.
38 amplitudes and reduced frequency of diabetic antrums.
40 ted, and tumors predominantly located in the antrum (32.35%), cardia (30.15%), and body (24.26%).
44 TB creates a gastroileal anastomosis in the antrum after the SG; nutrient transit is maintained in t
48 f duodenogastric juice with exclusion of the antrum and 72% of rats with reflux of duodenogastric jui
49 in proliferation in the proximal corpus and antrum and a multifocal reduction in parietal cell numbe
52 ve rate of biopsy-based tests at the gastric antrum and body were calculated in terms of degree of ga
53 rents for neurones projecting to the fundus, antrum and caecum were 149 +/- 38 (n = 25), 115 +/- 18 (
55 Proliferation was increased 2-fold in both antrum and corpus in H. pylori-positive patients, was no
57 two H. pylori strains were isolated from the antrum and corpus parts of the stomach, and comparisons
61 ssive acid blockade, biopsies of the stomach antrum and duodenum are normal, and the tissue inflammat
63 d predominantly in mucous gland cells of the antrum and in mucous neck cells of the glandular corpus.
64 ed with inflammation and atrophy both in the antrum and in the corpus, while homB status was associat
65 tudy we isolated ICC from the murine gastric antrum and investigated the Ca(2+)-dependent ionic condu
66 fibrillation, in addition to pulmonary vein antrum and posterior wall isolation, ablation of nonpulm
68 ritis with areas of mucosal dysplasia in the antrum and predominantly midsuperficial gastritis in the
71 ical proliferation foci in the mucosa of the antrum and pyloric junction at 4.5 and 6 months of age,
72 ds to gastric dysplasia and polyposis of the antrum and pyloric junction, but H. felis infection of t
74 5 and 6 months of age, whereas polyps of the antrum and pylorus were present in all mice, regardless
76 elium of the most distal stomach region, the antrum and pylorus; expression in the adult intestine is
78 that is primarily synthesized in the gastric antrum and stimulates the secretion of histamine from en
79 infiltration and gastric atrophy in both the antrum and the corpus by multiple linear regression anal
82 Afferents were also supplied to the distal antrum and the pylorus, with pyloric innervation consist
83 aracterize the ICC within the canine gastric antrum and to determine the site(s) of pacemaker activit
84 include the separate roles of the fundus and antrum and to include the complex interactions the stoma
86 oaded myocytes isolated from the rat gastric antrum and voltage clamped at -60 1r1rqmV1qusing the per
87 ica appearance with narrowing of the gastric antrum and/or body (n = 5), narrowing of the body and/or
88 inute (cpm) pacemaker activity in corpus and antrum, and a proximal-to-distal slow wave frequency gra
90 H. pylori behaves differently in the corpus, antrum, and corpus-antrum transition zone subregions of
91 udinal muscle layer of the murine corpus and antrum, and it revealed marked heterogeneity in the dist
92 regenerative compartments termed corpus and antrum, and our understanding of the transcriptional net
93 ty; regional gastric motility of the fundus, antrum, and pylorus; and tests of sensation and complian
94 on DMV neurones projecting to the fundus and antrum, and the alpha3beta4 nAChR subtype on DMV neurone
95 within the tunica muscularis of the gastric antrum, and these cells serve different physiological fu
96 elated to atrophy (PRC +/- SE; 0.87 +/- 0.4 [antrum] and 0.93 +/- 0.4 [corpus], P < 0.05), whereas be
97 ent [PRC] +/- SE), TT versus CC: 37.6 +/- 6 [antrum] and 32.1 +/- 6 [corpus] pg/mg protein (P < 0.001
98 ad the highest IL-1beta levels (82.9 +/- 12 [antrum] and 87.2 +/- 11 [corpus]) and showed a synergist
99 io x 100,000 = 650 versus 338, respectively [antrum], and 172 versus 40, respectively [corpus]) (P <
100 astric corpus including the fundus and body, antrum, angulus, or duodenum) were examined by endoscopy
101 y labeled by a viral tracer from the gastric antrum, are primarily excitatory as optogenetic activati
105 predominantly in corpus biopsies, rarely in antrum biopsies (95.2% vs 24.6%); they were found in 497
106 ow counts predominantly in corpus, rarely in antrum biopsies (95.2% vs. 24.6%); they were found in 49
107 -fixed, paraffin-embedded sections of rhesus antrum biopsy samples were stained with H&E, periodic ac
109 se from the stomach, mainly from its pyloric antrum, but a weaker input originated from the fundus re
110 d histamine secretion in human, dog, and rat antrum by activating sst2 receptors on gastrin and hista
111 in the level of DAG, whereas in the newborn antrum, CCK (10(-7) mol/L) caused a sustained increase i
112 s with tumors originating in the fundus/body/antrum compared with esophagus/cardia (13.4 v 10.8 month
113 ammation and proliferation in the corpus and antrum compared with uninfected or mice infected with th
114 ion via electrodes in the muscle wall of the antrum connected to a neurostimulator in an abdominal wa
117 wall of the lesser curvature of the pyloric antrum, corresponding to the predominant focus of H. mus
118 In neurones projecting to the fundus and the antrum, currents resistant to alpha-BGT were significant
121 to live in either gastric fundus or gastric antrum depending on the level of acidity at the gastric
122 present if: (1) the first pass around the PV antrum did not produce PV isolation and (2) subsequent a
124 Human intestinal-type gastric cancers in the antrum exhibited progressive TFF1 repression and promote
126 culture, Mutant follicles exhibited delayed antrum formation [indicative of follicle stimulant hormo
127 ith abundant granulosa cells and evidence of antrum formation that appeared arrested before ovulation
134 f the hormone gastrin in the distal stomach (antrum) has been known for almost 110 years, and the phy
135 Gastrin, produced by G cells in the gastric antrum, has been identified as the circulating hormone r
137 r gastrin-producing (G) cells in the stomach antrum, hypogastrinemia, and increased stomach luminal p
139 Preneoplasia formed progressively in the antrum in 35- to 80-week-old Villin-Cre(+);Klf4(fl/fl) m
140 ocated on the lesser curvature of the distal antrum in all patients and extended to the pylorus in 25
145 es should be obtained, with samples from the antrum/incisura and corpus placed in separately labeled
146 on; at a minimum, biopsies from the body and antrum/incisura should be obtained and placed in separat
150 IGLEs had recovered, the disorganization of antrum innervation had partially recovered, and some IMA
154 e within the esophagus during pulmonary vein antrum isolation (PVAI) and correlate these data with es
155 utcome of patients undergoing pulmonary vein antrum isolation (PVAI) for atrial fibrillation (AF).
156 atrial (LA) wall injury after pulmonary vein antrum isolation (PVAI) in patients with atrial fibrilla
157 of atrial flutter (AFL) after pulmonary vein antrum isolation (PVAI) in patients with previous cardia
158 lmonary vein ablation (CPVA), pulmonary vein antrum isolation (PVAI), and, if failed, PVAI using the
161 ated surface area (ISA) after pulmonary vein antrum isolation for paroxysmal atrial fibrillation (AF)
162 nsecutive patients undergoing pulmonary vein antrum isolation for persistent atrial fibrillation.
163 y-one patients presenting for pulmonary vein antrum isolation for treatment of AF underwent 3-dimensi
164 We report the outcome of pulmonary vein (PV) antrum isolation in paroxysmal atrial fibrillation (AF)
171 ts with paroxysmal AF undergoing extended PV antrum isolation, the rate of late recurrence is lower t
172 rence at least 6 months after pulmonary vein antrum isolation, with an average follow-up of 9.6+/-3.7
176 an academic hospital (HR, 0.85), fundus/body/antrum location (HR, 0.90), highest socioeconomic status
177 show that a +4 stem cell (SC) in the gastric antrum, marked by expression of Cck2r (a GPCR) and Delta
178 , and emergence of ectopic pacemakers in the antrum may be caused by "reprogramming" of the ICC pacem
179 line and 6-h postprandial ultrasound gastric antrum measurements, satiety visual analogue scales (VAS
181 odenogastric juice with the exclusion of the antrum (n = 53); esophagoduodenostomy with proximal gast
183 ficantly higher (P < 0.05) in the corpus and antrum of animals in the high-salt diet group compared w
184 (P < 0.05) at 4 and 8 WPI in the corpus and antrum of animals on the high-salt diet when compared wi
186 inogenic histological changes in the pyloric antrum of the gastric mucosa, progressing from gastritis
188 esion GPCRs in the esophagus, the corpus and antrum of the stomach, the proximal and distal parts of
193 We comprehensively examined the gastric antrums of Lep(ob) mice using functional, morphological,
195 001 for each), *1/*2 versus *1/*1: 24 +/- 8 [antrum] (P <0.01) and 36.5 +/- 7 [corpus] (P <0.001).
196 chemoreceptor is responsible for the corpus-antrum phenotypes, we infected mice with strains lacking
199 tion of these neurons evoke EPSCs in gastric-antrum-projecting neurons, are functionally coupled to e
200 mbryonic day 10.5 (E10.5) to adult in murine antrum, pyloric region, small intestine, and colon.
201 his pyloric metaplasia, corpus glands become antrum (pylorus)-like with loss of acid-secreting pariet
202 tal foregut derivatives, the gastric corpus, antrum, pylorus, and duodenum are distinct structures wi
204 contrast, few DMV neurones that supplied the antrum/pylorus (3/13), duodenum (4/18) or caecum (1/13)
205 cular smooth muscle, cardiac atrium, gastric antrum/pylorus, enteric neurones, and vagal and dorsal r
209 Pepsin started to accumulate in the pylorus/antrum region before concentrating in the body stomach b
213 ith gastritis predominantly localized to the antrum retain normal (or even high) acid secretion, wher
214 ctions in the jejunum migrated orally to the antrum (retrograde peristaltic contractions; RPCs).
215 colonization and gastric infections (cardia-antrum section) which were observed at 10 to 12 weeks af
216 ice were more susceptible to gastric (cardia-antrum section), anorectal, and acute systemic (intraven
217 quivalent functional roles in the corpus and antrum share similar transcriptional states including th
219 ements of muscle cells within the corpus and antrum showed that stretch-induced changes in slow-wave
220 set of electrical rhythmicity in the gastric antrum, small bowel and proximal colon of the mouse.
221 olar recording electrodes were placed on the antrum, small intestine, and the transverse and descendi
222 (STOCs), and membrane potentials of gastric antrum smooth muscle cells from wild-type and phospholam
223 itutively elevated in phospholamban-knockout antrum smooth muscle cells relative to wild-type cells.
224 the resting membrane potential of wild-type antrum smooth muscle cells to a greater extent than phos
225 membrane potential of phospholamban-knockout antrum smooth muscle cells was hyperpolarized by approxi
226 vity in wild-type and phospholamban-knockout antrum smooth muscle cells was inhibited by ryanodine, b
227 GMP increased the STOC activity of wild-type antrum smooth muscle cells, but had no effect on STOC ac
228 in STOC activity evoked by SNP in wild-type antrum smooth muscle cells, but had no effect on STOC ac
229 racellular Ca(2+) wave activity in wild-type antrum smooth muscle cells, but had no effect on the con
231 ficantly increased in phospholamban-knockout antrum smooth muscles compared to wild-type smooth muscl
236 arcinogenesis of both the gastric corpus and antrum, suggesting that gastrin is an essential cofactor
237 ween the corpus and the fundus than with the antrum, suggesting that physiological differences betwee
239 sis showed that inflammation is worse in the antrum than in the corpus in both wild-type and Che(-) m
240 nalysis and were both greater in the gastric antrum than in the gastric body of infected patients.
241 unitary potentials in the gastric fundus and antrum that contributes to the overall excitability of t
242 undus, 23% in the corpus, and only 8% in the antrum, the absolute number of vagally contacted GRP-IR
243 d intestinal-subtype tumors occurring in the antrum; these have the best overall prognosis and the lo
244 labelled premotor DMV neurons to the gastric-antrum through an increase in inhibitory post-synaptic c
247 edgehog signaling is strictly paracrine from antrum to colon throughout embryonic and adult life.
250 In contrast, in the antrum and the corpus-antrum transition zone, chemotaxis does not help initial
253 om the normal and pathological human gastric antrum using a least-squares minimization analysis appli
254 We cultured ICC from the murine gastric antrum, verified that cells were Kit immunoreactive, and
255 ) highly correlated in same-patient samples (antrum vs body, r = 0.85, P < .0001), and (4) inversely
259 egions in the mouse, forestomach, corpus and antrum, we first describe the existence of a "secondary
261 gment of the duodenum as well as the pyloric antrum were collected and processed with diaminobenzidin
262 atostatin and gastrin mRNA abundances in the antrum were depressed by about 35% by antral denervation
263 the distal esophagus and cardia, fundus, and antrum were evaluated for inflammation, H. pylori infect
267 Mucosal segments from human, dog, and rat antrum were superfused with various concentrations of so
268 ributions, with peak densities in the corpus-antrum, were similar in the three strains of mice and co
269 began to develop polyposis in the fundus and antrum when they were over 6 - 12 months old, and in the
270 itive vs. -negative gastritis and in gastric antrum, where bacterial density is greatest, suggest tha
271 , but has quite different functions from the antrum, which provides mixing and propulsion of contents
272 ic tone) and contractility of the fundus and antrum while administering five doses of i.v. nicotine a
273 d Rag2(-/-) mice colonized in the corpus and antrum with 10(5) to 10(6) H. pylori CFU/gram without as