ライフサイエンスコーパス: aortic aneurysm

1 e, and 1.93 (95% CI 1.47-2.53) for abdominal aortic aneurysm.

2 2-polarization in a mouse model of abdominal aortic aneurysm.

3 lesion, which was initially thought to be an aortic aneurysm.

4 sease is frequently accompanied by ascending aortic aneurysm.

5 air that had discordance of aortic valve and aortic aneurysm.

6 , which is commonly associated with thoracic aortic aneurysm.

7 associated with the occurrence of abdominal aortic aneurysm.

8 lopment and testing of pathogenic models for aortic aneurysm.

9 ue of doxycycline in blocking MFS-associated aortic aneurysm.

10 the treatment of intact descending thoracic aortic aneurysms.

11 can be recommended for the stabilization of aortic aneurysms.

12 nced our knowledge of the pathophysiology of aortic aneurysms.

13 a new technique for the repair of abdominal aortic aneurysms.

14 ent elective surgery for descending thoracic aortic aneurysms.

15 States underwent repair of intact abdominal aortic aneurysms.

16 aortic aneurysm repair (EVAR) for abdominal aortic aneurysms.

17 is upregulated in atherosclerotic plaque and aortic aneurysms.

18 e more likely to undergo repair for isolated aortic aneurysms.

19 the development and enlargement of thoracic aortic aneurysms.

20 e for reducing the growth of small abdominal aortic aneurysms.

21 o identify individuals at risk for abdominal aortic aneurysms.

22 but was inversely associated with abdominal aortic aneurysm (0.46 [0.35-0.59]) and subarachnoid haem

23 r disease causes, the ratio ranged from 1.4 (aortic aneurysm: 3.5 vs 5.1 deaths per 100000 persons) t

24 The abdominal aortic aneurysm (AAA) anatomy influences the technical s

25 12] is the most upregulated MMP in abdominal aortic aneurysm (AAA) and, hence, MMP-12-targeted imagin

26 o the EVAR-1 scan protocol reduced abdominal aortic aneurysm (AAA) deaths by 3% and increased electiv

27 teinases (MMPs) play a key role in abdominal aortic aneurysm (AAA) development.

28 To study their contribution to abdominal aortic aneurysm (AAA) formation and development, we dete

29 Abdominal aortic aneurysm (AAA) formation is characterized by infl

30 Abdominal aortic aneurysm (AAA) growth after endovascular aneurysm

31 Population screening for abdominal aortic aneurysm (AAA) has commenced in several countries

32 The role of resolvins in abdominal aortic aneurysm (AAA) has not been established.

33 Abdominal aortic aneurysm (AAA) is a common aortic disease with a

34 RATIONALE: Abdominal aortic aneurysm (AAA) is a complex disease with both gen

35 Abdominal aortic aneurysm (AAA) is a complex disease with both gen

36 Abdominal aortic aneurysm (AAA) is a degenerative disease characte

37 Abdominal aortic aneurysm (AAA) is a life-threatening vascular dis

38 Abdominal aortic aneurysm (AAA) is a local dilatation of the abdom

39 Abdominal aortic aneurysm (AAA) is a major cause of morbidity and

40 An abdominal aortic aneurysm (AAA) is a permanent and irreversible di

41 Abdominal aortic aneurysm (AAA) is a prevalent life-threatening di

42 Abdominal aortic aneurysm (AAA) is a severe aortic disease with a

43 Abdominal aortic aneurysm (AAA) is an important cause of cardiovas

44 Abdominal aortic aneurysm (AAA) is an inflammatory vascular diseas

45 Abdominal aortic aneurysm (AAA) is characterized by transmural inf

46 ations, the decision to operate on abdominal aortic aneurysm (AAA) is primarily on the basis of measu

47 An abdominal aortic aneurysm (AAA) is typically defined as aortic enl

48 egulated tryptophan metabolism and abdominal aortic aneurysm (AAA) is unknown.

49 2) axis plays an important role in abdominal aortic aneurysm (AAA) pathogenesis, with effects on dise

50 nmet need for treatments to reduce abdominal aortic aneurysm (AAA) progression.

51 in physically frail patients with abdominal aortic aneurysm (AAA) randomized to either early endovas

52 Abdominal aortic aneurysm (AAA) remains a fatal disease.

53 Abdominal aortic aneurysm (AAA) remains the second most frequent v

54 age 65 to 100 years who underwent abdominal aortic aneurysm (AAA) repair (n = 71,422), pulmonary res

55 tcomes after open and endovascular abdominal aortic aneurysm (AAA) repair are each well described sep

56 We identified patients undergoing abdominal aortic aneurysm (AAA) repair in the Vascular Quality Ini

57 Suprarenal aortic clamping during abdominal aortic aneurysm (AAA) repair results in ischemia-reperfu

58 mmon elective surgical procedures [abdominal aortic aneurysm (AAA) repair, colectomy, coronary artery

59 renal arteries in open juxtarenal abdominal aortic aneurysm (AAA) repair, the volume effect in these

60 ase (CKD) predicts mortality after abdominal aortic aneurysm (AAA) repair.

61 program on outcomes after elective abdominal aortic aneurysm (AAA) repair.

62 ce for patients requiring elective abdominal aortic aneurysm (AAA) repair.

63 The critical challenge in abdominal aortic aneurysm (AAA) research is the accurate diagnosis

64 Adventitial DCN is reduced in abdominal aortic aneurysm (AAA) resulting in vessel wall instabili

65 described in literature are due to abdominal aortic aneurysm (AAA) rupture into the left renal vein.

66 A general abdominal aortic aneurysm (AAA) screening program, targeting 65-ye

67 ative pain control during elective abdominal aortic aneurysm (AAA) surgery.

68 in the contemporary management of abdominal aortic aneurysm (AAA) with relation to recommended treat

69 SMCs is correlated with rupture of abdominal aortic aneurysm (AAA), an age-related vascular disease.

70 ve open and endovascular repair of abdominal aortic aneurysm (AAA), cost may be an important factor i

71 In the event of rupture of an abdominal aortic aneurysm (AAA), mortality is very high.

72 fluences the treatment outcomes of abdominal aortic aneurysm (AAA).

73 contributes to the development of abdominal aortic aneurysm (AAA).

74 a significant risk factor of human abdominal aortic aneurysm (AAA).

75 B1 to angiotensin (Ang) II-induced abdominal aortic aneurysm (AAA).

76 ase (COPD) might increase risk for abdominal aortic aneurysm (AAA).

77 e severity of periodontitis and of abdominal aortic aneurysm (AAA).

78 l inflammatory diseases, including abdominal aortic aneurysm (AAA).

79 y of geometric features for future abdominal aortic aneurysms (AAA) growth prediction.

80 e increases the risk of developing abdominal aortic aneurysms (AAA).

81 Abdominal aortic aneurysms (AAAs) are a deadly pathology with stro

82 graft devices for the treatment of abdominal aortic aneurysms (AAAs) are being increasingly used worl

83 Ruptured abdominal aortic aneurysms (AAAs) have mortality estimated at 81%.

84 Abdominal aortic aneurysms (AAAs) represent a potentially life-thr

85 urgical treatment for asymptomatic abdominal aortic aneurysms (AAAs) with a diameter of at least 55 m

86 n implicated in the development of abdominal aortic aneurysms (AAAs), but its effect on AAA growth ov

87 dality for the characterization of abdominal aortic aneurysms (AAAs).

88 artery disease (PAD; P=0.090) and abdominal aortic aneurysms (AAAs; P=0.12), and the variant associa

89 uscle isoform of alpha-actin, cause thoracic aortic aneurysms, acute aortic dissections, and occlusiv

90 Abdominal aortic aneurysms affect more than 3% of US older adults.

91 Remodeling of abdominal aortic aneurysms after EVAR is not uniform.

92 with age- and sex-matched controls (1:10 for aortic aneurysm and 1:100 for aortic dissection) using t

93 , 42 years) had a first-degree relative with aortic aneurysm and 7209 persons (mean age, 39 years) ha

94 o of 6.70 (95% CI, 5.96-7.52) for developing aortic aneurysm and a hazard ratio of 9.24 (95% CI, 5.53

95 Increased aortic size, a precursor of aortic aneurysm and a risk factor for dissection, cluste

96 Ruptured aortic aneurysm and aortic dissections are potentially p

97 Older patients with ascending aortic aneurysm and aortic insufficiency secondary to di

98 L-cholesterol is likely to prevent abdominal aortic aneurysm and aortic stenosis, in addition to CAD

99 published data on genes involved in thoracic aortic aneurysm and attempts to explain divergent hypoth

100 Sporadic aortic aneurysm and dissection (AAD), caused by progress

101 ars, first-degree relatives of patients with aortic aneurysm and dissection had a hazard ratio of 6.7

102 associated with valve dysfunction, ascending aortic aneurysm and dissection.

103 Sporadic aortic aneurysm and dissections (AADs) are common vascul

104 In the case of thoracic aortic aneurysm and dissections (thoracic aortic disease

105 The incidence rates of aortic aneurysm and dissections approach the incidence r

106 For both aortic aneurysm and dissections, the absolute event rate

107 vated amylase in the context of an abdominal aortic aneurysm and generalized atheromatosis.

108 effective in preclinical models of abdominal aortic aneurysm and show great potential for clinical tr

109 ied shortly after parturition from ascending aortic aneurysm and spontaneous hemorrhage.

110 We found a lower rate of repair of abdominal aortic aneurysms and a larger mean aneurysm diameter at

111 Thoracic aortic aneurysms and acute aortic dissections (TAADs) oc

112 of the most common aortic diseases, namely, aortic aneurysms and acute aortic syndromes.

113 successful medical stabilization of growing aortic aneurysms and aortic root stabilization in Marfan

114 ry of GenTAC (Genetically Triggered Thoracic Aortic Aneurysms and Cardiovascular Conditions) is a lon

115 itute GenTAC (Genetically Triggered Thoracic Aortic Aneurysms and Cardiovascular Conditions) Registry

116 aneurysms are much more common than thoracic aortic aneurysms and combined they account for >25 000 d

117 human atherosclerotic plaques and abdominal aortic aneurysms and correlated with decreased expressio

118 human atherosclerotic plaques and abdominal aortic aneurysms and correlated with decreased expressio

119 ceptor contributes to formation of ascending aortic aneurysms and dissections (AADs) induced by smoot

120 on genetic alterations for familial thoracic aortic aneurysms and dissections (TAAD) are missense mut

121 ase G1 (PRKG1, p.Arg177Gln) develop thoracic aortic aneurysms and dissections (TAAD) as young adults.

122 Thoracic aortic aneurysms and dissections (TAAD) represent a subs

123 Heritable thoracic aortic aneurysms and dissections (TAAD), including Marfa

124 he most prevalent cause of familial thoracic aortic aneurysms and dissections (TAAD).

125 on-syndromic hearing loss, familial thoracic aortic aneurysms and dissections, and multiple variation

126 with conditions that predispose to thoracic aortic aneurysms and dissections, including MFS.

127 the FBN1 locus associated with both thoracic aortic aneurysms and dissections.

128 e for 25% of families with familial thoracic aortic aneurysms and dissections.

129 responsible for a large family with thoracic aortic aneurysms and dissections.

130 nt protein kinase 1 (PKG1) leads to thoracic aortic aneurysms and dissections.

131 ular inflammation in patients with abdominal aortic aneurysms and predicts the rate of aneurysm growt

132 Aortic aneurysms and prophylactic aortic surgery are mor

133 thnic differences in prevalence of abdominal aortic aneurysms and showed more complex iliac anatomy i

134 from low-shear-rate conditions in abdominal aortic aneurysms and thoracic aortic dissections to thro

135 for first-degree relatives versus 11 to 13 (aortic aneurysm) and 2 to 3 (aortic dissections) per 100

136 metalloproteinases during the progression of aortic aneurysm, and provide new insights into the poten

137 ased the incidence and severity of abdominal aortic aneurysms, and caused aortic arch ruptures and di

138 ers have been implicated in the pathology of aortic aneurysm, aortic dissection, and more recently, v

139 f 7 surgical emergencies (ruptured abdominal aortic aneurysm, aortic dissection, appendicitis, perfor

140 .65, 2.7, 4.46, 3.22) for ruptured abdominal aortic aneurysm, aortic dissection, appendicitis, perfor

141 proteolytic cascades in enlarging abdominal aortic aneurysm are feasible.

142 Aortic aneurysms are a common vascular disease in Wester

143 Abdominal aortic aneurysms are associated with chronic inflammatio

144 Abdominal aortic aneurysms are much more common than thoracic aort

145 , peripheral arterial disease, and abdominal aortic aneurysms, are also briefly reviewed.

146 ility of penetrance of both BAV and thoracic aortic aneurysm as well as the variability of the associ

147 For centuries, physicians have recognized aortic aneurysms as an acute threat to life.

148 1-3) that frequently presents with ascending aortic aneurysm (AscAA)(4).

149 95% confidence intervals (CIs) of abdominal aortic aneurysm associated with physical activity.

150 was associated with enlargement of abdominal aortic aneurysms at 1 year, particularly in aneurysms sm

151 Ascending thoracic aortic aneurysm (ATAA) is caused by the progressive weak

152 elastic properties of two ascending thoracic aortic aneurysm (ATAA) patients from pre-operative gated

153 nted for each sex separately, with abdominal aortic aneurysms being assessed for aneurysm repair by e

154 congressional campaign to fund screening for aortic aneurysms brought the disease to national attenti

155 ng neoplasm in the acetylcysteine group, and aortic aneurysm, contusion, forearm fracture, and worsen

156 eractions between these different factors in aortic aneurysm development and identified a key role fo

157 emerged as a common molecular signature for aortic aneurysm development.

158 Baseline abdominal aortic aneurysm diameter (P<0.0001) and current smoking

159 el cohort study, 342 patients with abdominal aortic aneurysm (diameter >/=40 mm) were classified by t

160 sis of thoracic aortic disease and abdominal aortic aneurysm disease.

161 ich is characterized by a high risk of fatal aortic aneurysms/dissections, can occur secondarily to s

162 l valve prolapse, collagen vascular disease, aortic aneurysm, Down syndrome, sleep apnea, depression,

163 ong patients with small infrarenal abdominal aortic aneurysms, doxycycline compared with placebo did

164 up after endovascular treatment of abdominal aortic aneurysms (EVAR) is mainly aimed at detection of

165 ercholesterolemic diet led to development of aortic aneurysms exhibiting all the features of human di

166 proposed cellular mechanisms responsible for aortic aneurysm formation and identifies opportunities f

167 D-series resolvins inhibit murine abdominal aortic aneurysm formation and increase M2 macrophage pol

168 s et al. (2017) report that, in experimental aortic aneurysm formation, neutralization of interleukin

169 hat disrupt aortic wall homeostasis to cause aortic aneurysm formation.

170 od flow) lumen and the wall structure of the aortic aneurysm from CT angiograms (CTA) was compared ag

171 ic aneurysms (SaAAAs) and fusiform abdominal aortic aneurysms (FuAAAs) regarding patient characterist

172 ncreatitis, pancreatic pseudocyst, abdominal aortic aneurysm, generalized atheromatosis.

173 For abdominal aortic aneurysms, genetic associations have been identif

174 Discovery of novel biomarkers for abdominal aortic aneurysm growth (AAA) prediction.

175 EVAR for abdominal aortic aneurysm has an initial survival advantage over O

176 A genetic predisposition to thoracic aortic aneurysm has been established, and gene discovery

177 ween physical activity and risk of abdominal aortic aneurysm has been inconsistent with some studies

178 The treatment of aortic aneurysms has evolved dramatically in the past 3

179 disruption is involved in the development of aortic aneurysms has led to renewed investigations into

180 Traditionally, thoracic aortic aneurysms have been labeled as a degenerative dis

181 EVAR) versus open repair of intact abdominal aortic aneurysms have been shown in randomised trials, b

182 al activity may reduce the risk of abdominal aortic aneurysm, however, further studies are needed to

183 (HR: 1.78; 95% CI: 1.51 to 2.10), abdominal aortic aneurysm (HR: 1.72; 95% CI: 1.34 to 2.21), and no

184 ardiovascular diseases such as hypertension, aortic aneurysm, hypercholesterolaemia, atherosclerosis,

185 clinical studies in the context of abdominal aortic aneurysm identified hundreds of candidate strateg

186 imaging are 2 novel approaches to abdominal aortic aneurysm imaging evaluated in clinical trials.

187 that underwent TEVAR for descending thoracic aortic aneurysm in the MOTHER database and 231 in the Un

188 The management of abdominal aortic aneurysm in women needs improvement.

189 ine for repair of intact descending thoracic aortic aneurysms in Medicare beneficiaries.

190 To investigate the pathogenesis of aortic aneurysms in MFS, we generated a vascular model d

191 132 patients (0.6% of all operated abdominal aortic aneurysms in Sweden).

192 of repair of intact (nonruptured) abdominal aortic aneurysms, in-hospital mortality among patients w

193 Abdominal aortic aneurysm is a dynamic vascular disease characteri

194 Thoracic aortic aneurysm is a potentially life-threatening condit

195 nderstanding of the pathogenesis of thoracic aortic aneurysm is quite limited.

196 idline laparotomy in patients with abdominal aortic aneurysm is safe and effectively prevents the dev

197 Aortic aneurysm is the most life-threatening complicatio

198 se To assess whether the stability of murine aortic aneurysms is associated with the homogeneity of p

199 The etiology of aortic aneurysms is poorly understood, but it is associa

200 One patient developed a small aortic aneurysm late and has not required treatment.

201 FEB is downregulated in both human and mouse aortic aneurysm lesions.

202 aneurysm repair (EVAR) for mycotic abdominal aortic aneurysms (MAAAs).

203 deviation), 12 were women, 18 had ascending aortic aneurysms (maximal diameter > 4.0 cm), and 10 had

204 The primary outcome was change in abdominal aortic aneurysm maximum transverse diameter measured fro

205 , including atrial fibrillation (Northwest), aortic aneurysm (Midwest), and endocarditis (Mountain We

206 Prognosis for women with abdominal aortic aneurysm might be worse than the prognosis for me

207 ication and lead to a reduction in abdominal aortic aneurysm morbidity and mortality.

208 In cardiovascular pathologies, such as aortic aneurysm, new knowledge on the involvement of cel

209 -cause mortality in patients after repair of aortic aneurysms of the descending thoracic aorta thorac

210 omes the primary treatment modality for most aortic aneurysms, open repair remains an essential treat

211 idence rates and hazard ratios of developing aortic aneurysm or dissection among first-degree relativ

212 n among first-degree relatives of those with aortic aneurysm or dissection, in comparison with age- a

213 lly predicted LDL-cholesterol with abdominal aortic aneurysm (OR, 1.75 [95% CI, 1.40-2.17]) and aorti

214 [95% CI, 1.00-1.37]; P=0.050), and abdominal aortic aneurysm (OR, 2.60 [95% CI, 1.15-5.89]; P=0.022)

215 (OR: 1.28; 95% CI: 1.23 to 1.32), abdominal aortic aneurysms (OR: 1.28; 95% CI: 1.20 to 1.37), and s

216 nd allergic rhinitis, mitral valve disorder, aortic aneurysm, or depression (P > 0.1 for all comparis

217 s the development of specific treatments for aortic aneurysms over time and more broadly addresses ho

218 lation and flutter, rheumatic heart disease, aortic aneurysm, peripheral arterial disease, endocardit

219 ) and three who received deferred treatment (aortic aneurysm, pneumonia, and unknown cause); all four

220 ogical processes involved in later stages of aortic aneurysm progression.

221 neurysm repair (EVAR) for ruptured abdominal aortic aneurysm (r-AAA) requires advanced infrastructure

222 ences in timely repair of ruptured abdominal aortic aneurysm (rAAA) were associated with excess risk

223 patients hospitalized for ruptured abdominal aortic aneurysms (rAAA) by conducting a retrospective an

224 For the management of descending thoracic aortic aneurysms, recent evidence has suggested that out

225 Current management of aortic aneurysms relies exclusively on prophylactic oper

226 Dissections or ruptures of aortic aneurysms remain a leading cause of death in the

227 A robust estimate of the frequency of aortic aneurysms remains to be determined as the majorit

228 owest cost-of-rescue hospitals for abdominal aortic aneurysm repair ($60456 vs $23261; P < .001), col

229 evascularization (19%-IVSR vs. 16%-VSF), and aortic aneurysm repair (13%-IVSR vs. 13%-VSF) procedures

230 lowing general elective surgeries: abdominal aortic aneurysm repair (AAA), coronary artery bypass gra

231 y (CT) or ultrasonography after endovascular aortic aneurysm repair (EVAR) for abdominal aortic aneur

232 Endovascular aortic aneurysm repair (EVAR), left ventricular assist d

233 aged 65 to 100 years who underwent abdominal aortic aneurysm repair (n = 69207), colectomy for cancer

234 the existence and outcomes of open abdominal aortic aneurysm repair (OAR) and carotid endarterectomy

235 atients were most likely to undergo isolated aortic aneurysm repair (W:76%, B:59%, A:68%, H:76%, P <

236 ic aneurysm repair, or thoracic endovascular aortic aneurysm repair at 20 sites in North America.

237 c aneurysm repair, and thoracic endovascular aortic aneurysm repair devices.

238 he outcomes of patients undergoing abdominal aortic aneurysm repair in a vascular network in the Sout

239 The process of centralization of abdominal aortic aneurysm repair in a vascular network was safe fo

240 Selection of patients for abdominal aortic aneurysm repair is currently based on aneurysm si

241 idence of incisional hernias after abdominal aortic aneurysm repair is high.

242 8%), and percutaneous endovascular abdominal aortic aneurysm repair or thoracic endovascular aortic a

243 5 carotid, 21,428 lower extremity, and 5,800 aortic aneurysm repair procedures.

244 omes following carotid, lower extremity, and aortic aneurysm repair procedures.

245 ed on patients undergoing elective abdominal aortic aneurysm repair through a midline laparotomy (Cli

246 tic aneurysm repair or thoracic endovascular aortic aneurysm repair was performed in 53 (20.2%).

247 uled for lower limb bypass or open abdominal aortic aneurysm repair were randomly assigned, on hemogl

248 7% (total hip replacement) to 77% (abdominal aortic aneurysm repair), and most patients were white.

249 , coronary artery bypass grafting, abdominal aortic aneurysm repair, abdominal aortic aneurysm repair

250 acement, percutaneous endovascular abdominal aortic aneurysm repair, and thoracic endovascular aortic

251 ng outcomes of patients undergoing abdominal aortic aneurysm repair, based on prospectively entered N

252 1 of 7 common surgical procedures-abdominal aortic aneurysm repair, colectomy, cystectomy, prostatec

253 edures (colectomy, lung resection, abdominal aortic aneurysm repair, coronary artery bypass graft, ao

254 acement, percutaneous endovascular abdominal aortic aneurysm repair, or percutaneous thoracic endovas

255 acement, percutaneous endovascular abdominal aortic aneurysm repair, or thoracic endovascular aortic

256 All patients undergoing elective abdominal aortic aneurysm repair, registered in the Dutch Surgical

257 abdominal aortic aneurysm repair, abdominal aortic aneurysm repair, total hip arthroplasty, total kn

258 epair, or percutaneous thoracic endovascular aortic aneurysm repair.

259 the percentage of patients who had abdominal aortic aneurysm-repair without intraoperative complicati

260 tic aneurysm ruptures, 126 (36.8%) abdominal aortic aneurysm repairs, and 48 (14.0%) deaths occurred.

261 ogy after abdominal surgery for repair of an aortic aneurysm requiring multiple therapeutic paracente

262 antation for midaortic syndrome and multiple aortic aneurysms, respectively underwent renal transplan

263 Elective endovascular repair of an abdominal aortic aneurysm results in lower perioperative mortality

264 es and intensities of activity and abdominal aortic aneurysm risk.

265 he IL-6 signaling pathway in mouse models of aortic aneurysm rupture or dissection.

266 ow-up (1005+/-280 days), 17 (5.0%) abdominal aortic aneurysm ruptures, 126 (36.8%) abdominal aortic a

267 ifferences between saccular-shaped abdominal aortic aneurysms (SaAAAs) and fusiform abdominal aortic

268 maximal diameter and volume of an abdominal aortic aneurysm sac can be used for temporal monitoring

269 structed centerline; volume of the abdominal aortic aneurysm sac; and volume from the lowest renal ar

270 assess changes in the size of the abdominal aortic aneurysm sack using CT angiography (CTA) after su

271 sion of TFEB was measured in human and mouse aortic aneurysm samples.

272 Although thoracic and abdominal aortic aneurysms share some common characteristics, incl

273 role to atherosclerotic plaque and abdominal aortic aneurysm stability are poorly understood.

274 n treatment is necessary for a comprehensive aortic aneurysm surgery practice.

275 , drawing on evidence from thoraco-abdominal aortic aneurysm surgery.

276 using a major paradigm shift in the field of aortic aneurysm surgery.

277 , drawing on evidence from thoraco-abdominal aortic aneurysm surgery.

278 (AoD) is a serious complication of thoracic aortic aneurysm (TAA).

279 Genetic aortopathy (GA) underlies thoracic aortic aneurysms (TAA) in younger adults.

280 Data on thoracic aortic aneurysms (TAA), type B aortic dissections (TBAD)

281 Thoracoabdominal aortic aneurysm (TAAA) remains a challenging problem.

282 lower with endovascular repair of abdominal aortic aneurysm than with open repair, but the survival

283 e the most common cause of familial thoracic aortic aneurysms that lead to dissection (TAAD).

284 Abdominal aortic aneurysm tissue reveals a high M1/M2 ratio in whi

285 Translating iPSC findings into clinical aortic aneurysm tissue samples highlights the potential

286 f the initiation and progression of thoracic aortic aneurysms to contrast key predisposing risk facto

287 ssigned patients with asymptomatic abdominal aortic aneurysms to either endovascular repair or open r

288 This allows aortic aneurysms to serve as a case study for exploring

289 Successful aortic aneurysm treatment depends on either open replace

290 eing assessed for repair of intact abdominal aortic aneurysm using data from study periods after the

291 Thresholds for repair of abdominal aortic aneurysms vary considerably among countries.

292 e the technical details of the management of aortic aneurysms vary greatly depending on the location

293 , as compared with open repair, of abdominal aortic aneurysm was associated with a substantial early

294 wn to be associated with BAV and/or thoracic aortic aneurysm was performed.

295 In patients with abdominal aortic aneurysm, we assessed whether USPIO-enhanced MRI

296 clerotic carotid artery specimens, abdominal aortic aneurysms) were obtained from patients undergoing

297 Cobinamide could be a treatment for aortic aneurysms where oxidative stress contributes to t

298 can lead to severe malformations, including aortic aneurysms, which are frequently associated with i

299 on patients with intact descending thoracic aortic aneurysms who underwent TEVAR or open surgical re

300 dynamic regimes, acting on sealed abdominal aortic aneurysms, with references to real case studies.