ライフサイエンスコーパス: aortic calcification

1 ng (psoas area, psoas density, and abdominal aortic calcification).

2 xpression per se is not sufficient to induce aortic calcification.

3 Ps with biochemical parameter in relation to aortic calcification.

4 with significant differences in coronary or aortic calcification.

5 coronary artery calcification and abdominal aortic calcification.

6 e culture and mouse model, respectively, for aortic calcification.

7 rmation by micro-CT of anatomic location and aortic calcification.

8 n CKD, perhaps explaining this inhibition of aortic calcification.

9 have contributed to their protection against aortic calcification.

10 d reduced flow-mediated dilation and greater aortic calcification.

11 coronary artery calcification, and abdominal aortic calcification.

12 calcification, but higher dosages stimulated aortic calcification.

13 ty-five patients had at least one measurable aortic calcification.

14 Little is known about the heritability of aortic calcification.

15 role in the presence and extent of abdominal aortic calcification.

16 ormalized to height (in meters squared), and aortic calcification.

17 all shear stress in relation to distribution aortic calcifications.

18 Purpose To determine if abdominal aortic calcification (AAC) at CT predicts cardiovascular

19 ry artery calcification (CAC), and abdominal aortic calcification (AAC) at the first follow-up visit

20 easured at the proximal femur, and abdominal aortic calcification (AAC) was quantified (8-points scor

21 Abdominal aortic calcification (AAC), preceding CAC, may predict c

22 Aortic calcification (AC) in the entire aorta was assess

23 se; little is known, however, about thoracic aortic calcification (AC).

24 vestigate the relationship between abdominal aortic calcification and AAA growth via a secondary coho

25 ts, 57.5% females aged 60-80 y had abdominal aortic calcification and clinical assessments performed

26 enetic difference was found only between the aortic calcification and control group with VEGF SNP -25

27 ew also analyzes the frequent coexistence of aortic calcification and coronary artery disease in term

28 were significantly different in control and aortic calcification and could enhance the aortic calcif

29 n, mice lacking HDAC9 had a 40% reduction in aortic calcification and improved survival.

30 Secondary outcomes included abdominal aortic calcification and serum and urine markers of mine

31 tudy was to investigate the relation between aortic calcification and VEGF SNPs (-2578C>A, -1154G>A a

32 both of which also associated with abdominal aortic calcifications and smaller global reflection coef

33 65 days were small body surface area, severe aortic calcification, and falls within the past 6 months

34 Lmna(G609G/+) mice showed excessive aortic calcification, and primary aortic vascular smooth

35 e-dependent increases in serum phosphate and aortic calcification associated with increased risk of m

36 and RAP1GAP, were associated with abdominal aortic calcification at a genome-wide level (P < 5.0 x 1

37 locity was 10.8 m/s, and 81.3% had abdominal aortic calcification at baseline.

38 No SNPs were associated with thoracic aortic calcification at the genome-wide threshold.

39 t quantifies skeletal muscle, abdominal fat, aortic calcification, bone density, and solid abdominal

40 comes included augmentation index, abdominal aortic calcification, BP, physical function, and blood m

41 a high-phosphate medium, warfarin increased aortic calcification but only in the absence of pyrophos

42 iol and paricalcitol were protective against aortic calcification, but higher dosages stimulated aort

43 Aortic calcification, collagen accumulation, and wall th

44 6 weeks did not affect arterial stiffness or aortic calcification compared with placebo.

45 ted dilation and greater coronary artery and aortic calcification compared with women without hot fla

46 inflammation in health, and renal injury and aortic calcification despite hyperphosphatemia in CKD.

47 d aortic calcification and could enhance the aortic calcification development.

48 rent genotypes of VEGF SNPs which may induce aortic calcification development.

49 vidually measured sites, baseline infrarenal aortic calcification had the strongest negative associat

50 al fat, muscle area, liver fat fraction, and aortic calcification improved predictive performance, yi

51 DR activators calcitriol and paricalcitol on aortic calcification in a mouse model of chronic kidney

52 e established as aggravating factors for the aortic calcification in association with different VEGF

53 nversely, parathyroid hormone (PTH) inhibits aortic calcification in low-density lipoprotein receptor

54 t the first and twelfth month after KTx: the aortic calcification index (ACI), fibroblast growth fact

55 Aortic calcification is an important independent predict

56 Aortic calcification is developed due to accumulation of

57 ification (n = 9,417) or descending thoracic aortic calcification (n = 8,422).

58 SNPs associated with the extent of abdominal aortic calcification (n = 9,417) or descending thoracic

59 ricardial fat, was associated with abdominal aortic calcification (odds ratio 1.32, 95% confidence in

60 dilation (beta=-0.97; SE, 0.44; P=0.03) and aortic calcification (odds ratio, 1.63; 95% confidence i

61 -/-) (LRP6-VKO) siblings exhibited increased aortic calcification on high-fat diet without changes in

62 ith no known CV risk factors but significant aortic calcification on preoperative imaging will have i

63 ation (P=0.02) and 17% decrease in abdominal aortic calcification (P=0.03).

64 eam tomography to assess coronary artery and aortic calcification, reported hot flashes (any/none, pr

65 Aortic calcification scores at CT colonography are signi

66 coronary artery calcification and abdominal aortic calcification scores.

67 nificantly between groups, nor did abdominal aortic calcification, serum phosphate, parathyroid hormo

68 Conclusion CT-based abdominal aortic calcification was a strong predictor of future ca

69 Aortic calcification was diagnosed by examining the post

70 Moderate aortic calcification was present on prior chest X-ray.

71 vo study with injury-induced local abdominal aortic calcification, we showed that elastin antibody-co

72 ent levels of uremia, hyperphosphatemia, and aortic calcification were induced by feeding nephrectomi

73 , 2.87; 95% confidence interval: 1.22, 6.72) aortic calcifications were associated with leakage.

74 ed to the progression of coronary artery and aortic calcification, whereas sevelamer attenuated or ar