戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 terial compliance, and, notably, significant aortic dilatation.
2 r cell apoptosis, all of which contribute to aortic dilatation.
3 receptor leads to a significant reduction in aortic dilatation.
4 rd elucidating 1 gene defect responsible for aortic dilatation.
5 sought on the screened prevalence of BAV and aortic dilatation.
6 lls and extracellular matrix associates with aortic dilatation.
7 ng techniques, with the finding of ascending aortic dilatation (4.7 to 9 cm) in all 9 and significant
8 ts outside the central region presented with aortic dilatation (55.0% versus 9.9%; P<0.001) more ofte
9 atives with BAV (29.2%; 95% CI 15.3%-45.1%), aortic dilatation alongside tricuspid aortic valves was
10        However, the prevalence of BAV and of aortic dilatation amongst family members is uncertain.
11                            The prevalence of aortic dilatation amongst relatives was 9.4% (95% CI 5.7
12      The role of atherosclerosis in thoracic aortic dilatation and aneurysm formation is poorly defin
13 ive analysis included patients with thoracic aortic dilatation and at least two CT angiographic exami
14 on of the aortic wall leading to progressive aortic dilatation and eventual rupture.
15  mutant mMFS:Il11ra1(-/)(-) mice had reduced aortic dilatation and exhibited lesser fibrosis, inflamm
16         Enhanced HBP activity contributed to aortic dilatation and medial degeneration via vascular s
17                                              Aortic dilatation and other structural cardiac abnormali
18 d the effects of losartan and doxycycline on aortic dilatation and survival in Fbn1(mgR/mgR) mice.
19  problems, especially mitral valve prolapse, aortic dilatation, and aortic dissection, has resulted i
20                       These data demonstrate aortic dilatation, aortic medial degeneration, and alter
21               No patient in either group had aortic dilatation at any level of >20% or more than mild
22 sic aortic abnormalities in BAVs account for aortic dilatation beyond that caused by valvular hemodyn
23                                  Progressive aortic dilatation, defined as an increase in diameter >0
24                                The extent of aortic dilatation (DeltaAD) was determined with AAAs def
25 relation coefficient > 0.9) and diagnosis of aortic dilatation did not demonstrate significant interf
26 ion of children at risk of developing severe aortic dilatation during their pediatric follow-up is st
27 o determine the natural history of ascending aortic dilatation following AVR.
28 is for patients with and without progressive aortic dilatation following surgical replacement of the
29                    Patients with progressive aortic dilatation had higher luminal pressure difference
30 imed to assess the effects of fenofibrate on aortic dilatation in a mouse model of AAA.
31  of aortic regurgitation are associated with aortic dilatation in BAV patients, intrinsic pathology a
32 ine the effects of irbesartan on the rate of aortic dilatation in children and adults with Marfan syn
33 s associated with a reduction in the rate of aortic dilatation in children and young adults with Marf
34 f this study was to identify risk factors of aortic dilatation in children with BAV.
35 s to summarize the recent data pertaining to aortic dilatation in congenital heart disease (CHD) and
36      It has been shown that losartan reduces aortic dilatation in patients with Marfan syndrome.
37 g that atherosclerosis plays a minor role in aortic dilatation in the population.
38 ltiple factors predispose this population to aortic dilatation, including underlying genetics, cellul
39 n aortic VSMCs to cause ERK-related thoracic aortic dilatation, inflammation, and fibrosis.
40                                              Aortic dilatation is a common finding in CHD.
41  elastic blood vessel, we show that abnormal aortic dilatation is associated with a transition from s
42                                     Although aortic dilatation is common in CHD, data about adverse l
43 able aortic diameters (N = 9) and those with aortic dilatation (N = 8).
44 4 versus 3.9+/-0.6 cm, p=NS) and progressive aortic dilatation occurred in only 10 patients (9.3%).
45                  No complications related to aortic dilatation occurred in this cohort.
46 bioavailability is associated with ascending aortic dilatation, outflow tract malrotation, overriding
47 eurysms (P<0.05), while A2AR agonism limited aortic dilatation (P<0.05).
48                        It is unknown whether aortic dilatation precedes acute aortic dissection in TS
49                    No patients with baseline aortic dilatation (range, 3.5 to 5.3 cm) dilated beyond
50  the aorta of 75 subjects: BAV patients with aortic dilatation stratified by leaflet fusion pattern (
51 ic plaques are weakly associated with distal aortic dilatation, suggesting that atherosclerosis plays
52 mended threshold of aortic root or ascending aortic dilatation that would justify surgical interventi
53                                              Aortic dilatation was accompanied by changes in the elas
54                                              Aortic dilatation was monitored by ultrasound imaging.
55                     Whilst the prevalence of aortic dilatation was particularly high in relatives wit
56                                              Aortic dilatation was present in 29% of the patients, an
57  treatment and posttreatment with rTMD123 on aortic dilatation were measured using the CaCl2-induced
58 r inhibitor, in Marfan syndrome might reduce aortic dilatation, which is associated with dissection a
59 to establish a reproducible porcine model of aortic dilatation, which recapitulates the structural an