ライフサイエンスコーパス: aortic insufficiency

1 s, pacemaker implantation rates, or moderate aortic insufficiency.

2 rrhage, thromboembolic event, or progressive aortic insufficiency.

3 continuous-flow VADs, including bleeding and aortic insufficiency.

4 he aortic cusps in aortic root dilation with aortic insufficiency.

5 long-term follow-up suggests an increase in aortic insufficiency.

6 One study had aortic insufficiency.

7 in the descending aorta after creation of an aortic insufficiency.

8 The second patient developed new moderate aortic insufficiency.

9 e aortic valve: 2 for endocarditis and 3 for aortic insufficiency.

10 d severe, 4 moderate, and 3 mild-to-moderate aortic insufficiency.

11 re laboratory-adjudicated moderate-to-severe aortic insufficiency (4.2% versus 13.7%; P=0.04), compar

12 Valve morphology, aortic stenosis (AS), and aortic insufficiency (AI) have been proposed as potentia

13 Aortic insufficiency (AI) is increasingly recognized as

14 lationship between neo-aortic root size, neo-aortic insufficiency (AI), and reintervention at mid-ter

15 ients to right-sided heart failure (RHF) and aortic insufficiency (AI), especially as patients live l

16 s with symptomatic and asymptomatic isolated aortic insufficiency (AI).

17 e in 41 patients: aortic stenosis (AS; n=3), aortic insufficiency (AI; n=11), and AS/AI (n=27).

18 her incidence of residual moderate or severe aortic insufficiency among patients with bicuspid AV (2.

19 lure (HF) was induced in rabbits by combined aortic insufficiency and aortic constriction.

20 s associated with greater risk of developing aortic insufficiency and aortic valve reoperation than r

21 F rabbit ventricular myocytes (HF induced by aortic insufficiency and constriction).

22 ical approach for following up patients with aortic insufficiency and helps to determine the optimal

23 aging, demonstrating low rates of mortality, aortic insufficiency and re-intervention.

24 al but also appear to have a reduced risk of aortic insufficiency and thromboembolic complications.

25 ding to bilateral hip replacement by age 30, aortic insufficiency, and hypertension.

26 Urgent or emergent surgery, aortic insufficiency, and perioperative stroke or renal

27 Rate of aortic insufficiency at 15 years was 7.9 +/- 3.3%, lower

28 SVD presented mostly as acute, severe aortic insufficiency attributed to leaflet tear (77.8%).

29 rhage, thromboembolic event, and progressive aortic insufficiency, create substantial morbidity and m

30 ersus 96.3; P=0.001) and the incidence of 2+ aortic insufficiency declined (14.0% versus 2.7%; P<0.00

31 (hazard ratio: 1.1; p = 0.01), pre-operative aortic insufficiency (hazard ratio: 2.7; p = 0.002), and

32 < 0.001) and pre-operative presence of pure aortic insufficiency (HR: 1.74; 95% CI: 1.13 to 2.68; p

33 Sensitivity analyses excluded pure aortic insufficiency, intermediate/high risk (STS predic

34 Aortic insufficiency is a valvular disease characterized

35 ic rupture, or congestive heart failure from aortic insufficiency occur.

36 Adults presenting with aortic insufficiency or a dilated aortic annulus or asce

37 ons (OR, 1.89; CI, 1.10-3.24; P = 0.02), and aortic insufficiency (OR, 1.85; CI, 1.26-2.71; P = 0.002

38 95% +/- 4% for subvalvar aortic stenosis or aortic insufficiency (p = 0.01).

39 cranial hemorrhage (P=0.015) and progressive aortic insufficiency (P=0.078) but not for thromboemboli

40 patients with ascending aortic aneurysm and aortic insufficiency secondary to dilated sinotubular ju

41 No patient had aortic insufficiency, significant coronary artery diseas

42 e cumulative incidence of moderate or severe aortic insufficiency was 14.5% (95% CI: 9.5%-22.0%), reo

43 Tricuspid regurgitation and aortic insufficiency were the predominant echocardiograp

44 Severe aortic insufficiency with leaflet tear is the major mode