ライフサイエンスコーパス: aortic regurgitation

1 predictors of valve surgery in asymptomatic aortic regurgitation.

2 e subset of patients with no post-procedural aortic regurgitation.

3 es across the aortic valve and the degree of aortic regurgitation.

4 lower prosthesis-patient mismatch, and more aortic regurgitation.

5 alve during the index procedure for residual aortic regurgitation.

6 gradient, with no worsening of paravalvular aortic regurgitation.

7 ondiscriminatory in relation to post-TAVR PV aortic regurgitation.

8 Hg, and 95% of patients had </=+1 degree of aortic regurgitation.

9 eries, dilatation of the neoaortic root, and aortic regurgitation.

10 6 pigs, one being excluded because of severe aortic regurgitation.

11 not involve prosthetic heart valves or acute aortic regurgitation.

12 mination who had no more than mild mitral or aortic regurgitation.

13 lmonary stenosis, pulmonary hypertension, or aortic regurgitation.

14 tion and 43 patients (86%) had at least mild aortic regurgitation.

15 erienced no change in severity of mitral and aortic regurgitation.

16 in a significantly higher incidence of mild aortic regurgitation.

17 >6 months and predominantly results in mild aortic regurgitation.

18 ay be heterogeneous in patients with chronic aortic regurgitation.

19 cular systolic or diastolic dysfunction, and aortic regurgitation.

20 gnosed cardiac-valve disorders, particularly aortic regurgitation.

21 quals mitral inflow volume in the absence of aortic regurgitation.

22 gitation, ventricular septal defect flow and aortic regurgitation.

23 cally in eight sheep with surgically induced aortic regurgitation.

24 valve positioning and reduction of residual aortic regurgitation.

25 onary (n = 3) leaflet was excised to produce aortic regurgitation.

26 forward stroke volume remained unchanged in aortic regurgitation.

27 rofile method for evaluating the severity of aortic regurgitation.

28 afterload mismatch dominates the response in aortic regurgitation.

29 ration in asymptomatic patients with chronic aortic regurgitation.

30 thod can be used to evaluate the severity of aortic regurgitation.

31 mia, hemolysis, device failure, or worsening aortic regurgitation.

32 There was no residual moderate or severe aortic regurgitation.

33 mm Hg and 40+/-14 mm Hg, whereas 50% had >2+ aortic regurgitation.

34 vascular complications and less paravalvular aortic regurgitation.

35 not occur in the 26 patients with persistent aortic regurgitation.

36 strokes, and incidence of moderate or severe aortic regurgitation.

37 rwent additional balloon dilation to correct aortic regurgitation.

38 of CT-ADP in order to identify patients with aortic regurgitation.

39 ost pronounced in patients without post-TAVI aortic regurgitation.

40 nd low New York Heart Association class with aortic regurgitation.

41 +/-8 and 53+/-17 mm Hg, whereas 28% had >II+ aortic regurgitation.

42 rease in MVA than did those with trace or no aortic regurgitation (0.19 vs. 0.086 cm2/year, p < 0.05)

43 HMW multimers normalized in patients without aortic regurgitation (137 patients).

44 gher rate of moderate or severe paravalvular aortic regurgitation (15.3%) than in-range (6.5%) or abo

45 ere was a higher prevalence of any degree of aortic regurgitation (17.0 percent vs. 11.8 percent, P=0

46 ; 95% CI, 1.02-2.29), and moderate to severe aortic regurgitation (22.4% vs 14.7%; HR, 2.05; 95% CI,

47 ears), including 46 normal subjects, 52 with aortic regurgitation, 253 with mitral regurgitation and

48 and a higher incidence of moderate or severe aortic regurgitation (3.5% vs. 0.5%) and pacemaker impla

49 surgery group had at least mild paravalvular aortic regurgitation (33.3% vs. 6.3%).

50 hree [1%]) and moderate or severe prosthetic aortic regurgitation (34 [9%] vs ten [3%]) were more com

51 y lower frequency of residual more-than-mild aortic regurgitation (4.1% vs 18.3%; RR, 0.23; 95% CI, 0

52 ) tracts, 50 normal volunteers without MR or aortic regurgitation (44+/-5 years, 31 male) were studie

53 , 1179 with tricuspid regurgitation, 817 had aortic regurgitation, 471 with aortic stenosis, and 193

54 ave aortic root dilatation (92% versus 84%), aortic regurgitation (55% versus 36%), and to have under

55 ation had significantly more mild or greater aortic regurgitation (60% vs. 4%, p < 0.0001), moderate

56 on, with a significantly higher frequency of aortic regurgitation (622 [33.1%] vs 57 [24.1%], P < .00

57 More TAVR patients had moderate/severe total aortic regurgitation (8.2% vs. 0.0%, p<0.001) and a new

58 Aortic regurgitation (a previously unreported problem) w

59 vasodilator therapy in patients with chronic aortic regurgitation, a reduction in left ventricular vo

60 ently associated with the presence of severe aortic regurgitation, abscess, embolization before surgi

61 ho had at least mild mitral regurgitation or aortic regurgitation after exposure to fenfluramines on

62 aphy resulted in lower rates of paravalvular aortic regurgitation after self-expanding transcatheter

63 ADP, were each predictive of the presence of aortic regurgitation after TAVR and were associated with

64 4; p < 0.001; presence of moderate or severe aortic regurgitation after TAVR, HR: 2.79, 95% CI: 1.82

65 Patients with aortic regurgitation after the initial implantation, as

66 mensional (2D) TEE as methods for predicting aortic regurgitation after transcatheter aortic valve re

67 late-onset ND abnormalities are common; 10) aortic regurgitation and aortic root dilation are well t

68 in one patient and ventricular dysfunction, aortic regurgitation and atrioventricular valve regurgit

69 cular arrhythmia, mitral or aortic stenosis, aortic regurgitation and chronic obstructive pulmonary d

70 alve were significantly associated with mild aortic regurgitation and elevated peak velocities across

71 In patients with severe aortic regurgitation and LV dysfunction, older age and h

72 s, whereas TAVR had higher rates of residual aortic regurgitation and need for pacemaker implantation

73 No changes in residual aortic regurgitation and no cases of structural valve fa

74 diography for diagnosis of acute significant aortic regurgitation and pericardial tamponade was 100%.

75 e) with isolated moderately severe to severe aortic regurgitation and preserved left ventricular (LV)

76 enosis in combination with at least moderate aortic regurgitation and preserved left ventricular func

77 Classic physical findings such as aortic regurgitation and pulse deficit were noted in onl

78 Greater than or equal to III+ aortic regurgitation and severe aortic stenosis were see

79 Aortic valve stenosis (with or without aortic regurgitation and without associated mitral steno

80 ic aortic valves (with or without associated aortic regurgitation and without associated mitral valve

81 aortic stenosis (with or without associated aortic regurgitation) and without associated mitral sten

82 ired normal left ventricular function, </=2+ aortic regurgitation, and >/=2 echocardiograms performed

83 .29; 95% confidence interval, 2.46-11.4) for aortic regurgitation, and 19.4% versus 9.6% (odds ratio,

84 In 18 sheep (4 normal, 6 with aortic regurgitation, and 8 with old myocardial infarcti

85 ction <30%), the presence of moderate/severe aortic regurgitation, and chronic obstructive pulmonary

86 ortic stenosis populations, in patients with aortic regurgitation, and in patients with bicuspid aort

87 idual gradients, no cases of moderate-severe aortic regurgitation, and none-trace residual aortic reg

88 disease, previous acute HF, grade III or IV aortic regurgitation, and pulmonary hypertension both at

89 ociety of Thoracic Surgeons score, degree of aortic regurgitation, and right ventricular systolic pre

90 , hypertension, hyperlipidemia, medications, aortic regurgitation, and right ventricular systolic pre

91 Inherited aortopathies, moderate to severe aortic regurgitation, and severe aortic stenosis were se

92 severe aortic stenosis, moderate and severe aortic regurgitation, and uncorrected coarctation of the

93 d management of the valve disease, including aortic regurgitation, aortic root dilation, hypertension

94 trol patients with matched valvular lesions (aortic regurgitation, aortic stenosis, or mixed lesions)

95 More patients had none/trivial aortic regurgitation (AR) (47.5% vs. 33%), and fewer had

96 Aortic regurgitation (AR) after transcatheter aortic val

97 gitation (MR) is common in those with severe aortic regurgitation (AR) and can predispose to atrial f

98 ve replacement (AVR) in patients with severe aortic regurgitation (AR) and left ventricular (LV) dysf

99 CMR) imaging allow quantification of chronic aortic regurgitation (AR) and mitral regurgitation (MR).

100 ptomatic/minimally symptomatic patients with aortic regurgitation (AR) and normal left ventricular (L

101 (S3-THV) incorporates new features to reduce aortic regurgitation (AR) and vascular complications in

102 MR) plus hemodynamically-significant chronic aortic regurgitation (AR) are mostly unknown.

103 patients with mitral regurgitation (MR) and aortic regurgitation (AR) continues to elicit uncertaint

104 ient reduction was 54+/-26%, and significant aortic regurgitation (AR) developed in 15% of patients.

105 procedure, or more than mild intraprosthetic aortic regurgitation (AR) either new or worsening from 3

106 ons for surgery in patients with significant aortic regurgitation (AR) focus on symptoms and left ven

107 Residual aortic regurgitation (AR) following transcatheter aortic

108 Post-implantation aortic regurgitation (AR) grade >/= 2 occurred in 28.4%

109 (MAVD) and isolated aortic stenosis (AS) or aortic regurgitation (AR) has not been performed, making

110 Chronic severe aortic regurgitation (AR) imposes significant volume and

111 (AS) in 2152 patients (41.2% of native VHD), aortic regurgitation (AR) in 279 (5.3%), mitral stenosis

112 o determine the prevalence and correlates of aortic regurgitation (AR) in a population-based sample g

113 rdiography (TEE) to define the mechanisms of aortic regurgitation (AR) in acute type A aortic dissect

114 -sectional area to determine the severity of aortic regurgitation (AR) in an animal model.

115 (3D) method for determining the severity of aortic regurgitation (AR) in an experimental animal mode

116 ography, angiography, and measurement of the aortic regurgitation (AR) index, which is calculated as

117 Significant postprocedural aortic regurgitation (AR) is observed in 10% to 20% of c

118 The natural history of stage B aortic regurgitation (AR) is unknown.

119 in patients with hemodynamically significant aortic regurgitation (AR) is unknown.

120 Neo-aortic root dilation (ARD) and neo-aortic regurgitation (AR) may be progressive after arter

121 valve (MV) enlargement occurring in chronic aortic regurgitation (AR) prevents functional mitral reg

122 The causes of aortic regurgitation (AR) severe enough to warrant aorti

123 lor flow vena contracta (VC) measurements of aortic regurgitation (AR) severity by comparing them to

124 s), elevated transaortic flow velocities and aortic regurgitation (AR) was determined.

125 The percentage of moderate or severe aortic regurgitation (AR) was low and not statistically

126 undergoing aortic valve surgery for chronic aortic regurgitation (AR), we sought to: 1) compare surv

127 function-either aortic stenosis (AS) or pure aortic regurgitation (AR)-something not previously under

128 nappreciated cause of both acute and chronic aortic regurgitation (AR).

129 ranscatheter aortic valve replacement (TAVR) aortic regurgitation (AR).

130 to 2D-TEE for the prediction of paravalvular aortic regurgitation (AR).

131 therapy on survival in patients with severe aortic regurgitation (AR).

132 es and outcomes of patients with significant aortic regurgitation (AR).

133 for neo-aortic root size and severity of neo-aortic regurgitation (AR).

134 fibrosis is common in patients with chronic aortic regurgitation (AR).

135 ve replacement (AVR) in patients with severe aortic regurgitation (AR).

136 g, but this method has not been validated in aortic regurgitation (AR).

137 on of effective regurgitant orifice (ERO) of aortic regurgitation (AR).

138 oms on survival after surgical correction of aortic regurgitation (AR).

139 gic stresses and energetic adaptation during aortic regurgitation (AR).

140 lacement (TAVR) in patients with pure native aortic regurgitation (AR).

141 s the multimodality diagnostic evaluation of aortic regurgitation (AR).

142 Using FDA criteria (aortic regurgitation [AR] > or =mild and/or mitral regur

143 Although more severe degrees of aortic regurgitation are associated with aortic dilatati

144 Residual gradient and residual aortic regurgitation are major predictors of midterm and

145 troke, nonfemoral access, and postprocedural aortic regurgitation are predictors of adverse outcome,

146 ogression of discrete subaortic stenosis and aortic regurgitation, as well as reoperation.

147 for symptomatic moderate-to-severe or severe aortic regurgitation at 30 centres in the USA.

148 versus 3.9%; P=0.01), and moderate or severe aortic regurgitation at 30 days (10% versus 3%; P=0.002)

149 was associated with more moderate or severe aortic regurgitation at 30 days and cardiac death at 30

150 Paravalvular aortic regurgitation at 30 days was moderate or higher i

151 with BAV had more frequently moderate/severe aortic regurgitation at first presentation compared with

152 ation at any level of >20% or more than mild aortic regurgitation at up to 4 years of follow-up.

153 1) undergoing operation for severe isolated aortic regurgitation between 1980 and 1989 were compared

154 and 198 men undergoing surgery for isolated aortic regurgitation between 1980 and 1989.

155 The VCW is a reliable measure of mitral and aortic regurgitation, but its value in measuring TR is u

156 for the quantification of aortic stenosis or aortic regurgitation by application of Doppler technique

157 Aortic regurgitation can be difficult to diagnose and qu

158 Chronic aortic regurgitation can lead to significant morbidity a

159 the outcome of aortic valve replacement for aortic regurgitation complicated by extreme left ventric

160 The outcome of aortic regurgitation conservatively followed in clinical

161 optimal timing of surgical repair in chronic aortic regurgitation continues to be a topic of interest

162 r aortic valve implantation patients without aortic regurgitation (correction) and transcatheter aort

163 wer rates of moderate or severe paravalvular aortic regurgitation: DAR </=10%, 17.6%; DAR 10% to 15%,

164 This study, using quantified aortic regurgitation, demonstrates that the flow converg

165 on size, valve prosthesis type, and residual aortic regurgitation determined an increased risk.

166 46 patients with severe or moderately severe aortic regurgitation diagnosed by color Doppler echocard

167 days, the incidence of moderate paravalvular aortic regurgitation did not increase, and no associatio

168 sment of hemostasis could be used to monitor aortic regurgitation during TAVR.

169 points included the presence of paravalvular aortic regurgitation evaluated by an independent echocar

170 ilar rate of moderate-to-severe paravalvular aortic regurgitation (Evolut R/PRO 10.5% versus Sapien 3

171 mes spanning from aortic forward velocity to aortic regurgitation fraction.

172 o 27.2, 95% CI 1.2 to 619.6, p = 0.0386) and aortic regurgitation (GEE risk ratio 2.4, 95% CI 1.3 to

173 Aortic regurgitation grade > or = 3 (RR 4.27, p = 0.04)

174 (p = 0.02), and post-implant periprosthetic aortic regurgitation grade >/=2 of 4 (p < 0.001).

175 Post-procedure aortic regurgitation grade I or lower was present in 34

176 ent (10.0+/-3.4 vs 9.7+/-4.1 mm Hg; P=0.58), aortic regurgitation >/=2 of 4 (19.0% vs 14.9%; P=0.54),

177 Aortic regurgitation >/=mild after TAVR was associated w

178 Preimplantation aortic regurgitation >/=mild was associated with reduced

179 ore TAVR and mean aortic gradient >20 mm Hg, aortic regurgitation >= grade 2, and percutaneous corona

180 Patients with associated aortic regurgitation had a higher rate of decrease in MV

181 After adjustment for aortic velocities, aortic regurgitation had no significant impact on surviv

182 Doppler assessment of the progression of aortic regurgitation has been shown in larger studies th

183 than a century, numerous eponymous signs of aortic regurgitation have been described in textbooks an

184 Indications for surgical correction of aortic regurgitation have been established mostly in men

185 ft ventricular dilation and symptom onset in aortic regurgitation have been performed, with the data

186 asive methods for evaluating the severity of aortic regurgitation have not been accepted widely nor c

187 score (hazard ratio, 1.35), higher grades of aortic regurgitation (hazard ratio, 1.29), and higher ri

188 fidence interval [CI], 1.006-1.13; P = .03), aortic regurgitation (HR, 10.2; 95% CI, 3.2-32.2; P < .0

189 e subset of patients with no post-procedural aortic regurgitation (HR: 1.88; p = 0.02).

190 rative to precisely quantify the severity of aortic regurgitation immediately after valve implantatio

191 ast one grade in 17 patients (P = 0.001) and aortic regurgitation improved by at least one grade in 1

192 50 mm Hg in 65% of neonates and significant aortic regurgitation in 14%.

193 re aortic regurgitation (none-trace and mild aortic regurgitation in 76% and 24% of patients, respect

194 cardiography results demonstrated mild or no aortic regurgitation in 99% (73 of 74) with a mean gradi

195 Patients diagnosed with severe aortic regurgitation in clinical practice incur excess m

196 ence was primarily due to more frequent mild aortic regurgitation in dexfenfluramine patients (6.3% v

197 The indication for TV-in-TV was significant aortic regurgitation in most patients, often due not onl

198 urgitation in three (0.5%) of 569 and severe aortic regurgitation in none.

199 OA, decreased ejection fraction, and greater aortic regurgitation in TAVR patients; and smaller left

200 ortic regurgitation, and none-trace residual aortic regurgitation in the majority of patients.

201 implantation in 127 (21.6%) of 589, moderate aortic regurgitation in three (0.5%) of 569 and severe a

202 ith pulmonary atresia and moderate or severe aortic regurgitation in univariate analyses, but no inde

203 Aortic regurgitation increased from trivial (none to mil

204 with aortic stenosis and nearly 5 years with aortic regurgitation; independent correlates included sm

205 Development of transcatheter approaches for aortic regurgitation is challenging, owing to the absenc

206 Moderate or severe aortic regurgitation is common after TAVR and an adverse

207 low tract gradient slowly increases and mild aortic regurgitation is common, although generally nonpr

208 tors, although stroke risk is equivalent and aortic regurgitation is less.

209 Acute aortic regurgitation is life-threatening with few nonsur

210 t textbook support of the eponymous signs of aortic regurgitation is not matched by the literature.

211 reduced ejection fraction, and isolated pure aortic regurgitation is now under investigation in clini

212 Extreme left ventricular dilation due to aortic regurgitation is observed in male patients and is

213 Similarly, moderate or severe aortic regurgitation is uncommon.

214 of surgery in asymptomatic mild to moderate aortic regurgitation may be predicted by indices of left

215 Forty-five patients with chronic aortic regurgitation (mean age 50+/-14 years) were studi

216 rupt onset of chest or back pain) and signs (aortic regurgitation murmur or pulse deficits) of dissec

217 aortic stenosis (n=14), dilated root without aortic regurgitation (n=15), and dilated root with aorti

218 regurgitation (n=15), and dilated root with aortic regurgitation (n=20).

219 ance Imaging in the Assessment of Mitral and Aortic Regurgitation; NCT04038879).

220 ts with residual high shear or with residual aortic regurgitation, no recovery of HMW multimers was o

221 -4 mm Hg) and the absence of moderate-severe aortic regurgitation (none-trace and mild aortic regurgi

222 Among the 46 patients with aortic regurgitation, normalization occurred in 20 patie

223 Aortic regurgitation occurred in 10% of a sample group o

224 Moderate or severe aortic regurgitation occurred in 40 (14%) of 280 patient

225 Aortic regurgitation occurred more often in the last 20

226 Postprocedural aortic regurgitation occurs in 10 to 20% of patients und

227 annular sizing approach resulted in less PV aortic regurgitation of grade worse than mild after TAVR

228 eria of the Food and Drug Administration for aortic regurgitation of mild or greater severity and mit

229 djusted odds ratio compared with controls of aortic regurgitation of mild or greater severity increas

230 These analyses showed that aortic regurgitation of mild or greater severity occurre

231 Aortic regurgitation of mild or greater severity, mitral

232 Aortic regurgitation (of any degree) was present in 13.8

233 FDA-grade regurgitation (at least mild aortic regurgitation or at least moderate mitral regurgi

234 eter aortic valve implantation patients with aortic regurgitation or balloon valvuloplasty patients (

235 sion was more often associated with moderate aortic regurgitation or greater (odds ratio 2.4, 95% con

236 c regurgitation vs 56.6% for those with mild aortic regurgitation or less; p=0.003).

237 Drug Administration criteria (at least mild aortic regurgitation or moderate mitral regurgitation).

238 No patient had significant aortic regurgitation or other valvular disease.

239 type A IMH were less likely to present with aortic regurgitation or pulse deficits and were more lik

240 nd 1999 with ejection fraction > or =50% and aortic regurgitation or stenosis, absent or mild.

241 tenosis, severe mitral regurgitation, severe aortic regurgitation, or subaortic stenosis.

242 rgical criteria defined for aortic stenosis, aortic regurgitation, or the aorta are reached.

243 (p = 0.019), and more total and paravalvular aortic regurgitation (p < 0.0001).

244 Older patients more often had post-dilation aortic regurgitation (p < 0.001).

245 05) and there was a higher incidence of mild aortic regurgitation (p < 0.05).

246 in both dexfenfluramine groups had decreased aortic regurgitation (P = 0.003 for the dexfenfluramine

247 at aortic diameters increased with worsening aortic regurgitation (P:<0.001) and advancing age (P:<0.

248 imensions for the prediction of paravalvular aortic regurgitation (PAR) following transcatheter aorti

249 Paravalvular aortic regurgitation (PAR) negatively affects the progno

250 point-of-care assessment of peri-prosthetic aortic regurgitation (periAR) during transcatheter aorti

251 was a predictor for progression to moderate aortic regurgitation postoperatively.

252 aphic findings at discharge (grade III or IV aortic regurgitation, pulmonary hypertension) identified

253 Echocardiographic evidence suggests that aortic regurgitation regresses in some previously treate

254 y of requiring 2 valves and leaving residual aortic regurgitation remain important considerations.

255 echocardiography in the surgical timing for aortic regurgitation remains a matter of debate because

256 In asymptomatic aortic regurgitation, resting LV strain, resting RV stra

257 between the popliteal-brachial gradient and aortic regurgitation severity.

258 lity, suggesting that surgical correction of aortic regurgitation should be considered at an earlier

259 f mortality, strokes, and moderate or severe aortic regurgitation, TAVR was both non-inferior (pooled

260 tic valve dysfunction, men had more frequent aortic regurgitation than women (33.8% versus 22.2%, P<0

261 had baseline bicuspid aortic valve and mild aortic regurgitation that progressed to moderate regurgi

262 an important study in patients with chronic aortic regurgitation, the optimal timing of surgery in a

263 er of the aortic root; the rate of change in aortic regurgitation; the time to aortic dissection, aor

264 r adaptation occurs in patients with chronic aortic regurgitation to maintain left ventricular (LV) p

265 There were six cases of aortic regurgitation, two cases of mitral regurgitation,

266 chronic animal model with surgically created aortic regurgitation using a new semiautomated color Dop

267 the TAVR group (72.4% for moderate or severe aortic regurgitation vs 56.6% for those with mild aortic

268 as 8.5 +/- 5.6 mm Hg, and moderate or severe aortic regurgitation was 1.9% at discharge.

269 The prevalence of moderate or severe aortic regurgitation was 3.5% (95% confidence interval,

270 as 4.1%, and the incidence of periprosthetic aortic regurgitation was 64.5%.

271 Aortic regurgitation was assessed by color flow Doppler

272 For each steady state, the severity of aortic regurgitation was assessed by measurement of the

273 ultiple CT annular measures for post-TAVR PV aortic regurgitation was compared with 2D echocardiograp

274 itral regurgitation jet length>/=2 cm or any aortic regurgitation was considered best suited to be FC

275 At the end of the follow-up, trivial or no aortic regurgitation was demonstrated in 33.3%, mild in

276 nd Drug Administration criteria, significant aortic regurgitation was detected in 15 subjects (6.6%)

277 0.001), although moderate or severe residual aortic regurgitation was higher in TAVR patients (6.8% v

278 cedural months (19% versus 12%; P=0.01); but aortic regurgitation was less (34% versus 52% mild and 8

279 h severe aortic stenosis and complex anatomy aortic regurgitation was less than moderate in 99% of pa

280 frequency of moderate or severe paravalvular aortic regurgitation was lower 12 months after self-expa

281 Rate of mild aortic regurgitation was lower with the repositionable a

282 decrease, 35 mm Hg), and acute post-dilation aortic regurgitation was moderate or greater in 70 patie

283 spite relief of the obstruction, progressive aortic regurgitation was noted at follow-up after 14 pro

284 higher rate of moderate-severe paravalvular aortic regurgitation was observed in the Evolut R/PRO gr

285 Mild aortic regurgitation was present in 68% but generally di

286 Mild or greater aortic regurgitation was present in 8.8% of treated pati

287 Aortic regurgitation was quantified using standard crite

288 Moderate to severe aortic regurgitation was reduced from 45.1% at pre-TAVR

289 Eight to 20 weeks after aortic regurgitation was surgically induced in six sheep

290 but progressive valve disease (predominantly aortic regurgitation) was identified in 17% of adult and

291 Nonfemoral access and postprocedural aortic regurgitation were also significant predictors of

292 AS gradient and lower grade of post-dilation aortic regurgitation were associated with longer freedom

293 ary artery disease, or greater than moderate aortic regurgitation were excluded.

294 Echocardiographic changes in aortic regurgitation were observed in 8 controls (7 [1.7

295 d to FDA-grade regurgitation or any grade of aortic regurgitation were older age, higher diastolic bl

296 es mellitus, and moderate to severe residual aortic regurgitation were significantly associated with

297 an gradient and incidence of moderate/severe aortic regurgitation were similar in both groups at 1 ye

298 Mean gradient, valve area, and residual aortic regurgitation were stable during follow-up.

299 ic valve replacement has been recommended in aortic regurgitation with extreme left ventricular dilat

300 f 92.3%, 92.4%, and 98.6%, respectively, for aortic regurgitation, with similar results in the valida

301 ement or repair, and 31 patients with native aortic regurgitation without coexisting aortic stenosis.