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1 potonia, hyperelastic skin, hearing loss and aortic rupture.
2 r PKCbeta prevented death due to spontaneous aortic rupture.
3 argement, aneurysm formation, dissection and aortic rupture.
4 s a poor prognosis owing to the high risk of aortic rupture.
5 dysfunction and 30% lethality from abdominal aortic rupture.
6 ionally includes myopathy, hearing loss, and aortic rupture.
7 e that increase the risk of life-threatening aortic rupture.
8 n results in AAA formation with incidence of aortic rupture.
9  with or without nitroprusside in preventing aortic rupture.
10  efficacy of antihypertensives in preventing aortic rupture.
11 ihypertensive regimen eliminated in-hospital aortic rupture.
12 re is a lack of effective therapy preventing aortic rupture.
13 cative of impending topologic catastrophe or aortic rupture.
14  or hybrid interventions to treat or prevent aortic rupture.
15 isease, complications of reinterventions, or aortic rupture.
16 fitness and an elevated risk of aneurysm and aortic rupture.
17  or enlarging aortic aneurysms, and 11 acute aortic ruptures.
18 ection, 0.95 (95% CI, 0.61 to 1.49); and for aortic rupture, 0.81 (95% CI, 0.49 to 1.35).
19         There have been 23 reported cases of aortic rupture after endovascular treatment for CoA, inc
20 ase causes dilation of the aorta, leading to aortic rupture and death if not treated early.
21 ood pressure, aneurysm diameter, and time to aortic rupture and death.
22 uctural alterations, which frequently led to aortic rupture and death.
23  celecoxib also showed significantly reduced aortic rupture and mortality.
24 risk of periprocedural complications such as aortic rupture and tamponade was low (<1%) and similar b
25 n, 35 had intramural aortic hematoma, 18 had aortic rupture, and 10 had penetrating aortic ulcer.
26 exacerbated AAA, compromised survival due to aortic rupture, and inflammation in the abdominal aorta.
27 giotensin II (Ang II)-induced AAA formation, aortic rupture, and reduced survival.
28 nding aortic dissections, 1033 (9%) thoracic aortic ruptures, and 185 (2%) traumatic aortic tears.
29                                      ARM and aortic rupture are uncommon after elective FB-EVAR of as
30  days or in hospital) or late mortality from aortic rupture, dissection, organ or limb malperfusion a
31 However, a subset of these patients develops aortic rupture due to further dilatation of the residual
32 re no deaths and no instances of aneurysm or aortic rupture during the subsequent average follow-up p
33                                    Traumatic aortic rupture has received significant attention over t
34 extent I-III TAAAs were associated with late aortic rupture (hazard ratio, 5.85 [95% CI, 1.31-26.2];
35                             The incidence of aortic rupture in Ang II-infused GV DKO mice (10%) was s
36 Ehlers-Danlos syndrome, a disease leading to aortic rupture in early adult life.
37 s severe aortic pathology and mortality from aortic rupture in young mutant mice.
38            Delayed repair of acute traumatic aortic rupture is safe under appropriate treatment and s
39                                     Although aortic rupture is the major cause of mortality in MFS, p
40       LAEs were defined as fatal or nonfatal aortic rupture, new refractory hypertension or pain, org
41 n of the aorta (>=1 cm/y), fatal or nonfatal aortic rupture, new refractory pain, uncontrollable hype
42                                Fourteen late aortic ruptures occurred; 5-year cumulative incidence wa
43 uires surgery to prevent death from proximal aortic rupture or malperfusion.
44 hic complications such as aortic dissection, aortic rupture, or congestive heart failure from aortic
45 creased risk of aortic aneurysm formation or aortic rupture, particularly in the thoracic aorta.
46 rse events were defined as fatal or nonfatal aortic rupture, rapid aortic growth (>10 mm/y), aneurysm
47 ed AAA expansion and significant increase of aortic rupture rate.
48 tential for the noninvasive detection of the aortic rupture site prior to dilation of the aorta and t
49 ysms, and a higher incidence of death due to aortic rupture than Apoe(-/-) controls.
50 f pericardial tamponade or intra-pericardial aortic rupture, to resect the primary entry tear, to cor
51 verse aortic events (AAEs)-that is, thoracic aortic ruptures, type A aortic dissections, and thoracic
52                           Incidence of early aortic rupture was 0.4% (n=4).
53                        The major sign of the aortic rupture was apparent bleeding in 4 patients and f
54                     Unexpectedly, death from aortic rupture was significantly higher in Mmp12(-/-)/Ap
55                   MRI immediately identified aortic rupture when oversized devices were tested.
56 ciency increased SMC apoptosis and ascending aortic rupture with increased aortic pressure.