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1 eristic impedance (Zc), a measure of central aortic stiffness.
2 s likely account for the greater increase in aortic stiffness.
3 carotid and femoral arteries as a measure of aortic stiffness.
4 lapril, did not significantly reduce central aortic stiffness.
5 (cfPWV) is regarded as the gold standard for aortic stiffness.
6 mmation in AA, leading to ECM deposition and aortic stiffness.
7 sity may accelerate age-related increases in aortic stiffness.
8  microvascular function, and does not impair aortic stiffness.
9 tic DL-powered evaluation of the MRI-derived aortic stiffness.
10   The male mice had progressive, age-related aortic stiffness.
11 ion and vasodilatation responses, suggesting aortic stiffness.
12 se wave velocity (aPWV), a robust measure of aortic stiffness.
13 nt of alterations in angiotensin II-mediated aortic stiffness.
14 ident stroke independently of CV factors and aortic stiffness.
15 stroke risk prediction beyond Framingham and aortic stiffness.
16 n is predominantly correlated with increased aortic stiffness.
17  this effect correlates with the decrease in aortic stiffness.
18 sociations were also observed with follow-up aortic stiffness.
19  cancer therapy adversely increases thoracic aortic stiffness, a known independent predictor of adver
20 thesized that anthracycline use may increase aortic stiffness, a known predictor of CV events.
21                                    Increased aortic stiffness, a major mechanical factor predicting C
22 content and improved its solubility, reduced aortic stiffness, ameliorated diabetic nephrosclerosis,
23                         Rationale: Increased aortic stiffness, an important feature of many vascular
24                              DEL-1 prevented aortic stiffness and abolished the progression of hypert
25                      The association between aortic stiffness and all-cause mortality in kidney trans
26 es a measure of systemic arterial stiffness, aortic stiffness and central arterial pressure.
27 (PMAP), peripheral vascular resistance, AIx, aortic stiffness and central pulse pressure, but only an
28 ess associations between changes in proximal aortic stiffness and changes in fitness, fatness, and ot
29 ur results suggest that associations between aortic stiffness and CVD events are mediated by pathways
30  analyses, 8% to 13% of the relation between aortic stiffness and CVD events was mediated by hyperemi
31                       WD feeding resulted in aortic stiffness and endothelial dysfunction as determin
32 onsistent with the hypothesis that increased aortic stiffness and excessive flow pulsatility damage t
33 ysis showed that 34% of the relation between aortic stiffness and GFR was mediated by pulsatility ind
34  for pulsatility index, the relation between aortic stiffness and GFR was no longer significant (P=0.
35            However, the relationship between aortic stiffness and HCM has not been studied previously
36 at preceded left atrial dilatation linked to aortic stiffness and increased fibrosis in myocardium an
37          Our findings indicate that abnormal aortic stiffness and increased pressure pulsatility are
38 ediates adverse hemodynamics through related aortic stiffness and increased pulsatile load.
39                  Both training modes reduced aortic stiffness and increased whole body insulin sensit
40  and determine their relationship to central aortic stiffness and left ventricular (LV) remodeling.
41 ions of total arterial and proximal thoracic aortic stiffness and LV mass.
42                            Three measures of aortic stiffness and pressure pulsatility (carotid-femor
43            For example, higher levels of all aortic stiffness and pressure pulsatility measures (beta
44                                              Aortic stiffness and pressure pulsatility measures were
45           Adult OHF animals showed increased aortic stiffness and reduced endothelium-dependent relax
46                                              Aortic stiffness and small-artery structure and function
47 in lamellae and is responsible for increased aortic stiffness and subsequent cardiovascular events.
48 arable with EDS patients, they had decreased aortic stiffness and tensile strength and hyperextensibl
49         HFpEF is characterized by heightened aortic stiffness and unfavorable pulsatile left ventricu
50                           Inter-relations of aortic stiffness and vasodilator function with incident
51 ators of vascular phenotype severity such as aortic stiffness and vertebral tortuosity index have bee
52 which indexes such as aortic distensibility, aortic stiffness, and aortic compliance can be obtained
53 ining reversed the reduction in E/A, reduced aortic stiffness, and eliminated impairment of coronary
54 yocardial inflammation, myocardial fibrosis, aortic stiffness, and pericardial fat volume.
55 ntral pulse pressure (PP), PP amplification, aortic stiffness, and wave reflections.
56 prevalence and correlates of abnormally high aortic stiffness are incompletely understood.
57 nd without myocardial fibrosis, have altered aortic stiffness as assessed by magnetic resonance imagi
58 d whether this association is independent of aortic stiffness as estimated by carotid-femoral pulse w
59                                    Increased aortic stiffness, as indicated by increased PWV, is evid
60                                              Aortic stiffness, as represented by the pressure strain
61                                       Higher aortic stiffness assessed by PWV is associated with incr
62 creased filling pressures were correlated to aortic stiffness (augmentation pressure and index, P<0.0
63 i-corrected p < 0.0125) and higher follow-up aortic stiffness (B = -1.12, 95% CI -1.95, -0.29, Bonfer
64 terial tonometry, we evaluated 3 measures of aortic stiffness: brachial pulse pressure; carotid-femor
65             We assessed the relation between aortic stiffness by cardiac MRI, surgical root replaceme
66 were observed for cardiorespiratory fitness, aortic stiffness, circulating glucose, lipids and inflam
67 P amplification, mainly related to increased aortic stiffness, contributes to the significant increas
68                              Aging increases aortic stiffness, contributing to cardiovascular risk ev
69                       In addition, increased aortic stiffness correlates with myocardial fibrosis, an
70 al pulse wave velocity, a measure of central aortic stiffness, decreased after cranberry juice (8.3 +
71                                      Because aortic stiffness depends on the prevailing blood pressur
72 and small-sized arteries, showed that, while aortic stiffness did not change with increasing degree o
73 inear relationship of PMAP with both AIx and aortic stiffness did not differ significantly between dr
74 intensity reduces central blood pressure and aortic stiffness-equivalent to a ~4-year reduction in va
75 n is a promising tool for measuring regional aortic stiffness for non-invasive cardiovascular disease
76                       In this cohort, higher aortic stiffness, FWA, and augmentation index were assoc
77 ansplant recipients (KTRs) is uncertain, and aortic stiffness has not yet been incorporated into risk
78 ditionally, pulse pressure (PP), a marker of aortic stiffness, has been associated with increased ris
79                                     Abnormal aortic stiffness implies an unfavorable prognosis and ha
80 of microvascular structure and function, and aortic stiffness in 2045 participants (1107 women, mean
81 aortic arch is related to increased proximal aortic stiffness in individuals without cardiovascular d
82 rcise alone may not be sufficient to improve aortic stiffness in older adults with obesity.
83 mmation during the acute phase and increased aortic stiffness in the early postacute phase.
84 elationship between telomere length (TL) and aortic stiffness in well-characterized, younger and olde
85              PWV was higher (i.e., increased aortic stiffness) in HCM patients with myocardial fibros
86 nt for age, important correlates of abnormal aortic stiffness included higher mean arterial pressure,
87 n the participants receiving anthracyclines, aortic stiffness increased markedly (relative to baselin
88 eys (Macaca fascicularis) (n=7/group), where aortic stiffness increases by 200% in vivo.
89                   The prevalence of abnormal aortic stiffness increases steeply with advancing age in
90                                              Aortic stiffness increases with age and is a robust pred
91                                              Aortic stiffness increases with age and vascular risk fa
92    Increased pulse pressure, a reflection of aortic stiffness, increases cardiac load and may increas
93 - 1.6 x 10(-3) mm Hg, p < 0.001) and greater aortic stiffness index (26.7 +/- 25.8 and 55.9 +/- 76.8
94                                Thus, whereas aortic stiffness is a known independent predictor of mor
95                                              Aortic stiffness is associated with clinical hallmarks o
96                                       Higher aortic stiffness is associated with higher rates of surg
97                                    Increased aortic stiffness is associated with numerous common dise
98                                              Aortic stiffness is closely linked with cardiovascular d
99                             In other models, aortic stiffness is improved after the reduction of infl
100 t that the burden of disease attributable to aortic stiffness is likely to increase considerably over
101 atrilat compared with enalapril suggest that aortic stiffness is maintained by specific, partially re
102                                              Aortic stiffness is reduced through supervised training
103                    The resultant increase in aortic stiffness likely further worsens systolic hyperte
104 ese analyses provide the first evidence that aortic stiffness may contribute to lower GFR by transfer
105 dation of the role this novel locus plays in aortic stiffness may facilitate development of therapeut
106 ch as renal dysfunction and enhanced central aortic stiffness may play an important role in the devel
107   Being a factor of various severe diseases, aortic stiffness may play an important role in the early
108                                              Aortic stiffness, measured by pulse wave velocity, was a
109 ng adults with a CTD was performed to derive aortic stiffness measures (strain, distensibility, and b
110                                  None of the aortic stiffness measures changed significantly in the a
111                               We evaluated 2 aortic stiffness measures, carotid-femoral pulse wave ve
112 partum hypertensive stimuli develop enhanced aortic stiffness, microvascular myogenic tone, AngII con
113 erations in left ventricular function and in aortic stiffness occur during the early phase of aneurys
114         CONCLUSION A significant increase in aortic stiffness occurs within 4 months of exposure to a
115 id profile, adipose tissue inflammation, and aortic stiffness of LCR rats.
116  individuals (<50 years), whereas changes in aortic stiffness per se are more marked in older individ
117                             These changes in aortic stiffness persisted after accounting for age, sex
118 es included measures of myocardial fibrosis, aortic stiffness, physical functioning, quality of life
119                     In healthy participants, aortic stiffness positively correlated with age (rho = 0
120 in left ventricular stroke volume (LVSV) and aortic stiffness predict future pulmonary edema.
121                               Assessments of aortic stiffness (pulse pressure and characteristic impe
122                                      In vivo aortic stiffness (pulse wave velocity) increased progres
123 E/A), myocardial performance index (MPI) and aortic stiffness (pulse wave velocity; PWV) were evaluat
124 n IVRT, a 64% decrease in E/A, and increased aortic stiffness (PWV: 6.36 +/- 0.47 vs.4.89 +/- 0.41, O
125 ral hemodynamic parameters involve increased aortic stiffness, reduced wave reflections, and increase
126 sis factor-alpha therapy and correlates with aortic stiffness reduction.
127 cognitive impairment in the presence of high aortic stiffness remain unclear.
128      Results At the 4-month follow-up visit, aortic stiffness remained similar to baseline in the con
129 te this, the role of FFAs in obesity-related aortic stiffness remains unclear.
130 teractions with the immune cells to mitigate aortic stiffness remains unknown.
131 were independently associated with increased aortic stiffness, renal injury, and incident cardiovascu
132                              The increase in aortic stiffness results in increased cardiac workload a
133 lying clinical conditions known to influence aortic stiffness, such as hypertension or diabetes (P <
134 st, however, after additional adjustment for aortic stiffness, suggesting a shared causal pathway.
135                                              Aortic stiffness tended to decrease, but FMD was not cha
136 CR leads to greater improvements in proximal aortic stiffness than exercise alone.
137                                    Increased aortic stiffness that decreases LVSV during adrenergic s
138 h IFG to prevent LV hypertrophy and abnormal aortic stiffness that is observed in middle-aged and old
139 e velocity (CFPWV) is a heritable measure of aortic stiffness that is strongly associated with increa
140 ected five highly significant QTLs affecting aortic stiffness: two interacting QTLs (AS-m1 on chromos
141 included central (aortic) blood pressure and aortic stiffness using cardiovascular magnetic resonance
142                               The WD-induced aortic stiffness was associated with enhanced endothelia
143                                       Higher aortic stiffness was associated with higher LV mass, ind
144  cardiovascular disease risk factors, higher aortic stiffness was associated with increased LV mass (
145                                       Higher aortic stiffness was associated with older age, higher s
146                     Further, higher baseline aortic stiffness was associated with poor semantic fluen
147  for mean arterial pressure, each measure of aortic stiffness was associated with reduced hyperemic f
148                                              Aortic stiffness was corelated with left ventricular aft
149                                              Aortic stiffness was evaluated as the product of Young's
150                                Assessment of aortic stiffness was evaluated by measuring pulse wave v
151                                              Aortic stiffness was independently associated with HFpEF
152 modify arterial stiffness, proximal thoracic aortic stiffness was not increased in those with IFG com
153 ted for age, sex, heart rate, and body size, aortic stiffness was related to GFR (Slope of regression
154  pulse wave velocity, which reflects central aortic stiffness, was statistically significantly lower
155 in which factors known to influence thoracic aortic stiffness were included as covariates in the mode
156                                Correlates of aortic stiffness were similar if we used age-specific ra
157 trophy, diastolic dysfunction, and increased aortic stiffness, which are independent predictors of ca
158                        Findings suggest that aortic stiffness, which is potentially modifiable, may b
159 rmal (9.2 +/- 2.2 m/s), indicating increased aortic stiffness, which strongly correlated with NT-proB
160 H patients have increased apparent ascending aortic stiffness, which was strongly associated with the
161 anges in vascular properties responsible for aortic stiffness with aging would be greater in old male
162 as to discuss the methodology used to assess aortic stiffness, with particular emphasis on radiologic
163                                     Proximal aortic stiffness (Z(c)) is greater in women than men, an

 
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