コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 Evolut R/PRO or Sapien 3 valves in bicuspid aortic valve.
2 magnetic resonance imaging sequences of the aortic valve.
3 imal diameter > 4.0 cm), and 10 had bicuspid aortic valves.
4 integrate transcriptomic data from 233 human aortic valves.
5 c analyses were performed in human tricuspid aortic valves.
6 erogenesis, is upregulated in human calcific aortic valves.
7 at the aortic side of leaflets of explanted aortic valves.
8 d progression of calcium volume score in the aortic valve (14% [95% CI, 5-24] versus 98% [95% CI, 77-
9 individuals with Marfan syndrome or bicuspid aortic valve, a family history of AD was associated with
10 3 and Evolut R/PRO implantation in bicuspid aortic valve anatomy; a higher rate of moderate-severe p
11 nohistochemistry analyses, and assessment of aortic valve and cardiac function were determined by ech
12 rlson score. EE more frequently involved the aortic valve and prosthesis (64.3% vs. 46.7%; p < 0.001;
15 s with medically-managed isolated severe AS (aortic valve area < 1 cm(2)) and preserved LVEF (>50%) w
16 atients (50.4% men; mean age, 77 years) with aortic valve area <1.3 cm(2) and analyzed the occurrence
17 -five patients with LG severe AS (defined by aortic valve area <=1 cm(2) or aortic valve area indexed
18 7+/-5.6 versus 11.6+/-5.0 mm Hg; P=0.12) and aortic valve area (1.72+/-0.37 versus 1.76+/-0.42 cm(2);
20 ports a shared genetic etiology with between aortic valve area and birth weight along with other card
21 cestry participants, we estimated functional aortic valve area by planimetry from prospectively obtai
22 produce an accurate and precise estimate of aortic valve area in patients with severe aortic stenosi
24 S (defined by aortic valve area <=1 cm(2) or aortic valve area indexed to body surface area <=0.6 cm(
25 ls without imaging demonstrated that smaller aortic valve area is predictive of increased risk for ao
27 , indexed left ventricular ventricular mass, aortic valve area, and aortic valve replacement as a tim
28 We constructed a polygenic risk score for aortic valve area, which in a separate cohort of 311 728
29 sease, causes a progressive narrowing of the aortic valve as a consequence of thickening and calcific
35 eased use of TAVR for patients with bicuspid aortic valve (BAV) stenosis despite the exclusion of bic
37 FRANCE-TAVI nationwide registry (Registry of Aortic Valve Bioprostheses Established by Catheter) incl
39 e progression of coronary artery calcium and aortic valve calcification in patients with end-stage ki
42 work formalizes a deep learning baseline for aortic valve classification and outlines a general strat
43 rence between the interval from QRS onset to aortic valve closure midline, as derived for continuous-
44 pericardium were compared to native porcine aortic valve cusps in a rat subcutaneous model for up to
46 ted tomography (CT) can detect bioprosthetic aortic valve degeneration and predict valve dysfunction.
49 athways constituting biomarkers for calcific aortic valve disease (CAVD), including extra-cellular ma
50 lve area is predictive of increased risk for aortic valve disease (odds ratio, 1.14; P=2.3x10(-6)).
51 ssions Database, we identified patients with aortic valve disease admitted 2012 to 2016 for SAVR, TAV
52 ltiple links between the polygenic score for aortic valve disease and key health-related comorbiditie
57 ons of HF (including ischemic heart disease, aortic valve disease, atrial fibrillation, congenital he
58 ation to investigate the genetic etiology of aortic valve disease, perform clinical prediction, and u
59 verload, commonly caused by hypertension and aortic valve disease, promotes remodelling of the myocar
61 valve (BAV) have a higher risk of developing aortic valve dysfunction and progressive proximal aorta
62 ly increased in patients with more than mild aortic valve dysfunction but was independent from BAV le
63 Homozygous knockout mice for Adamts19 show aortic valve dysfunction, recapitulating aspects of the
64 s and their impact on myocardial remodeling, aortic valve flow patterns, and clinical progression.
71 days and 1 year, had significantly increased aortic valve gradients at 1 year (17.8 +/- 2.2 mm Hg vs.
72 nificantly affect aortic valve hemodynamics (aortic valve gradients or area), and was associated with
73 months, patients in the TAVR group had lower aortic-valve gradients than those in the surgery group (
74 and reduced leaflet motion of bioprosthetic aortic valves have been documented by four-dimensional c
75 t, but these findings did not correlate with aortic valve hemodynamic status after aortic valve repla
77 linical events, did not significantly affect aortic valve hemodynamics (aortic valve gradients or are
84 er Transfusion Requirements in Transcatheter Aortic Valve Implantation (TRITAVI) registry retrospecti
87 ecutive patients who underwent transcatheter aortic valve implantation using new-generation Evolut R/
88 ral Versus Local Anesthesia in Transcatheter Aortic Valve Implantation) trial is a multicenter, open-
89 d Ventricular Remodeling After Transcatheter Aortic Valve Implantation) trial is an ongoing randomize
91 th an antiplatelet agent after transcatheter aortic-valve implantation (TAVI) have not been well stud
92 nd thromboembolic events after transcatheter aortic-valve implantation (TAVI) in patients who do not
94 rmalities (such as Marfan syndrome, bicuspid aortic valve, inflammatory vasculitis, atherosclerosis a
95 onal and phenotypic changes occurring in the aortic valve interstitial cells (VICs) during osteogenic
100 re associated with 1-year mortality, whereas aortic valve involvement (OR, 0.47; 95% CI, .22-.98; P =
102 TAVR) for degenerated surgical bioprosthetic aortic valves is associated with favorable early outcome
104 exhibited reduced valve calcification, lower aortic valve leaflet area, increased M2 macrophage polar
108 histology, and morphometric analyses during aortic valve morphogenesis and in aged animals in multip
109 e of 65.5 years (range, 60-75 years) who had aortic valve or root infection, or both, with M. chimaer
110 major vascular complications, endocarditis, aortic valve re-intervention, and New York Heart Associa
111 were grouped on the basis of the severity of aortic valve regurgitation (AVR) and aortic valve stenos
112 significant serositis and severe mitral and aortic valve regurgitation, controlled with adalimumab,
113 e (BVF) was defined as: valve-related death, aortic valve reintervention, or severe hemodynamic SVD.
114 Rates of procedural secondary outcomes (eg, aortic valve reintervention, pacemaker rates) were more
117 Of 614 patients who underwent transcatheter aortic valve replacement (11.8% PPMI rate), we included
118 ent for age, sex, and surgical/transcatheter aortic valve replacement (as time-dependent covariates);
119 g coronary artery bypass grafting (CABG) and aortic valve replacement (AVR) and the relationship betw
120 porary data on loss in life expectancy after aortic valve replacement (AVR) are scarce, particularly
122 of anticoagulation (AC) after bioprosthetic aortic valve replacement (AVR) on valve hemodynamics and
123 1.21-1.97]) and after further adjustment for aortic valve replacement (AVR; adjusted HR, 1.47 [95% CI
124 lar biopsies (10x1x1 mm(3)) were obtained at aortic valve replacement (HFpEF(AVR), n=5; and HFrEF(AVR
125 isk aortic stenosis to undergo transcatheter aortic valve replacement (n = 221) or surgery (n = 214).
126 e replacement (TAVR) is supplanting surgical aortic valve replacement (SAVR) and medical therapy (MT)
127 noninferior and may be superior to surgical aortic valve replacement (SAVR) for mortality, stroke, a
128 rged as a reasonable alternative to surgical aortic valve replacement (SAVR) for patients with severe
129 aortic valve replacement (TAVR) or surgical aortic valve replacement (SAVR) for real-world propensit
130 aortic valve replacement (TAVR) to surgical aortic valve replacement (SAVR) in patients 70 years or
131 acement (TAVR) is an alternative to surgical aortic valve replacement (SAVR) in patients with severe
132 aortic valve replacement (TAVR) or surgical aortic valve replacement (SAVR) in the PARTNER (Placemen
133 aortic valve replacement (TAVR) or surgical aortic valve replacement (SAVR) is preferred for patient
134 s with severe aortic stenosis after surgical aortic valve replacement (SAVR) or transcatheter aortic
135 c valve replacement (TAVR) and redo surgical aortic valve replacement (SAVR) represent the 2 treatmen
136 aortic valve replacement (TAVR) or surgical aortic valve replacement (SAVR) results in similar rates
139 supports the widespread use of transcatheter aortic valve replacement (TAVR) among patients who are a
141 ular assist device (LVAD), and transcatheter aortic valve replacement (TAVR) are expensive cardiovasc
142 Conduction disturbances after transcatheter aortic valve replacement (TAVR) are often transient.
143 on 276,316 patients undergoing transcatheter aortic valve replacement (TAVR) at sites in all U.S. sta
151 ical trial results showed that transcatheter aortic valve replacement (TAVR) is noninferior and may b
155 dy was to assess the effect of transcatheter aortic valve replacement (TAVR) on hospitalizations in s
158 h severe aortic stenosis given transcatheter aortic valve replacement (TAVR) or surgical aortic valve
159 ogists often determine whether transcatheter aortic valve replacement (TAVR) or surgical aortic valve
160 surgical risk, treatment with transcatheter aortic valve replacement (TAVR) or surgical aortic valve
161 is similar after transfemoral transcatheter aortic valve replacement (TAVR) or surgical aortic valve
162 surgical risk, treatment with transcatheter aortic valve replacement (TAVR) results in lower rates o
165 d expanding the indication for transcatheter aortic valve replacement (TAVR) to low-risk patients wit
166 OTION) was designed to compare transcatheter aortic valve replacement (TAVR) to surgical aortic valve
167 Expanding the indication of transcatheter aortic valve replacement (TAVR) toward lower-risk and yo
169 t on coronary events following transcatheter aortic valve replacement (TAVR), and no study has determ
170 ause coronary occlusion during transcatheter aortic valve replacement (TAVR)-in-TAVR and present chal
183 tic valve replacement [TAVR] versus surgical aortic valve replacement [SAVR]) using trial-adjudicated
184 red across treatment arms (eg, transcatheter aortic valve replacement [TAVR] versus surgical aortic v
185 m/s/year] p = 0.019), and increased risk for aortic valve replacement and death (n = 145; hazard rati
186 cute kidney injury early after transcatheter aortic valve replacement and is an independent predictor
187 esenting a better alternative to concomitant aortic valve replacement and lung transplant in elderly
188 view of reported outcome after bioprosthetic aortic valve replacement and to translate this to age-sp
189 VI had an early safety benefit over surgical aortic valve replacement and was associated with faster
190 lar ventricular mass, aortic valve area, and aortic valve replacement as a time-dependent covariate,
191 an indication for transfemoral transcatheter aortic valve replacement as agreed by the heart team wer
192 adjustment for outcome predictors including aortic valve replacement as time-dependent covariate, lo
194 ll, 34 893 patients undergoing transcatheter aortic valve replacement at 445 hospitals were analyzed.
197 ysiological mechanisms of post-transcatheter aortic valve replacement complications and provide updat
198 hed patients who had undergone transcatheter aortic valve replacement for aortic stenosis, patients w
199 ess the efficacy and safety of transcatheter aortic valve replacement for bicuspid aortic stenosis.
200 evaluated patients undergoing transcatheter aortic valve replacement from November 1, 2011 to June 3
201 on individual end points after transcatheter aortic valve replacement has been conducted to date.
202 ave increased every year, exceeding surgical aortic valve replacement in 2019 (72,991 vs. 57,626), an
203 nd SURTAVI trials (Surgical or Transcatheter Aortic Valve Replacement in Intermediate-Risk Patients)
205 Background To support decision-making in aortic valve replacement in nonelderly adults, we aim to
206 e with aortic valve hemodynamic status after aortic valve replacement in patients at low risk for sur
207 nvalvular atrial fibrillation; transcatheter aortic valve replacement in patients with symptomatic se
210 ter aortic valve replacement versus surgical aortic valve replacement in the whole cohort and within
213 ith end-stage renal disease (ESRD), surgical aortic valve replacement is associated with higher early
214 This report suggests that transcatheter aortic valve replacement may favorably impact lung trans
215 eter Valves) and registry the outcomes after aortic valve replacement of the 4 flow-gradient groups.
218 s from 68 male/46 female patients undergoing aortic valve replacement surgery were obtained at baseli
219 and extra-large aortic annuli, transcatheter aortic valve replacement using 29-mm Sapien-3 and 34-mm
221 tes of clinical events between transcatheter aortic valve replacement versus surgical aortic valve re
222 onths, the survival benefit of transcatheter aortic valve replacement was also greater in the joint m
223 r the primary analysis cohort, transcatheter aortic valve replacement was performed in 210 (79.8%), a
224 large aortic annuli underwent transcatheter aortic valve replacement with 29-mm Sapien-3 (n=640) or
225 for papers reporting clinical outcome after aortic valve replacement with currently available biopro
226 and vascular access method, to transcatheter aortic valve replacement with the first generation Porti
228 d analysis of the FRAILTY-AVR study (Frailty Aortic Valve Replacement) was performed to analyze the d
229 ave compared bioprostheses for transcatheter aortic valve replacement, and no trials have compared bi
230 er self-expanding transcatheter and surgical aortic valve replacement, but these findings did not cor
231 mbosis has been reported after bioprosthetic aortic valve replacement, characterized using 4-dimensio
232 created by current generation transcatheter aortic valve replacement, percutaneous endovascular abdo
233 igation in patients undergoing transcatheter aortic valve replacement, percutaneous endovascular abdo
234 ters is well established after transcatheter aortic valve replacement, the role of Tei has not been e
235 patients with severe symptomatic AS awaiting aortic valve replacement, there has been a trend of incr
236 enosis undergoing transfemoral transcatheter aortic valve replacement, use of CS compared with GA res
237 consisted of a total of 22 876 referrals for aortic valve replacement, with (N=8098) TAVR and (N=14 7
254 italization at 1 year compared with surgical aortic valve replacement; however, the effect of treatme
255 ed patients after transfemoral transcatheter aortic valve replacement; propensity score-matching iden
256 rosthetic-valve function after transcatheter aortic-valve replacement (TAVR) as compared with surgica
257 During the introduction of transcatheter aortic-valve replacement (TAVR) in the United States, re
260 replacement (TAVR) as compared with surgical aortic-valve replacement in patients with severe aortic
262 s, monocytes, while passing through stenotic aortic valves result in proinflammatory effects that are
265 tional associations of obesity with incident aortic valve stenosis (n = 1,215) and replacement (n = 4
266 (OR, 1.04 [95% CI, 0.77-1.39]; P=0.810), and aortic valve stenosis (OR, 1.03 [95% CI, 0.56-1.90]; P=0
267 cluding subjects with a medical diagnosis of aortic valve stenosis (remaining n=308 683 individuals),
268 x was associated with causal risk ratios for aortic valve stenosis and replacement, respectively, of
272 g triglycerides is likely to prevent CAD and aortic valve stenosis but may increase thromboembolic ri
276 nd the age and sex-adjusted hazard ratio for aortic valve stenosis was 1.3 (95% confidence interval [
277 pandable valve for the treatment of bicuspid aortic valve stenosis) registry included 353 consecutive
278 gh shear stress, as present in patients with aortic valve stenosis, activates multiple monocyte funct
283 lacement in patients with symptomatic severe aortic valve stenosis; and antiplatelet agents vorapaxar
284 itral (2.18; 95% CI, 1.71-2.77) and mitral + aortic valve surgery (1.85; 95% CI, 1.33-2.58) and lowes
286 ndary Ross procedure performed after initial aortic valve surgery achieves superior long-term surviva
287 of initiating public outcomes reporting for aortic valve surgery on rates of valve surgery and in-ho
288 New York Heart Association functional class, aortic valve surgery, LV ejection fraction, LV end-systo
292 xpanding intra-annular Portico transcatheter aortic valve system (Abbott Structural Heart, St Paul, M
293 ial (the Portico Re-sheathable Transcatheter Aortic Valve System US Investigational Device Exemption
295 ome 2.0 arrays in calcified and noncalcified aortic valve tissue from 58 patients with AS (mean age,
296 late the behavior of thinner, more compliant aortic valve tissues in a physiologically realistic syst
298 for BAV patient with a normally functioning aortic valve was estimated at 0.05 Z score unit per year
299 area between the anterior mitral leaflet and aortic valve was inspected at myectomy in 106 consecutiv
300 t n-3 PUFA incorporation into human stenotic aortic valves was higher in noncalcified regions compare