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1 were significantly (P<0.001) elevated in the aortic-banded (1.25+/-0.12 microg/L, n=12) compared with
2 ined by serial echocardiograms), hearts from aortic banded and age-matched control rabbits were perfu
3 ed myocardial hypertrophy group (n = 5), and aortic-banded and cyclosporine A- treated cardiomyopathy
4 t was significantly (P<0.05) elevated in the aortic-banded animals.
5 nic mice but did not significantly change in aortic-banded G alpha q-25 mice.
6  (100 nmol/L) in isolated ejecting hearts of aortic-banded guinea pigs.
7 syndecan-4 in the left ventricle lysate from aortic-banded heart failure (ABHF) rats compared with SH
8 inned left ventricular fibers from untreated aortic-banded hypertrophied rats relative to age-matched
9          The effects of 2DG were compared in aortic-banded (LVH, n=5) and sham-operated (control, n=5
10 eight/tibial length ratio of vehicle-treated aortic-banded mice was increased by 34.4+/-3.6% compared
11 roups were evaluated: control group (n = 5), aortic-banded myocardial hypertrophy group (n = 5), and
12 tricular papillary muscles were excised from aortic-banded or sham-operated guinea-pig hearts.
13                     Sham-operated (SHAM) and aortic banded rat hearts (HYP) were perfused with buffer
14 r attenuated the hypertrophic response in an aortic-banded rat model of left ventricular hypertrophy,
15 ed adult ventricular myocytes from ascending aortic banded rats and age-matched controls.
16 e progression of load-induced hypertrophy in aortic banded rats with or without cyclosporine.
17 reated aortic-banded rats (n = 9), untreated aortic-banded rats (n = 9), and normal age-matched contr
18 ventricular myocytes from fosinopril-treated aortic-banded rats (n = 9), untreated aortic-banded rats
19 ly reversed pressure-overload hypertrophy in aortic-banded rats after 14 days.
20 ypertrophied adult ventricular myocytes from aortic-banded rats and age-matched controls.
21 ics in myocytes from control rats (SHAM) and aortic-banded rats exhibiting diastolic dysfunction.
22 myocytes from control sham operated rats and aortic-banded rats exhibiting diastolic dysfunction.
23 ization of maximal Ca(2+)-activated force in aortic-banded rats subjected to persistent pressure over
24 t ventricular pressure overload in ascending aortic-banded rats with left ventricular hypertrophy dur
25 2+]i (P < .05 versus myocytes from untreated aortic-banded rats).
26                   In myocytes from untreated aortic-banded rats, peak systolic [Ca2+]i was higher tha
27 d controls but not in the fosinopril-treated aortic-banded rats.