ライフサイエンスコーパス: aortitis

1 luding single-organ vasculitis (eg, isolated aortitis).

2 f multimodality imaging in the evaluation of aortitis.

3 for the pathologic diagnosis of IgG4-related aortitis.

4 h a newly-recognized subset of noninfectious aortitis.

5 ed by medical autopsy findings of syphilitic aortitis.

6 ignificant detection of VZV in granulomatous aortitis.

7 noglobulin G4-related aortitis, and isolated aortitis.

8 ctive inhibitor of CSF1 receptor, attenuated aortitis.

9 py and progresses over decades as smoldering aortitis.

10 l vasculitides are the most common causes of aortitis.

11 prognosis of noninfectious surgical thoracic aortitis.

12 uding 217 (3.8%) with noninfectious thoracic aortitis (118 clinically isolated aortitis, 57 giant cel

13 its forme fruste variety, 15 patients (10%); aortitis, 12 patients (8%), and in the remaining 91 pati

14 vasculitides (49.6%), followed by idiopathic aortitis (39.3%).

15 s thoracic aortitis (118 clinically isolated aortitis, 57 giant cells arteritis, 21 Takayasu arteriti

16 ic inflammation and fibrosis and can include aortitis and periaortitis with resultant complications s

17 ology of giant cell arteritis, granulomatous aortitis, and intracerebral varicella zoster virus (VZV)

18 ayasu's arteritis, immunoglobulin G4-related aortitis, and isolated aortitis.

19 oke, giant cell arteritis, and granulomatous aortitis, and it develops after virus reactivates from g

20 cute with rupture, acute dissection, mycotic aortitis, and trauma.

21 ain infections and vasculopathies, including aortitis, atherosclerosis, and Wegener's granulomatosis.

22 t- and long-term management of noninfectious aortitis, because of the frequent good response of IgG4-

23 eritis and Takayasu arteritis, with isolated aortitis being increasingly recognised in the literature

24 n 10 French centers, including patients with aortitis between 1 January 2014 and 31 December 2019.

25 icant fraction of thoracic lymphoplasmacytic aortitis cases, about 40% of inflammatory abdominal aort

26 rocedure, while treatment with statins after aortitis diagnosis (HR: 0.47; 95% CI: 0.24-0.90; P = 0.0

27 Aortitis due to hypervirulent Klebsiella pneumoniae (hvK

28 inal aortic aneurysm secondary to infectious aortitis following solid organ transplantation is a rare

29 nts with Takayasu arteritis and IgG4-related aortitis have also been successfully treated with the B-

30 Aortic arch aortitis (HR: 2.08; 95% CI: 1.26-3.44; P = 0.005) was in

31 Descending thoracic aortitis (HR: 2.35; 95% CI: 1.11-4.96; P = 0.031) and ao

32 ing, in particular to distinguish infectious aortitis (IA) and noninfectious aortitis (NIA).

33 s vascular remodeling and spontaneous severe aortitis in mice.

34 in four, bicuspid aortic valve in four, and aortitis in one.

35 g used to recognize the presence of isolated aortitis in patients with unexplained systemic disease.

36 e+Wnk1(lox/lox) mice spontaneously developed aortitis in the infrarenal abdominal aorta, which extend

37 ated systemic disease as a cause of thoracic aortitis, inflammatory abdominal aortic aneurysm or peri

38 nized disorder that may manifest as thoracic aortitis, inflammatory abdominal aortic aneurysm or retr

39 Aortitis is a group of disorders characterized by the in

40 Inflammatory aortitis is most often caused by large vessel vasculitis

41 Determining the etiology of aortitis is often challenging, in particular to distingu

42 the need for a change in the way idiopathic aortitis is regarded.

43 se should be considered in all patients with aortitis judged to be of unknown etiology.

44 Aortitis long-term outcomes are not well known.

45 se claudication and tissue gangrene, whereas aortitis may lead to aneurysm formation and dissection,

46 Treatment of the aortitis may prevent progression of the IgG4-related sys

47 h infectious aortitis (IA) and noninfectious aortitis (NIA).

48 ll arteritis and Takayasu arteritis, and the aortitis of Cogan syndrome and relapsing polychondritis.

49 and the disease itself may be accompanied by aortitis or a Takayasu's-like or medium-sized vessel vas

50 ascending aortic aneurysm (unassociated with aortitis or acute dissection), the aortic valve was cong

51 d be considered in any patient found to have aortitis or periaortitis.

52 of 3.9 years, 19 (16.1%) clinically isolated aortitis patients developed features of a systemic infla

53 that 82% of noninfectious surgical thoracic aortitis patients will experience a vascular complicatio

54 s, sclerosing cholangitis, lymphoplasmacytic aortitis, salivary gland involvement, orbital pseudotumo

55 The presence of VZV antigen in granulomatous aortitis was highly significant (P = .0001) as compared

56 Histopathologically, severe arthritis and aortitis were noted only in mice infected with isolate B

57 ntrast-enhanced CT imaging revealed thoracic aortitis with carotid occlusion, coronary artery stenosi

58 omputed tomography (CT) results demonstrated aortitis with periaortic tissue thickening.