ライフサイエンスコーパス: aortopulmonary

1 A 10-mm, Damus-Kaye-Stansel-type aortopulmonary anastomosis was created in 10 fetal sheep

2 Major aortopulmonary collateral arteries (MAPCAs) are congenit

3 Major aortopulmonary collateral arteries (MAPCAs) are rare con

4 lmonary atresia or severe stenosis and major aortopulmonary collateral arteries (MAPCAs) that aims to

5 tervention for tetralogy of Fallot and major aortopulmonary collateral arteries at Lucile Packard Chi

6 Tetralogy of Fallot with major aortopulmonary collateral arteries is a complex and hete

7 The presence of major aortopulmonary collateral arteries, older operative age,

8 ously operated tetralogy of Fallot and major aortopulmonary collateral arteries.

9 k factors for waitlist death included higher aortopulmonary collateral burden, >1 hospitalization in

10 tients, 23 vessels) and coil embolization of aortopulmonary collateral channels (8 patients, 31 colla

11 flow and facilities simultaneous coiling of aortopulmonary collateral channels and access for branch

12 monary atresia: There is a high incidence of aortopulmonary collateral channels, arborization abnorma

13 ors for early mortality included presence of aortopulmonary collateral vessels and prior thoracic sur

14 lemental oxygen requirement, and presence of aortopulmonary collateral vessels.

15 r, and origin of true pulmonary arteries and aortopulmonary collateral vessels; for stenosis; for thr

16 ly stent infants required unifocalization of aortopulmonary collaterals (17%).

17 to identify echocardiographic predictors of aortopulmonary collaterals (APCs) in infants with tetral

18 raphy measurements of pulmonary arteries and aortopulmonary collaterals (APCs).

19 th ventricular septal defect (VSD) and major aortopulmonary collaterals (MAPCAs) is a complex lesion

20 f Fallot with pulmonary atresia and multiple aortopulmonary collaterals and familial cholestasis.

21 d CMR were utilized in combination to assess aortopulmonary collaterals or the need for an interventi

22 utflow tract, pulmonary arteries, aorta, and aortopulmonary collaterals, and on its ability to quanti

23 s, right ventricular morphology, presence of aortopulmonary collaterals, or Fontan pressures on multi

24 together for interventions and assessment of aortopulmonary collaterals.

25 tal lambs underwent in utero placement of an aortopulmonary graft (shunt).

26 e of transbronchial biopsy of subcarinal and aortopulmonary lymph nodes.

27 ntricular septal defect, truncus arteriosus, aortopulmonary septal defect, and totally anomalous pulm

28 ding thin-walled myocardium, ventricular and aortopulmonary septal defects, and abnormal smooth muscl

29 st cells into the developing OFT to form the aortopulmonary septum.

30 r (A) or pulmonary blood flow supplied by an aortopulmonary shunt (B) or by a cavopulmonary connectio

31 y blood flow (after in utero placement of an aortopulmonary shunt) and 6 age-matched control lambs.

32 blood flow (Qp) was provided through a 5-mm aortopulmonary shunt.

33 %), 9 palliated by Fontan operation and 2 by aortopulmonary shunts: d-transposition of the great arte

34 tal lambs underwent in utero placement of an aortopulmonary vascular graft (shunt) and were studied 8

35 By using aortopulmonary vascular graft placement in the fetal lam

36 absent pulmonary valve syndrome (n = 24) and aortopulmonary window (n = 3).

37 enabled distinction between adenomas in the aortopulmonary window and those in the thymus.

38 Ectopic parathyroid glands in the aortopulmonary window are usually detected at sestamibi

39 ght adenomas, one hyperplastic gland) in the aortopulmonary window were examined with ultrasound (US)

40 ial anomalous pulmonary venous return, and 1 aortopulmonary window) who failed conventional therapy (

41 lar septal defect, pulmonary valve agenesis, aortopulmonary window, and double-outlet left ventricle.