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1 migraine, hypotension, and obstructive sleep apnea syndrome.
2 , 697 (68 percent) had the obstructive sleep apnea syndrome.
3 sleep in patients with the obstructive sleep apnea syndrome.
4 ant in the pathogenesis of obstructive sleep apnea syndrome.
5 diovascular abnormality in obstructive sleep apnea syndrome.
6 usion which results in the obstructive sleep apnea syndrome.
7 tested as pharmacologic treatments for sleep apnea syndrome.
8 tus (T2DM), smoking, hypertension, and sleep apnea syndrome.
9 or exclusion criterion was obstructive sleep apnea syndrome.
10 cs with moderate to severe obstructive sleep apnea syndrome.
11 io of %EFV to %VAF loss decreased with sleep apnea syndrome (1.34+/-0.3 vs. 0.52+/-0.08, p<0.05).
12  effective in remission of obstructive sleep apnea syndrome and metabolic dysfunction-associated stea
13 regarding the evolution of obstructive sleep apnea syndrome and persistence of abnormal pharyngeal se
14 rders of ventilatory control including sleep apnea syndromes and obesity hypoventilation syndrome.
15 tive apneas in patients with the obstructive apnea syndrome are accompanied by transient limb vasocon
16 ypertension, diabetes, and obstructive sleep apnea syndrome between September 2007 and July 2017.
17 uded obesity hypoventilation syndrome, sleep apnea syndrome, hypertension, gastroesophageal reflux, d
18 nt condition for the production of the sleep apnea syndrome in normal individuals.
19  significant prevalence of obstructive sleep apnea syndrome in patients in waiting list for LT, and L
20 surgical treatment for the obstructive sleep apnea syndrome in school-age children did not significan
21 hort (PLSC; N = 6,367), and "Impact of Sleep Apnea syndrome in the evolution of Acute Coronary syndro
22 ne patients with untreated obstructive sleep apnea syndrome in wakefulness and sleep.
23                            Obstructive sleep apnea syndrome involves abnormal upper airway sensory in
24                            Obstructive sleep apnea syndrome is a highly prevalent disease resulting i
25                            Obstructive sleep apnea syndrome is a well recognized cause of excessive s
26 firm previous reports that obstructive sleep apnea syndrome is associated with reduced parasympatheti
27                                        Sleep apnea syndrome is one of a series of sleep-related breat
28 reathing, particularly the obstructive sleep apnea syndrome, is common during childhood.
29 es have suggested that the obstructive sleep apnea syndrome may be an important risk factor for strok
30 f these findings for the management of sleep apnea syndrome must be verified by appropriate clinical
31 ommon disabling symptom in obstructive sleep apnea syndrome.Objectives: To evaluate the efficacy and
32  independent effect of the obstructive sleep apnea syndrome on the composite outcome of stroke or dea
33 ody mass index [BMI] >35), obstructive sleep apnea syndrome, or other causes of respiratory failure.
34         Many patients with obstructive sleep apnea syndrome (OSA) living near sea level travel to alt
35 23%) than in subjects with obstructive sleep apnea syndrome (OSAS) (0.06%), parasomnia (0.7%), restle
36 s modestly associated with obstructive sleep apnea syndrome (OSAS) among young children, but strongly
37                            Obstructive sleep apnea syndrome (OSAS) and nonalcoholic fatty liver disea
38  hypoxia (CIH) occurs with obstructive sleep apnea syndrome (OSAS) and provokes systemic endothelial
39 treatment in children with obstructive sleep apnea syndrome (OSAS) associated with significant hypoxe
40              Children with obstructive sleep apnea syndrome (OSAS) have more collapsible airways comp
41 er airway in children with obstructive sleep apnea syndrome (OSAS) have not been established.
42          The prevalence of obstructive sleep apnea syndrome (OSAS) in patients with nonarteritic ante
43                        The obstructive sleep apnea syndrome (OSAS) is associated with cardiovascular
44                            Obstructive sleep apnea syndrome (OSAS) is associated with intermittent hy
45 thophysiology of pediatric obstructive sleep apnea syndrome (OSAS) is suggested by the observation th
46                            Obstructive sleep apnea syndrome (OSAS) is usually diagnosed with overnigh
47                            Obstructive sleep apnea syndrome (OSAS) leads to neurocognitive and autono
48 tudies suggesting that the obstructive sleep apnea syndrome (OSAS) may compromise optic nerve head pe
49                            Obstructive sleep apnea syndrome (OSAS) represents a substantial disease o
50 te the correlation between obstructive sleep apnea syndrome (OSAS) risk with periodontal disease and
51  patients with concomitant obstructive sleep apnea syndrome (OSAS) seems to have a favorable impact o
52                            Obstructive sleep apnea syndrome (OSAS), a disorder characterized by episo
53  reported in patients with obstructive sleep apnea syndrome (OSAS), and these two chronic conditions
54 primary snoring through to obstructive sleep apnea syndrome (OSAS), may cause compromise of respirato
55 le in treating snoring and obstructive sleep apnea syndrome (OSAS).
56 cally in children with the obstructive sleep apnea syndrome (OSAS).
57 essure (CPAP) titration in obstructive sleep apnea syndrome (OSAS).
58 s-dependent cognition, and obstructive sleep apnea syndrome (OSAS).
59 athing in 10 children with obstructive sleep apnea syndrome (OSAS; age, 4.3 +/- 2.3 years) and 10 mat
60 allampati score III or IV, obstructive sleep apnea syndrome, reduced mobility of cervical spine, limi
61 ion, and hypertension, the obstructive sleep apnea syndrome retained a statistically significant asso
62  sleep problems, and comorbidities for sleep apnea syndrome (SAS) in COVID-19 and influenza (FLU) inf
63 ome (FES) and body mass index (BMI) in sleep apnea syndrome (SAS) patients compared to normal subject
64 isease, morbid obesity associated with sleep apnea syndrome, sickle cell disease, and polycythemic st
65                        The obstructive sleep apnea syndrome significantly increases the risk of strok
66 o 9 years of age, with the obstructive sleep apnea syndrome to early adenotonsillectomy or a strategy
67              Prevalence of obstructive sleep apnea syndrome was 38% before the LT, 86% at 6 months, a
68 n unadjusted analysis, the obstructive sleep apnea syndrome was associated with stroke or death from
69       The diagnosis of the obstructive sleep apnea syndrome was based on an apnea-hypopnea index of 5
70 that, in children with the obstructive sleep apnea syndrome without prolonged oxyhemoglobin desaturat
71 ormed in children with the obstructive sleep apnea syndrome, yet its usefulness in reducing symptoms