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1 raphy to determine SDB severity (obstructive apnea-hypopnea index).
2 es in leg fluid volume and either DeltaNC or apnea-hypopnea index.
3  obstructive sleep apnea was assessed by the apnea-hypopnea index.
4 en the groups and correlated negatively with apnea-hypopnea index.
5        Nocturnal polysomnography to evaluate apnea-hypopnea index.
6 andard overnight sleep study to determine an apnea-hypopnea index.
7 ifferent gradations of OSA severity based on apnea-hypopnea index.
8 provement in nocturnal oxygen saturation and apnea/hypopnea index.
9 s were matched for age, body mass index, and apnea/hypopnea index.
10        Seventy-one subjects (ages, 55-76 yr; apnea-hypopnea index, 0.2-96.6 events/h) were evaluated
11  with coronary artery disease [CAD] and OSA [apnea-hypopnea index 15 events/h] with Epworth Sleepines
12 ith paroxysmal AF and moderate to severe SA (apnea-hypopnea index 15).
13 (29 male) with Cheyne-Stokes breathing (mean apnea-hypopnea index 19.8 [SD 2.6] and stable symptomati
14 ft tissue structures in 48 control subjects (apnea-hypopnea index, 2.0 +/- 1.6 events/hour) and 48 pa
15 ; mean [SD] age, 61.2 [8.7] years; mean [SD] apnea-hypopnea index, 31.2 [17] events per hour; 71% wit
16 ), and had severe OSA at baseline (mean [SD] apnea-hypopnea index, 33.1 [14.9] events/h).
17      We recruited 30 obese control subjects (apnea-hypopnea index, 4.7 +/- 3.1 events per hour) and 7
18 ge, 54 [10] yr; median [interquartile range] apnea-hypopnea index, 41 [35-53]; mean [SD] Epworth slee
19                          Seventeen subjects (apnea-hypopnea index, 42.6 +/- 6.2 [SEM]) were studied d
20 our) and 72 obese patients with sleep apnea (apnea-hypopnea index, 43.5 +/- 28.0 events per hour).
21 ents/hour) and 48 patients with sleep apnea (apnea-hypopnea index, 43.8 +/- 25.4 events/hour).
22 tive sleep apnea (75% male; mean age, 52 yr; apnea-hypopnea index, 49/h; baseline sleepiness score, 1
23  A 64-year-old male patient with severe OSA (Apnea-Hypopnea Index, 54 events/h) underwent implantatio
24               OSA prevalence was 65% (median apnea-hypopnea index, 7.2; range, 0-93), 40% of which we
25   Home sleep testing was used to measure the apnea-hypopnea index, a measure of SDB severity.
26                Nasal occlusion increased the apnea hypopnea index (AHI) (occlusion mean = 6.6 +/- 8.0
27                  SDB was quantified with the apnea hypopnea index (AHI) and oxygen desaturation measu
28  metabolites and their associations with the apnea hypopnea index (AHI) and with moderate-severe OSA
29      Apnea-predominant OSA was defined as an apnea hypopnea index (AHI) greater than 2 with more than
30 association of head and facial form with the apnea hypopnea index (AHI) in 364 white individuals and
31 re was a significant correlation between the apnea hypopnea index (AHI) measured by polysomnography a
32 d by urinary cotinine levels and obstructive apnea hypopnea index (AHI) scores.
33                       A greater reduction in apnea hypopnea index (AHI) was strongly associated with
34 ure mapping on three primary OSA traits [the apnea hypopnea index (AHI), overnight average oxyhemoglo
35 vided by height in meters squared), sex, and apnea hypopnea index (AHI).
36 leep fragmentation, all participants with an apnea-hypopnea index (AHI) > or = 1 were analyzed separa
37 structive sleep apnea was assessed using the apnea-hypopnea index (AHI) (>=4% oxygen saturation crite
38      Two SDB definitions were considered: 1) apnea-hypopnea index (AHI) 5 and 2) oxygen desaturation
39       The association between post-treatment apnea-hypopnea index (AHI) and arousal threshold percent
40 r the OSA-associated quantitative phenotypes apnea-hypopnea index (AHI) and body mass index (BMI).
41 on sessions for all participants with a high apnea-hypopnea index (AHI) and by 37% (P = 0.008) among
42             Between-group differences on the apnea-hypopnea index (AHI) and Epworth sleepiness scale
43 of portable monitors, or association between apnea-hypopnea index (AHI) and health outcomes among com
44               A polysomnographically derived apnea-hypopnea index (AHI) and hypoxemia index (percent
45                                          The apnea-hypopnea index (AHI) and oxygen desaturation index
46                                              Apnea-hypopnea index (AHI) and percent nighttime with ox
47 d significant dose-relationships between REM apnea-hypopnea index (AHI) and prevalent hypertension.
48 rospectively evaluate the association of the apnea-hypopnea index (AHI) and sleep-related hypoxia wit
49                SDB was categorized using the apnea-hypopnea index (AHI) and the hypoxemia index (perc
50  risk for all-cause mortality, with FEV1 and apnea-hypopnea index (AHI) as the primary exposure indic
51                       The main outcomes were apnea-hypopnea index (AHI) change and nightly device use
52 sleep-disordered breathing, depending on the apnea-hypopnea index (AHI) cutoff, ranged from 40 to 60%
53 signals in a one-dimensional (1D) format for Apnea-Hypopnea Index (AHI) estimation in pediatric subje
54 imary outcomes were safety and the change in apnea-hypopnea index (AHI) from baseline to 12 months po
55  The primary end point was the change in the apnea-hypopnea index (AHI) from baseline to the interven
56 moderate to severe SDB, defined as having an Apnea-Hypopnea Index (AHI) greater than 15 as assessed b
57      In fully adjusted models, patients with apnea-hypopnea index (AHI) greater than 30 had a 30% hig
58 week, on a nondialysis day, subjects with an apnea-hypopnea index (AHI) greater than or equal to 20 h
59 revalence of periodontal disease between the apnea-hypopnea index (AHI) groups, with a negligible Spe
60 ociation analyses in independent cohorts for apnea-hypopnea index (AHI) in a total of 7,708 individua
61 o detect SDB events and estimate whether the apnea-hypopnea index (AHI) is >= 15, indicative of moder
62                                Women with an apnea-hypopnea index (AHI) less than 10 comprised the co
63                                Women with an apnea-hypopnea index (AHI) less than 10 were the control
64                             Patients with an apnea-hypopnea index (AHI) less than 15 were the control
65 o 12.9 years with snoring and an obstructive apnea-hypopnea index (AHI) less than 3 enrolled at 7 US
66                  Nonetheless, an obstructive apnea-hypopnea index (AHI) of 1 or greater is often used
67  patients with resistant hypertension and an apnea-hypopnea index (AHI) of 15 or higher.
68 ricular ejection fraction of 45% or less, an apnea-hypopnea index (AHI) of 15 or more events (occurre
69         A total of 14.7% of subjects with an apnea-hypopnea index (AHI) of 15 or more had a diagnosis
70 th recently diagnosed (<6 months) OSA and an apnea-hypopnea index (AHI) of 20 events/hour or more.
71 patients with moderate to severe OSA with an apnea-hypopnea index (AHI) of 20 to 65 events per hour a
72            OSA was defined as a nightly mean apnea-hypopnea index (AHI) of more than 15 events/h.
73  50% reduction or greater in preimplantation Apnea-Hypopnea Index (AHI) score and postimplantation AH
74                            Outcomes were the apnea-hypopnea index (AHI) score, ascertained via polyso
75           The prevalence varies based on the apnea-hypopnea index (AHI) threshold used for the evalua
76                                         Mean apnea-hypopnea index (AHI) was 52.6 +/- 28.2 (SD) events
77                                          The apnea-hypopnea index (AHI) was derived from standardized
78 SE).Measurements and Main Results: Change in apnea-hypopnea index (AHI) was measured.
79 ed with a tomograph and corneal topographer, apnea-hypopnea index (AHI) with polysomnography, and ser
80 e and BUI intensity were associated with the apnea-hypopnea index (AHI), a measure of severity of apn
81    For OSA, facial shape correlated with the apnea-hypopnea index (AHI), and DS individuals with seve
82 nce of OSA, physiological data including the apnea-hypopnea index (AHI), and nocturnal oxygen saturat
83 r, OAs have variable success at reducing the apnea-hypopnea index (AHI), and predicting responders is
84 ated with hsCRP and MRP 8/14 levels and with apnea-hypopnea index (AHI), BMI z score, and apolipoprot
85      Our main exposures were the obstructive apnea-hypopnea index (AHI), central apnea index (CAI >/=
86 ed into four severity groups on the basis of apnea-hypopnea index (AHI), followed by comparisons of c
87                          In combination with apnea-hypopnea index (AHI), identification of "respirato
88 g the value of the ODI as a surrogate to the apnea-hypopnea index (AHI), it was shown that the value
89 nography study that obtained measurements of apnea-hypopnea index (AHI), peripheral oxygen saturation
90 es included oxygen desaturation index (ODI), apnea-hypopnea index (AHI), subjective sleepiness (Epwor
91                               Rationale: The apnea-hypopnea index (AHI), used for the diagnosis of ob
92 y was associated with a 55% reduction in the apnea-hypopnea index (AHI), which decreased from a prefl
93                 Logistic regression assessed apnea-hypopnea index (AHI)-adjusted associations between
94 p-disordered breathing was quantified by the apnea-hypopnea index (AHI)-the average number of apneas
95 e sleep apnea (OSA) have been defined by the apnea-hypopnea index (AHI).
96 oss individuals and are not predicted by the apnea-hypopnea index (AHI).
97 for the diagnosis of OSAS and calculation of Apnea-Hypopnea Index (AHI).
98 rence, hours of use, mask leak, and residual apnea-hypopnea index (AHI).
99 e changes would correlate with reductions in apnea-hypopnea index (AHI).Methods: A total of 67 indivi
100              OSA severity was defined by the apnea-hypopnea index (AHI): severe >30, moderate >15-30,
101                                              Apnea-hypopnea index (AHI, the average number of apneas
102  The primary end point was the change in the apnea-hypopnea index (AHI, the number of apneas and hypo
103                    Outcome measures included apnea-hypopnea index (AHI; average number of apneas plus
104            Primary outcome measures were the apnea-hypopnea index (AHI; ie, the number of apnea and h
105        The primary outcome measures were the apnea-hypopnea index (AHI; the number of apnea or hypopn
106                       OSAS was defined by an apnea/hypopnea index (AHI) greater than or equal to 1 ev
107  a resolution of symptoms and a reduction in Apnea/Hypopnea Index (AHI) to < 5/h, and partial respons
108 p was divided into 3 sub-groups based on the apnea/hypopnea index (AHI): mild, moderate, or severe OS
109 ne 30, 1997, and had to have normal results (apnea hypopnea index [AHI] <5).
110 al of 126 patients with moderate-severe OSA (apnea hypopnea index [AHI], 25.6 [SD 12.3]) were randoml
111 of hypersomnolence in 741 patients with SDB (apnea-hypopnea index [AHI] >/= 10 events/h).
112                          Mild to severe OSA (apnea-hypopnea index [AHI] >=5) was associated with poor
113                    The prevalence of severe (apnea-hypopnea index [AHI] 30) and nonsevere (AHI <30) O
114  surgical responses (>/=50% reduction in the apnea-hypopnea index [AHI] and <10 events/hour) in patie
115 participants were identified as free of OSA (apnea-hypopnea index [AHI] of <5 events/h and not treate
116  frequency of apneas and hypopneas using the Apnea-Hypopnea Index [AHI]) and sleep-related hypoxemia
117 terval 1.00 to 1.21] per 10-unit increase in apnea-hypopnea index [AHI]) but not in older men or in w
118 in 68 patients (55 males, 13 females; median apnea-hypopnea index [AHI], 35) not receiving hypotensiv
119         We examined 55 sleep apnea probands (apnea-hypopnea index [AHI]: 43.2 +/- 26.3 events/h), 55
120 antimuscarinic (oxybutynin) on OSA severity (apnea-hypopnea index [AHI]; primary outcome) and geniogl
121   A total of 25 adults with positional OSAS (apnea-hypopnea index [AHI]supine:AHInon-supine >/= 2) we
122 both sleep laboratory and clinical criteria (apnea/hypopnea index [AHI] > or = 10 and the presence of
123      The 24-h prevalence of predominant CAs (apnea/hypopnea index [AHI] >/=5 events/h, with CA of >50
124 ecutive new outpatients diagnosed with SAHS (apnea/hypopnea index [AHI] >or= 5/hour, and >or= 2 sympt
125 ore episodes of apnea and hypopnea per hour (apnea-hypopnea index, AHI).
126 y of apneas and hypopneas per hour of sleep (apnea-hypopnea index, AHI).
127  was observed between either the obstructive apnea-hypopnea index and any aggregation parameter, but
128 on was used to model the association between apnea-hypopnea index and echocardiographic measures whil
129 subjects, which directly correlated with the apnea-hypopnea index and hypoxemic indices.
130    For each interval, we assessed the median apnea-hypopnea index and the relative risk of sudden dea
131 ents per hour or <15/h, respectively, on the apnea-hypopnea index) and the median PWAD index.
132                             Body mass index, apnea-hypopnea index, and cognitive abilities were model
133 iac biomarker expression, functional status, apnea-hypopnea index, and overnight hypoxemia.
134  of sleep time with oxygen saturation < 90%, apnea-hypopnea index, and oxygen desaturation index-did
135 ea or Cheyne-Stokes respiration, obstructive apnea-hypopnea index, and percentage of sleep time with
136 rs11691765, GPR83, P = 1.90 x 10(-8) for the apnea-hypopnea index, and rs35424364; C6ORF183/CCDC162P,
137 en saturation as measured by pulse oximetry, apnea-hypopnea index, and the fraction of events that we
138  (OSA) is variable when considering only the Apnea Hypopnea Index as the treatment end point.
139 e four-year follow-up study according to the apnea-hypopnea index at base line were estimated after a
140                 The effects of severe apnea (apnea-hypopnea index, at least 30 episodes/h), which occ
141 om 12-channel home polysomnography, were the apnea-hypopnea index (average number of apneas/hypopneas
142 s were metaanalyzed for association with the apnea-hypopnea index, average oxygen saturation during s
143 incident CVD experienced larger increases in apnea-hypopnea index between polysomnograms.
144  as experiencing at least a 50% reduction in apnea-hypopnea index between sham and active treatment.
145    The relation of incident CVD to change in apnea-hypopnea index between the 2 polysomnograms was te
146 ion was assessed for baseline sleep indices: apnea-hypopnea index, central sleep apnea (central apnea
147              The difference in adjusted mean apnea-hypopnea index change between subjects with and wi
148 rdiac causes during other intervals, and the apnea-hypopnea index correlated directly with the relati
149        Mean number of events measured by the apnea-hypopnea index decreased from 35.7/h to 2.1/h at 6
150                                              Apnea hypopnea index, defined as number of apneas and hy
151                                          The apnea-hypopnea index does not capture the range of physi
152 his hypothesis, we correlated loop gain with apnea-hypopnea index during supine, nonrapid eye movemen
153                                  We used the apnea-hypopnea index (events per hour) to define obstruc
154                              STM reduced the apnea-hypopnea index from 55.2 to 33.0 events/h (-41.0%)
155 ntification of all children with obstructive apnea-hypopnea index greater than 5/hour total sleep tim
156 e participants with obstructive sleep apnea (apnea-hypopnea index &gt; 5 events/hr) were administered an
157 ss quality of life in 122 patients with SDB (apnea-hypopnea index &gt; or = 5 events/hour), this study f
158  with polysomnography between 1992 and 2004 (apnea-hypopnea index &gt; or =15) who subsequently underwen
159  overweight/obese matched patients with OSA (apnea-hypopnea index &gt;/= 15 events per hour) and 11 norm
160                                         OSA (apnea-hypopnea index &gt;/= 5 events/h) was assessed using
161                     SDB was characterized by apnea-hypopnea index &gt;/=15 events per hour (polysomnogra
162 derate-to-severe sleep-disordered breathing (apnea-hypopnea index &gt;/=15%) was significantly higher in
163 ients with newly revascularized CAD and OSA (apnea-hypopnea index &gt;/=15/h) without daytime sleepiness
164 with paroxysmal AF (43 with >/=moderate OSA [apnea-hypopnea index &gt;/=15] and 43 without OSA [apnea-hy
165  best predicted by age >60 years (HR: 5.53), apnea-hypopnea index &gt;20 (HR: 1.60), mean nocturnal O2sa
166             To evaluate the presence of OSA (apnea-hypopnea index &gt;= 15 events . h(-1)), all patients
167 .Methods: Data from 1,207 patients with OSA (apnea-hypopnea index &gt;= 15 events/h) were used to evalua
168 20 participants with moderate-to-severe OSA (apnea-hypopnea index &gt;=15 events per hour) were randomiz
169              The presence of SDB (defined as apnea-hypopnea index &gt;=15/h) was assessed with a portabl
170 ed that, for clinically defined sleep apnea (apnea/hypopnea index &gt; or = 10 and daytime symptoms), me
171 women diagnosed with moderate to severe OSA (apnea-hypopnea index, &gt;/=15) in 19 Spanish sleep units.
172 eepiness: severe sleep-disordered breathing (apnea-hypopnea index, &gt;30 episodes/hr), self-report of p
173 M and non-REM sleep was quantified using the apnea-hypopnea index in REM (AHIREM) and non-REM sleep (
174  correlation was found between loop gain and apnea-hypopnea index in the atmospheric group only (r =
175                        At baseline, the mean apnea-hypopnea index in the patients with the syndrome w
176 for mortality, but its diagnostic metric-the apnea-hypopnea index-is a poor risk predictor.
177  18 overweight/obese subjects without apnea (apnea-hypopnea index &lt; 15 events per hour) with 25 overw
178 ife in normal subjects (n = 15) without SDB (apnea-hypopnea index &lt; 5 events/hour) recruited from the
179  When compared with individuals without OSA (apnea-hypopnea index &lt; 5), significantly increased risk
180 to moderate degrees of sleep apnea (5 < or = apnea-hypopnea index &lt; or = 30).
181 ears with snoring and mild oSDB (Obstructive Apnea-Hypopnea Index &lt;3 with tonsillar hypertrophy [Brod
182 lectomy for mSDB (snoring and an obstructive apnea-hypopnea index &lt;3) between June 29, 2016, and Febr
183 ea-hypopnea index >/=15] and 43 without OSA [apnea-hypopnea index &lt;5]), right atrial and left atrial
184  of 1 to each criterion that was satisfied: (apnea-hypopnea index, &lt;30 events per hour) + (nadir oxyg
185 aphic factors (mean age, 63 yr; 52% female), apnea-hypopnea index (mean, 13.8; SD, 15.0), smoking, an
186 derate to severe sleep-disordered breathing (apnea-hypopnea index, measured as events/hour, >/=15) ar
187 es, death, sleep study measures (such as the Apnea-Hypopnea Index), measures of cardiovascular status
188 ith mild SDB, all of whom had an obstructive apnea-hypopnea index (OAHI) no greater than 3.
189 ged between 2 to 4 years with an Obstructive Apnea-Hypopnea Index (OAHI) score of 2 or greater and le
190 omnography, and severe SDB was defined as an apnea-hypopnea index of >30 per hour of sleep.
191     Relative to the reference category of an apnea-hypopnea index of 0 events per hour at base line,
192 t confidence interval, 1.13 to 1.78) with an apnea-hypopnea index of 0.1 to 4.9 events per hour at ba
193 d adjusted rate ratio [caffeine/placebo] for apnea-hypopnea index of 0.89 [95% CI = 0.55-1.43]; P = 0
194 Sleep-disordered breathing was defined as an apnea-hypopnea index of 15 or more events per hour of sl
195 he prevalence of sleep-disordered breathing (apnea-hypopnea index of 15 or more) among hormone users
196                         Participants with an apnea-hypopnea index of 15 to 50 events per hour were ra
197 t confidence interval, 1.46 to 5.64) with an apnea-hypopnea index of 15.0 or more events per hour.
198 the syndrome was 35, as compared with a mean apnea-hypopnea index of 2 in the comparison group.
199 onsecutive patients were enrolled who had an apnea-hypopnea index of 20 h(-1) or greater and an Epwor
200 ructive sleep apnea syndrome was based on an apnea-hypopnea index of 5 or higher (five or more events
201 founders.Measurements and Main Results: SDB (apnea-hypopnea index of 5 or more) and insomnia (Women's
202 t confidence interval, 1.29 to 3.17) with an apnea-hypopnea index of 5.0 to 14.9 events per hour, and
203 e or more events per hour); patients with an apnea-hypopnea index of less than 5 served as the compar
204                                           An apnea-hypopnea index of more than 10 per hour was signif
205 omes were total sleep time on actigraphy and apnea-hypopnea index on polysomnography.
206 leep-disordered breathing was ascertained by apnea-hypopnea index or clinical diagnosis.
207 al apnea (OR, 6.31; 95% CI, 1.94-20.51), and apnea-hypopnea index (OR, 1.22; 95% CI, 1.08-1.39 [per 5
208  associated with significant improvements in apnea-hypopnea index (P < 0.001); microarousal index (P
209 idal carbon dioxide were determinants of the apnea-hypopnea index (P value range = 0.04-0.001).
210 versely with DeltaNC (r=-0.755, P<0.001) and apnea-hypopnea index (r=-0.765, P<0.001), it did not in
211 ificant positive correlation between SAA and apnea-hypopnea index (r=0.40, P=0.03).
212                                          The apnea-hypopnea index reduction threshold of 50% was reac
213 saturation (SaO2); right putamen tCho/Cr and apnea hypopnea index; right putamen GABA/Cr and baseline
214 mes were nocturnal oxygen saturation and the apnea/hypopnea index; secondary outcomes were sleep stru
215 us mean diffusivity correlated with a higher apnea hypopnea index (Spearman's r = -0.50, p = 0.008) a
216 idnight to 6 a.m. had a significantly higher apnea-hypopnea index than those with sudden death from c
217 leep-disordered breathing, as defined by the apnea-hypopnea index (the number of episodes of apnea an
218     SDB classified via polysomnography using apnea-hypopnea index thresholds, with subtypes including
219  classified based on conventionally accepted apnea/hypopnea index thresholds: >=5.0/h (OSA(>=5)), >=1
220 oke or cardiovascular events with increasing apnea-hypopnea index values.
221                                       Median apnea hypopnea index was 6.0 (IQR, 3.1-10.3) events per
222                                     The mean apnea-hypopnea index was 16 (95% CI, 13-19), and the mea
223                                   Mean (SEM) apnea-hypopnea index was also significantly higher in pa
224                     Each 10-unit increase in apnea-hypopnea index was associated with 0.2 (95% CI, 0.
225                               Initially, the apnea-hypopnea index was determined by polysomnography f
226 e, diabetes mellitus, and creatinine levels, apnea-hypopnea index was independently associated with i
227                          The mean (+/-SD) of apnea-hypopnea index was significantly higher in patient
228 bjects (1,839 in fully adjusted models), the apnea-hypopnea index was used to classify OSA as none (0
229 and 2590 m than placebo and autoCPAP: median apnea/hypopnea index was 5.8 events per hour (5.8/h) (IQ
230 y of apneas and hypopneas per hour of sleep (apnea-hypopnea index) was determined by unattended, sing
231 uch as age, race and ethnicity, obesity, and apnea-hypopnea index, were not associated with improveme
232  studies focused on traits defined using the apnea-hypopnea index, which contains limited information
233  (Epworth Sleepiness Scale score >10) and an apnea-hypopnea index with 3% desaturation and from 5 to
234                                              Apnea-hypopnea index with a 4% desaturation criterion (p
235                                              Apnea-hypopnea index with a 4% desaturation criterion de

 
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