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1 ry effects cannot be explained by changes in appetitive behavior.
2 via MOR, within the CeA promote this form of appetitive behavior.
3  cortex (IL) in the environmental control of appetitive behavior.
4 s capable of supporting distinct theories of appetitive behavior.
5 volved in stimulant-related learning driving appetitive behavior.
6 of hypothalamic nuclei in the stimulation of appetitive behavior.
7 hat have helped elucidate dopamine's role in appetitive behavior.
8 egion that is important in the regulation of appetitive behavior.
9 ated with emotion regulation, attention, and appetitive behavior.
10 in parasympathetic outflow, and decreases in appetitive behavior.
11 ning drug levels into increased drug-related appetitive behavior.
12 tions to the parabrachial nucleus to promote appetitive behavior.
13 LHA(Vglut2)) negatively regulate feeding and appetitive behavior.
14 epresentations of space to promote effective appetitive behavior.
15 viors and dissect the BLA-to-CeA circuit for appetitive behaviors.
16  signaling and examined several aversive and appetitive behaviors.
17 o produce different conditioned defensive or appetitive behaviors.
18 mprise a network likely involved in approach/appetitive behaviors.
19 CeA neurons define a pathway for suppressing appetitive behaviors.
20 een proposed to be selectively active during appetitive behaviors.
21 or a common neural mechanism associated with appetitive behaviors.
22 or determining their contribution to learned appetitive behaviors.
23 output nucleus of the amygdala implicated in appetitive behaviors.
24 n the prelimbic cortex (PL), which regulates appetitive behaviors.
25 ry and sufficient for spatial memory-related appetitive behaviors.
26  for developing treatments for cue-triggered appetitive behaviors.
27 nt of sex-based treatments for cue-triggered appetitive behaviors.
28 forming bottom-up sensory signals to mediate appetitive behaviors.
29 ect of the somatostatin analog octreotide on appetitive behavior among patients after ES.
30 ts in the amygdala that promote and suppress appetitive behaviors analogous to the direct and indirec
31 c strength are hypothesized to contribute to appetitive behavior and addiction.
32 ocrine and nonendocrine functions, including appetitive behavior and carbohydrate substrate utilizati
33 ion in nucleus accumbens contributes to both appetitive behavior and fearful behavior that is generat
34 ral hypothalamus (LHA) integrates reward and appetitive behavior and is composed of many overlapping
35                            The NAcc mediates appetitive behavior and is critically modulated by dopam
36 ormones are linked to mechanisms that govern appetitive behavior and its suppression.
37 el hypothalamic-AcSh circuit that influences appetitive behavior and mediates the antidepressant acti
38 nship through which the central clock drives appetitive behavior and metabolic homeostasis and the pa
39 inct populations of CeA neurons that mediate appetitive behaviors and dissect the BLA-to-CeA circuit
40 ent with roles in the performance of learned appetitive behaviors and in positive reinforcement, resp
41 e been studied extensively, CeA circuits for appetitive behaviors and their relationship to threat-re
42 examine gustatory physiology, tastant-evoked appetitive behavior, and food ingestion to understand cl
43 g trait anxiety, contextual fear memory, and appetitive behavior, and is known to be sensitive to str
44 e within a distributed network that controls appetitive behavior, and neuromodulation of the VS has d
45 rain circuits that play a role in attention, appetitive behavior, and reward processing.
46 ient deficiency, how sensory signals trigger appetitive behaviors, and how food intake is regulated t
47 ed whole body energy demands into subsequent appetitive behavior are incompletely understood.
48 ion, but dissociates from stable conditioned appetitive behavior as this signal returns to preconditi
49 egulating the balance of limbic control over appetitive behavior at the point of learning.
50 Performance levels and lipping intensity (an appetitive behavior) both showed that the monkeys' motiv
51 e and ingestive behavior, opaline can elicit appetitive behavior but can also inhibit ingestion and e
52 may be a reinforcer that elicits conditioned appetitive behavior, but its reinforcing properties stro
53 tions to the ventral tegmental area (VTA) in appetitive behaviors, but these circuits have not been c
54   The results suggest that the inhibition of appetitive behavior by energy state signals may depend,
55  hippocampus is involved with the control of appetitive behavior by interoceptive "hunger" and "satie
56 sed behavior and reveals mechanisms by which appetitive behavior can go awry.
57  variety of physiologic functions, including appetitive behaviors, cognitive functions and metabolism
58 nd was followed by a variety of approach and appetitive behaviors, consistent with electrophysiologic
59 ditioned reflexes discussed by Pavlov or the appetitive behavior discussed by Craig; they have only u
60 evented DNQX microinjections from generating appetitive behavior (eating) in rostral shell, and equal
61 ion can be quite variable between studies of appetitive behavior, even within the same species.
62 cells may underlie its inhibitory effects on appetitive behavior for rewarding stimuli.
63 ated that serotonin can increase or decrease appetitive behavior in an odor-specific manner.
64 f the neural circuit that regulates a sexual appetitive behavior in C. elegans.
65 h natural reward, are less responsive during appetitive behavior in familiar conditions, and are inse
66 supports a multitude of functions related to appetitive behavior in humans and animals, and it has be
67  regulation of competing limbic control over appetitive behavior in mice, we hereby examined the effe
68 pothesis that activity of circuits promoting appetitive behavior in the core of the nucleus accumbens
69 rates, thereby regulating both defensive and appetitive behaviors in a frequency-dependent manner.
70 amate disruptions typically generate intense appetitive behaviors in rats, but the disruption increme
71  the causal role for CeA circuits underlying appetitive behaviors is poorly understood.
72  on a range of conditioned and unconditioned appetitive behaviors known to depend on mesolimbic DA ac
73                                              Appetitive behaviors of rats were monitored in a runway
74 es associated with hedonic taste evaluation, appetitive behavior, oromotor coordination, and inhibito
75 rrive in the CeA eliciting both aversive and appetitive behaviors, our understanding of the anatomy o
76 like behaviors, but not in some aversive and appetitive behaviors previously ascribed to CeA neurons.
77 and anatomically-defined CeA neurons play in appetitive behavior remain unclear.
78                                              Appetitive behaviors require complex decision making tha
79 of them reduced sensory-induced aversive and appetitive behaviors, respectively.
80 Ac to likely account for CRF facilitation of appetitive behaviors.SIGNIFICANCE STATEMENT Although the
81 rimary role for these neurons in controlling appetitive behaviors such as foraging that promote the d
82 ce of neural systems-level information about appetitive behavior that could be used in responsive neu
83  Although compelling within the framework of appetitive behavior, the view that illicit drugs hijack
84                                 A measure of appetitive behavior, this task requires subjects to unde
85  dopamine (DA) is phasically released during appetitive behaviors, though there is substantive disagr
86 ormone response is associated with increased appetitive behavior toward a sweet-fat stimulus among pa
87 ly more trials than nondepleted rats; hence, appetitive behavior was mildly potentiated by depletion,
88          Silencing neuronal activity reduces appetitive behaviors, whereas inducible activation resul
89 ) CeA neurons define a pathway for promoting appetitive behaviors, while R-spondin 2(+) BLA pyramidal