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1 corneal lipid film spread and distributed in aqueous tear deficiency (ATD) dry eye.
2 oups included meibomian gland disease (MGD), aqueous tear deficiency (ATD), Sjogren syndrome, non-Sjo
3 and disease (MGD) or Sjogren's syndrome (SS) aqueous tear deficiency (ATD).
4 sfunction (meibomian gland disease [n = 11], aqueous tear deficiency [n = 10]-without (n = 7) and wit
5 link rates were significantly higher in both aqueous tear deficiency and conjunctivochalasis compared
6 s, and pharmacodynamics in a canine model of aqueous tear deficiency and keratoconjunctivitis sicca.
7                Patients with DE symptoms and aqueous tear deficiency had higher tear serotonin levels
8 ndrome-like pathology in lacrimal tissue, or aqueous tear deficiency in nonautoimmune animals and hum
9 sory thresholds were significantly higher in aqueous tear deficiency using either Cochet-Bonnet (3.6
10 er orchiectomy and pituitary removal, and no aqueous tear deficiency was apparent in patients receivi
11                        Of these, 22 (8%) had aqueous tear deficiency, 124 (47%) lipid tear deficiency
12 l correlations were analyzed among age, sex, aqueous tear deficiency, dry eye, ocular demodicosis, fo
13 gland disease (MGD), Sjogren's syndrome (SS) aqueous tear deficiency, or neurotrophic keratopathy (NK
14 pain, irritation, and anxiety in response to aqueous tear deficiency.
15 rface disease that develops in patients with aqueous tear deficiency.
16 on, a Sjogren's syndrome-like pathology, and aqueous tear deficiency.
17 al (TB) bulbar conjunctiva in control versus aqueous tear deficient eyes (ATD) and evaluate correlati
18                                              Aqueous tear-deficient dry eye is a multifactorial chron
19 lipid film is also dictated by the amount of aqueous tear fluid.
20 e interaction between the lipid film and the aqueous tear fluid.
21                                              Aqueous tear production and clearance were inhibited by
22    Compensatory mechanisms such as increased aqueous tear production and dilation of MGs make early d
23 emonstrate any evidence of dry eyes based on aqueous tear production and had normal conjunctival gobl
24 eviated the inhibitory effect of IL-1beta on aqueous tear production and lacrimal gland protein secre
25 ystrophy) and 13 normal subjects with normal aqueous tear production and no irritation symptoms.
26                                              Aqueous tear production and volume, tear clearance, and
27         Cotton threads were used to evaluate aqueous tear production and volume.
28  injection into the lacrimal gland inhibited aqueous tear production by 52% and inhibited neurally an
29                                              Aqueous tear production by means of the cotton thread te
30 within 3 days, and a significant decrease in aqueous tear production that persisted for 1 month.
31                                              Aqueous tear production was inhibited by applying transd
32                                              Aqueous tear production was measured using phenol red-im
33 ges in stromal nerve fiber density (NFD) and aqueous tear production were determined.
34 neas exhibited significantly reduced NFD and aqueous tear production, and increased inflammatory cell
35                                              Aqueous tear production, tear fluorescein clearance and
36 rison to that in age-matched NRTN(+/+) mice, aqueous tear production, tear fluorescein clearance, and
37 eal neurotoxicity, inflammation, and reduced aqueous tear production.
38 inhibit lacrimal gland protein secretion and aqueous tear production.
39 ts into groups based on both DE symptoms and aqueous tear production; serotonin concentrations were s
40 venting key signs of dry eye disease such as aqueous tear secretion, conjunctival goblet cells, epith