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2 oups included meibomian gland disease (MGD), aqueous tear deficiency (ATD), Sjogren syndrome, non-Sjo
4 sfunction (meibomian gland disease [n = 11], aqueous tear deficiency [n = 10]-without (n = 7) and wit
5 link rates were significantly higher in both aqueous tear deficiency and conjunctivochalasis compared
6 s, and pharmacodynamics in a canine model of aqueous tear deficiency and keratoconjunctivitis sicca.
8 ndrome-like pathology in lacrimal tissue, or aqueous tear deficiency in nonautoimmune animals and hum
9 sory thresholds were significantly higher in aqueous tear deficiency using either Cochet-Bonnet (3.6
10 er orchiectomy and pituitary removal, and no aqueous tear deficiency was apparent in patients receivi
12 l correlations were analyzed among age, sex, aqueous tear deficiency, dry eye, ocular demodicosis, fo
13 gland disease (MGD), Sjogren's syndrome (SS) aqueous tear deficiency, or neurotrophic keratopathy (NK
17 al (TB) bulbar conjunctiva in control versus aqueous tear deficient eyes (ATD) and evaluate correlati
22 Compensatory mechanisms such as increased aqueous tear production and dilation of MGs make early d
23 emonstrate any evidence of dry eyes based on aqueous tear production and had normal conjunctival gobl
24 eviated the inhibitory effect of IL-1beta on aqueous tear production and lacrimal gland protein secre
28 injection into the lacrimal gland inhibited aqueous tear production by 52% and inhibited neurally an
34 neas exhibited significantly reduced NFD and aqueous tear production, and increased inflammatory cell
36 rison to that in age-matched NRTN(+/+) mice, aqueous tear production, tear fluorescein clearance, and
39 ts into groups based on both DE symptoms and aqueous tear production; serotonin concentrations were s
40 venting key signs of dry eye disease such as aqueous tear secretion, conjunctival goblet cells, epith