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1  drug resistance or demonstrated presence of arachnoid adhesions respectively.
2 breakage of a catheter, pump malfunction and arachnoid adhesions.
3 in the more internal connective tissues (pia-arachnoid and endoneurium-perineurium) was also found in
4 cal parenchyma may be transmitted to the pia-arachnoid and might then serve in the induction of neuro
5 be VE-cadherin at intercellular junctions of arachnoid and pia mater cells that form the leptomeninge
6 ers and T cells into the SAS bordered by the arachnoid and pia mater during health and neuroinflammat
7 olecular structure and function of the dura, arachnoid and pia mater in the context of conventional v
8                                    The dura, arachnoid and pia mater, as the constituent layers of th
9 pear as the meninges differentiate into pia, arachnoid, and dura.
10 tral retinal vessels, and in the dura mater, arachnoid, and pia mater of the meningeal sheath surroun
11 ined by meninges, classically known as dura, arachnoid, and pia mater.
12 tures for fibroblasts in the embryonic dura, arachnoid, and pia.
13 he central nervous system (CNS)- dura mater, arachnoid, and piamater - possess unique features that a
14                                      How the arachnoid barrier balances separation and communication
15                     Here, we show that mouse arachnoid barrier cell specification requires the repres
16 noid beneath and separate from E-cadherin(+) arachnoid barrier cells.
17                                 Although the arachnoid barrier creates a partition, communication bet
18                                          The arachnoid barrier delineates the border between the cent
19 rive arachnoid barrier formation, highlights arachnoid barrier fetal functions, and provides novel to
20 identifies fundamental mechanisms that drive arachnoid barrier formation, highlights arachnoid barrie
21                        We also show that the arachnoid barrier is functional prenatally and, in its a
22 on of layer-specific markers, mapping of the arachnoid barrier maturation trajectory, and joint analy
23                                          The arachnoid barrier, a component of the blood-cerebrospina
24 ell types of the pia, inner arachnoid, outer arachnoid barrier, and dural border layer; and contrast
25 e traced the developmental trajectory of the arachnoid barrier, detailing the transcriptomic shifts a
26  create discontinuities where they cross the arachnoid barrier, forming structures that we termed ara
27 tructures that function as routes across the arachnoid barrier.
28  arachnoid layers to one another and for the arachnoid barrier.
29 a layer of Prox1(+) cells located within the arachnoid beneath and separate from E-cadherin(+) arachn
30 eling of cells of the outer meninges (mainly arachnoid), blood vessels (arteries, veins, and capillar
31             The endothelial, epithelial, and arachnoid brain barriers work in concert to selectively
32 ptic nerve US to determine the dimensions of arachnoid bulk (DAB) ratio and ONSD before invasive ICP
33  central nervous system tumors, arising from arachnoid cap cells and typically following a benign cli
34 nical stimulation of adult human and rat pia-arachnoid cell cultures (loaded with calcium indicator d
35            Gap junction coupling between pia-arachnoid cells and astrocytes was shown by dye transfer
36                         Calcium waves in pia-arachnoid cells could invade contiguous astrocytes, and
37                       The differentiation of arachnoid cells in the mutant meninges is also abnormal.
38 th aged human datasets identified two unique arachnoid clusters specific to ageing leptomeninges.
39 ospinal fluid (CSF) efflux structures termed arachnoid cuff exit (ACE) points and speculated that the
40                                              Arachnoid cuff exit (ACE) points, anatomical discontinui
41 d barrier, forming structures that we termed arachnoid cuff exit (ACE) points.
42  dural layer and calvaria (via the so-called arachnoid cuff exit points).
43 els surrounding bridging veins terminate at "arachnoid cuff exit" (ACE) points, which enable waste ef
44 after a trauma, spontaneous hemorrhage in an arachnoid cyst is a rare and serious complication with a
45 mental abnormalities of the corpus callosum, arachnoid cyst, abnormalities of the septum pellucidum a
46 me loss (45%), mega cisterna magna (36%) and arachnoid cysts (27%).
47                                     Cerebral arachnoid cysts (ACs) are one of the most common and poo
48 ach, we reveal several genes associated with arachnoid cysts and identify four phenotypic subtypes of
49  displayed elevated rates of posterior fossa arachnoid cysts and mega cisterna magna findings indepen
50                                              Arachnoid cysts are congenital or developmental intra-ar
51                                              Arachnoid cysts are congenital, benign and intra-arachno
52                          A great majority of arachnoid cysts are congenital.
53                                              Arachnoid cysts are mostly asymptomatic and they can dev
54  cerebellar dysplasia, ventriculomegaly, and arachnoid cysts are nearly invariant.
55                                              Arachnoid cysts form 1% of the non-traumatic lesions whi
56 here is a male dominance at a rate of 3/1 in arachnoid cysts which locate mostly in the middle fossa.
57 has proved useful, such as ventriculomegaly, arachnoid cysts, and abdominal masses, are described.
58 sts and identify four phenotypic subtypes of arachnoid cysts, the severity of which correlates with t
59 e is an uncommon and serious complication of arachnoid cysts, which can give rise to atypical feature
60 bdural hygroma is a rare complication of the arachnoid cysts.
61 ghest penetration ratios were choroid plexus/arachnoid (dolutegravir and tenofovir), CSF (lamivudine)
62 ion defects leading to disruption of the pia-arachnoid, ectopia of fibroblasts in the cortex, and rea
63 enhancement patterns are characterized: dura-arachnoid enhancement and pia-subarachnoid space enhance
64  in the meninges is reduced, causing loss of arachnoid fibroblasts that express Raldh2, an enzyme req
65 ptomeninges, consisting of the pia mater and arachnoid, form a connective tissue investment and barri
66                      This study examines the arachnoid fossae (AF) in crania from early modern Wrocla
67                                              Arachnoid granulations (AG) are poorly investigated.
68 an ex vivo model of CSF outflow across human arachnoid granulations (AGs) as an approximation of in v
69  lytic lesions on skull radiographs, seen as arachnoid granulations fovea in CT.
70                  Notably, we also identified arachnoid granulations involved in cerebrospinal fluid r
71 rents, or decreased CSF reabsorption via the arachnoid granulations or other pathways.
72 e cranial bone, which arise as the result of arachnoid granulations' (AG) protruding and causing the
73 esorption into the blood circulation through arachnoid granulations.
74 hus representing the mouse equivalent of the arachnoid granulations.
75 ood vessels and later within neurons and pia arachnoid (> or =3 hours), particularly within piriform
76 sive mononeuropathies, 2 (CI: 2, 3); and sub-arachnoid haemorrhage, 1 (CI: 0.8, 2).
77 xtent that the injury is associated with sub-arachnoid hemorrhage and hematoma.
78 ral nerve root sleeve tear through which the arachnoid herniates, which contrasted the common percept
79 aining was detected in the pia mater, in the arachnoid, in venous sinuses, and among the layers of th
80 vessels in the subarachnoid space and in the arachnoid layer of the meninges.
81 over trigeminal sensory neurons and over the arachnoid layers surrounding trigeminal ganglia supports
82 both TNFR subtypes was also present over the arachnoid layers surrounding trigeminal ganglia.
83 n of cell types responsible for adherence of arachnoid layers to one another and for the arachnoid ba
84 oid space (enclosed by the meningeal pia and arachnoid layers), and the outmost meningeal dural layer
85 E) points, anatomical discontinuities in the arachnoid mater around bridging veins, serve as key site
86 widespread infection of cells of the pia and arachnoid mater of the leptomeninges over large areas of
87 , a triple layer of membranes-the pia mater, arachnoid mater, and dura mater-surround the CNS, encomp
88 ake mechanisms, GABA transporters in the pia-arachnoid may help to regulate the amount of GABA availa
89 r indirectly across the choroid plexuses and arachnoid membrane (blood-CSF barrier) into the CSF and
90 outflow may occur in a similar manner in the arachnoid membrane adjacent to the granulations, in addi
91           We also found discontinuity of the arachnoid membrane near the cribriform plate, which prov
92  suggesting the need for a model using human arachnoid membrane with granulations for the study of co
93                                  The brain's arachnoid membrane with granulations is an important bio
94  can develop anywhere in the brain along the arachnoid membrane.
95  a result of splitting or duplication of the arachnoid membrane.
96 y of CSF but also of metabolites through the arachnoid membrane.
97                                          The arachnoid membranes thicken.
98          Meningiomas (MNs), arising from the arachnoid/meningeal layer, are nonresponsive to chemothe
99 ell lines were implanted between the pia and arachnoid meninges as well as in the sciatic nerve to mi
100 tor outer segments, and in the fascicles and arachnoid of the optic nerve.
101 ically distinct cell types of the pia, inner arachnoid, outer arachnoid barrier, and dural border lay
102 2-MRM; this "bud-on-branch" sign reflects an arachnoid outpouching herniating through a lateral dural
103                                              Arachnoid outpouching was confirmed intraoperatively in
104  data were reviewed for lateral dural tears, arachnoid outpouching, and ruptured spinal meningeal div
105                                     The dura-arachnoid pattern consists of curvilinear enhancement ov
106  in vertebrates comprise three layers (dura, arachnoid, pia mater), representing an important barrier
107 overmigrated neurons into the developing pia-arachnoid, scattering its mesenchymal cells throughout t
108          The causes of hyperintensity in the arachnoid space in this sequence can be divided into two
109 igration of neurons and glial cells into the arachnoid space results in the formation of cortical dys
110 ir transport of fluorescent solutes from sub-arachnoid space to brain in mice or rats.
111 , and Scleral Thickness (at the Anterior Sub-arachnoid space) were calculated for each ONH.
112  accumulate in the ventricles, meninges, sub-arachnoid spaces, and injection site.
113 Meningiomas are tumors that originate in the arachnoid villi and are the most common non-glial neopla
114                                              Arachnoid villi occlude and degenerate.
115                                      The pia-arachnoid waves were blocked by either octanol or apyras
116  3 was almost entirely restricted to the pia-arachnoid, whereas mGATs 1 and 4 were present only in sp

 
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