ライフサイエンスコーパス: arsenite

1 ned for sensitive and selective detection of arsenite.

2 ic accumulation and increased sensitivity to arsenite.

3 lectively neddylated at Lys85 in response to arsenite.

4 nic speciation due to its selectivity toward arsenite.

5 reducing the central arsenic atom (As(V)) to arsenite.

6 er pentavalent organoarsenicals or inorganic arsenite.

7 ollution of groundwater, which is largely by arsenite.

8 ditions of severe stress induced with sodium arsenite.

9 s may explain the limited phloem mobility of arsenite.

10 Plants chemically reduce arsenate to arsenite.

11 n SGs following exposure to the stress agent arsenite.

12 M arsenite with a linear range up to 100 muM arsenite.

13 were both unstable and partially reduced to arsenite.

14 ation of the arsB mutant restored the MIC of arsenite.

15 resulted in a 16-fold increase in the MIC of arsenite.

16 rease, then decrease of monothioarsenate and arsenite.

17 s showed that arxA was strongly induced with arsenite.

18 to isolate cells with defective response to arsenite.

19 nothioarsenate can have higher mobility than arsenite.

20 for the proatherogenic effects of inorganic arsenite.

21 sts fail to properly form SGs in response to arsenite.

22 ed by exposing p53-knockdown HBECs to sodium arsenite (2.5 muM) for 16 weeks.

23 vations indicating that GADD34 is induced by arsenite, a thiol-directed oxidative stressor, in the ab

24 Arsenite, a trivalent form of arsenic, is an element tha

25 Exposure to arsenite also diminished the recruitment of BRCA1 and RA

26 Treatments with arsenite also led to a dose-dependent decrease in the le

27 Arsenite also recruited a TDP-43 kinase, casein kinase-1

28 Arsenite also stabilized GADD34 protein, slowing its deg

29 Sodium arsenite, an agent that induces oxidative stress, promot

30 e we have studied four stress agents: sodium arsenite, an oxidative agent; Gramicidin, eliciting K(+)

31 sed sensitivities of 0.91 +/- 0.07 mV/mM for arsenite and 0.98 +/- 0.02 mV/mM for arsenate.

32 ted limits of detection (LODs) of 13 muM for arsenite and 132 muM for arsenate.

33 el signaling networks largely accounting for arsenite and antimonite action.

34 of cultured human epidermal keratinocytes to arsenite and antimonite in contrast to comparisons of ar

35 The striking parallels between responses to arsenite and antimonite indicate the skin carcinogenic r

36 rs and affected signaling pathways following arsenite and antimonite treatments.

37 ansgenic lines showed increased tolerance to arsenite and arsenate and a greater capacity for arsenat

38 ion was higher at pH 7.0 compared to 4.5 for arsenite and arsenate and vice versa for monothioarsenat

39 ion, desorption, and readsorption of aqueous arsenite and arsenate by CuO-NP.

40 We report the first example of arsenite and arsenate removal from water by incorporatio

41 The kinetics and efficiencies of arsenite and arsenate removal from water were evaluated

42 rs emitting bioluminescence as a response to arsenite and arsenate was applied during a field campaig

43 worms from F0 and F1 generations accumulated arsenite and arsenate when F0 L4 larvae were exposed to

44 -through fractionation of inorganic arsenic (arsenite and arsenate) in environmental solids in combin

45 arsenate, and compared it to the sorption of arsenite and arsenate, in suspensions containing 2-line

46 studies often focus on inorganic arsenic as arsenite and arsenate, neglecting the organoarsenicals,

47 ed in 8- and 4-fold reduction in the MICs of arsenite and arsenate, respectively, and complementation

48 cell, yielding a self-powered biosensor for arsenite and arsenate.

49 two genes, contributes to the resistance to arsenite and arsenate.

50 significantly different decay rates between arsenite and control treatment.

51 ls were further increased by the addition of arsenite and GSH to the medium, indicating that GGCT2;1

52 stress and found that the oxidative stressor arsenite and heat shock-activated stress responses evide

53 f the selective intracellular recognition of arsenite and its pumping out from the cell.

54 ta indicate that signaling events induced by arsenite and oxidative stress may regulate LRRK2 functio

55 Arsenite and phosphite were confirmed to be the best cat

56 es in the presence of the reduced precursors arsenite and sulfide.

57 The kinetic extraction profiles of arsenite and total inorganic arsenic are obtained for ea

58 Arsenite and total inorganic arsenic in each subfraction

59 in situ applicability for treatment of both arsenites and arsenates, and contrary to all known compe

60 s (hydrogen peroxide), a heavy metal stress (arsenite) and an amino acid analogue (azetidine-2-carbox

61 ilarity between trivalent inorganic arsenic (arsenite) and antimony (antimonite), we hypothesized tha

62 exposure of cells to the environmental toxin arsenite, and in a proteasome mutant, loss of Cuz1 enhan

63 53 whose steady-state levels were altered by arsenite, and of these, only 4 exhibited significantly d

64 i linked to growth traits under heat stress, arsenite, and paraquat, the majority of which were best

65 ure to noncytotoxic concentrations of sodium arsenite, and we confirmed some of these changes using r

66 electron donor for reduction of arsenate to arsenite; and d) As has a high affinity for sulfhydryl g

67 Thus, sodium arsenite appears to promote SG formation and TDP-43 modi

68 By equilibrating arsenite, arsenate, and monothioarsenate with purified m

69 rsenic (TUA): TUA1 was defined as the sum of arsenite, arsenate, monomethylarsonic acid, and dimethyl

70 efficiencies of inorganic oxoanions such as arsenite, arsenate, phosphite, phosphate, and borate is

71 luoride, persulphate, acetate, thiosulphate, arsenite, arsenate, sulphite, and iodide.

72 Our results also suggest arsenite as a general inhibitor for RING finger E3 ubiqu

73 'Photoarsenotrophy', the use of arsenite as an electron donor for anoxygenic photosynthe

74 On the other hand, arsenite As(III) is significantly adsorbed on goethite,

75 The reversible inhibition of laccase by arsenite (As(3+)) and arsenate (As(5+)) is reported for

76 of arsenate (As(V) ) generated by microbial arsenite (As(III) ) oxidation is poorly understood.

77 of yeast with the toxic trivalent metalloid arsenite (As(III)) also activates Hog1 as part of a prot

78 We found that CeO(2) NPs adsorbed both arsenite (As(III)) and arsenate (As(V)) ions and the ads

79 U-As(IMM), of urinary inorganic As species, arsenite (As(III)) and arsenate (As(V)), and their metab

80 mine drainage (AMD) and are most harmful as arsenite (As(III)) and hexavalent (Cr(VI)).

81 e (S(-II)) with NOM and its consequences for arsenite (As(III)) binding.

82 lence and compound speciation, and inorganic arsenite (As(III)) compounds are the most toxic to human

83 robes biotransform both arsenate (As(V)) and arsenite (As(III)) into more toxic methylated metabolite

84 Oxidation of arsenite (As(III)) is a critical yet often weak link in

85 The presence of the arsenite (As(III)) methyltransferase gene (arsM) in soil

86 The redox chemistry of chromate (Cr(VI)) and arsenite (As(III)) on the iron oxyhydroxide, ferrihydrit

87 methylarsenite (MAs(III)) by methylation of arsenite (As(III)) or reduction of methylarsenate (MAs(V

88 onmental degradation to more toxic inorganic arsenite (As(III)) that contaminates crops and drinking

89 Many microbes methylate inorganic arsenite (As(III)) to more toxic and carcinogenic methyl

90 Binding of arsenite (As(III)) to sulfhydryl groups (Sorg(-II)) play

91 accompanied by the simultaneous oxidation of arsenite (As(III)) was achieved using an electrochemical

92 us suspension of goethite in the presence of arsenite (As(III)) was investigated with X-ray absorptio

93 lucidated the molecular-scale interaction of arsenite (As(III)) with Fe(III)-NOM complexes under redu

94 methylation of As is catalyzed by the enzyme arsenite (As[III]) S-adenosylmethionine methyltransferas

95 I)] to less toxic and carcinogenic inorganic arsenite [As(III)] by C-As bond cleavage.

96 Arsenite [As(III)] can be oxidized to arsenate [As(V)] b

97 strate that Yap8 directly binds to trivalent arsenite [As(III)] in vitro and in vivo and that approxi

98 ability to rice plants, whereas oxidation of arsenite [As(III)] results in As immobilization.

99 Some arsenite [As(III)]-oxidizing bacteria exhibit positive c

100 onmental degradation to more toxic inorganic arsenite [As(III)].

101 used by the reduction of arsenate [As(V)] to arsenite [As(III)].

102 Arsenite, as well as other agents, triggered relocalizat

103 and the valence state of inorganic arsenic (arsenite, As(III) vs. arsenate As(V)) can be modulated b

104 itions and mechanism(s) for the reduction of arsenite, As(III), by pyrite are incompletely understood

105 adsorption mechanism of arsenate, As(V), and arsenite, As(III), on MNPs by macroscopic adsorption exp

106 tion near-edge spectroscopy showed arsenate, arsenite, As-(GS)3, and As-PCs with varying ratios in va

107 Microbial arsenite (AsIII) oxidation forms a critical piece of the

108 t of barley (Hordeum vulgare) seedlings with arsenite (AsIII) rapidly induced physiological and trans

109 -MS)-for the detection of arsenic(III) ions (arsenite, AsO2(-)) in aqueous solution.

110 rsenate because of partial transformation to arsenite at pH 4.5.

111 the root, facilitating efflux of arsenic as arsenite back into the soil to limit both its accumulati

112 etects bacterial biomethylation of inorganic arsenite both in vivo and in vitro with detection limits

113 specifically suppressed ER stress induced by arsenite but not tunicamycin.

114 wed that monothioarsenate is less toxic than arsenite, but more toxic than arsenate at concentrations

115 For arsenite, but not for sorbitol, quenching oxidative stre

116 disassembled canonical SGs induced by sodium arsenite, but not those induced by hydrogen peroxide, le

117 ontaminated drinking water and food, and the arsenite can accumulate in the human tissues.

118 of As(V)/AsSum (total combined arsenate and arsenite concentrations) (0.59-0.78), coupled with high

119 d to high dose of arsenite through consuming arsenite-contaminated drinking water and food, and the a

120 our study unveiled, for the first time, that arsenite could alter epigenetic signaling by targeting t

121 Herein, we found that arsenite could bind directly to the RING finger domains

122 Herein, we found that arsenite could bind directly to the zinc fingers of Tet

123 t a variety of weak acids (silicate, borate, arsenite, cyanide, carbonate, and sulfide) cannot only b

124 f beta-galactosidase is under control of the arsenite-derepressable arsR-promoter.

125 t pH 7.0 suggest mobilization to proceed via arsenite desorption, reaction with dissolved or surface-

126 roducible response generating matrix for the arsenite detection at an ultratrace concentration in aqu

127 development of a whole-cell based sensor for arsenite detection coupling biological engineering and e

128 Arsenite detection is demonstrated with three different

129 Arsenic trioxide and sodium arsenite did not directly modify or inhibit the activity

130 on of the As-hyperaccumulator Pteris vittata arsenite efflux (PvACR3), on As tolerance, accumulation,

131 enite efflux, arsenate reduction followed by arsenite efflux and arsenite methylation.

132 pressed the yeast (Saccharomyces cerevisiae) arsenite efflux transporter ACR3 into Arabidopsis to eva

133 Known mechanisms include arsenite efflux, arsenate reduction followed by arsenite

134 sistance to arsenate is reduction coupled to arsenite efflux.

135 te DNA damage with a high-fat diet or sodium arsenite exacerbated adipocyte senescence and metabolic

136 )-(oxyhydr)oxides and that both arsenate and arsenite exclusively formed monodentate-binuclear ("brid

137 In arsenite-exposed cells, 186 probe set-identified transcr

138 tive strategies delineating the link between arsenite exposure and metabolic disorders.

139 , transgenerational reproductive toxicity by arsenite exposure and the underlying mechanisms in C. el

140 Our study demonstrates that maternal arsenite exposure causes transgenerational reproductive

141 impact and genetic circuitry involved during arsenite exposure in bacteria.

142 e of C. elegans was significantly reduced by arsenite exposure in F0 and that this reduction in brood

143 ng mechanisms underlying the implications of arsenite exposure in metabolic disorders.

144 We conducted a literature search focused on arsenite exposure in vivo and in vitro, using relevant e

145 Mechanistically, arsenite exposure significantly inhibited autophagy nece

146 aggregation and proteolysis after prolonged arsenite exposure, GADD34-bound CK1 catalyzed TDP-43 pho

147 00637 human skin fibroblast cells induced by arsenite exposure.

148 The extent of complexation was highest for arsenite, followed by monothioarsenate and arsenate.

149 Together with the preferential formation of arsenite following sample dilution, these data provide e

150 d arsenate when F0 L4 larvae were exposed to arsenite for 24 h.

151 king water with 0, 250 ppb, or 25 ppm sodium arsenite for 5 wk and then challenged intratracheally wi

152 Some arsenite formed intermediately, which was subsequently a

153 uent generations (F1-F5) were cultured under arsenite-free conditions.

154 ts lacking HAC1 lose their ability to efflux arsenite from roots, leading to both increased transport

155 Irradiation of the arsenite/goethite under conditions where dissolved oxyge

156 s may be involved, our findings support that arsenite has effects on whole-body glucose homeostasis,

157 ferent exogenous stressors, including sodium arsenite, heat, and hippuristanol.

158 particulate matter (PM), i.e., the inorganic arsenite iAs(III) and arsenate iAs(V), and the methylate

159 toxic levels of arsenic, including inorganic arsenite (iAs3+, </= 5 muM), inorganic arsenate (</= 20

160 tINT2 in Xenopus laevis oocytes also induced arsenite import.

161 technique can determine the concentration of arsenite in a few min with a detection limit of 0.1 ppb

162 Predominance of arsenite in control experiments and no evidence of surfa

163 nges in mRNA stability in response to sodium arsenite in human fibroblasts.

164 Oral administration of arsenite in mice resulted in heavy accumulation in brown

165 proved that the intermediate accumulation of arsenite in the drainage channel is microbially catalyze

166 C1 therefore functions to reduce arsenate to arsenite in the outer cell layer of the root, facilitati

167 eover, GO-SH demonstrates selectivity toward arsenite in the presence of arsenate.

168 oring a possibility of the quantification of arsenite in the ultratrace concentration range, the Ru N

169 expression and activity are up-regulated by arsenite, in a manner dependent on activating transcript

170 aled that As was mobilized, predominantly as arsenite, in all treatments with relative mobilization i

171 As concentrations, the percentage of unbound arsenite increased in the vein and mesophyll of young ma

172 Cells exposed to 10muM sodium arsenite increased the stability of HIPK1 and HIPK2 prot

173 Similarly, in dopaminergic MN9D cells, arsenite induced the export of endogenous Nurr1, resulti

174 culture (SILAC) to assess quantitatively the arsenite-induced alteration of global kinome in human ce

175 Arsenite-induced dephosphorylation is accompanied by los

176 Here, we identify genes required for arsenite-induced ER stress response in a genome-wide RNA

177 r screen discovered several genes modulating arsenite-induced ER stress, including sodium-dependent n

178 el are assessed against experimental data of arsenite-induced genotoxic damage to human hepatocytes;

179 Interestingly, we found that arsenite-induced genotoxic stress causes a PLK1-dependen

180 ibitor, flavopiridol, partially restored the arsenite-induced growth inhibition of human skin fibrobl

181 The majority of the arsenite-induced L-glutamate influx was via a high-affin

182 e 1 (PLK1) as one of the kinases involved in arsenite-induced NOTCH1 down-modulation.

183 Here, we report that arsenite-induced oxidative stress differs from thapsigar

184 dylation promotes SG assembly in response to arsenite-induced oxidative stress.

185 d that the deletion of p50 (p50-/-) impaired arsenite-induced p53 protein expression, which could be

186 dylatable SRSF3 (K85R) mutant do not prevent arsenite-induced polysome disassembly, but fails to supp

187 ine norovirus (MNV) infection did not impact arsenite-induced SG assembly or G3BP1 integrity, suggest

188 l-time RT-PCR confirmed a major, significant arsenite-induced stabilization of the mRNA encoding delt

189 ermined that ZIKV disrupted the formation of arsenite-induced stress granules and changed the subcell

190 remodeling of the RNA-bound proteome during arsenite-induced stress, distinct from autophagy-related

191 m target histone-3 are phosphorylated during arsenite-induced stress.

192 Arsenite induces oxidative stress, which is linked to me

193 We have identified arsenite- inducible regulatory particle-associated prote

194 -1), encoded by a previously uncharacterized arsenite-inducible gene in budding yeast.

195 titutive P (trc) promoter (Ptrc-cfrA) or the arsenite-inducible promoter P (arsB) (Pars-cfrA).

196 inger AN1-type domain 2a gene, also known as arsenite-inducible RNA-associated protein (AIRAP), was r

197 AIRAPL (arsenite-inducible RNA-associated protein-like) is an ev

198 icals, including arsenic trioxide and sodium arsenite, inhibited activation of the NLRP1, NLRP3, and

199 Enrichment cultures converted 63% of arsenite into methylated products, with dimethylarsinic

200 d-type background showed increased efflux of arsenite into the external medium.

201 roscope and useful to estimate the amount of arsenite ions in various water samples.

202 a differential pulse voltammetric technique, arsenite is oxidized to arsenate leading to its quantita

203 were pursued with an oxidative agent (sodium arsenite), K-releasing agent (Gramicidin) and a metal io

204 ects of targeting telomeres with sodium meta-arsenite (KML001) (an agent undergoing early clinical tr

205 s, inositol transporters are responsible for arsenite loading into the phloem, the key source of arse

206 Following arsenite maternal exposure of the F0 generation, subsequ

207 Thus, sodium arsenite may confer its cytotoxic effect partly through

208 ent study revealed, for the first time, that arsenite may exert its carcinogenic effect by targeting

209 t into gels/solids in response to transient, arsenite-mediated stress.

210 te reduction followed by arsenite efflux and arsenite methylation.

211 a previously uncharacterized, human-specific arsenite methyltransferase (AS3MT) isoform (AS3MT(d2d3))

212 e (AS3MT) isoform (AS3MT(d2d3)), which lacks arsenite methyltransferase activity and is more abundant

213 We conclude that arsenite modification of mRNA stability is relatively un

214 osure to cadmium chloride (CdCl2) and sodium arsenite (NaAsO2).

215 Pregnant CD-1 mice were exposed to sodium arsenite [none (control), 10 ppb, or 42.5 ppm] in drinki

216 ular heme caused by the massive induction by arsenite of heme oxygenase mRNA (HMOX1; 68-fold increase

217 rrent knowledge gaps in our understanding of arsenite on diabetes development.

218 cing, we characterized the global effects of arsenite on gene expression in Agrobacterium tumefaciens

219 When exposed to 25 muM arsenite or arsenate overnight, most inorganic arsenic w

220 ooded conditions, and rice plants exposed to arsenite or DMA(V) were grown to maturity in nonsterile

221 Here, we show that arsenite or H2O2-induced stresses promote loss of Ser(91

222 n FCV-infected cells that were stressed with arsenite or hydrogen peroxide.

223 The arsenite oxidase (AioBA) is regulated by a three-compone

224 nes faecalis) that was an early isolate with arsenite oxidase activity.

225 es encoding both subunits of the respiratory arsenite oxidase AioA and the dissimilatory arsenate red

226 Finally, we demonstrate that anaerobic arsenite oxidase and respiratory arsenate reductase cata

227 ncient lineage of DMSORs compared to aerobic arsenite oxidase catalytic subunits, which evolved from

228 A new arsenite oxidase clade, ArxA, represented by the haloalk

229 nature of the molybdenum active site of the arsenite oxidase from the Alphaproteobacterium Rhizobium

230 CIB 8687, the betaproteobacterium from which arsenite oxidase had its structure solved and the first

231 Arsenite oxidase is thought to be an ancient enzyme, ori

232 ly arsenate respiratory reductase, ArrA, and arsenite oxidase, AioA (formerly referred to as AroA and

233 ere arxA is predicted to encode for the sole arsenite oxidase.

234 These results suggest that ArxA-type arsenite oxidases appear to be widely distributed in the

235 ces as distinct members of the ArxA clade of arsenite oxidases.

236 Arsenite oxidation decreases arsenic toxicity and mobili

237 Arsenotrophy, growth coupled to autotrophic arsenite oxidation or arsenate respiratory reduction, oc

238 The role of arxA in photosynthetic arsenite oxidation was confirmed by disrupting the gene

239 um, resulting in the loss of light-dependent arsenite oxidation.

240 acNaV derived from NaVAe1, a BacNaV from the arsenite oxidizer Alkalilimnicola ehrlichei found in Mon

241 L15 is an alphaproteobacterium isolated from arsenite-oxidizing biofilms whose draft genome contains

242 ation studies using E. coli or C. glutamicum arsenite permease mutants clearly show that CgAcr3-1 is

243 Arsenite preconcentrates onto the Ru surface just by dip

244 hat CgAcr3-1 is an antiporter that catalyzes arsenite-proton exchange with residues Cys129 and Glu305

245 treatment of cells with puromycin or sodium arsenite, reagents that arrest translation, also resulte

246 Here, we demonstrate that ARS2 (arsenite-resistance protein 2), a zinc finger protein th

247 Here, we show that menin interacts with arsenite-resistant protein 2 (ARS2), a component of the

248 40 in vitro and in cells, and treatment with arsenite resulted in substantially impaired H2B ubiquiti

249 volatilization can be mediated by the enzyme arsenite S-adenosylmethionine methyltransferase (ArsM) o

250 Thus formed Ru NPs have the arsenite selective surface and conducting core that is i

251 Six "target" oxo-anion pollutants (arsenate, arsenite, selenate, selenite, chromate, and perchlorate)

252 n a proteasome mutant, loss of Cuz1 enhances arsenite sensitivity.

253 eover, cells induced to undergo apoptosis by arsenite show increased R-CRT on their cell surface.

254 Our study revealed that arsenite significantly reduced differentiation of murine

255 urface just by dipping the RuNPs/GC into the arsenite solution as it interacts chemically with Ru NPs

256 mpetitive effect of silicate on arsenate and arsenite sorption increased with increasing silicate pre

257 polymerization was slowest, was arsenate and arsenite sorption not affected by the presence of silica

258 Highly selective separation of arsenite species [As(III)] was achieved by chelation wit

259 e RNAs in stress granules and P-bodies under arsenite stress and compare those results to those for t

260 erall changes in Ago2-mRNA interactions upon arsenite stress by cross-linking immunoprecipitation (CL

261 nscript to be translationally induced during arsenite stress conditions.

262 Arsenite stress quickly and reversibly decreased asymmet

263 Arsenite stress stimulates LRRK2 self-association and as

264 tress granule breakdown during recovery from arsenite stress, indicating a possible role for hYVH1 in

265 mRNAs under nonstress conditions, but during arsenite stress, when translation is globally repressed,

266 ished RNA localization in the cytosol during arsenite stress.

267 sporter, LAT1, suppressed mTOR activation by arsenite, supporting a role for these transporters in mo

268 When exposed to arsenite, the endodermis was again a site of accumulatio

269 rsenate, which is then reduced by PvGSTF1 to arsenite, the form of arsenic stored in the vacuoles of

270 ons cause a greater proportion of pore-water arsenite, the more toxic form of arsenic, in the rhizosp

271 Humans are exposed to high dose of arsenite through consuming arsenite-contaminated drinkin

272 nd seed arsenic concentrations when fed with arsenite through the leaves.

273 ed arsenic concentrations in plants fed with arsenite through the roots, relative to wild-type plants

274 Results suggest an effective oxidation of arsenite to arsenate on the surface of CuO-NP.

275 with varying concentrations of poly(I:C) or arsenite to induce the ISR, we provide additional proof

276 by coupling the intracellular recognition of arsenite to the generation of an electrochemical signal.

277 0.44, 0.74, 0.15, 0.17 and 0.67 ng L(-1) for arsenite, total inorganic, mono-, dimethylated As and tr

278 nts in the highly conserved ASNA1 gene (arsA arsenite transporter, ATP-binding, homolog), which encod

279 e stress-induced translational repression in arsenite-treated cells expressing either wild-type or am

280 a PERK-independent eIF2alpha kinase through arsenite treatment and is independent of activating tran

281 response to hyperosmolarity, heat shock, and arsenite treatment but rapidly dephosphorylated after ox

282 in and accumulates in stress granules during arsenite treatment of human cells.

283 reversible: oxidative stress resulting from arsenite treatment transformed large IBs into a scatteri

284 BP1 co-localize to stress granules following arsenite treatment, but not during mitosis.

285 on also occurs in cells during heat-shock or arsenite treatment, when poly-ubiquitinated proteins acc

286 in steady-state ALAS1 mRNA levels seen after arsenite treatment.

287 ed peat (2.80 +/- 0.02 angstrom) compared to arsenite treatments (2.73 +/- 0.01 angstrom).

288 In sulfidic solutions containing arsenite, two thioarsenic species with S/As ratios of 2:

289 Thioarsenates form from arsenite under sulfate-reducing conditions, e.g., in ric

290 This study suggests that arsenite was predominantly complexed with carboxylic gro

291 d that this As was mostly arsenate, although arsenite was present on the edge of the Fe deposit.

292 Arsenite was present only as a minor species (3-5%) in t

293 Here, treatment of human erythrocytes with arsenite was shown to induce the uptake of L-glutamate a

294 anism against toxic arsenic species, such as arsenite, which consists of the selective intracellular

295 d biosensor has a detection limit of ~40 muM arsenite with a linear range up to 100 muM arsenite.

296 and antimonite in contrast to comparisons of arsenite with other metal compounds.

297 ed as an intermediate by abiotic reaction of arsenite with sulfide.

298 hich grows by coupling arsenate reduction to arsenite with the oxidation of sulfide to sulfate.

299 onothioarsenate forms by abiotic reaction of arsenite with zerovalent sulfur.

300 was generally lower compared to arsenate and arsenite, with the exception of the near instantaneous a