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1 of 25-um microspheres recovered in systemic arterial blood.
2 .72), or partial carbon-dioxide pressure in arterial blood (-0.3 mm Hg; 95% CI, -0.8 to 0.2 mm Hg; P
4 s that die after successful reperfusion with arterial blood actually are killed by changes initiated
5 , blood gases, and plasma-free hemoglobin in arterial blood, as well as blood entering and exiting th
10 stimulate breathing when oxygenation of the arterial blood decreases; and pulmonary arterial smooth
14 trated by observations of faster recovery of arterial blood flow and large numbers of newly formed ar
15 serelaxin for 120 min increased total renal arterial blood flow by 65% (95% CI 40%, 95%; p < 0.001)
16 ge-field capacitor technology, for measuring arterial blood flow in both contact and non-contact mode
19 In IUGR baboons there was increased carotid arterial blood flow velocity during late systole and dia
21 , deep-tissue and ultrafast imaging of mouse arterial blood flow with an unprecedented frame rate of
22 tional microvascular density, independent of arterial blood flow, while disturbance of the microcircu
25 nd-tidal alveolar dead space fraction (first arterial blood gas after intubation) (per 0.1 unit incre
31 exercise maintained alveolar ventilation and arterial blood gas homeostasis but at the expense of ear
32 echanically ventilated patients may not have arterial blood gas measurements available at relevant ti
39 een blood eosinophil count on admission with arterial blood gas values, duration of mechanical ventil
40 O2 and VCO2), heart rate, cardiac output and arterial blood gas variables at peak exercise on a cycle
41 ompared with the baseline period, unadjusted arterial blood gas, chest radiograph, and RBC utilizatio
42 ased provider financial incentives targeting arterial blood gas, chest radiograph, and RBC utilizatio
43 ventilation, for each protocol, we recorded arterial blood gas, respiratory mechanics, alveolar recr
44 ygen saturation by pulse oximetry (SpO(2) ), arterial blood gas, spirometry, and contrast-enhanced ec
49 in Sherpa compared to lowlanders we measured arterial blood gases and global cerebral blood flow (dup
50 he respiratory center's output to changes in arterial blood gases and pH, is one of the most importan
51 rcised at approximately 85% of maximum while arterial blood gases and work of breathing were assessed
52 very (CDO(2) ), oesophageal temperature, and arterial blood gases during exposure to three commonly e
54 r OHS is not very high (<20%) but to measure arterial blood gases in patients strongly suspected of h
55 arterial blood gases from 703 patients; 650 arterial blood gases were associated with SpO2 less than
58 dmissions of ventilated TBI patients who had arterial blood gases within 24 h of admission to the ICU
59 e intervention was associated with 128 fewer arterial blood gases, 73 fewer chest radiographs, and 16
60 imate diaphragm energy expenditure (effort), arterial blood gases, airway pressure, tidal volume and
61 ntaris muscles while monitoring respiration, arterial blood gases, and blood glucose in mice exposed
62 genation macrocirculation, echocardiography, arterial blood gases, and microcirculation parameters di
63 the last minutes of each phase, we measured arterial blood gases, changes in end-expiratory lung vol
64 t program designed to decrease the avoidable arterial blood gases, chest radiographs, and RBC utiliza
65 primary outcome was the number of orders for arterial blood gases, chest radiographs, and RBCs per pa
67 3 +/- 2 kg m(-2) ), FMD (Duplex ultrasound), arterial blood gases, Hct and [Hb], blood viscosity, and
68 Toward the end of each phase, we measured arterial blood gases, inspiratory effort, and work of br
69 e end-expiratory pressure level, we assessed arterial blood gases, respiratory mechanics, ventilation
74 rinsulinemic (4x basal) hyperglycemic clamp (arterial blood glucose 146 +/- 2 mg/dL) with portal GLC
75 od leaving the CNS capillary bed compared to arterial blood, indicating efflux from the CNS into the
76 brain arterio-venous malformations, shunting arterial blood into the brain's deep venous system throu
78 We suggest that the rapid rise pattern of arterial blood nicotine concentration stimulates and the
79 arotid body is a sensory organ for detecting arterial blood O2 levels and reflexly mediates systemic
83 ts (44.8%) had lactic acidosis defined as an arterial blood pH less than 7.35 and a lactate concentra
84 control rate (defined as achievement of mean arterial blood pressure >=65 mm Hg, with urine flow >=0.
85 etting by low systolic (</=90 mm Hg) or mean arterial blood pressure (</=65 mm Hg) accompanied by sig
87 er 9th, 2014), with continuous monitoring of arterial blood pressure (ABP) and intracranial pressure
90 pertonic NaCl produces a greater increase in arterial blood pressure (ABP) than equi-osmotic mannitol
92 lin is a ubiquitous peptide that can elevate arterial blood pressure (ABP) yet understanding of the m
93 n of central chemoreceptors by CO2 increases arterial blood pressure (ABP), sympathetic nerve activit
94 red whether the methods identify the optimal arterial blood pressure (ABPopt) and lower limit of auto
95 calculated by transfer function analysis of arterial blood pressure (BP) and cerebral blood flow vel
96 al mechanisms responsible for maintenance of arterial blood pressure (BP) during haemorrhage in human
98 can predict the limb BCG in responses to the arterial blood pressure (BP) waves in the aorta was deve
103 from t = 0-70 min plus hemorrhage to a mean arterial blood pressure (MAP) of 30 mmHg from t = 40-70
105 heart rate (HR) and digital heart-level mean arterial blood pressure (MAP) were continuously recorded
106 gulation (CA) is often expressed by the mean arterial blood pressure (MAP)-cerebral blood flow (CBF)
108 transient tachycardia and a biphasic caudal arterial blood pressure (PCA) response that are in direc
109 vessels to appropriately react to changes in arterial blood pressure (pressure reactivity) is impaire
110 ion coefficient = -0.29; p = 0.015) and mean arterial blood pressure (repeated-measures correlation c
111 dichotomized subjects into two groups: mean arterial blood pressure 70-90 and greater than 90 mm Hg.
112 er than 90 mm Hg (42%) as compared with mean arterial blood pressure 70-90 mm Hg (15%) (absolute risk
116 Fetuses were first instrumented to measure arterial blood pressure and carotid artery blood flow an
117 if cardiopulmonary resuscitation-targeted to arterial blood pressure and coronary perfusion pressure
118 n system (RAS) is a principal determinant of arterial blood pressure and fluid and electrolyte balanc
119 er hemodilution, treated animals show higher arterial blood pressure and have a stable body temperatu
120 thetic control circuits to increase systemic arterial blood pressure and heart rate with the purpose
121 [OH]D) concentration is associated with high arterial blood pressure and hypertension risk, but wheth
122 gical or biochemical measure, including mean arterial blood pressure and inotrope use during the 48 h
123 on of AIP into the PVN significantly reduced arterial blood pressure and lumbar sympathetic nerve dis
124 he association of systolic and mean invasive arterial blood pressure and noninvasive blood pressure w
125 difference between clinically observed mean arterial blood pressure and optimal mean arterial blood
126 difference between clinically observed mean arterial blood pressure and optimal mean arterial blood
127 difference between clinically observed mean arterial blood pressure and optimal mean arterial blood
129 tor-PKC activity in the hypothalamus reduces arterial blood pressure and sympathetic nerve discharges
130 arterial blood pressure outside optimal mean arterial blood pressure and the absolute difference betw
133 perfusion pressure (CPP), calculated as mean arterial blood pressure at midbrain level minus ICP, was
134 Metaboreceptor function, defined as mean arterial blood pressure at the end of postexercise circu
135 diopulmonary bypass did not differ, the mean arterial blood pressure at the limit of autoregulation a
136 rial blood pressure was defined as that mean arterial blood pressure at the lowest cerebral oximetry
138 terial blood pressure closer to optimal mean arterial blood pressure calculated by bedside multimodal
139 ure have worse outcomes than those with mean arterial blood pressure closer to optimal mean arterial
144 spital cardiopulmonary resuscitation with 1) arterial blood pressure during cardiopulmonary resuscita
145 01 to 0.37 +/- 0.01 mm (P < 0.001), and mean arterial blood pressure from 83 +/- 1 to 78 +/- 2 mmHg (
146 ean arterial blood pressure and optimal mean arterial blood pressure greater than 10 mm Hg and durati
147 bility of good neurologic outcome, with mean arterial blood pressure greater than 110 mm Hg having th
148 n 10 mm Hg and duration outside optimal mean arterial blood pressure greater than 80% had increased m
149 significantly higher in patients with a mean arterial blood pressure greater than 90 mm Hg (42%) as c
150 el adjusting for potential confounders, mean arterial blood pressure greater than 90 mm Hg was associ
152 mean arterial pressure outside optimal mean arterial blood pressure have worse outcomes than those w
153 The apelin-apelin receptor system affects arterial blood pressure homeostasis; however, the centra
154 (100 mg/kg i.p.) significantly lowered mean arterial blood pressure in normotensive and hypertensive
155 mortality (p < 0.001) than systolic invasive arterial blood pressure in the same range (</=70 mm Hg).
157 n sympathetic nerve activity, heart rate and arterial blood pressure induced by reductions in cerebra
159 othesis that elevated postresuscitation mean arterial blood pressure is associated with neurologic ou
162 py-based bedside calculation of optimal mean arterial blood pressure is feasible and might be a promi
163 EY POINTS: Dysfunctions in CNS regulation of arterial blood pressure lead to an increase in sympathet
164 Orthostatic intraocular pressure and mean arterial blood pressure may be a helpful early screening
165 st compressions for >/=1 minute and invasive arterial blood pressure monitoring before and during CPR
166 ant association between ventilation rate and arterial blood pressure occurred in children 1 year old
168 zard analysis, duration outside optimal mean arterial blood pressure of greater than 80% of monitorin
169 ean arterial blood pressure and optimal mean arterial blood pressure of more than 10 mm Hg (adjusted
171 s with no significant changes in either mean arterial blood pressure or heart rate, consistent with t
172 Neither losartan nor divalinal affected arterial blood pressure or significantly altered the amy
173 s associated with a shorter duration of mean arterial blood pressure outside optimal mean arterial bl
174 in rats by withdrawing blood until the mean arterial blood pressure reached 27 +/- 1 mm Hg for the f
175 bilateral wrist) and, when available, intra-arterial blood pressure readings (IABP) were included.
177 de a novel method for precisely guiding mean arterial blood pressure targets during cardiopulmonary b
178 t rest led to significantly higher values of arterial blood pressure than without muscle loading, and
179 patients (97%), and the median optimal mean arterial blood pressure was 89.7 mm Hg (84.6-100 mm Hg).
180 s of an ICU admission, the minimum diastolic arterial blood pressure was a hemodynamic variable that
181 on and the duration and degree to which mean arterial blood pressure was below the autoregulation thr
187 ased analysis of cerebral blood velocity and arterial blood pressure waveforms in 11 astronauts befor
191 vity, adrenal sympathetic nerve activity and arterial blood pressure whereas equi-osmotic mannitol/so
192 ed vasoactive drugs to achieve a target mean arterial blood pressure with 82 centers (68.9%) employin
193 d to determine if targeting an elevated mean arterial blood pressure would improve neurologic outcome
194 by peak exercise cardiac power output (mean arterial blood pressure x cardiac output) and functional
197 orrelated with diastolic, systolic, and mean arterial blood pressure, a surrogate marker for arterial
199 No differences were found in temperature, arterial blood pressure, and oxygenation between alpha-s
200 Cerebral blood flow (CBF) is controlled by arterial blood pressure, arterial CO2, arterial O2, and
202 mean occlusive divided by mean nonocclusive arterial blood pressure, both subtracted by central veno
203 ct brain perfusion in the face of changes in arterial blood pressure, but little is known about indiv
205 art rate variability, intracranial pressure, arterial blood pressure, cerebral perfusion pressure, an
206 hAT expression in CD4(+) cells have elevated arterial blood pressure, compared to littermate controls
208 ide (ETCO2), oxygen saturation (SaO2), intra-arterial blood pressure, electrocardiography (EKG), and
209 vasodilation, SIL unexpectedly elevated mean arterial blood pressure, failed to inhibit MFS aortic ro
210 observed in controls; normalization of mean arterial blood pressure, heart rate, and increased survi
213 All patients had continuous monitoring of arterial blood pressure, intracranial pressure, and cere
214 For every period, mean values (+/- SDs) of arterial blood pressure, intracranial pressure, pressure
215 concomitant measurement of continuous intra-arterial blood pressure, the gold standard for shock mon
226 o compare survival outcomes and intra-arrest arterial blood pressures between children receiving card
227 ered (strain B) or decreased (strain C) mean arterial blood pressures compared to their corresponding
228 nalysis of exosomes purified from fetal cord arterial blood revealed a total of 328 proteins, among w
230 ty-shear rate relationship was obtained from arterial blood samples analyzed using a standard viscosi
240 pted microcatheter aspiration of 3 different arterial blood samples: (1) within the core of the occlu
243 n rhesus macaques were acquired for 2 h with arterial blood sampling and metabolite analysis to measu
245 Methods: A 90-min dynamic PET scan with arterial blood sampling and metabolite analysis was acqu
249 and time activity curve were assessed using arterial blood sampling and served as measures for recep
250 netic analysis of a 90-min dynamic scan with arterial blood sampling is recommended for the quantific
251 standardized uptake values, suggesting that arterial blood sampling may not be necessary for modelin
254 dynamic (18)F-JNJ-64413739 PET/MRI scan with arterial blood sampling to determine the appropriate kin
255 inyl)acetamide) PET scans were acquired with arterial blood sampling to estimate the metabolite-corre
256 culation, a dynamic (18)F-FHNP PET scan with arterial blood sampling was acquired from rats treated w
263 a input functions were obtained using online arterial blood sampling with metabolite corrections deri
277 put functions were obtained using continuous arterial blood-sampling as well as using image-derived m
278 citabine directly into the pancreas, via its arterial blood supply, has a superior therapeutic effect
281 The human circulatory system consists of arterial blood that delivers nutrients to tissues, and v
283 ted hypoxemia (partial pressure of oxygen in arterial blood to fraction of inspired oxygen ratio [PFR
284 ohimbine with 90-min dynamic PET and sampled arterial blood to measure intact (11)C-yohimbine in plas
287 hamber) RV volume (RV(EV)), distal pulmonary arterial blood vessel volume (arterial BV5: vessels <5 m
288 nature and span a wide range of scales, from arterial blood vessels and bronchial mucus transport in
292 Sufficient blood flow to tissues relies on arterial blood vessels, but the mechanisms regulating th
295 he bolus at the infusion rate = 60 min), and arterial blood was collected for data quantification.
298 iably reduce CBF or CDO(2) Oxygen content in arterial blood was fully restored with acclimatisation t