ライフサイエンスコーパス: arterial calcification

1 naling events associated with toxin-mediated arterial calcification.

2 ays are causally implicated in toxin-induced arterial calcification.

3 Cardiovascular risk factors were related to arterial calcification.

4 ed, and 50 (83.3%) had mild to severe breast arterial calcification.

5 eritable risk for CAD/MI is mediated through arterial calcification.

6 tients present a more severe, often prenatal arterial calcification.

7 stal deposition underlies the development of arterial calcification.

8 t an additional protective mechanism against arterial calcification.

9 f the osteoclastic V-H+-ATPase, will inhibit arterial calcification.

10 e decoy receptor also crucial for preventing arterial calcification.

11 that estrogen may play a regulatory role in arterial calcification.

12 potential role of osteoregulatory factors in arterial calcification.

13 c studies in three families with symptomatic arterial calcifications.

14 ic PP(i) levels and induces life-threatening arterial calcifications.

15 scular calcification, particularly Abdominal Arterial Calcifications (AAC), holds predictive value fo

16 whether vitamin K1 supplementation inhibits arterial calcification activity in individuals with diab

17 Arterial calcification Agatston scores and maximum trans

18 Breast arterial calcification and calcium scores were determine

19 , studies favor a correlation between breast arterial calcification and cardiovascular risk factors o

20 Clinical evidence links arterial calcification and cardiovascular risk.

21 The pathogenesis of arterial calcification and chondrocalcinosis has become

22 Lower fetuin-A levels are associated with arterial calcification and death in end-stage renal dise

23 mutation as causing SMS and leading to early arterial calcification and dental inflammation and resor

24 on of proteins involved in the inhibition of arterial calcification and has been suggested to reduce

25 rt disease, through the evaluation of breast arterial calcification and identification of opportuniti

26 is review we address potential mechanisms of arterial calcification and, in particular, ways in which

27 e supports the universal reporting of breast arterial calcifications and personalized patient-provide

28 Phosphate excess induces arterial calcification, and although elevated FGF23 help

29 creased fibroblast growth factor 23 (FGF23), arterial calcification, and cardiovascular mortality.

30 rotegerin (OPG) is a marker and regulator of arterial calcification, and it is related to cardiovascu

31 Intracranial large and small arterial calcifications are a common incidental finding

32 Arterial calcifications are associated with increased ca

33 icient in both MGP and OPN had twice as much arterial calcification as MGP(-/-) OPN(+/+) at 2 wk, and

34 To assess whether estrogen modulates arterial calcification as well as bone calcification, we

35 Femoral arterial calcification at baseline and 6 mo was measured

36 fy histological pathologies present, such as arterial calcification (AUROC = 0.93) and identify missi

37 In a murine model, Sort1-deficiency reduced arterial calcification but did not affect bone mineraliz

38 Arterial calcification, common in atherosclerosis, is as

39 a marker of atherosclerotic inflammation, to arterial calcification detected by contemporaneous CT.

40 In addition, arterial calcification due to CD73 deficiency (ACDC) occ

41 alized arterial calcification of infancy and arterial calcification due to CD73 deficiency.

42 alized arterial calcification of infancy and arterial calcification due to CD73 deficiency.

43 lized arterial calcification of infancy, and arterial calcification due to CD73 deficiency.

44 neralized arterial calcification of infancy, arterial calcification due to deficiency of CD73, ankylo

45 Breast arterial calcification has emerged as a potential risk s

46 Idiopathic infantile arterial calcification (IIAC; OMIM 208000) is characteri

47 High fat diets induce diabetes and arterial calcification in male low density lipoprotein r

48 ough adulthood does prevent the formation of arterial calcification in OPG(-/-) mice.

49 tery stiffness, which is caused, in part, by arterial calcification in patients with chronic kidney d

50 ) that share the ability to potently inhibit arterial calcification in the rat, the V-H+-ATPase inhib

51 play a prominent role in the pathogenesis of arterial calcification, including duration of dialysis a

52 fferences of two distinct pathomechanisms of arterial calcifications: intimal associated with atheros

53 Arterial calcification is a common feature of atheroscle

54 Arterial calcification is a phenotype of vascular repair

55 In patients with diabetes mellitus, arterial calcification is accelerated and calcification

56 Medial arterial calcification is accelerated in patients with C

57 e process, there is increasing evidence that arterial calcification is actually an active, regulated

58 Arterial calcification is associated with cardiovascular

59 ried out to further test the hypothesis that arterial calcification is linked to bone resorption by d

60 Arterial calcification is modulated by transition of art

61 Arterial calcification is ubiquitous in vascular disease

62 gility after estrogen loss (osteocalcin) and arterial calcification linked to cardiovascular disease

63 Medial arterial calcification (MAC) characterizes human arteria

64 Medial arterial calcification (MAC) is a chronic systemic vascu

65 Medial arterial calcification (MAC) is a common outcome in diab

66 Elastin-specific medial arterial calcification (MAC) is an arterial disease comm

67 Medial arterial calcification (MAC) often occurs in ageing arte

68 ropathy (CN); however, the links with medial arterial calcification (MAC) seen in people with CN are

69 ning with mammography, measurement of breast arterial calcification may offer a personalized, noninva

70 ealed a common pattern associating meningeal arterial calcifications, necrotic and calcified areas in

71 OPG over a 4-wk period could not reverse the arterial calcification observed in OPG(-/-) mice.

72 development in individuals with generalized arterial calcification of infancy (GACI) due to loss-of-

73 Generalized arterial calcification of infancy (GACI) is a rare genet

74 Generalized arterial calcification of infancy (GACI) is a rare genet

75 Generalized arterial calcification of infancy (GACI) is an autosomal

76 Other conditions include generalized arterial calcification of infancy (GACI), characterized

77 m lethal orphan diseases such as generalized arterial calcification of infancy (GACI), to common dise

78 cum (PXE) and, in some patients, generalized arterial calcification of infancy (GACI).

79 rs, with phenotypic overlap with generalized arterial calcification of infancy and arterial calcifica

80 apping clinical features include generalized arterial calcification of infancy and arterial calcifica

81 decreased bone mineralization in generalized arterial calcification of infancy caused by ENPP1 mutati

82 Generalized arterial calcification of infancy is an intractable ecto

83 nosis, pseudoxanthoma elasticum, generalized arterial calcification of infancy, and arterial calcific

84 (asj) mutant mouse as a model of generalized arterial calcification of infancy, and we have now explo

85 ation: pseudoxanthoma elasticum, generalized arterial calcification of infancy, arterial calcificatio

86 for pseudoxanthoma elasticum and generalized arterial calcification of infancy, diseases that current

87 udoxanthoma elasticum and type 2 generalized arterial calcification of infancy, heritable ectopic min

88 m (PXE) as well as some cases of generalized arterial calcification of infancy, which is otherwise ca

89 bute to the calcification in PXE/generalized arterial calcification of infancy.

90 homa elasticum and some cases of generalized arterial calcification of infancy.

91 homa elasticum and some cases of generalized arterial calcification of infancy.

92 al to the pathophysiology of PXE/generalized arterial calcification of infancy.

93 lished reports on prevalence of intracranial arterial calcifications on computed tomography imaging a

94 Because lack of MGP also causes arterial calcification, our findings demonstrate that th

95 Arterial calcification predicts accelerated restenosis a

96 , recent developments in the pathogenesis of arterial calcification provide valuable information pert

97 ntrast, Klotho was inversely correlated with arterial calcification (r = - 0.388 [- 0.578 to - 0.159]

98 FGF-23 was positively correlated with arterial calcification (r = 0.446 [0.254-0.611], p < 0.0

99 Arterial calcification severity was assessed by computed

100 Arterial calcification severity was assessed using compu

101 ude hemostasis, apoptosis, bone development, arterial calcification, signal transduction, and growth

102 , preexisting cardiovascular disease status, arterial calcification status, and concomitant chemother

103 Arterial calcification, thickness, and stiffness were de

104 sion, FGF-23 and Klotho were associated with arterial calcification, thickness, and stiffness, clarif

105 emodeling across recent studies, in terms of arterial calcification, thickness, and stiffness.

106 alvulopathies, calciphylaxis, and idiopathic arterial calcification, vascular calcification is now re

107 lished that metformin administration reduces arterial calcification via autophagy; however, whether m

108 We found that arterial calcification was associated with a similar los

109 Arterial calcification was induced by subcutaneous injec

110 In contrast, the prevalence of coronary arterial calcification was similar between patients with

111 tion to assess the association of the breast arterial calcification were performed.

112 erm but die within two months as a result of arterial calcification which leads to blood-vessel ruptu